1- Define the word conundrum and explain why the term is appropriate w.pdf
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1. Define the word conundrum and explain why the term is appropriate with regard to prescription of Levodopa. 2. Dr. Sayer made a connection between Parkinson's disease and a form of encephalitis. How are Parkinson's disease and the effects of encephalitis lethargica similar? How are they different? 3. What was it that Leonard responded to during in his EEG that allowed Dr. Sayer to confirm that Leonard was capable of brain activity? 4. From lecture and your text, what are the two named regions in the brain that are heavily under the control of dopaminergic neurons? Give an example of an event that demonstrated the activity of each region after being stimulated by the L-DOPA from the movie Awakenings. 5. Watch the following video and share a good definition of tardive dyskinesia, then describe how Leonard Lowe exhibited this in a scene from the movie. What caused his dyskinesia?.
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Awakenings (Guide for Movie Response Paper) 1. Describe the symp.docx
Awakenings (Guide for Movie Response Paper)
1. Describe the symptoms of the character “Lenny” in childhood as well as adulthood.
2. Describe Lucy’s symptoms.
3. In Lenny’s, Lucy’s and the other similar cases, how do their symptoms compare with those of people with schizophrenia?
4. Describe Dr. Sayer’s “will” theory in regard to his patients’ reactions to the ball. Relate this to the “will” of some people with schizophrenia.
5. What various stimuli make these patients react? (i.e., what reaches them?)
6. Why might these patients have been diagnosed as suffering from “atypical schizophrenia”?
7. Unlike the case of schizophrenia, we know what caused these patients’ disorder. What was it?
8. The older doctor says these patients aren’t thinking, “the virus didn’t spare their higher faculties.” Dr. Sayer asks how he knows that. The older doctor replies, “Because the alternative is unthinkable.” What does he mean by that? What effect did this point of view have on these patients’ diagnosis and treatment? Can you relate this to schizophrenia or mental illness in general?
9. Why does it bother Dr. Sayer that Lucy was drawn to the window rather than to the drinking fountain? Can you relate this to Maslow?
10. The EEG indicates what about Lenny?
11. Dr. Sayer uses an unusual technique to assess his patients. What is it?
12. What finally moves Burt?
13. Why does the poem, “The Panther,” have special significance for Lenny?
14. What is L-Dopa? It is used in the treatment of what disorder?
15. Dr. Sayer parallels Parkinson’s and his patients’ disorder. Describe this.
16. The dopamine circuit is affected in opposite ways in Parkinson’s and schizophrenia. Phenothiazines taken for a long period produce Parkinson-like symptoms in the person with schizophrenia. Knowing this and knowing the effect on phenothiazine on the dompamine circuit, explain why you think L-Dopa “awakes” Dr. Sayer’s patients.
17. What are the various psychological reactions the patients have to their “awakenings”?
18. What Parkinson-like symptoms does Lenny exhibit when he develops tolerance to LDopa?
19. One doctor describes Lenny as being paranoid. How does Dr. Sayer explain this? Describe this in terms of labeling theory.
20. What ethical questions are raised in this movie regarding treatment? What is your opinion?
21. What is the second “awakening” Dr. Sayer refers to at the end of the movie? What does this suggest about the treatment of mental patients?
.
How Brain Activity Monitoring can Help Manage Asperger’s Syndrome
Using electroencephalography (a neuroimaging technique) to track mood, anxiety, stimulation level, cognitive functioning, concentration, and stress. Using feedback based on information provided by brain data (neurofeedback) to recommend therapies for Asperger's. Training and improving coping responses to states of overstimulation in Asperger's using neurofeedback-assisted mindfulness meditation. Sharing of self-collected brain data with medical professionals to improve Asperger's treatment.
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Awakenings (Guide for Movie Response Paper) 1. Describe the symp.docx
Awakenings (Guide for Movie Response Paper)
1. Describe the symptoms of the character “Lenny” in childhood as well as adulthood.
2. Describe Lucy’s symptoms.
3. In Lenny’s, Lucy’s and the other similar cases, how do their symptoms compare with those of people with schizophrenia?
4. Describe Dr. Sayer’s “will” theory in regard to his patients’ reactions to the ball. Relate this to the “will” of some people with schizophrenia.
5. What various stimuli make these patients react? (i.e., what reaches them?)
6. Why might these patients have been diagnosed as suffering from “atypical schizophrenia”?
7. Unlike the case of schizophrenia, we know what caused these patients’ disorder. What was it?
8. The older doctor says these patients aren’t thinking, “the virus didn’t spare their higher faculties.” Dr. Sayer asks how he knows that. The older doctor replies, “Because the alternative is unthinkable.” What does he mean by that? What effect did this point of view have on these patients’ diagnosis and treatment? Can you relate this to schizophrenia or mental illness in general?
