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12-Lead EKG Interpretation
Judith M. Haluka
BS, RCIS, EMT-P
ECG Grid
• Left to Right = Time/duration
• Vertical – measure of voltage (amplitude)
– Expressed in mm
P-Wave
• Depolarization of atrial muscle
• Low voltage (2-3mm in amplitude)
• Duration <.11 seconds
Abnormal P Waves
• P – Pulmonale
– Tall Peaked
– Right atrial enlargement secondary to
pulmonary HTN (COPD)
• P-Mitrale
– Broad notched
– LA enlargement secondary to mitral valve
disease
P-wave Abnormalities
• Wolfe – Parkinson-White
– Ventricles activated early
– Short PR Interval
– Delta Wave
QRS Complex
• Depolarization of ventricles
• Larger Muscle Mass
• Amplitude as high as 25mm
• Duration with Normal Conduction <.10
• Amplitudes >25mm can mean chamber
enlargement as in ventricular hypertrophy
QRS Complex
• Low Amplitude
– Diffuse, severe coronary artery disease
– Pericardial Effusion
– Hypothyroid
QRS Complex
• 1st Negative deflection = Q Wave
• 1st Positive deflection = R wave
• Negative deflection after R wave = S wave
• Positive deflection after R wave = R Prime
• Negative deflection after S wave = S Prime
ST Segment
• Time between completion of depolarization
and onset of repolarization
– Normally isoelectric & gently blends into
upslope of T wave
– Point where ST takes off from QRS= J point
• Plays important role in diagnosis of
ischemic heart disease
ST Segment
• ST Elevation = hallmark of AMI
• Slight elevation across entire tracing is
normal especially in young males
• ST DEPRESSION – indicative of a # of
conditions . . . Ischemia, ventricular
hypertrophy
T - Wave
• Repolarization of the ventricles
• Same direction as predominant QRS
deflection
• Abnormalities – usually inversion with
BBB, hypertrophy or AMI
QT Interval
• Beginning of QRS to end of T Wave
• Normal variations with HR and gender
• Abnormalities
– Prolonged – commonly from drugs like Procan
or Quinidine or electrolyte imbalance
– Increased opportunity for R on T, ventricular
re-entry rhythms and sudden death
Vectors and Lead Systems
• Arrows represent direction as well as
amplitude
Vectors
• Vector 1 – depolarization of atrial
(corresponds to P wave)
• Vector 2 – Ventricular Septum (1st
deflection of QRS)
• Vector 3 – Bulk of ventricular muscle
• Vector 4 – Repolarization of ventricular
muscle
Limb Leads
• Look at heart in Frontal Planes
• Used to locate axis
• V Leads look at heart in Transverse Plane
Lead Placement
• Correct placement a must
• Small changes in height of R wave are
important
• Can be produced with slight movement of
leads
Lead Placement
• V1 – Right Sternal Border – 4th ICS
• V2 – Left Sternal Border – 4th ICS
• V3 Midway Between V2 and V4
• V4 Midclavicular line – 5th ICS
• V5 Anterior Axillary line – 5th ICS
• V6 Mid axillary line – 5th ICS
Determining Axis
• Impulse toward electrode = Positive
• Impulse away from electrode = Negative
• The more directly toward or away the
greater the amplitude either positive or
negative
Axis Deviation
• Normal Axis = 60 Degrees (0-90)
• Further counter clockwise than 0 = Left
Axis Deviation
• Further clockwise than 90 = Right Axis
Deviation
• > -30 Marked LAD
• >-120 Marked RAD
Axis Deviation
• Determined by Tallest R Wave
– Normal is Lead II
• PVC’s or VT from Right Ventricle = LAD
• PVC’s or VT from Left Ventricle = RAD
R-Wave Progression
• V1 is small – progressively increasing from
right to left until QRS fully upright in V5
and V6
• Point where QRS becomes biphasic =
transition zone
• R wave progression is frequently lost in
Anterior Wall Infarction
Bundle Branch Blocks
• Incomplete – Conducts slowly
– QRS between .10 and .12
• Complete – Total failure of affected bundle
to conduct impulse
– QRS >.12
Bundle Branch Blocks
• Right Bundle Branch Blocks
