2. PULMONARY EMBOLISM
Pulmonary embolism is a blockage of an artery in the lungs by a
substance that has moved from elsewhere in the body through the
bloodstream (embolism).
Occurs when a thrombus forms (most commonly in a deep vein),
detaches, travels to the right side of the heart, and then lodges in a
branch of the pulmonary artery
4. ETIOLOGICAL FACTORS
Clients prone to pulmonary embolism are those at risk for deep
vein thrombosis, including those with prolonged immobilization,
surgery, obesity, pregnancy, heart failure, advanced age, or a
history of thromboembolism.
Fat embolican occur as a complication following fracture of a
long bone and can cause pulmonary emboli.
5.
6. CLINICAL MANIFESTATION
Apprehension and restlessness
Blood-tinged sputum
Chest pain
Cough
Crackles and wheezes on auscultation
Cyanosis ▪ Distended neck veins
7. CLINICAL MANIFESTATION
Dyspnea accompanied by anginal and pleuritic pain, exacerbated
by inspiration
Feeling of impending doom
Hypotension
Petechiae over the chest and axilla
Shallow respirations
Tachypnea and tachycardia
8. DIAGNOSTIC EVALUATION
Detailed history collection
Physical examination
Spo2
ABG analysis
ECG
CT pulmonary angiography
Lung ventilation/perfusion
scan
Ultrasound of the legs may
confirm the diagnosis.
Deep vein thrombosis and pe
are known as venous
thromboembolism
9. MANAGEMENT
Notify the Rapid Response Team and healthcare provider (HCP).
Reassure the client and elevate the head of the bed.
Prepare to administer oxygen.
Obtain vital signs and check lung sounds.
Prepare to obtain an arterial blood gas.
Prepare for the administration of heparin therapy or other
therapies.
Document the event, interventions taken, and the client’s
response to treatment.
10. Treatment is thinning the blood with anticoagulants such as
heparin, low molecular weight hepar, direct thrombin inhibitors
The oral blood thinner, warfarin. Immediately acting intravenous
or injected blood thinner must be administered right away.
The oral blood thinner, warfarin, takes about five days to be
come effective to prevent the development of a recurrent PE.
In addition to blood thinners, more aggressive therapies include
“clot buster” drugs such as TPA or catheter-based or surgical
embolectomy to remove the PE.