9. Why does it bother Dr. Sayer that Lucy was drawn to the window rather than to the drinking fountain? Can you relate this to Maslow?
10. The EEG indicates what about Lenny?
11. Dr. Sayer uses an unusual technique to assess his patients. What is it?
12. What finally moves Burt?
13. Why does the poem, “The Panther,” have special significance for Lenny?
14. What is L-Dopa? It is used in the treatment of what disorder?
15. Dr. Sayer parallels Parkinson’s and his patients’ disorder. Describe this.
16. The dopamine circuit is affected in opposite ways in Parkinson’s and schizophrenia. Phenothiazines taken for a long period produce Parkinson-like symptoms in the person with schizophrenia. Knowing this and knowing the effect on phenothiazine on the dompamine circuit, explain why you think L-Dopa “awakes” Dr. Sayer’s patients.
17. What are the various psychological reactions the patients have to their “awakenings”?
18. What Parkinson-like symptoms does Lenny exhibit when he develops tolerance to LDopa?
19. One doctor describes Lenny as being paranoid. How does Dr. Sayer explain this? Describe this in terms of labeling theory.
20. What ethical questions are raised in this movie regarding treatment? What is your opinion?
21. What is the second “awakening” Dr. Sayer refers to at the end of the movie? What does this suggest about the treatment of mental patients?
.
How Brain Activity Monitoring can Help Manage Asperger’s Syndrome
Using electroencephalography (a neuroimaging technique) to track mood, anxiety, stimulation level, cognitive functioning, concentration, and stress. Using feedback based on information provided by brain data (neurofeedback) to recommend therapies for Asperger's. Training and improving coping responses to states of overstimulation in Asperger's using neurofeedback-assisted mindfulness meditation. Sharing of self-collected brain data with medical professionals to improve Asperger's treatment.
________ is NOT a common cause for maintenance insomnia. Stress Eat.pdf
________ is NOT a common cause for maintenance insomnia. Stress Eating late at night
Substance abuse Psychological disorder RLS is characterized by differences in functioning.
endorphin GAQA serotonin dopamine Muscle paralysis with lingers with narcolepsy once the
person awakes can be resolved by. feeding the person touching the person letting The person
smell the scent of lavender whispering to the person After a period of most coma patients
recover, move to a vegetative state, or die. 2 to 3 days 5 days 1 week 2 to 4 weeks
Solution
13) Is not a common cause for maintenance insomnia
B)eating late at night
Insomnia is a sleep disorder, people have trouble sleeping. Psychological stress, chronic pain,
heart failure, heartburn, restless leg syndrome, hyperthyroidism, menopause, drugs such as
caffeine, alcohol and nicotine etc are responsible for insomnia. So, eating late at night cannot
cause insomnia.
14) Muscle paralysis that lingers with narcolepsy once the person awakes can be resolved by
B) Touching the person
Patients with narcolepsy will experience sleep paralysis or muscle paralysis, when the person is
awakens, just touch them to end the paralysis.
17) RLS is characterized by differences in functioning
D) Dopamine
Here the person will have uncomfortable sensations deep in the legs developing at rest. He
cannot move and is worst at night with sleep disturbance. It is believed that RLS is due to
functional disturbance of dopamine neurotransmission in case of subcortical areas of the brain.
18)After a period of------most coma patients recover, move to a vegetative state or die
D)2 to 4 weeks
It has been observed that coma rarely lasts more than 4 weeks. During this period some patients
move from coma to the vegetative state or some may regain partial consciousness or some may
die..
My psychology 1st edition pomerantz test bank
My Psychology 1st Edition Pomerantz Test Bank
Full download: https://goo.gl/poEGwQ
1. Receptors are where transduction occurs in a sensory system. For .pdf
1. Receptors are where transduction occurs in a sensory system. For the visual system, the
receptors are called photoreceptors. There are two types of photoreceptors. What are they called?
2. What is transduction? Use your own words (not the Chapter Outline’s or the textbook’s) to
give a definition as you understand it. 6. Receptors are where transduction occurs in a sensory
system. For the visual system, the receptors are called photoreceptors. There are two types of
photoreceptors. What are they called? 7. What is transduction? Use your own words to give a
definition as you understand it
Solution
1.The two types of photo receptors seen are Rods and Cones.