– Reverse normal pattern of negative QRS in V1
– RSR in V1
– Wide S wave in V5 and V6
Left Bundle Branch Block
• RSR in V5 and V6
• Deep negative QS in V1 and V2
• Causes Widespread ST Changes
• Non-Diagnostic for ischemia and infarction
Myocardial Infarction
Coronary Anatomy
Myocardial Infarction
• Usually result of clot formation at site of
fixed lesion
Hallmark of Infarction
• Transmural – full thickness of myocardial
wall
– ST Elevation
– T Wave Inversion
– Q Wave Formation
Inferior Wall Infarction
• Leads II, III and aVF
• Reciprocal Changes in Anterior Wall
• Most common Presentation is Bradycardia
• Can be associated with RV Infarction
Old Inferior Wall MI
Anterior Wall Infarction
• V2, V3 through V4
• Loss of R Wave progression
• Reciprocal Depression in leads of inferior
wall
Lateral Wall
• I and aVL
• V5 and V6
• Usually associated with another infarction
ST Elevation
• QRS Dos not return to baseline (J-point)
• 2 or more leads looking at the same wall
• Acute Event
T Wave Inversion
• Frequently bi-phasic
• Same leads as ST elevation
• Still in “process of infarcting”
Q Wave
• Ceases to depolarize
• Essentially electrically inert
• Permanent
Sneaky Causes of ST Elevation
• PERICARDITIS
– Widespread
– No reciprocal changes
– PR Segment Depression

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12-lead EKG Interpretation1.pdf

  • 1. 12-Lead EKG Interpretation Judith M. Haluka BS, RCIS, EMT-P
  • 2. ECG Grid • Left to Right = Time/duration • Vertical – measure of voltage (amplitude) – Expressed in mm
  • 3. P-Wave • Depolarization of atrial muscle • Low voltage (2-3mm in amplitude) • Duration <.11 seconds
  • 4. Abnormal P Waves • P – Pulmonale – Tall Peaked – Right atrial enlargement secondary to pulmonary HTN (COPD) • P-Mitrale – Broad notched – LA enlargement secondary to mitral valve disease
  • 5.
  • 6. P-wave Abnormalities • Wolfe – Parkinson-White – Ventricles activated early – Short PR Interval – Delta Wave
  • 7.
  • 8. QRS Complex • Depolarization of ventricles • Larger Muscle Mass • Amplitude as high as 25mm • Duration with Normal Conduction <.10 • Amplitudes >25mm can mean chamber enlargement as in ventricular hypertrophy
  • 9. QRS Complex • Low Amplitude – Diffuse, severe coronary artery disease – Pericardial Effusion – Hypothyroid
  • 10. QRS Complex • 1st Negative deflection = Q Wave • 1st Positive deflection = R wave • Negative deflection after R wave = S wave • Positive deflection after R wave = R Prime • Negative deflection after S wave = S Prime
  • 11.
  • 12. ST Segment • Time between completion of depolarization and onset of repolarization – Normally isoelectric & gently blends into upslope of T wave – Point where ST takes off from QRS= J point • Plays important role in diagnosis of ischemic heart disease
  • 13. ST Segment • ST Elevation = hallmark of AMI • Slight elevation across entire tracing is normal especially in young males • ST DEPRESSION – indicative of a # of conditions . . . Ischemia, ventricular hypertrophy
  • 14. T - Wave • Repolarization of the ventricles • Same direction as predominant QRS deflection • Abnormalities – usually inversion with BBB, hypertrophy or AMI
  • 15. QT Interval • Beginning of QRS to end of T Wave • Normal variations with HR and gender • Abnormalities – Prolonged – commonly from drugs like Procan or Quinidine or electrolyte imbalance – Increased opportunity for R on T, ventricular re-entry rhythms and sudden death
  • 16. Vectors and Lead Systems • Arrows represent direction as well as amplitude
  • 17. Vectors • Vector 1 – depolarization of atrial (corresponds to P wave) • Vector 2 – Ventricular Septum (1st deflection of QRS) • Vector 3 – Bulk of ventricular muscle • Vector 4 – Repolarization of ventricular muscle
  • 18.
