3. Crush Injury Syndrome
• Crush injury is compression of extremities or other parts of the body that
causes muscle swelling and/or neurological disturbances in the affected
areas of the body, while crush syndrome is localized crush injury with
systemic manifestations
4.
5. Clinical feature
The compromised limb is pulseless and becomes red, swollen and
blistered; sensation and muscle power may be lost
Treatment
Fasciotomy is indicated → Compartment syndrome
Excision of dead muscle must be radical to avoid sepsis
Open wound should be managed aggressively.
7. Fat Emboli: Fat particles or droplets that travel
through the circulation
Fat Embolism: A process by which fat emboli passes
into the bloodstream and lodges within a blood
vessel.
Fat Embolism Syndrome (FES): serious
manifestation of fat embolism occasionally
causes multi system dysfunction, the lungs are always
involved and next is brain
9. Pulmonary
Hypoxia, rales, pleural friction rub
CXR usually normal early on, later may show
‘snowstorm’ pattern- diffuse bilateral infiltrates
CT chest: ground glass opacification with interlobular
septal thickening.
10. Neurological findingsUsually occur after respiratory symptoms
Incidence- 80% patients with FES
Minor global dysfunction is most common-ranges from mild
delirium to coma.
Seizures/focal deficits
Transient and reversible in most cases.
CT Head: general edema, usually nonspecific
MRI brain: Low density on T1, and high intensity T2 signal, correlates
to degree of impairment.
11. Dermatological findings
Petechie
Usually on conjunctiva, neck, axilla, upper limbs.
Results from occlusion of dermal capillaries by fat
globules and then extravasations of RBC.
Resolves in 5-7 days. Usually fast resolving.
Pathognomic, but only present in 20-50% of patients.
15. Treatment and management
Immobilization and early internal fixation of
fracture.
Continuous pulse oximeter monitoring in high- risk
patients may help in detecting desaturation early,
allowing early institution of oxygen
High dose steroid