2. Transduction in lay man language refers to transfer of foreign DNA to some specific cell by
means of vector. Generally here the vector used is Virus.
6. Please refer to my answer in question 1. it sates that rodes and cones are two photoreceptors.
7. same as the answer in question 2..
Approach to Coma - April 2023 x
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Neurotransmission, Neuropsychiatry, and Neuropharmacology 2013
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a) What happens to language and communication after brain damage of different types?
b) How did the ability to communicate and the ability to use language develop in the evolution of the species? How can we relate this development to the evolution of the brain?
c) How do children learn to communicate and use language? How can we relate their acquisition of language to the development of their brains?
d) How can we measure and visualize processes in the brain that are involved in language and communication?
e) How can we make good models of language and communication processes that will help us to explain the linguistic phenomena that we study?
f) How can we make computer simulations of language processing, language development and language loss?
g) How can we make experiments that will allow us to test our models and hypotheses about language processing?
ACQUIRED LANGUAGE DISORDERS:
• Aphasia- language loss due to brain damage, cause can be infarction, hemorrhage or head trauma
• Either comprehension or expression of language or both is effected
• Aphasiology or linguistics aphasiology is the dominant branch of neurolinguistics. Aphasia is an acquired language disorder, a focal lesion. Another cause of acquired disorders can e progressive neurological disease, such as, dementia. Language and memory are closely connected and interdependent, especially in higher cognitive functions.
DEVELOPMENTAL LANGUAGE DISORDERS:
• Disorders that are found in children who have not experienced any specific lesion event.
• Neurolinguists compare developmental language disorders (like SLI, dyslexia) to similar acquired disorders with the view of language acquisition and plasticity (ability to be moulded) of young brains, they also study the language develooment of children with non specific developmental disorders affecting language.
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• Neurolinguists study development of language and speech, their prerequisites in the evolution of species. The changes in the structure and function f the brain are compared to different species ways of living.
• Experiments are being carried out with primates that are being taught human communication systems.
An overview of déjà vu
Explaining what déjà vu is and a very brief review over scientific findings and speculation concerning the déjà vu experience.
#36420 Topic Discussion 6Number of Pages 1 (Double Spaced).docx
#36420 Topic: Discussion 6
Number of Pages: 1 (Double Spaced)
Number of sources: 1
Writing Style: APA
Type of document: Essay
Academic Level:Master
Category: Psychology
Language Style: English (U.S.)
Order Instructions:
I will upload the instruction
Module Chapter 5 wk6 p630
chapter 5 Methods and Strategies of Research
Outline
· ■ Experimental Ablation
Evaluating the Behavioral Effects of Brain Damage
Producing Brain Lesions
Stereotaxic Surgery
Histological Methods
Tracing Neural Connections
Studying the Structure of the Living Human Brain
Section Summary
· ■ Recording and Stimulating Neural Activity
Recording Neural Activity
Recording the Brain’s Metabolic and Synaptic Activity
Stimulating Neural Activity
Section Summary
· ■ Neurochemical Methods
Finding Neurons That Produce Particular Neurochemicals
Localizing Particular Receptors
Measuring Chemicals Secreted in the Brain
Section Summary
· ■ Genetic Methods
Twin Studies
Adoption Studies
Genomic Studies
Targeted Mutations
Antisense Oligonucleotides
Section Summary
In July 1982, several young people began showing up at neurology clinics in northern California displaying dramatic, severe symptoms (Langston et al., 1983). The most severely affected patients were almost totally paralyzed. They were unable to speak intelligibly, they drooled constantly, and their eyes were open with a fixed stare. Others, less severely affected, walked with a slow, shuffling gait and moved slowly and with great difficulty. The symptoms looked like those of Parkinson’s disease, but that disorder has a very gradual onset. In addition, it rarely strikes people before late middle age, and the patients were all in their twenties or early thirties.
The common factor linking these patients was intravenous drug use; all of them had been taking a “new heroin,” a synthetic opiate related to meperidine (Demerol). Some detective work revealed that the illicit drug was contaminated with MPTP, a toxic chemical that damaged dopaminergic neurons and caused the neurological symptoms. Because the symptoms looked like those of Parkinson’s disease, the patients were given L-DOPA, the drug that is used to treat this disease, and they all showed significant improvement in their symptoms. Unfortunately, the improvement was temporary; the drug lost its effectiveness.
