This document provides information on spinal cord and column injuries. It begins with objectives and epidemiology, then covers anatomy including the vertebrae, discs, ligaments, spinal cord and vessels. Mechanisms of injury are discussed as well as initial management including assessment, diagnostics, fractures/dislocations and SCIWORA. Long-term management includes prevention of respiratory, cardiovascular, gastrointestinal, urinary and skin complications. Musculoskeletal issues like spasticity, contractures and heterotrophic ossification are also addressed.
Spinal cord injuries complete topic about it and how to make good rehabilitation for the patient with spinal cord injuries .
wish it help people
my pleasure :)
Mostafa shakshak
Dr.MD.Monsur Rahman,PT
MPT-Musculoskeletal Disorders
Maharishi Markandeshwar Institute Of Physiotherapy And Rehabilitation, Maharishi Markandeshwar (Deemed to be University), Mullana - Ambala,133-207 (Haryana)
Spinal cord injuries complete topic about it and how to make good rehabilitation for the patient with spinal cord injuries .
wish it help people
my pleasure :)
Mostafa shakshak
Dr.MD.Monsur Rahman,PT
MPT-Musculoskeletal Disorders
Maharishi Markandeshwar Institute Of Physiotherapy And Rehabilitation, Maharishi Markandeshwar (Deemed to be University), Mullana - Ambala,133-207 (Haryana)
Spinal stenosis is a degenrative spine disorder in which the AP and transverse diameter are decreased causing neural compression and symptoms of chronic & acute nerve compression
A spinal cord injury (SCI) is damage to the spinal cord that causes temporary or permanent changes in its function. Symptoms may include loss of muscle function, sensation, or autonomic function in the parts of the body served by the spinal cord below the level of the injury.
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Spinal stenosis is a degenrative spine disorder in which the AP and transverse diameter are decreased causing neural compression and symptoms of chronic & acute nerve compression
A spinal cord injury (SCI) is damage to the spinal cord that causes temporary or permanent changes in its function. Symptoms may include loss of muscle function, sensation, or autonomic function in the parts of the body served by the spinal cord below the level of the injury.
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Defecation
Normal defecation begins with movement in the left colon, moving stool toward the anus. When stool reaches the rectum, the distention causes relaxation of the internal sphincter and an awareness of the need to defecate. At the time of defecation, the external sphincter relaxes, and abdominal muscles contract, increasing intrarectal pressure and forcing the stool out
The Valsalva maneuver exerts pressure to expel faeces through a voluntary contraction of the abdominal muscles while maintaining forced expiration against a closed airway. Patients with cardiovascular disease, glaucoma, increased intracranial pressure, or a new surgical wound are at greater risk for cardiac dysrhythmias and elevated blood pressure with the Valsalva maneuver and need to avoid straining to pass the stool.
Normal defecation is painless, resulting in passage of soft, formed stool
CONSTIPATION
Constipation is a symptom, not a disease. Improper diet, reduced fluid intake, lack of exercise, and certain medications can cause constipation. For example, patients receiving opiates for pain after surgery often require a stool softener or laxative to prevent constipation. The signs of constipation include infrequent bowel movements (less than every 3 days), difficulty passing stools, excessive straining, inability to defecate at will, and hard feaces
IMPACTION
Fecal impaction results from unrelieved constipation. It is a collection of hardened feces wedged in the rectum that a person cannot expel. In cases of severe impaction the mass extends up into the sigmoid colon.
DIARRHEA
Diarrhea is an increase in the number of stools and the passage of liquid, unformed feces. It is associated with disorders affecting digestion, absorption, and secretion in the GI tract. Intestinal contents pass through the small and large intestine too quickly to allow for the usual absorption of fluid and nutrients. Irritation within the colon results in increased mucus secretion. As a result, feces become watery, and the patient is unable to control the urge to defecate. Normally an anal bag is safe and effective in long-term treatment of patients with fecal incontinence at home, in hospice, or in the hospital. Fecal incontinence is expensive and a potentially dangerous condition in terms of contamination and risk of skin ulceration
HEMORRHOIDS
Hemorrhoids are dilated, engorged veins in the lining of the rectum. They are either external or internal.
