oesophagus injuries
perforation
Sumit kamma
Roll no-99
Introduction
● Any oesophageal perforations , also known as a ruptured or torn oesophagus
means a hole or tear in the oesophagus.
● The tube that carries food and liquid from the mouth to the stomach.
● This is a serious condition that require immediate medical attention and open
surgical repair.
● It is associated with high morbidity and mortality rates
● It is an emergency and prompt treatment Should be instituted because
delayed diagnosis and treatment are associated with a marked Increase in
mortality rate
Aetiology
1. Iatrogenic perforation
2. Spontaneous perforation
3. Blunt trauma
4. Corrosive Ingestion
5. Foreign body ingestion
Iatrogenic perforation
● Most common
● Commonly in upper ⅓ (pharyngoesophageal junction
● Increased risk:endoscopy procedures such as EMR/ ESD/POEM
● Features:chest pain / abdominal pain post endoscopy
● IOC:
stable Patient: CECT
Unstable Patient: contrast study
Management:
● In stable patients/Small perforation/Cervical esophagus
● Conservative management
1. Npo( nil per oral)
2. Iv fulid
3. Iv antibiotics
4. Pain killer's
In unstable patients/large Perforation/ Thoracic esophagus
● Perfer surgery
Thoracotomy
+
Repair esophagus
+
Chest tube insert
Spontaneous perforation
● Aka Boerhaave’s syndrome
● results from a sudden Increase in oesophageal pressure against a closed
glottis from vomiting ( MC in alcoholics)
● Mc site : left posterolateral wall of lower ⅓ of
● Clinical Features:Mackler’s triad
Chest pain + Retching+subcutaneous emphysema
● Heart auscultation: Hamman’s crunch due to pneumomediastinum
● Investigation of choice:
Stable patients: CECT
Unstable Patients: contrast study
X- ray:
● Pneumomediastenum sign:spinnaker sign
● Left side pleural effusion ( as perforation is common in left)
Management of Boerhaave’s syndrome
● Patients present early ( within 12 hr)
● With minimal Sepsis &
● Stable Patient
Thoracotomy
+
Repair the tear
+
Chest Tube
● Patient present late
● Sepsis
● Unstable Patient
Emergency Thoracotomy done
● Neck esophagostomy( proximal end connected to neck)
● Gastrotomy for nutrition:
After 10 to 12 wks anastomosis 2end
Blunt trauma
● Blunt external trauma rarely causes oesophageal perforations .
● Perforations from penetrating trauma is rare as the Oesophagus is deep
seated organ.
Foreign body perforations
● Foreign body ingestion, especially with sharp objects, may perforated
oesophagus.
● Clinical Features: dysphagia (early) ,pain,breathing difficulty
● Ioc : x- ray , AP view- FB lies in coronal plane
Lateral view- FB is posterior
● FB in trachea is differentiate by clinical Features as well as by x-ray
● Clinical Features: stridor ,breathing difficulty
● On x-ray, AP view - FB lies in sagittal plane
Lateral view- FB is anterior
Management
● FB in esophagus
● Asymptomatic patient
● FB beyond C6 level
Observation
Serial x- ray ( to check for distal migration of FB)
Check stools for FB
● Patient is symptomatic
● FB impacted at C6 level
● FB is button batteries
Endoscopic removal prefer
● Button batteries can disintegrate ,which can perforated the oesophagus
Corrosive esophageal perforation
● Corrosive Ingestion can also lead to transmutation necrosis and disruption of
the oesophageal wall.
● Corrosive are two type - Acid & Alkali
● Alkali penetration in oesophagus is deep than Acid
● Alkali penetrate through Saponification mechanism
● Acid penetrate through Coagulation mechanism
● Alkali lesser damage to stomach
● Acid more damage to stomach by pylorospasm
● Most caustic ingestion Occur in children accidentally, in adults with suicidal
intent.
● It is mild injury, but also is potentially lethal.
● The severity Of injury depends on the type, pH, quantity and duration of
exposure.
● Clinical Features: pain in neck,throat, chest.
● Drooling of saliva,dysphagia can be present
● Hoarseness of voice is an important sign as it may signify laryngeal injury
and potential airway obstruction.
● Diagnosis by injurious agent pH testing.
Initial management:
● Resuscitation with iv fluid
● Pain management
● Nasogastric tubes should not be inverted blindly ( can causes perforation)
● No use of prophylactic antibiotics
● No role of gastric lavage
Management after patient stabilised
● Early skilled endoscopy must done
● No role of steroids
● Definite Treatment: long term management of stricture ,as it is complication
● A nasogastric / duodenal tube can be placed with endoscopy guidance.
● Use for alimentary nutritional support.
● Most caustic injury managed Conservatively with supportive measures.
Management of stricture:-
1. Regular dilation
2. Long complete stricture: Oesophagectomy
Normally when Oesophagus is removed, stomach used as replacement.
But in corrosive/ caustic injury, colon or jejunum used as replacement
Summary:oesophageal perforations
● Potentially lethal condition due to sepsis
● Surgical emphysema, chest pain and vomiting constitute the classic triad of
Boerhaave’s syndrome
● Treatment aims at adequate drainage,closer of perforation site if possible and
supportive measure
● Delayed diagnosis and management lead to high morbidity and mortality
rates.
Refrance:-
● Bailey & love’s short practice of surgery
Thank you

Surgery -osephageal injuries by final yearstudent

  • 1.
  • 2.
