POSTOPERATIVE
VISION LOSS (POVL)
PRESENTED BY :
DR.ANKITA MADAN
 Partial or complete vision loss of vision occuring after
general anaesthesia for nonocular surgery.
 Uncommon complication
 Associated more with cardiac surgery or procedures on
the spine done in prone position.
PATHOPHYSIOLOGY
 Ischemia to visual pathway particulary the optic
nerve and the retina.
 Major source of blood supply of retina and optic
nerve is the Opthalmic artery branch of internal
carotid artery.
 Branches : Central retinal artery and long and
short posterior arteries – blood supply of retina
Posterior portion of optic nerve more susceptible to poor perfusion in
presence of hypotension due to comparatively less vascular supply.
CAUSES OF POVL
 Ischemic optic neuropathy (ION)
 Central retinal artery occlusion (CRAO)
 Cortical blindness
 Posterior reversible encephalopathy syndrome
(PRES)
VISION LOSS AFTER SPINAL
SURGERY
 Anterior ischemic optic neuropathy
 Posterior ischemic optic neuropathy
 Retinal vascular occlusion
ISCHEMIC OPTIC NEUROPATHY(ION)
Irreversible painless loss of vision
Spontaneously without any warning signs
Usually seen after cardiac surgery , spine
surgery , head and neck surgery , orthopedic
joint procedures and surgery on nose or
sinuses.
Two types - anterior or posterior ION
AION –Most commonly after cardiac
surgery
PION –After spine surgery in prone
postion or radical neck dissection.
MECHANISM FOR ION
 Increased venous pressure in the globe and
interstitial edema during prone position
 Increase in intraocular pressure
 Compression of vessels supplying optic nerve
 Hypoxia of optic nerve
RETINAL ISCHAEMIA
 CENTRAL RETINAL ARTERY OCCLUSION
decreases blood supply to the entire retina
 BRANCH RETINAL ARTERY OCCLUSION
affects only a portion of the retina.
 Following ocular trauma , embolic phenomenon
following carotid surgery and vasospastic
episodes.
 In spine surgery , it is due improper patient
positioning and external compression of eye.
POVL AFTER CARDIAC
SURGERY
 Risk factors :
 Lower postoperative haematocrit
 Presence of clinically significant vascular diseae
 Long duration of CPB
 Red cell transfusions
 Use of other blood components
POVL AFTER HEAD AND NECKSURGERY
 ION after neck dissection
 CRAO after neck and nasal or sinus surgery
 Orbital hemorrhage from blunt trauma during the
procedure
PERIOPERATIVE VISUAL LOSSIN OTHERSURGERIES
 After robotic and laparoscopic surgeries especially
after laparoscopic nephrectomies and robotic
prostatectomies.
 During robotic prostatectomy, patient in steep
trendelenburg position for a prolonged time and co2
insufflation of the abdomen increases the CVP , the
intrathoracic pressure and the intraocular pressure.
OTHER CAUSES OF POVL
CORTICAL BLINDNESS
 Result of decreased perfusion to the occipital
cortex due to hypoperfusion and embolic
phenomenon.
 Normal light reflex and fundoscopic examination.
 Prevention by maintaining normal perfusion
pressure and hematocrit of about 30%.
POSTERIORREVERSIBLE ENCEPHALOPATHY
SYNDROME (PRES)
 SEIZURES
 HEADACHE
 VOMITING
 VISION DEFECTS
 DECREASED LEVEL OF CONSCIOUSNESS.
Reported after severe hypertension ,
chemotherapy , immune suppression ,
renal disease , vasculitis and eclampsia ,
lumbar spine fusion , hysterectomy and
video assisted thoracoscopic surgery .
Treatment is symptomatic.
ASA PRACTICE ADVISORY
 Inform patients undergoing spine surgery of long
duration and expected excessive blood lossabout
small and unpredictable risk of POVL.
 Systemic BP to be monitored in high risk patients.
Deliberate hypotension should be practiced on case
by case basis.