FLATULENCE
As gas accumulates in the lumen of the intestines, the bowel wall stretches and distends (flatulence). It is a common cause of abdominal fullness, pain, and cramping. Normally intestinal gas escapes through the mouth (belching) or the anus (passing of flatus)
FECAL INCONTINENCE
Fecal incontinence is the inability to control passage of feces and gas from the anus. Incontinence harms a patient’s body image
PREPARATION AND GIVING OF LAXATIVESACCORDING TO POTTER AND PERRY,
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ACCORDING TO apic.org,
Antimicrobial stewardship is a coordinated program that promotes the appropriate use of antimicrobials (including antibiotics), improves patient outcomes, reduces microbial resistance, and decreases the spread of infections caused by multidrug-resistant organisms.
ACCORDING TO pewtrusts.org,
Antibiotic stewardship refers to efforts in doctors’ offices, hospitals, long term care facilities, and other health care settings to ensure that antibiotics are used only when necessary and appropriate
According to WHO,
Antimicrobial stewardship is a systematic approach to educate and support health care professionals to follow evidence-based guidelines for prescribing and administering antimicrobials
In 1996, John McGowan and Dale Gerding first applied the term antimicrobial stewardship, where they suggested a causal association between antimicrobial agent use and resistance. They also focused on the urgency of large-scale controlled trials of antimicrobial-use regulation employing sophisticated epidemiologic methods, molecular typing, and precise resistance mechanism analysis.
Antimicrobial Stewardship(AMS) refers to the optimal selection, dosing, and duration of antimicrobial treatment resulting in the best clinical outcome with minimal side effects to the patients and minimal impact on subsequent resistance.
According to the 2019 report, in the US, more than 2.8 million antibiotic-resistant infections occur each year, and more than 35000 people die. In addition to this, it also mentioned that 223,900 cases of Clostridoides difficile occurred in 2017, of which 12800 people died. The report did not include viruses or parasites
VISION
Being proactive
Supporting optimal animal and human health
Exploring ways to reduce overall use of antimicrobials
Using the drugs that prevent and treat disease by killing microscopic organisms in a responsible way
GOAL
to prevent the generation and spread of antimicrobial resistance (AMR). Doing so will preserve the effectiveness of these drugs in animals and humans for years to come.
being to preserve human and animal health and the effectiveness of antimicrobial medications.
to implement a multidisciplinary approach in assembling a stewardship team to include an infectious disease physician, a clinical pharmacist with infectious diseases training, infection preventionist, and a close collaboration with the staff in the clinical microbiology laboratory
to prevent antimicrobial overuse, misuse and abuse.
to minimize the developme
3. Objectives
At the conclusion of this presentation
the participant will be able to:
• Identify the components of the spine
• Assess for spine and spinal cord injury
• Discuss the initial management of the
spinal cord injured patient
• Evaluate the long term needs of the spinal
cord injured patient
• Describe effects of spinal cord injury on
the rest of the body
4. Epidemiology
• Approx 12,000 new cases per year
• Average age 40.7 years
• 80.7% male
• Increased incidence among African
Americans (27%) and Asians (2%)
• Most common causes - MVC (41%),
Falls, Violence
15. Mechanisms of Injury
McQuillan, K., Von Rueden, K.,
Hartsock, R., Flynn, M., & Whalen,
E. (eds.). (2002). Trauma Nursing:
From Resuscitation Through
Rehabilitation. Philadelphia: W. B.
Saunders Company. Reprinted
with permission.