Fetal transplantation, an experimental neurosurgical method of treating parkinsonism, has shown some promise. The rationale for the procedure is this: The symptoms of parkinsonism, whether from Parkinson’s disease or the toxic effects of MPTP, are caused by the lack of dopamine in the caudate nucleus and putamen. There is at present no way to induce the brain to grow new dopaminergic neurons. However, if dopamine-secreting neurons can be introduced into the caudate nucleus and putamen and if they survive and secrete dopamine, then perhaps the parkinsonian symptoms will diminish. Because the implanted neurons must be healthy and vigorous and not trigger the .
How Pornography Affects Developing Brains - Dr. Jennifer Brown
Dr. Jennifer Brown has done extensive research on the effects of pornography on children's developing brains. This PowerPoint presentation details some of the main points of her study. Dr. Brown presented this information to the Prevent Child Abuse Utah Joining Forces Conference in October 2014. This research was also instrumental in passing S.B. 227 in Utah to allow a judge to reduce or restrict custody if a parent has intentionally exposed their child to pornography.
#36421 Topic Discussion 7Number of Pages 1 (Double Spaced).docx
#36421 Topic: Discussion 7
Number of Pages: 1 (Double Spaced)
Number of sources: 1
Writing Style: APA
Type of document: Essay
Academic Level:Master
Category: Psychology
Language Style: English (U.S.)
Order Instructions: ATTACHED
I will upload the instruction
Module Chapter 5 wk6 p630
chapter 5 Methods and Strategies of Research
Outline
· ■ Experimental Ablation
Evaluating the Behavioral Effects of Brain Damage
Producing Brain Lesions
Stereotaxic Surgery
Histological Methods
Tracing Neural Connections
Studying the Structure of the Living Human Brain
Section Summary
· ■ Recording and Stimulating Neural Activity
Recording Neural Activity
Recording the Brain’s Metabolic and Synaptic Activity
Stimulating Neural Activity
Section Summary
· ■ Neurochemical Methods
Finding Neurons That Produce Particular Neurochemicals
Localizing Particular Receptors
Measuring Chemicals Secreted in the Brain
Section Summary
· ■ Genetic Methods
Twin Studies
Adoption Studies
Genomic Studies
Targeted Mutations
Antisense Oligonucleotides
Section Summary
In July 1982, several young people began showing up at neurology clinics in northern California displaying dramatic, severe symptoms (Langston et al., 1983). The most severely affected patients were almost totally paralyzed. They were unable to speak intelligibly, they drooled constantly, and their eyes were open with a fixed stare. Others, less severely affected, walked with a slow, shuffling gait and moved slowly and with great difficulty. The symptoms looked like those of Parkinson’s disease, but that disorder has a very gradual onset. In addition, it rarely strikes people before late middle age, and the patients were all in their twenties or early thirties.
The common factor linking these patients was intravenous drug use; all of them had been taking a “new heroin,” a synthetic opiate related to meperidine (Demerol). Some detective work revealed that the illicit drug was contaminated with MPTP, a toxic chemical that damaged dopaminergic neurons and caused the neurological symptoms. Because the symptoms looked like those of Parkinson’s disease, the patients were given L-DOPA, the drug that is used to treat this disease, and they all showed significant improvement in their symptoms. Unfortunately, the improvement was temporary; the drug lost its effectiveness.
Fetal transplantation, an experimental neurosurgical method of treating parkinsonism, has shown some promise. The rationale for the procedure is this: The symptoms of parkinsonism, whether from Parkinson’s disease or the toxic effects of MPTP, are caused by the lack of dopamine in the caudate nucleus and putamen. There is at present no way to induce the brain to grow new dopaminergic neurons. However, if dopamine-secreting neurons can be introduced into the caudate nucleus and putamen and if they survive and secrete dopamine, then perhaps the parkinsonian symptoms will diminish. Because the implanted neurons must be healthy and vigorous and not t.
#36417 Topic Discussion 3Number of Pages 1 (Double Spaced).docx
#36417 Topic: Discussion 3
Number of Pages: 1 (Double Spaced)
Number of sources: 1
Writing Style: APA
Type of document: Essay
Academic Level:Master
Category: Psychology
Language Style: English (U.S.)
