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A CASE STUDY OF GID IN GOAT
Clinical practice:
 Case Presentation
Presented by : Chet Raj Pathak (Roll No.
09)
B.V.Sc.&A.H. VIIIth Semester,
TU/IAAS, Rampur, Chitwan
1
 Name of the owner : Kashi Tamang
 Address : Fulbari-7,chitwan
 Date : 2071-09-17
 Case species : Caprine
 Breed : khari X
 Age : 18 months
2
Case history
 Circling movements since two weeks
 Slight decreased in feed intake
 Body weight reduced relatively
 Rest by keeping head against the wall
 Pastured in local fields
 Deworming was not adopted since 6
months
3
Clinical signs
 Restlessness : circling movement (often)
 Rest : pressing head against the wall
 Appetite : reduced (slightly)
 Body : emaciated, dehydrated
 Body temperature :102.2⁰ F
 Pulse rate: 68 per minute
 Respiratory rate: 25 per minute
4
 Tentative diagnosis:
“Coenurosis”
 The case was followed up to 2 months
 Death : seriously injured in an accident
and hip joint dislocation
5
Postmortem findings
peritonea
l layer
6
Coenurus cyst
mesentery
7
Intestinal
walls
8
Diagnosis
 “Coenurosis” was diagnosed
On the basis of history , clinical signs and
PM findings
By Dr. Shyam Bdr. Raut
9
Treatment
 FENTAS PLUS
Sig: 1 tab orally at haust
 LIV-52 VET
Sig: 10 ml OD orally* 7 days
10
R
Discussion
Introduction:
 Disease of the central nervous system of
ruminants ,horses, pigs and humans
 caused by Coenurus cerebralis, the
larvalstage of the (Taenia multiceps )
 Tapeworm inhabits the small intestine of
the definitive hosts like dogs and wild
canids, such as foxes, jackals and
coyotes
Soulsby 1982
11
Etiology
 Coenurus cerebralis (coenurus cyst)
larval stage/ metacestode of T.multiceps
Cyst measures 0.5–5cm in diameter
- Translucent walls: containing large
fluid with several hundred protoscolices
- Lying free on the leptomeninges or
partly penetrating the brain tissue and spinal
cord (primary site)
-may present in intramuscular and
subcutaneous tissues
12
Host related facts
 Adult stage of parasite is harbored by
canine species in the GIT (they acts as
definitive hosts)
 Larval stage is found in small
ruminants(most commonly in sheep CNS
and IM/SC in goats)
 Other species like bovine, equine, swine
and human may be infected with the
metacestode of parasite
13
Life cycle of parasite
Meat
containing
coenurus
cyst
Ingestion by
canids (farm
or wild
Definitive
hosts
:Developm
ent of adult
tapewormGravid
segment of
parasite
source of
egg
shedding
Contaminat
ed pasture
Ingestion of
pasture
Intermediate
hosts
14
Risk factors
 Contamination level of environment
 Dispersion of eggs
 Eggs survival
 Age of host
 Immune response of host
 Heterologous infection
15
Pathogenesis and clinical signs
Ingestion of
contaminated
pasture
large number of
immature stages
migrate in the brain
an acute
meningoencephalitis
Chronic Infection is
seen in1-6 months
by increase in
coenurus size and
destruction of brain
tissue
16
The cyst is
situated in
peripheral
region in one
of cerebral
hemisphere
Animal holds
its head to
one side and
turns in circle
towards the
affected side
Blind in the
eye opposite
to the side of
infection
17
Cont..
cont..
Parasite in the
anterior part
Head is held against
chest and animal
steps high or may
walk in a straight line
till it meets obstacle
and remain
motionless
18
Cont..
Invasion of
cerebellum:
hyperesthesia
,jerky or
staggers
movements
and may
prostration
In spinal
cord:
progressive
paresis of
one or both
limbs
On the
surface of
brain :
atrophied or
perforated
skull bone
19
20
Fig :- small foci or extensive
areas of purulent exudate
usually adjacent to the cysts in
the leptomeninges
Fig :-Two cysts : one in the
parietal and one in the
occipital lobe
A third cyst : attached to the
tectum.