20. Reflex Assessment
• Test for sensory/motor
sparing
• Major deep tendon reflexes
(DTR) assessed
• Biceps (C5)
• Brachioradialis (C5-6)
• Triceps (C7-8)
• Quadriceps (knee-jerk)
(L3-4)
• Achilles (S1-2)
• Scoring 0 to ++++
++
++
++
++
++
++
++
++
++
++
21. Superficial Reflex Assessment
Abdominal - umbilicus pulls toward
stimulus
Cremasteric - scrotum pulls up with
stoking inner thigh
Bulbocavernosus - anal sphincter
contraction with stimulus
Superficial anal – anal sphincter
contraction with stroking peri-anal area
Priapism – results with tugging on
catheter
22. Spinal Cord Injury
• Primary
• From the time of initial mechanism of injury
• Secondary
• Any incidence of hypotension or hypoxia can
result in further injury to the spinal cord
23. Spinal Cord Injury
• ASIA Impairment scale
• Complete (A) – lack of motor/sensory function in
sacral roots (S4-5)
• Incomplete (B) – sensory preservation, motor
loss below injury including S4-5
• Incomplete (C) – motor preservation below injury,
more than ½ muscle groups motor strength <3
• Incomplete (D) - motor preservation below injury,
at least 50% muscle groups motor strength >3
• Normal (E) – all motor/sensory function present
24. Cord Syndromes
• Central Cord
• Typically fall with
hyperextension
• Elderly
• Presents with weak
upper extremities,
variable bowel and
bladder dysfunction,
disproportionately
functional lower
extremities
25. Cord Syndromes
• Anterior Cord
• Primarily a
hyperflexion
mechanism
• Anterior segment of
spinal cord controls
motor function
below the injury
26. Cord Syndromes
• Brown-Sequard
• Hemisection of
the cord usually
from penetrating
injury
• Loss motor on
side of injury
• Loss of
sensation on the
opposite side
Image found on Wikimedia.org
27. Cord Syndromes
• Conus Medullaris
• S4-5 exit at L1; may have L1
fracture
• Areflexic bowel and bladder,
flaccid anal sphincter
• Variable lower extremity loss
• Cauda Equina
• Lumbar sacral nerve roots, with
or without fracture
• Variable loss; areflexia; radicular
pain
28. Complete Cord Injury
• Quadriplegia
(Tetraplegia)
• Loss of function below the
level of injury
• Includes sacral roots
(bowel and bladder)
• C1-T1
• Paraplegia
• Loss of function below the
level of injury
• Below T1
29. Diagnostics
• Plain films
• Lateral, A/P, odontoid; C-T-L spines
• May be used for rapid identification of
gross deformity
• CT Scan
• Comprehensive, cervical through
sacral
• Demonstrates degree of compression
and cord canal impingement
• MRI Scan
• Demonstrates ligamentous, spinal
cord injury
30. Diagnostics
• Clearing the Cervical Spine
• Awake, alert, and oriented
• NO distracting injuries
• NO drugs or alcohol that alter
experience
• NO pain or tenderness
• Clearing spine with films, CT,
MRI
• Complaints of neck pain
• Neurologic deficit
• Altered level of consciousness,
ventilator
31. Fractures-Dislocations
• Atlanto-occipital dissociation
• Complete injury; death
• Atlanto-axial dislocation
• Complete injury; death
• Jumped, Jump-locked facets
• Require reduction; may
impinge on cord; unstable
due to ligamentous injury
34. SCIWORA
• Spinal Cord Injury without
Radiographic Abnormality
• Most frequently children
• Dislocation occurs with spontaneous
relocation
• Cord injury evident
• Radiographs negative
35. Management
• Airway
• C1-4 injuries require definitive airway
• Injuries below C4 may also require airway
due to
• Work of breathing
• Weak thoracic musculature
• Breathing
• Adequacy of respirations
• SpO2
• Tidal volume
• Effort
• Pattern
36. Management
• Circulation
• Neurogenic shock
• Injuries above T6
• Hypotension
• Bradycardia –treat symptomatic only
• Warm and dry
• Poikilothermic – keep warm
• Fluid resuscitation