Order Instructions:
I will upload the instruction
Module Chapter 5 wk6 p630
chapter 5 Methods and Strategies of Research
Outline
· ■ Experimental Ablation
Evaluating the Behavioral Effects of Brain Damage
Producing Brain Lesions
Stereotaxic Surgery
Histological Methods
Tracing Neural Connections
Studying the Structure of the Living Human Brain
Section Summary
· ■ Recording and Stimulating Neural Activity
Recording Neural Activity
Recording the Brain’s Metabolic and Synaptic Activity
Stimulating Neural Activity
Section Summary
· ■ Neurochemical Methods
Finding Neurons That Produce Particular Neurochemicals
Localizing Particular Receptors
Measuring Chemicals Secreted in the Brain
Section Summary
· ■ Genetic Methods
Twin Studies
Adoption Studies
Genomic Studies
Targeted Mutations
Antisense Oligonucleotides
Section Summary
In July 1982, several young people began showing up at neurology clinics in northern California displaying dramatic, severe symptoms (Langston et al., 1983). The most severely affected patients were almost totally paralyzed. They were unable to speak intelligibly, they drooled constantly, and their eyes were open with a fixed stare. Others, less severely affected, walked with a slow, shuffling gait and moved slowly and with great difficulty. The symptoms looked like those of Parkinson’s disease, but that disorder has a very gradual onset. In addition, it rarely strikes people before late middle age, and the patients were all in their twenties or early thirties.
The common factor linking these patients was intravenous drug use; all of them had been taking a “new heroin,” a synthetic opiate related to meperidine (Demerol). Some detective work revealed that the illicit drug was contaminated with MPTP, a toxic chemical that damaged dopaminergic neurons and caused the neurological symptoms. Because the symptoms looked like those of Parkinson’s disease, the patients were given L-DOPA, the drug that is used to treat this disease, and they all showed significant improvement in their symptoms. Unfortunately, the improvement was temporary; the drug lost its effectiveness.
Fetal transplantation, an experimental neurosurgical method of treating parkinsonism, has shown some promise. The rationale for the procedure is this: The symptoms of parkinsonism, whether from Parkinson’s disease or the toxic effects of MPTP, are caused by the lack of dopamine in the caudate nucleus and putamen. There is at present no way to induce the brain to grow new dopaminergic neurons. However, if dopamine-secreting neurons can be introduced into the caudate nucleus and putamen and if they survive and secrete dopamine, then perhaps the parkinsonian symptoms will diminish. Because the implanted neurons must be healthy and vigorous and not trigger the.
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1- How could Esquel achieve their ambitious sustainability- people- an.pdf
1. How could Esquel achieve their ambitious sustainability, people, and community-oriented
objectives in a highly competitive industry when most of its competitors were using a low-cost
production strategy?
2. How should Esquel prepare itself for the risks from this Integral project? What kind of hurdles
could they face as they engage more deeply in this venture and effort?
3. Would the Integral project yield financial returns? Discuss the potential scenarios both in the
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well as long run tangible and/or intangible benefits.
4. How should Esquel plan for the changing labor force and cost pressures in China? What are
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Similar to 1- Define the word conundrum and explain why the term is appropriate w.pdf
________ is NOT a common cause for maintenance insomnia. Stress Eat.pdf
________ is NOT a common cause for maintenance insomnia. Stress Eating late at night
Substance abuse Psychological disorder RLS is characterized by differences in functioning.
endorphin GAQA serotonin dopamine Muscle paralysis with lingers with narcolepsy once the
person awakes can be resolved by. feeding the person touching the person letting The person
smell the scent of lavender whispering to the person After a period of most coma patients
recover, move to a vegetative state, or die. 2 to 3 days 5 days 1 week 2 to 4 weeks
Solution
13) Is not a common cause for maintenance insomnia
B)eating late at night
Insomnia is a sleep disorder, people have trouble sleeping. Psychological stress, chronic pain,
heart failure, heartburn, restless leg syndrome, hyperthyroidism, menopause, drugs such as
caffeine, alcohol and nicotine etc are responsible for insomnia. So, eating late at night cannot
cause insomnia.
14) Muscle paralysis that lingers with narcolepsy once the person awakes can be resolved by
B) Touching the person
Patients with narcolepsy will experience sleep paralysis or muscle paralysis, when the person is
awakens, just touch them to end the paralysis.
17) RLS is characterized by differences in functioning
D) Dopamine
Here the person will have uncomfortable sensations deep in the legs developing at rest. He
cannot move and is worst at night with sleep disturbance. It is believed that RLS is due to
functional disturbance of dopamine neurotransmission in case of subcortical areas of the brain.
18)After a period of------most coma patients recover, move to a vegetative state or die
D)2 to 4 weeks
It has been observed that coma rarely lasts more than 4 weeks. During this period some patients
move from coma to the vegetative state or some may regain partial consciousness or some may
die..
My psychology 1st edition pomerantz test bank
My Psychology 1st Edition Pomerantz Test Bank
Full download: https://goo.gl/poEGwQ
1. Receptors are where transduction occurs in a sensory system. For .pdf
1. Receptors are where transduction occurs in a sensory system. For the visual system, the
receptors are called photoreceptors. There are two types of photoreceptors. What are they called?