Source: google
Diagnosis
 Diagnosis by specific serological test is
not available
 An early diagnosis prior to clinical sign
development is done by ophthalmoscopy
of eye
 Perforated part can localize by painful
manipulation or for accurate localization
radiography is suggested
21
Treatments
 In many cases Rx is useless
 If cyst is present in the brain : surgical
removal is possible after general
anaesthesia and recovery is good
especially if localization of cyst is
accurate
 Combination of the praziquental and
fenbendazole may helpful
 It is found that many farmers may
choose to slaughter those fit for
marketing for economic reasons and
euthanise those in poor condition.
22
Control
 Use of tenicidal drug for all farm dogs to
break life cycle of parasite
 Carcass of animal infected with cyst
should be disposed safely and should not
be available to dogs
23
Conclusion
 Since, the critical clinical stage the
disease is incurable
 But the disease can be controlled by the
alteration of pasture
 Effective deworming of both definitive and
intermediate hosts of parasite
24
References
 Tirgari, M. et al. ,(1987) : Vet.Rec. 120:173.
 Chakrabarti, A.(2012) : A Textbook of Preventive
Veterinary Medicine, 5th Edt. Kalyani Publishers,
New Delhi.
 Soulsby, E.J.L.(1982) : Helminthes, Arthropods
and protozoa of Domesticated Animals, 7th
Edit. The English Language Book Society
and Bailliera tindaii London.
 Radostits, O.M., et al., (2000) : Veterinary medicine,
9th Edit. W.B. Saunders.
 Sausan, E.A. (1998) : The Mercks Veterinary
Manual, 8th Edition
25
Acknowledgements
 Assoc. Prof. Dr. Krishna Kaphle(Dept. of
Theriogenology , TU/IAAS, Rampur, Chitwan
 Dr. Shyam Bdr. Raut (Senior Doctor of VTH Rampur)
 Ishwor dhakal, B.V.Sc. & A.H. 8th semester
 Dr. Dipak kathayet, Aabhas Paudel ,Suresh Nepali,
Suhel K.C.
26
27

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A CASE STUDY OF COENUROSIS IN GOAT VTH CHITWAN

  • 1. A CASE STUDY OF GID IN GOAT Clinical practice:  Case Presentation Presented by : Chet Raj Pathak (Roll No. 09) B.V.Sc.&A.H. VIIIth Semester, TU/IAAS, Rampur, Chitwan 1
  • 2.  Name of the owner : Kashi Tamang  Address : Fulbari-7,chitwan  Date : 2071-09-17  Case species : Caprine  Breed : khari X  Age : 18 months 2
  • 3. Case history  Circling movements since two weeks  Slight decreased in feed intake  Body weight reduced relatively  Rest by keeping head against the wall  Pastured in local fields  Deworming was not adopted since 6 months 3
  • 4. Clinical signs  Restlessness : circling movement (often)  Rest : pressing head against the wall  Appetite : reduced (slightly)  Body : emaciated, dehydrated  Body temperature :102.2⁰ F  Pulse rate: 68 per minute  Respiratory rate: 25 per minute 4
  • 5.  Tentative diagnosis: “Coenurosis”  The case was followed up to 2 months  Death : seriously injured in an accident and hip joint dislocation 5
  • 9. Diagnosis  “Coenurosis” was diagnosed On the basis of history , clinical signs and PM findings By Dr. Shyam Bdr. Raut 9
  • 10. Treatment  FENTAS PLUS Sig: 1 tab orally at haust  LIV-52 VET Sig: 10 ml OD orally* 7 days 10 R
  • 11. Discussion Introduction:  Disease of the central nervous system of ruminants ,horses, pigs and humans  caused by Coenurus cerebralis, the larvalstage of the (Taenia multiceps )  Tapeworm inhabits the small intestine of the definitive hosts like dogs and wild canids, such as foxes, jackals and coyotes Soulsby 1982 11
  • 12. Etiology  Coenurus cerebralis (coenurus cyst) larval stage/ metacestode of T.multiceps Cyst measures 0.5–5cm in diameter - Translucent walls: containing large fluid with several hundred protoscolices - Lying free on the leptomeninges or partly penetrating the brain tissue and spinal cord (primary site) -may present in intramuscular and subcutaneous tissues 12
