the presentation includes basic information of septic arthritis and gives an idea about its general management, microorganism causing it, and general symptoms. The information is categorically placed into different subtitles.
2. INTRODUCTION
• Pathological joint space invasion
by microbes followed by
inflammation
OR
• Inflammation of synovial
membrane due to infection
which results in purulent
effusion in joint capsule
4. SPREAD OF INFECTION TO JOINT CAPSULE
INFLAMMATORY REACTION WITH SEROPURULENT EXUDATE
INCREASE IN SYNOVIAL FLUID
ARTICULAR CARTILAGE IS ERODED AND
DESTROYED
5.
6. CAUSATIVE MICROORGANISM
STAPH. AUREUS (MC)
NEONATES AND YOUNGER INFANTS GROUP B STREPTOCOCCI AND GRAM NEGATIVE
ENTERIC BACILLI
SEXUALLY ACTIVE ADOLESCENTS AND ADULTS N. GONORRHOEA
IV DRUG ABUSER, IMMUNOCOMPRAMISED GRAM NEGATIVE BACILLI LIKE E. COLI AND
PSEUDOMONAS
SICKLE CELL ANEMIA SALMONELLA
PROSTHETIC JOINT RECEIPIENTS CoNS E. g. STAPH EPIDERMIS
POLYARTICULAR STAPHYLOCOCCUS > STREPTOCOCCI GROUP G
OTHER M. TUBERCULOSIS
8. CLINICAL FEATURES
INFANT
FEATURES OF SEPTICEMIA ARE MORE AS
COMPARED TO JOINT PAIN
JOINT IS WARMTH, RESISTANT TO
MOVEMENT, TENDERNESS
CHILDREN
MONOARTICULAR
REDNESS AND SUPERFICIAL JOINT
SWELLING VISIBLE
LOCAL WARMTH AND TENDERNESS
RESTRICTION OF MOVEMENTS
9. CLINICAL FEATURES
ADULTS
• JOINT IS PAINFUL, SWOLLEN AND INFLAMED
• DECREASED ABILITY TO MOVE THE JOINT
• SYSTEMIC SYMPTOMS LIKE FEVER WITH CHILLS,
MALAISE, POOR APPETITE, IRRITABILITY
• SUPERFICIAL JOINTS ARE AFFECTED MORE
11. KOCHER’S CRITERIA
VARIABLE INCLUDES
1. NON WEIGHT BEARING ON AFFECTED SIDE
2. ERYTHROCYTE SEDIMENTATION RATE > 40
3. FEVER > 38.5 C
4. WHITE BLOOD CELL COUNT > 12000
SCORE LIKELIHOOD OF SEPTIC ARTHRITIS
1 3 %
2 40 %
3 93 %
4 99%
12. TOM SMITH ARTHRITIS
• SEPTIC ARTHRITIS OF HIP IN INFANCY
• HEAD OF FEMUR IS RAPIDLY DESTROYED BY PYOGENIC PROCESS.
• CAUSE:
- METAPHYSIS IS INTRASYNOVIAL
- PRESENCE OF TRANSEPIPHYSEAL VESSEL IN CHILDREN TILL THE AGE OF 18 MONTHS
• PATHOPHYSIOOGY
- INCREASED INTRAARTIULAR PRESSURE
- DIRECT DESTRUCTIVE ACTION OF PUS ON ARTICULAR CARTILAGE
- ISCHEMIA TO GROWTH PLATE DUE TO THROMBOSIS ON THE BOTH SIDE OF THE PLATE
• TREATMENT
- AS SOON AS PUS IS ASPIRATED, DRAINAGE OF JOINT TO PREVENT DAMAGE TO HIP
- RECONSTRUCTIVE HIP PROCEDURE PLANNED LATER
13. TUBERCULAR ARTHRITIS
• LESS COMMON
• IT CAN RESULT FROM
1. Actual infection from M. Tuberculosis which
presents as monoarticular infection
2. From an allergic phenomenon which present as
polyarthritis.
14. INVESTIGATIONS
1. X RAY
NON SPECIFIC
INITIAL STAGE- X RAY ARE NORMAL, GROUND GLASS APPEARANCE
WITH LOSS OF SOFT TISSUE PLANES SEEN
16. 2. USG
• More reliable
• It can diagnose joint effusion in early stages.
• Detects effusion and pus in joint with synovial thickening.
• It can identify plane of collection so that we can differentiate it from
cellulitis and bursitis.
• ECHO FREE – TRANSIENT SYNOVITIS
• POSITIVE ECHOGENICITY – SEPTIC ARTHRITIS
17. 3. SYNOVIAL FLUID ANALYSIS
• NORMAL: HIGH VISCOUS, COLOURLESS, TRANSPARENT
• SEPTIC ARTHITIS:
GROSS : HIGH VISCOSITY AND COLOUR YELLOW OR STROW YELLOW,
OPAQUE
LAB EXAMINATION: WBC COUNT INCREASES,
CULTURE MAY BE POSITIVE
SUGAR LEVEL REDUCED
18. TREATMENT
• SPLINTAGE
The joint must be held in most comfortable position either on splint or
plaster
• GENERAL CARE
Analgesics and iv fluids
19. ANTIBIOTICS
Once the samples are obtained, antibiotics should be started without
without waiting for result
Later they should be changed as per the final report
NEISSERIA (GRAM NEGATIVE DIPLOCOCCI) CEFTRIAXONE
MSSA CEFTRIAXONE
MRSA VANCOMYCIN OR CLINDAMYCINE OR LINEZOLID
STREPTOCOCCUS CEFTRIAXONE
E. COLI, PROTEUS, SERRATIA PIPTAZ
CEFTAZIDIME
PROSTHESIS RELATED (CoNS) THIRD GENERATION CEPHALOSPORIN + VANCOMYCIN