2. What is transduction? Use your own words (not the Chapter Outline’s or the textbook’s) to
give a definition as you understand it. 6. Receptors are where transduction occurs in a sensory
system. For the visual system, the receptors are called photoreceptors. There are two types of
photoreceptors. What are they called? 7. What is transduction? Use your own words to give a
definition as you understand it
Solution
1.The two types of photo receptors seen are Rods and Cones.
2. Transduction in lay man language refers to transfer of foreign DNA to some specific cell by
means of vector. Generally here the vector used is Virus.
6. Please refer to my answer in question 1. it sates that rodes and cones are two photoreceptors.
7. same as the answer in question 2..
Approach to Coma - April 2023 x
This ppt is created as part of faculty class for medicine and neurology residents , neurologist and neurosurgeons . Especially while attending a case of coma in emergency. How to approach a case of coma
Neurotransmission, Neuropsychiatry, and Neuropharmacology 2013
Neurotransmission, Neuropsychiatry, and Neuropharmacology 2013 Postgraduate Course at Aarhus University includes historical information on Masters of Neuropsychopharmacology
Neurolinguistics MASLP
a) What happens to language and communication after brain damage of different types?
b) How did the ability to communicate and the ability to use language develop in the evolution of the species? How can we relate this development to the evolution of the brain?
c) How do children learn to communicate and use language? How can we relate their acquisition of language to the development of their brains?
d) How can we measure and visualize processes in the brain that are involved in language and communication?
e) How can we make good models of language and communication processes that will help us to explain the linguistic phenomena that we study?
f) How can we make computer simulations of language processing, language development and language loss?
g) How can we make experiments that will allow us to test our models and hypotheses about language processing?
ACQUIRED LANGUAGE DISORDERS:
• Aphasia- language loss due to brain damage, cause can be infarction, hemorrhage or head trauma
• Either comprehension or expression of language or both is effected
• Aphasiology or linguistics aphasiology is the dominant branch of neurolinguistics. Aphasia is an acquired language disorder, a focal lesion. Another cause of acquired disorders can e progressive neurological disease, such as, dementia. Language and memory are closely connected and interdependent, especially in higher cognitive functions.
DEVELOPMENTAL LANGUAGE DISORDERS:
• Disorders that are found in children who have not experienced any specific lesion event.
• Neurolinguists compare developmental language disorders (like SLI, dyslexia) to similar acquired disorders with the view of language acquisition and plasticity (ability to be moulded) of young brains, they also study the language develooment of children with non specific developmental disorders affecting language.
LANGUAGE AND SPEECH:
• Neurolinguists study development of language and speech, their prerequisites in the evolution of species. The changes in the structure and function f the brain are compared to different species ways of living.
• Experiments are being carried out with primates that are being taught human communication systems.
An overview of déjà vu
Explaining what déjà vu is and a very brief review over scientific findings and speculation concerning the déjà vu experience.
#36420 Topic Discussion 6Number of Pages 1 (Double Spaced).docx
#36420 Topic: Discussion 6
Number of Pages: 1 (Double Spaced)
Number of sources: 1
Writing Style: APA
Type of document: Essay
Academic Level:Master
Category: Psychology
Language Style: English (U.S.)
Order Instructions:
I will upload the instruction
Module Chapter 5 wk6 p630
chapter 5 Methods and Strategies of Research
Outline
· ■ Experimental Ablation
Evaluating the Behavioral Effects of Brain Damage
Producing Brain Lesions
Stereotaxic Surgery
Histological Methods
Tracing Neural Connections
Studying the Structure of the Living Human Brain
Section Summary
· ■ Recording and Stimulating Neural Activity
Recording Neural Activity
Recording the Brain’s Metabolic and Synaptic Activity
Stimulating Neural Activity
Section Summary
· ■ Neurochemical Methods
Finding Neurons That Produce Particular Neurochemicals
Localizing Particular Receptors
Measuring Chemicals Secreted in the Brain
Section Summary
· ■ Genetic Methods
Twin Studies
Adoption Studies
Genomic Studies
Targeted Mutations
Antisense Oligonucleotides
Section Summary
In July 1982, several young people began showing up at neurology clinics in northern California displaying dramatic, severe symptoms (Langston et al., 1983). The most severely affected patients were almost totally paralyzed. They were unable to speak intelligibly, they drooled constantly, and their eyes were open with a fixed stare. Others, less severely affected, walked with a slow, shuffling gait and moved slowly and with great difficulty. The symptoms looked like those of Parkinson’s disease, but that disorder has a very gradual onset. In addition, it rarely strikes people before late middle age, and the patients were all in their twenties or early thirties.