• Identify and control any source of bleeding
• Supplement with vasopressors
37. Neurogenic Shock
Injury to T6 and above
Loss of sympathetic innervation Increase in venous capacitance
Bradycardia Decrease in venous return
Hypotension
Decreased cardiac output
Decreased tissue perfusion
39. Management
• Deficit
• Spinal shock
• Flaccid paralysis
• Absence of cutaneous and/or
proprioceptive sensation
• Loss of autonomic function
• Cessation of all reflex activity below the
site of injury
• Identify level of injury
40. Management
• Pain
• Frequent physical and verbal
contact
• Explain all procedures to
patient
• Patient-family contact as soon
as possible
• Appropriate short-acting pain
medication and sedatives
• Foster trust
41. Management
• Communication
• Blink board
• Adapted call bell system
• Avoid clicking, provide a
better option
• Speech and occupational
therapy
• Prism glasses
• Setting limits/boundaries
for behavior
42. Management
• Special Treatment
• Hypothermia
• Recommends 33oC intravascular cooling
• Rapid application, Monitor closely
• Anecdotal papers
• No peer reviewed/ class I clinical research
studies to substantiate
• High dose methylprednisolone
• No longer considered standard of care
44. Management
• Reduction
• Cervical traction
• Halo
• Gardner-Wells tongs
• Surgical
• Stabilization
• Cervical collar – convert to
padded collar as soon as
possible
• CTO or TLSO for low
cervical, thoracic, lumbar
injuries
45. McQuillan, K., Von Rueden, K., Hartsock, R., Flynn, M., &
Whalen, E. (eds.). (2002). Trauma Nursing: From
Resuscitation Through Rehabilitation. Philadelphia: W. B.
Saunders Company. Reprinted with permission.
46. Management
• Rotational bed therapy
• Maintain alignment and traction
• Prevent respiratory complications of
immobility
47. Management
• Surgical
• Determined by
• Degree of deficit, location of injury, instability,
cord impingement
• Anterior vs. posterior decompression/ both
• Emergent
• Reserved for neurologic deterioration when
evidence of cord compression is present
• SSEP –during procedure to monitor
changes
• Limited to ascending sensory tracts esp..
dorsal columns
48. Complication Prevention
• Respiratory
• Complications of immobility
• Atelectasis, Pneumonia
• Pulmonary embolism
• Respiratory insufficiency/ failure
• Level of injury affects phrenic nerve,
intercostals
• Increased work of breathing, fatigue
• Rate and pattern are altered (accessory
muscle use)
• Monitor breath sounds
49. Respiratory
Ventilation
Early intubation to prevent hypoxia and fatigue
C1-4 injuries require tracheostomy and home ventilation
training
Quad cough training
Communication tools
Bronchoscopy
53. Cardiovascular
• Orthostatic hypotension
• Decreased BP, possibly increased heart
rate, dizziness or lightheadedness,
blurred vision, loss of consciousness
• Provide physical support with hose,
abdominal binder; salt tablets; Florinef;
sympathomimetics
• Slowly raise the head of the bed for
mobilization
• Turn slowly
• Prone to vasovagal response
54. Cardiovascular
• Poikilothermia
• Inability to shiver/sweat and
adjust body temperature
• Keep patient warm
• Warm the environment
• Monitor skin to prevent
burns or frostbite from
exposure
• Insensate skin
57. Gastrointestinal
• Nutrition
• Early enteral nutrition
• PO or tube feeding if ventilated
• Transpyloric tube if slow gastric
emptying
• Hypermetabolic rate
• Feed as with any critically
injured patient
58. Complication Prevention
• Venous thromboembolism
• Slightly higher risk the first 2-3 months post
injury
• Duplex ultrasonography evaluation
• Prevention (x 3months)
• LMWH
• Apply sequential compression devices
• Vena cava filter (in patients who cannot be anti-
coagulated or have failed anti-coagulation)