  • 19. Limb Leads • Look at heart in Frontal Planes • Used to locate axis • V Leads look at heart in Transverse Plane
  • 20. Lead Placement • Correct placement a must • Small changes in height of R wave are important • Can be produced with slight movement of leads
  • 21. Lead Placement • V1 – Right Sternal Border – 4th ICS • V2 – Left Sternal Border – 4th ICS • V3 Midway Between V2 and V4 • V4 Midclavicular line – 5th ICS • V5 Anterior Axillary line – 5th ICS • V6 Mid axillary line – 5th ICS
  • 22.
  • 23. Determining Axis • Impulse toward electrode = Positive • Impulse away from electrode = Negative • The more directly toward or away the greater the amplitude either positive or negative
  • 24.
  • 25. Axis Deviation • Normal Axis = 60 Degrees (0-90) • Further counter clockwise than 0 = Left Axis Deviation • Further clockwise than 90 = Right Axis Deviation • > -30 Marked LAD • >-120 Marked RAD
  • 26.
  • 27. Axis Deviation • Determined by Tallest R Wave – Normal is Lead II • PVC’s or VT from Right Ventricle = LAD • PVC’s or VT from Left Ventricle = RAD
  • 28.
  • 29. R-Wave Progression • V1 is small – progressively increasing from right to left until QRS fully upright in V5 and V6 • Point where QRS becomes biphasic = transition zone • R wave progression is frequently lost in Anterior Wall Infarction
  • 30.
  • 31.
  • 32. Bundle Branch Blocks • Incomplete – Conducts slowly – QRS between .10 and .12 • Complete – Total failure of affected bundle to conduct impulse – QRS >.12
  • 33. Bundle Branch Blocks • Right Bundle Branch Blocks – Reverse normal pattern of negative QRS in V1 – RSR in V1 – Wide S wave in V5 and V6
  • 34.
  • 35. Left Bundle Branch Block • RSR in V5 and V6 • Deep negative QS in V1 and V2 • Causes Widespread ST Changes • Non-Diagnostic for ischemia and infarction
  • 36.
  • 37.
  • 38.
  • 41. Myocardial Infarction • Usually result of clot formation at site of fixed lesion
  • 42. Hallmark of Infarction • Transmural – full thickness of myocardial wall – ST Elevation – T Wave Inversion – Q Wave Formation
  • 43.
  • 44. Inferior Wall Infarction • Leads II, III and aVF • Reciprocal Changes in Anterior Wall • Most common Presentation is Bradycardia • Can be associated with RV Infarction
  • 45.
  • 47. Anterior Wall Infarction • V2, V3 through V4 • Loss of R Wave progression • Reciprocal Depression in leads of inferior wall
  • 48.
  • 49.
  • 50.
  • 51. Lateral Wall • I and aVL • V5 and V6 • Usually associated with another infarction
  • 52. ST Elevation • QRS Dos not return to baseline (J-point) • 2 or more leads looking at the same wall • Acute Event
  • 53. T Wave Inversion • Frequently bi-phasic • Same leads as ST elevation • Still in “process of infarcting”
  • 54. Q Wave • Ceases to depolarize • Essentially electrically inert • Permanent
  • 55.
  • 56. Sneaky Causes of ST Elevation • PERICARDITIS – Widespread – No reciprocal changes – PR Segment Depression