2. it’s complex firmly attached aggregation of
bacteria to the tooth or other oral surface
clinically its yellow – grayish substance that
adheres to intra oral hard surfaces
4. Acquired
pellicle
• Adsorption of salivary glycoproteins
to tooth surface
colonization
• Bacteria attach to salivary
pellicle or to plaque
aggregation
Formation
of matrix
•Organic ( dextran & levan )
•Inorganic ( Ca , Ph. , Na , K , F )
maturation
6. Plaque induced
gingivitis
D.P only
With local
factors
Without
local factors
Modified by
systemic
factors
Endocrine
factor
puberty
Menstrual
cycle
pregnancy D . M
Blood
dyscrasias
medication
G.
enlargement
Induce
gingivitis
malnutrition
7. calculus Overhanging R Marginal R Ortho
RPD clasp Overcontoured R Mouth breathing malocclusion
8.
9.
10. GingivitisNormal gingiva
Reddish or bluish redPale/coral pinkcolor
- Swelling
( false pocket formation )
- Blunt & rounded edge
- Loss of adaptation
- Loss of scalloping
IDP : fills ID space
marginal G : envelops
the tooth and end in
knife edge
attached G : scalloped
Contour
/size
Loss of stipplingStippling 40%Surface
texture
Soft, loosely bound to bonefirmconsistency
Bleeding on probingNO to normal probing
Tendency
to Bleeding
Inflammatory lesion
reversible with plaque
removal
others
11.
12.
13.
14.
15. objective of scaling and root planing
is to restore gingival health by completely removing elements
that provoke gingival inflammation
16. the scraping of the gingival wall of a periodontal
pocket to separate diseased soft tissue
some degree of curettage is done unintentionally when scaling
and root planing is performed (inadvertent curettage )
Is the curettage essential after SRP ?Why ?
17. Open flap debridement for treatment of intrabony and
furcations defects
Remove inflamed periodontal tissues caused by
bacterial plaque Correct defect or anatomical
problems provoked by the disease To regenerate new
periodontal tissues