• Monitor for signs and symptoms
• Early mobilization, hydration
61. Urinary Tract Infection
• Signs and symptoms
• Fever, spontaneous voiding
between catheterizations,
Autonomic Dysreflexia, hematuria,
cloudy- foul-smelling urine, vague
abdominal discomfort, pyuria
• Prevention
• Remove indwelling catheter as
soon as clinically possible,
intermittent cath, hydration
62. Urinary
Renal calculi
• Chronic bacteriuria and sediment, long-
term indwelling catheters, urinary stasis,
chronic calcium loss
• Signs and symptoms – persistent UTI,
hematuria, unexplained Autonomic
Dysreflexia
• KUB x-ray, IVP with cystogram, passage
of stone
• Interventions - increased fluid intake,
dietary modifications, lithotripsy
63. Complication Prevention
Skin breakdown
• Pressure, insensate, dampness
• PREVENTION – frequent turning,
specialty beds, remove backboard asap;
proper fitting braces
• Nutrition, mobilization, cushions,
massage
• Early wound care specialist
• Surgery if deep
• Can cause delays in stabilization,
rehabilitation
64. Complication Prevention
Musculoskeletal
• Spasticity – flexor, extensor, alternating
• Reduce venous pooling, stabilize thorax, aids in
dressing and stand-pivot transfer
• Chronic pain, contractures, heterotrophic ossification,
skin breakdown
• ROM, positioning, weight-bearing, splinting,
pharmacologic management, surgery- neural severing
(permanent)
65. Musculoskeletal
Heterotrophic ossification
• Ectopic bone within
connective tissue
• Below spinal lesion
• More often complete
injuries with spasticity
• Redness, swelling, warmth,
pain, decreased ROM,
fever, positive bone scan
66. Musculoskeletal
Contractures
• Imbalance of muscle
innervation
• High level cord injury, skin
breakdown, concomitant
head injury, spasticity, HO,
fractures
• PREVENTION – aggressive
ROM, mobilization, PT/OT,
splinting, positioning, serial
casting, anti-spasmodics
67. Complication Prevention
A fluid filled cavity
which develops within
the spinal cord
Most common
symptom is pain
Serial monitoring
via MRI
Surgical
decompression
Neurologic - Post traumatic Syingomyelia
68. Complication Prevention
Autonomic dysreflexia
• An uncontrolled, massive sympathetic
reflex response to noxious stimuli, below
the level of the lesion
• Precipitating factors
• Full bladder
• Distended bowel
• Skin irritation, ingrown toenail
• UTI
• Uterine spasms, penile stimulation
• Tight clothing, wrinkled sheets
70. Autonomic Dysreflexia
• Sit patient upright to produce orthostatic
hypotension
• Monitor BP every 5 minutes
• Monitor neurologic status (GCS)
• Eliminate the offending stimulus
• Empty bladder, bowel; identify skin lesion
• Administer anti-hypertensives if the above
fails
• Education –family and patient
71. Psychologic
Pain/Depression
• Nocioceptive – noxious
stimuli to normally
innervated parts
• Neurogenic – nerve tissue
injury in CNS or PNS
• Evaluate for depression
associated with pain
• Counseling, ROM,
pharmacologic treatment,
TENS
73. Sexuality
Female
• Lack innervation to pelvic floor
• Maintain reflex lubrication/
congestion
• Loss psychogenic/ fantasy
response
• Fertility normal
• Pregnancy – loss of sensation,
increased BP, may precipitate AD
• Decreased respiratory excursion
• Alter GI/GU management
74. Rehabilitation
• Mobility
• Tendon transfer
• Functional electrical
stimulation
• Lower level of injury,
more functional
• Bowel and Bladder
Management
• Prevention of
complications
75. Summary
• Spinal cord injury occurrence is
decreased with safety equipment use
• Prevent secondary injury to result in
optimal neurologic recovery
• Spinal column fractures can occur
without long term effects
• Spinal cord injury requires diligence in
complication prevention