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1. Alzheimer’s Disease
Alzheimer’s disease (AD) is a nonreversible type of dementia
that progressively develops over many years.
Dementia is defined as multiple cognitive deficits that impair
memory and can affect language, motor skills, and/or abstract
thinking.
2. Risk Factors
Advanced age
Genetic predisposition
Environmental agents (herpes virus, metal, or toxic waste)
Previous head injury
Sex (female)
3. Clinical manifestation
AD is characterized by
Memory Loss,
Problems With Judgment
Changes In Personality
Forgetfulness
Difficulty in social or work situations
Loss of awareness of recent events and surroundings
movement is lost › Unrecognizable speech(at sever stage)
4. Investigation
Genetic testing for the presence of apolipoprotein E can determine if late-
onset dementia is due to AD.
There is no definitive diagnostic procedure, except brain tissue examination
upon death.
Magnetic resonance imaging (MRI), computed tomography (CT) and
electroencephalogram (EEG) may be performed to rule out other possible
causes of findings
5. Medications
Most medications for clients who have dementia attempt to target behavioral
and emotional problems, such as anxiety and depression.
These medications include antipsychotics, antidepressants, and anxiolytics.
Clients receiving these medications should be closely monitored for adverse
effects.
AD medications temporarily slow the course of the disease and do not work
for all clients.
If a client fails to improve with one medication, a trial of one of the other
medications is warranted.
6. Nursing Care
Assess cognitive status, memory, judgment, and personality changes.
Provide a safe environment.
Provide verbal and nonverbal ways to communicate with the client.
Check the client’s skin weekly for breakdown.
7. Cont.…
Offer varied environmental stimulations, such as walks, music, or craft
activities.
Keep a structured environment and introduce change gradually (client’s
daily routine or a room change).
Use a calendar to assist with orientation.
Use short directions when explaining an activity or care the client
needs, such as a bath.
8. Cerebral Aneurysm
A cerebral aneurysm is a balloon-like out-pouching caused by a
congenital or developed weakness in a cerebral artery.
Trauma, infection, or vessel wall lesions due to atherosclerosis
can all lead to the development of an aneurysm.
Increased pressure within the vessel lumen may cause the
aneurysm to rupture, causing significant intracranial bleeding
9.
10. SIGNS AND SYMPTOMS
Asymptomatic until rupture
Very bad headache due to hemorrhage and increased intracranial
pressure
Decreased level of consciousness due to increased intracranial
pressure from blood accumulating within the brain
11. INTERPRETING TEST RESULTS
CT scan shows the aneurysm unless it is very small.
Diffusion/perfusion MRI or MRA (magnetic resonance angiography)
shows vessel structure.
12. Medical TREATMENT
Surgical repair of the aneurysm.
Administer corticosteroid drugs to reduce inflammation:
Administer anticonvulsant drugs to reduce seizure risk due to
irritation of brain:
13. NURSING INTERVENTION
Monitor the patient’s neurological function for changes
typically use Glasgow Coma Scale or similar tool to grade response
to stimuli/
Monitor vital signs for changes
enlarged pulse pressure with bradycardia indicative of increased
intracranial pressure.
Explain to the patient:
Needs for homecare.
When to call healthcare provider.
14.
15. Hemiplegia
• Hemiplegia is total paralysis of the arm, leg, and trunk on the same
side of the body.
• Severe or complete loss of motor function on one side of the body.
• Hemiplegia is more severe than hemiparesis, wherein one half of
the body has less marked weakness.
• Hemiplegia may be congenital or acquired from an illness or stroke.
17. Signs & Symptoms
• Vary greatly from person to person.
Difficulty with gait
Difficulty with balance while standing or walking
Having difficulty with motor activities like holding, grasping or
pinching
Increasing stiffness of muscles
Muscle spasms
Difficulty with speech
18. Cont.…
Difficulty swallowing food
Significant delay during standing, smiling,
crawling or speaking
The majority of children who develop hemiplegia also
have abnormal mental development.
Behavior problems like anxiety, anger, irritability, lack of
concentration or comprehension
Emotions — depression
20. Management
◾ Frequent change of the patient's position (every 2 hours),
and of the bed sheets.
◾ Frequent wash of the skin of the back and pressure points.
◾ Suction of nasal and pharyngeal secretions.
◾ Oxygen administration in case of comatose
21. Cont.…
Tube feeding;- giving fruit juices, milk and pureed food,
besides I.V. fluids, in comatose patients.
Catheterization to prevent bed sore and urine retention
by subcutaneous injection of anticoagulant and wearing
elastic stocks to prevent DVT.
Treat specific causes of Hemiplegia accordingly.
23. DEFINITION
• Guillain Barre syndrome is a rare disorder in
which body's immune system attacks nerves and
causes damage to the peripheral nerves.
• The nerve injury often causes muscle weakness,
cause paralysis and sensitivity problems,
including pain, tingling or numbness.
26. Clinical Manifestations
• Hyporeflexia and weakness progress and may result in quadriplegia.
• Neuromuscular respiratory failure - demyelination of the nerves that
innervate the diaphragm and inter costal muscles results.
• Sensory dysfunction with abnormal proprioception, sensory ataxia can also
occur.
• Gloves and stocking paraesthesias with slight disturbances of sensibility.
27. • Weakness spreads to the arms and upper body.
• The weakness may increase until muscles cannot be used at all
and may result in paralysis.
• Inability to walk due to muscle weakness and paralysis.
• Difficult to speaking, chewing and swallowing, various muscles
required to form speech are weakened.
28. DIAGNOSIS
• Spinal tap (lumbar puncture). The fluid is tested for a type of
change that commonly occurs in people who have Guillain-Barre
syndrome.
• Electromyography. The electrodes measure nerve activity in the
muscles.
• Nerve conduction studies. Electrodes are taped to the
peripheral nerves. A small shock is passed through the nerve to
measure the speed of nerve signals.
• CSF analysis – elevated protein content upto 700 mg/dl. Normal-
15-45 mg/dl
29. MANAGEMENT
• Supportive care
• Ventiltory support
• Plasmapheresis (plasma exchange)– to remove
abnormal antibodies
• IV Immunoglobulin's – stop antibody damaging
cells.
30. NURSING MANAGEMENT
• Check for facial nerve paralysis.
• Inspect the patient’s face at rest and during conversation.
• Assess for any problems during swallowing, talking and chewing.
• Assess for any change in the vital signs.
• Maintaining respiratoryfunction.
31. Cont.…
Mechanical ventilation
Close monitoring and suctioning
Enhancing physical mobility
Providing adequate nutrition IV fluids and parenteral nutrition
monitors for the return of bowel sounds gastrostomy tube assesses the return
of the gag reflex
Monitoring and managing potential complications Thorough assessment of
respiratory function at regular intervals cardiac dysrhythmias, which
necessitate ECG monitoring
32. COMPLICATIONS
• Complications can also include:
– Persistent weakness, numbness, or other odd
sensations even after recovery
– Heart or blood pressure problems
– Pain
– Slow bowel or bladder function
– Blood clots and bedsores due to paralysis
Editor's Notes
Atherosclerosis is a hardening and narrowing of your arteries caused by cholesterol plaques lining the artery over time. It can put blood flow at risk as your arteries become blocked.
A transient ischemic attack (TIA) is a stroke that lasts only a few minutes. It happens when the blood supply to part of the brain is briefly blocked. Symptoms of a TIA are like other stroke symptoms, but do not last as long. They happen suddenly, and include: