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ECG Notes
Learning objectives
• Basics of ECG
• Establish consistent approach to interpreting ECGs
• Rate, rhythm, axis and identifying ischemia
• Review essential life- threatening cases
• Provide additional resources for future learning
2
Normal impulse conduction
• Sinoatrial node
• AV node
• Bundle of His
• Bundle branches
• Purkinje fibers
3
PQRST
• P wave = atrial depolarization = SA node
function
• PR interval = atrial depolarization + delay
in AV junction (AV node & bundle of His) =
delay allows time for the atria to contract
before the ventricles do
• QRS wave = ventricular depolarization
(muscle contraction)
• T wave = ventricular repolarization
4
Pacemakers of heart
• SA node: dominant pacemaker with an intrinsic rate of 60- 100 beats/ min
• AV node: back- up pacemaker with an intrinsic rate of 40- 60 beats/ min (= nodal rhythm = seen in
heart blocks)
• Ventricular cells: back- up pacemaker with an intrinsic rate of 20- 45 beats/ min (= last resort)
5
The ECG paper
• It is a thermal paper that records
electrical activity of the heart
• Horizontally we measure in seconds
(time)
• Vertically we measure in mm (amplitude)
• 1 small box = 0.04 seconds & 1 mm
• 1 big box = 0.2 seconds & 5 mm
• 1 big box has 5 small boxes up & down
6
Heart rate
• Every six seconds = 30 large boxes
• This helps when calculating the heart rate
• Heart rate = # of R waves x 10
• Normal resting HR:
• Newborn: 110- 150 bpm
• 2 years: 85- 125 bpm
• 4 years: 75- 115 bpm
• > 6 years (like an adult): 60- 100 bpm
• Adult: 50- 100 bpm
7
8
Steps of any rhythm analysis
1. Calculate rate: # R waves in 6 seconds x 10
2. Determine regularity: look at the distance between the R waves - use a caliper or marking on
pen/ paper. Are they equidistant apart, occasionally irregular, regularly irregular or irregularly
irregular?
3. Assess P waves
4. Determine PR interval
5. Determine QRS interval
9
Step 3: assess the P waves
• Are there P waves?
• Do all the P waves look alike?
• Do the P waves occur at a regular rate?
• Is there one P wave before each QRS?
• Normal = Normally shaped P waves with 1 P wave before each QRS
10
P mitrale = left atrial enlargement
• P mitrale indicates left atrial enlargement
• The wave is widened and has 2 peaks
• Indicates pulmonary hypertension and mitral stenosis
11
P pulmonale = right atrial enlargement
12
• May also occur in mitral stenosis or
pulmonary hypertension
• Note than in pulmonary HTN there
will also be inversion of T waves in V1
Step 4: determine PR interval
• Normal is 0.12- 0.20 seconds (3- 5
small boxes)
• PR should not be more than 5 small
squares
• Measure from start of P to start of R
13
Long PR interval
• First degree AV block
1. Drugs (all increase vagal tone or antagonize sympathetic system  can lead to sudden
death)
a. Calcium channel blockers
b. Beta blockers
c. Digoxin toxicity (can cause any arrhythmia, but mainly tachyarrhythmias in children &
bradyarrhythmias in adults)
d. Opioids
e. Clonidine
f. Sedative- hypnotics
2. Atrial surgery (scar tissue)
3. Acute rheumatic fever (minor Jones criteria)
4. Kawasaki disease
14
Short PR interval
1. Wolff- Parkinson- White syndrome
2. Pompe’s disease (glycogen storage disease IIa)
3. Fabry’s disease (heart + kidney)
4. GM- 1 gangliosides
5. Friedreich’s ataxia
6. Duchenne muscular dystrophy
15
WPW syndrome
16
Step 5: QRS wave
• Normal: 0.04- 0.12 seconds (1- 3 small boxes)
• QT interval will be short in digoxin toxicity and hypercalcemia
17
Long QT interval
• Congenital
1. Jervell- Lange- Nielsen syndrome: AR + deafness
2. Romano- Ward syndrome: AD + each generation will have a sudden death + normal hearing
• Acquired:
1. Metabolic:
a. Hypocalcemia
b. Hyperkalemia
c. Hypomagnesemia
d. Malnutrition
2. Drugs:
a. Type Ia and III anti- arrhythmics
b. Phenothiazines
c. TCAs
3. CNS trauma (brain edema  long QT  sudden death)
4. Myocardial ischemia or myocarditis
18
19
QTc calculation
• Normal must be < 0.40- 0.45 seconds
• Formula = Bazett formula
• < 0.45 s = normal
• 0.45- 0.48 s = doubtful
• 0.48- 0.50 s = highly suspicious of prolonged QT
• > 0.50 s = confirmed prolonged QT
• So just remember 1 small square = 0.04 s
• Your QT & RR in the formula will be the # of small
squares times 0.04   
20
21
22
Arrhythmia formation
• Arrhythmias can arise from problems in the:
• Sinus node
• Atrial cells
• AV junction
• Ventricular cells
• SA node may fire too slow or fast  sinus bradycardia or sinus tachycardia
• Atrial cell problems:
• Fire occasionally from a focus = premature atrial contractions (PACs)
• Fire continuously due to a looing re- entrant circuit = atrial flutter or paroxysmal SVT
• Block impulses coming from SA node = AV junctional blocks
• Ventricular cell problems:
• Fire occasionally from 1 or more foci = premature ventricular contractions (PVCs)
• Fire continuously from multiple foci = ventricular fibrillation
• Fire continuously due to a looping re- entrant circuit = ventricular tachycardia
23
Sinus tachycardia
24
Sinus bradycardia
25
Atrial flutter (always regular rhythm)
26
• Mechanism: Re- entrant pathway in the right atrium with every 2nd, 3rd or 4th impulse generating a
QRS (others are blocked in the AV node as the node re- polarizes)
Atrial fibrillation (always irregular rhythm)
27
Premature atrial, junctional and ventricular contractions
28
First degree heart block (fixed prolonged PR with NO drop beat)
29
Second degree Mobitz Type I heart block (progressive longer PR + drop)
30
Second degree Mobitz type II heart block (normal PR + drops)
31
Third degree heart block (complete dissociation between P and QRS, but
their inter- distances are fixed)
32
Paroxysmal SVT (fast HR + P wave absent + narrow QRS + ST changes)
33
More PSVT examples 
34
Ventricular tachycardia
35
Ventricular fibrillation
36
Torsade's de pointes
37
Ventricular Hypertrophy 
• Left ventricular hypertrophy
looks like:
• Deep R in V1
• High S in V 6
• Right ventricular
hypertrophy looks like:
• Deep R in V1
• Deep S in V6
38
39
40
Right bundle branch block 
• Basically V1 and V2 have rabbit ear kind of QRS
• Can be seen in ASD as right atrial enlargement will cause delay of signal conduction from there
41
42
Left bundle branch block (LBBB) 
• V1 and V2 have the same kind of wide predominantly negative QRS
43
LBBB 
44

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Pediatric ECG Notes

  • 2. Learning objectives • Basics of ECG • Establish consistent approach to interpreting ECGs • Rate, rhythm, axis and identifying ischemia • Review essential life- threatening cases • Provide additional resources for future learning 2
  • 3. Normal impulse conduction • Sinoatrial node • AV node • Bundle of His • Bundle branches • Purkinje fibers 3
  • 4. PQRST • P wave = atrial depolarization = SA node function • PR interval = atrial depolarization + delay in AV junction (AV node & bundle of His) = delay allows time for the atria to contract before the ventricles do • QRS wave = ventricular depolarization (muscle contraction) • T wave = ventricular repolarization 4
  • 5. Pacemakers of heart • SA node: dominant pacemaker with an intrinsic rate of 60- 100 beats/ min • AV node: back- up pacemaker with an intrinsic rate of 40- 60 beats/ min (= nodal rhythm = seen in heart blocks) • Ventricular cells: back- up pacemaker with an intrinsic rate of 20- 45 beats/ min (= last resort) 5
  • 6. The ECG paper • It is a thermal paper that records electrical activity of the heart • Horizontally we measure in seconds (time) • Vertically we measure in mm (amplitude) • 1 small box = 0.04 seconds & 1 mm • 1 big box = 0.2 seconds & 5 mm • 1 big box has 5 small boxes up & down 6
  • 7. Heart rate • Every six seconds = 30 large boxes • This helps when calculating the heart rate • Heart rate = # of R waves x 10 • Normal resting HR: • Newborn: 110- 150 bpm • 2 years: 85- 125 bpm • 4 years: 75- 115 bpm • > 6 years (like an adult): 60- 100 bpm • Adult: 50- 100 bpm 7
  • 8. 8
  • 9. Steps of any rhythm analysis 1. Calculate rate: # R waves in 6 seconds x 10 2. Determine regularity: look at the distance between the R waves - use a caliper or marking on pen/ paper. Are they equidistant apart, occasionally irregular, regularly irregular or irregularly irregular? 3. Assess P waves 4. Determine PR interval 5. Determine QRS interval 9
  • 10. Step 3: assess the P waves • Are there P waves? • Do all the P waves look alike? • Do the P waves occur at a regular rate? • Is there one P wave before each QRS? • Normal = Normally shaped P waves with 1 P wave before each QRS 10
  • 11. P mitrale = left atrial enlargement • P mitrale indicates left atrial enlargement • The wave is widened and has 2 peaks • Indicates pulmonary hypertension and mitral stenosis 11
  • 12. P pulmonale = right atrial enlargement 12 • May also occur in mitral stenosis or pulmonary hypertension • Note than in pulmonary HTN there will also be inversion of T waves in V1
  • 13. Step 4: determine PR interval • Normal is 0.12- 0.20 seconds (3- 5 small boxes) • PR should not be more than 5 small squares • Measure from start of P to start of R 13
  • 14. Long PR interval • First degree AV block 1. Drugs (all increase vagal tone or antagonize sympathetic system  can lead to sudden death) a. Calcium channel blockers b. Beta blockers c. Digoxin toxicity (can cause any arrhythmia, but mainly tachyarrhythmias in children & bradyarrhythmias in adults) d. Opioids e. Clonidine f. Sedative- hypnotics 2. Atrial surgery (scar tissue) 3. Acute rheumatic fever (minor Jones criteria) 4. Kawasaki disease 14
  • 15. Short PR interval 1. Wolff- Parkinson- White syndrome 2. Pompe’s disease (glycogen storage disease IIa) 3. Fabry’s disease (heart + kidney) 4. GM- 1 gangliosides 5. Friedreich’s ataxia 6. Duchenne muscular dystrophy 15
  • 17. Step 5: QRS wave • Normal: 0.04- 0.12 seconds (1- 3 small boxes) • QT interval will be short in digoxin toxicity and hypercalcemia 17
  • 18. Long QT interval • Congenital 1. Jervell- Lange- Nielsen syndrome: AR + deafness 2. Romano- Ward syndrome: AD + each generation will have a sudden death + normal hearing • Acquired: 1. Metabolic: a. Hypocalcemia b. Hyperkalemia c. Hypomagnesemia d. Malnutrition 2. Drugs: a. Type Ia and III anti- arrhythmics b. Phenothiazines c. TCAs 3. CNS trauma (brain edema  long QT  sudden death) 4. Myocardial ischemia or myocarditis 18
  • 19. 19
  • 20. QTc calculation • Normal must be < 0.40- 0.45 seconds • Formula = Bazett formula • < 0.45 s = normal • 0.45- 0.48 s = doubtful • 0.48- 0.50 s = highly suspicious of prolonged QT • > 0.50 s = confirmed prolonged QT • So just remember 1 small square = 0.04 s • Your QT & RR in the formula will be the # of small squares times 0.04    20
  • 21. 21
  • 22. 22
  • 23. Arrhythmia formation • Arrhythmias can arise from problems in the: • Sinus node • Atrial cells • AV junction • Ventricular cells • SA node may fire too slow or fast  sinus bradycardia or sinus tachycardia • Atrial cell problems: • Fire occasionally from a focus = premature atrial contractions (PACs) • Fire continuously due to a looing re- entrant circuit = atrial flutter or paroxysmal SVT • Block impulses coming from SA node = AV junctional blocks • Ventricular cell problems: • Fire occasionally from 1 or more foci = premature ventricular contractions (PVCs) • Fire continuously from multiple foci = ventricular fibrillation • Fire continuously due to a looping re- entrant circuit = ventricular tachycardia 23
  • 26. Atrial flutter (always regular rhythm) 26 • Mechanism: Re- entrant pathway in the right atrium with every 2nd, 3rd or 4th impulse generating a QRS (others are blocked in the AV node as the node re- polarizes)
  • 27. Atrial fibrillation (always irregular rhythm) 27
  • 28. Premature atrial, junctional and ventricular contractions 28
  • 29. First degree heart block (fixed prolonged PR with NO drop beat) 29
  • 30. Second degree Mobitz Type I heart block (progressive longer PR + drop) 30
  • 31. Second degree Mobitz type II heart block (normal PR + drops) 31
  • 32. Third degree heart block (complete dissociation between P and QRS, but their inter- distances are fixed) 32
  • 33. Paroxysmal SVT (fast HR + P wave absent + narrow QRS + ST changes) 33
  • 38. Ventricular Hypertrophy  • Left ventricular hypertrophy looks like: • Deep R in V1 • High S in V 6 • Right ventricular hypertrophy looks like: • Deep R in V1 • Deep S in V6 38
  • 39. 39
  • 40. 40
  • 41. Right bundle branch block  • Basically V1 and V2 have rabbit ear kind of QRS • Can be seen in ASD as right atrial enlargement will cause delay of signal conduction from there 41
  • 42. 42
  • 43. Left bundle branch block (LBBB)  • V1 and V2 have the same kind of wide predominantly negative QRS 43