The common factor linking these patients was intravenous drug use; all of them had been taking a “new heroin,” a synthetic opiate related to meperidine (Demerol). Some detective work revealed that the illicit drug was contaminated with MPTP, a toxic chemical that damaged dopaminergic neurons and caused the neurological symptoms. Because the symptoms looked like those of Parkinson’s disease, the patients were given L-DOPA, the drug that is used to treat this disease, and they all showed significant improvement in their symptoms. Unfortunately, the improvement was temporary; the drug lost its effectiveness.
Fetal transplantation, an experimental neurosurgical method of treating parkinsonism, has shown some promise. The rationale for the procedure is this: The symptoms of parkinsonism, whether from Parkinson’s disease or the toxic effects of MPTP, are caused by the lack of dopamine in the caudate nucleus and putamen. There is at present no way to induce the brain to grow new dopaminergic neurons. However, if dopamine-secreting neurons can be introduced into the caudate nucleus and putamen and if they survive and secrete dopamine, then perhaps the parkinsonian symptoms will diminish. Because the implanted neurons must be healthy and vigorous and not trigger the .
How Pornography Affects Developing Brains - Dr. Jennifer Brown
Dr. Jennifer Brown has done extensive research on the effects of pornography on children's developing brains. This PowerPoint presentation details some of the main points of her study. Dr. Brown presented this information to the Prevent Child Abuse Utah Joining Forces Conference in October 2014. This research was also instrumental in passing S.B. 227 in Utah to allow a judge to reduce or restrict custody if a parent has intentionally exposed their child to pornography.
#36421 Topic Discussion 7Number of Pages 1 (Double Spaced).docx
#36421 Topic: Discussion 7
Number of Pages: 1 (Double Spaced)
Number of sources: 1
Writing Style: APA
Type of document: Essay
Academic Level:Master
Category: Psychology
Language Style: English (U.S.)
Order Instructions: ATTACHED
I will upload the instruction
Module Chapter 5 wk6 p630
chapter 5 Methods and Strategies of Research
Outline
· ■ Experimental Ablation
Evaluating the Behavioral Effects of Brain Damage
Producing Brain Lesions
Stereotaxic Surgery
Histological Methods
Tracing Neural Connections
Studying the Structure of the Living Human Brain
Section Summary
· ■ Recording and Stimulating Neural Activity
Recording Neural Activity
Recording the Brain’s Metabolic and Synaptic Activity
Stimulating Neural Activity
Section Summary
· ■ Neurochemical Methods
Finding Neurons That Produce Particular Neurochemicals
Localizing Particular Receptors
Measuring Chemicals Secreted in the Brain
Section Summary
· ■ Genetic Methods
Twin Studies
Adoption Studies
Genomic Studies
Targeted Mutations
Antisense Oligonucleotides
Section Summary
In July 1982, several young people began showing up at neurology clinics in northern California displaying dramatic, severe symptoms (Langston et al., 1983). The most severely affected patients were almost totally paralyzed. They were unable to speak intelligibly, they drooled constantly, and their eyes were open with a fixed stare. Others, less severely affected, walked with a slow, shuffling gait and moved slowly and with great difficulty. The symptoms looked like those of Parkinson’s disease, but that disorder has a very gradual onset. In addition, it rarely strikes people before late middle age, and the patients were all in their twenties or early thirties.
The common factor linking these patients was intravenous drug use; all of them had been taking a “new heroin,” a synthetic opiate related to meperidine (Demerol). Some detective work revealed that the illicit drug was contaminated with MPTP, a toxic chemical that damaged dopaminergic neurons and caused the neurological symptoms. Because the symptoms looked like those of Parkinson’s disease, the patients were given L-DOPA, the drug that is used to treat this disease, and they all showed significant improvement in their symptoms. Unfortunately, the improvement was temporary; the drug lost its effectiveness.
Fetal transplantation, an experimental neurosurgical method of treating parkinsonism, has shown some promise. The rationale for the procedure is this: The symptoms of parkinsonism, whether from Parkinson’s disease or the toxic effects of MPTP, are caused by the lack of dopamine in the caudate nucleus and putamen. There is at present no way to induce the brain to grow new dopaminergic neurons. However, if dopamine-secreting neurons can be introduced into the caudate nucleus and putamen and if they survive and secrete dopamine, then perhaps the parkinsonian symptoms will diminish. Because the implanted neurons must be healthy and vigorous and not t.