  • 13. Host related facts  Adult stage of parasite is harbored by canine species in the GIT (they acts as definitive hosts)  Larval stage is found in small ruminants(most commonly in sheep CNS and IM/SC in goats)  Other species like bovine, equine, swine and human may be infected with the metacestode of parasite 13
  • 14. Life cycle of parasite Meat containing coenurus cyst Ingestion by canids (farm or wild Definitive hosts :Developm ent of adult tapewormGravid segment of parasite source of egg shedding Contaminat ed pasture Ingestion of pasture Intermediate hosts 14
  • 15. Risk factors  Contamination level of environment  Dispersion of eggs  Eggs survival  Age of host  Immune response of host  Heterologous infection 15
  • 16. Pathogenesis and clinical signs Ingestion of contaminated pasture large number of immature stages migrate in the brain an acute meningoencephalitis Chronic Infection is seen in1-6 months by increase in coenurus size and destruction of brain tissue 16
  • 17. The cyst is situated in peripheral region in one of cerebral hemisphere Animal holds its head to one side and turns in circle towards the affected side Blind in the eye opposite to the side of infection 17 Cont..
  • 18. cont.. Parasite in the anterior part Head is held against chest and animal steps high or may walk in a straight line till it meets obstacle and remain motionless 18
  • 19. Cont.. Invasion of cerebellum: hyperesthesia ,jerky or staggers movements and may prostration In spinal cord: progressive paresis of one or both limbs On the surface of brain : atrophied or perforated skull bone 19
  • 20. 20 Fig :- small foci or extensive areas of purulent exudate usually adjacent to the cysts in the leptomeninges Fig :-Two cysts : one in the parietal and one in the occipital lobe A third cyst : attached to the tectum. Source: google
  • 21. Diagnosis  Diagnosis by specific serological test is not available  An early diagnosis prior to clinical sign development is done by ophthalmoscopy of eye  Perforated part can localize by painful manipulation or for accurate localization radiography is suggested 21
  • 22. Treatments  In many cases Rx is useless  If cyst is present in the brain : surgical removal is possible after general anaesthesia and recovery is good especially if localization of cyst is accurate  Combination of the praziquental and fenbendazole may helpful  It is found that many farmers may choose to slaughter those fit for marketing for economic reasons and euthanise those in poor condition. 22
  • 23. Control  Use of tenicidal drug for all farm dogs to break life cycle of parasite  Carcass of animal infected with cyst should be disposed safely and should not be available to dogs 23
  • 24. Conclusion  Since, the critical clinical stage the disease is incurable  But the disease can be controlled by the alteration of pasture  Effective deworming of both definitive and intermediate hosts of parasite 24
  • 25. References  Tirgari, M. et al. ,(1987) : Vet.Rec. 120:173.  Chakrabarti, A.(2012) : A Textbook of Preventive Veterinary Medicine, 5th Edt. Kalyani Publishers, New Delhi.  Soulsby, E.J.L.(1982) : Helminthes, Arthropods and protozoa of Domesticated Animals, 7th Edit. The English Language Book Society and Bailliera tindaii London.  Radostits, O.M., et al., (2000) : Veterinary medicine, 9th Edit. W.B. Saunders.  Sausan, E.A. (1998) : The Mercks Veterinary Manual, 8th Edition 25
  • 26. Acknowledgements  Assoc. Prof. Dr. Krishna Kaphle(Dept. of Theriogenology , TU/IAAS, Rampur, Chitwan  Dr. Shyam Bdr. Raut (Senior Doctor of VTH Rampur)  Ishwor dhakal, B.V.Sc. & A.H. 8th semester  Dr. Dipak kathayet, Aabhas Paudel ,Suresh Nepali, Suhel K.C. 26
  • 27. 27