    Introduction ● Any oesophagealperforations , also known as a ruptured or torn oesophagus means a hole or tear in the oesophagus. ● The tube that carries food and liquid from the mouth to the stomach. ● This is a serious condition that require immediate medical attention and open surgical repair.
  • 3.
    ● It isassociated with high morbidity and mortality rates ● It is an emergency and prompt treatment Should be instituted because delayed diagnosis and treatment are associated with a marked Increase in mortality rate
  • 4.
    Aetiology 1. Iatrogenic perforation 2.Spontaneous perforation 3. Blunt trauma 4. Corrosive Ingestion 5. Foreign body ingestion
  • 5.
    Iatrogenic perforation ● Mostcommon ● Commonly in upper ⅓ (pharyngoesophageal junction ● Increased risk:endoscopy procedures such as EMR/ ESD/POEM ● Features:chest pain / abdominal pain post endoscopy ● IOC: stable Patient: CECT Unstable Patient: contrast study
  • 6.
    Management: ● In stablepatients/Small perforation/Cervical esophagus ● Conservative management 1. Npo( nil per oral) 2. Iv fulid 3. Iv antibiotics 4. Pain killer's
  • 7.
    In unstable patients/largePerforation/ Thoracic esophagus ● Perfer surgery Thoracotomy + Repair esophagus + Chest tube insert
  • 8.
    Spontaneous perforation ● AkaBoerhaave’s syndrome ● results from a sudden Increase in oesophageal pressure against a closed glottis from vomiting ( MC in alcoholics) ● Mc site : left posterolateral wall of lower ⅓ of
  • 9.
    ● Clinical Features:Mackler’striad Chest pain + Retching+subcutaneous emphysema ● Heart auscultation: Hamman’s crunch due to pneumomediastinum ● Investigation of choice: Stable patients: CECT Unstable Patients: contrast study
  • 10.
    X- ray: ● Pneumomediastenumsign:spinnaker sign
  • 11.
    ● Left sidepleural effusion ( as perforation is common in left)
  • 12.
    Management of Boerhaave’ssyndrome ● Patients present early ( within 12 hr) ● With minimal Sepsis & ● Stable Patient Thoracotomy + Repair the tear + Chest Tube
  • 13.
    ● Patient presentlate ● Sepsis ● Unstable Patient Emergency Thoracotomy done ● Neck esophagostomy( proximal end connected to neck) ● Gastrotomy for nutrition: After 10 to 12 wks anastomosis 2end
  • 14.
    Blunt trauma ● Bluntexternal trauma rarely causes oesophageal perforations . ● Perforations from penetrating trauma is rare as the Oesophagus is deep seated organ.
  • 15.
    Foreign body perforations ●Foreign body ingestion, especially with sharp objects, may perforated oesophagus. ● Clinical Features: dysphagia (early) ,pain,breathing difficulty ● Ioc : x- ray , AP view- FB lies in coronal plane Lateral view- FB is posterior
  • 17.
    ● FB intrachea is differentiate by clinical Features as well as by x-ray ● Clinical Features: stridor ,breathing difficulty ● On x-ray, AP view - FB lies in sagittal plane Lateral view- FB is anterior
  • 19.
    Management ● FB inesophagus ● Asymptomatic patient ● FB beyond C6 level Observation Serial x- ray ( to check for distal migration of FB) Check stools for FB
  • 20.
    ● Patient issymptomatic ● FB impacted at C6 level ● FB is button batteries Endoscopic removal prefer ● Button batteries can disintegrate ,which can perforated the oesophagus
  • 21.
    Corrosive esophageal perforation ●Corrosive Ingestion can also lead to transmutation necrosis and disruption of the oesophageal wall. ● Corrosive are two type - Acid & Alkali ● Alkali penetration in oesophagus is deep than Acid ● Alkali penetrate through Saponification mechanism ● Acid penetrate through Coagulation mechanism ● Alkali lesser damage to stomach ● Acid more damage to stomach by pylorospasm
  • 22.
    ● Most causticingestion Occur in children accidentally, in adults with suicidal intent. ● It is mild injury, but also is potentially lethal. ● The severity Of injury depends on the type, pH, quantity and duration of exposure. ● Clinical Features: pain in neck,throat, chest. ● Drooling of saliva,dysphagia can be present ● Hoarseness of voice is an important sign as it may signify laryngeal injury and potential airway obstruction. ● Diagnosis by injurious agent pH testing.
  • 23.
    Initial management: ● Resuscitationwith iv fluid ● Pain management ● Nasogastric tubes should not be inverted blindly ( can causes perforation) ● No use of prophylactic antibiotics ● No role of gastric lavage
  • 24.
    Management after patientstabilised ● Early skilled endoscopy must done ● No role of steroids ● Definite Treatment: long term management of stricture ,as it is complication ● A nasogastric / duodenal tube can be placed with endoscopy guidance. ● Use for alimentary nutritional support. ● Most caustic injury managed Conservatively with supportive measures.
  • 27.
    Management of stricture:- 1.Regular dilation 2. Long complete stricture: Oesophagectomy Normally when Oesophagus is removed, stomach used as replacement. But in corrosive/ caustic injury, colon or jejunum used as replacement
  • 29.
    Summary:oesophageal perforations ● Potentiallylethal condition due to sepsis ● Surgical emphysema, chest pain and vomiting constitute the classic triad of Boerhaave’s syndrome ● Treatment aims at adequate drainage,closer of perforation site if possible and supportive measure ● Delayed diagnosis and management lead to high morbidity and mortality rates.
  • 30.
    Refrance:- ● Bailey &love’s short practice of surgery
  • 31.