#36417 Topic Discussion 3Number of Pages 1 (Double Spaced).docx
#36417 Topic: Discussion 3
Number of Pages: 1 (Double Spaced)
Number of sources: 1
Writing Style: APA
Type of document: Essay
Academic Level:Master
Category: Psychology
Language Style: English (U.S.)
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I will upload the instruction
Module Chapter 5 wk6 p630
chapter 5 Methods and Strategies of Research
Outline
· ■ Experimental Ablation
Evaluating the Behavioral Effects of Brain Damage
Producing Brain Lesions
Stereotaxic Surgery
Histological Methods
Tracing Neural Connections
Studying the Structure of the Living Human Brain
Section Summary
· ■ Recording and Stimulating Neural Activity
Recording Neural Activity
Recording the Brain’s Metabolic and Synaptic Activity
Stimulating Neural Activity
Section Summary
· ■ Neurochemical Methods
Finding Neurons That Produce Particular Neurochemicals
Localizing Particular Receptors
Measuring Chemicals Secreted in the Brain
Section Summary
· ■ Genetic Methods
Twin Studies
Adoption Studies
Genomic Studies
Targeted Mutations
Antisense Oligonucleotides
Section Summary
In July 1982, several young people began showing up at neurology clinics in northern California displaying dramatic, severe symptoms (Langston et al., 1983). The most severely affected patients were almost totally paralyzed. They were unable to speak intelligibly, they drooled constantly, and their eyes were open with a fixed stare. Others, less severely affected, walked with a slow, shuffling gait and moved slowly and with great difficulty. The symptoms looked like those of Parkinson’s disease, but that disorder has a very gradual onset. In addition, it rarely strikes people before late middle age, and the patients were all in their twenties or early thirties.
The common factor linking these patients was intravenous drug use; all of them had been taking a “new heroin,” a synthetic opiate related to meperidine (Demerol). Some detective work revealed that the illicit drug was contaminated with MPTP, a toxic chemical that damaged dopaminergic neurons and caused the neurological symptoms. Because the symptoms looked like those of Parkinson’s disease, the patients were given L-DOPA, the drug that is used to treat this disease, and they all showed significant improvement in their symptoms. Unfortunately, the improvement was temporary; the drug lost its effectiveness.
Fetal transplantation, an experimental neurosurgical method of treating parkinsonism, has shown some promise. The rationale for the procedure is this: The symptoms of parkinsonism, whether from Parkinson’s disease or the toxic effects of MPTP, are caused by the lack of dopamine in the caudate nucleus and putamen. There is at present no way to induce the brain to grow new dopaminergic neurons. However, if dopamine-secreting neurons can be introduced into the caudate nucleus and putamen and if they survive and secrete dopamine, then perhaps the parkinsonian symptoms will diminish. Because the implanted neurons must be healthy and vigorous and not trigger the.
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RPMS TEMPLATE FOR SCHOOL YEAR 2023-2024 FOR TEACHER 1 TO TEACHER 3
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Pride Month Slides 2024 David Douglas School District
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The simplified electron and muon model, Oscillating Spacetime: The Foundation...
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Thesis Statement for students diagnonsed withADHD.ppt
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Chapter 4 - Islamic Financial Institutions in Malaysia.pptx
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A Survey of Techniques for Maximizing LLM Performance.pptx
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Exploiting Artificial Intelligence for Empowering Researchers and Faculty, In...
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How to Build a Module in Odoo 17 Using the Scaffold Method
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Executive Directors Chat Leveraging AI for Diversity, Equity, and Inclusion
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Normal Labour/ Stages of Labour/ Mechanism of Labour
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Mule 4.6 & Java 17 Upgrade | MuleSoft Mysore Meetup #46
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Digital Artifact 2 - Investigating Pavilion Designs
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- 1. 1. Define the word conundrum and explain why the term is appropriate with regard to prescription of Levodopa. 2. Dr. Sayer made a connection between Parkinson's disease and a form of encephalitis. How are Parkinson's disease and the effects of encephalitis lethargica similar? How are they different? 3. What was it that Leonard responded to during in his EEG that allowed Dr. Sayer to confirm that Leonard was capable of brain activity? 4. From lecture and your text, what are the two named regions in the brain that are heavily under the control of dopaminergic neurons? Give an example of an event that demonstrated the activity of each region after being stimulated by the L-DOPA from the movie Awakenings. 5. Watch the following video and share a good definition of tardive dyskinesia, then describe how Leonard Lowe exhibited this in a scene from the movie. What caused his dyskinesia?