 Central venous pressure must be monitored in high
risk patients.
To maintain hematocrit of above 28%
Use of vasopressors on case by case basis.
During positioning , direct compression on eyes
must be avoided and head should be maitained
in neutral position at the level or higher than the
level of heart.
Staging of surgical procedure must be given
consideration in high risk patients.
Vision must be tested of high risk patients for
POVL immediately after they are awake and
ophthalmology opinion must be taken urgently if
there is any concern.
Optimization of Hb level , hemodynamic status
and oxygenation may be addressed
THANKS 

Postoperative vision loss

  • 1.
  • 2.
     Partial orcomplete vision loss of vision occuring after general anaesthesia for nonocular surgery.  Uncommon complication  Associated more with cardiac surgery or procedures on the spine done in prone position.
  • 3.
    PATHOPHYSIOLOGY  Ischemia tovisual pathway particulary the optic nerve and the retina.  Major source of blood supply of retina and optic nerve is the Opthalmic artery branch of internal carotid artery.  Branches : Central retinal artery and long and short posterior arteries – blood supply of retina
  • 5.
    Posterior portion ofoptic nerve more susceptible to poor perfusion in presence of hypotension due to comparatively less vascular supply.
  • 7.
    CAUSES OF POVL Ischemic optic neuropathy (ION)  Central retinal artery occlusion (CRAO)  Cortical blindness  Posterior reversible encephalopathy syndrome (PRES)
  • 8.
    VISION LOSS AFTERSPINAL SURGERY  Anterior ischemic optic neuropathy  Posterior ischemic optic neuropathy  Retinal vascular occlusion
  • 9.
    ISCHEMIC OPTIC NEUROPATHY(ION) Irreversiblepainless loss of vision Spontaneously without any warning signs Usually seen after cardiac surgery , spine surgery , head and neck surgery , orthopedic joint procedures and surgery on nose or sinuses.
  • 10.
    Two types -anterior or posterior ION AION –Most commonly after cardiac surgery PION –After spine surgery in prone postion or radical neck dissection.
  • 12.
    MECHANISM FOR ION Increased venous pressure in the globe and interstitial edema during prone position  Increase in intraocular pressure  Compression of vessels supplying optic nerve  Hypoxia of optic nerve
  • 13.
    RETINAL ISCHAEMIA  CENTRALRETINAL ARTERY OCCLUSION decreases blood supply to the entire retina  BRANCH RETINAL ARTERY OCCLUSION affects only a portion of the retina.  Following ocular trauma , embolic phenomenon following carotid surgery and vasospastic episodes.  In spine surgery , it is due improper patient positioning and external compression of eye.
  • 14.
    POVL AFTER CARDIAC SURGERY Risk factors :  Lower postoperative haematocrit  Presence of clinically significant vascular diseae  Long duration of CPB  Red cell transfusions  Use of other blood components
  • 15.
    POVL AFTER HEADAND NECKSURGERY  ION after neck dissection  CRAO after neck and nasal or sinus surgery  Orbital hemorrhage from blunt trauma during the procedure
  • 16.
    PERIOPERATIVE VISUAL LOSSINOTHERSURGERIES  After robotic and laparoscopic surgeries especially after laparoscopic nephrectomies and robotic prostatectomies.  During robotic prostatectomy, patient in steep trendelenburg position for a prolonged time and co2 insufflation of the abdomen increases the CVP , the intrathoracic pressure and the intraocular pressure.
  • 17.
    OTHER CAUSES OFPOVL CORTICAL BLINDNESS  Result of decreased perfusion to the occipital cortex due to hypoperfusion and embolic phenomenon.  Normal light reflex and fundoscopic examination.  Prevention by maintaining normal perfusion pressure and hematocrit of about 30%.
  • 18.
    POSTERIORREVERSIBLE ENCEPHALOPATHY SYNDROME (PRES) SEIZURES  HEADACHE  VOMITING  VISION DEFECTS  DECREASED LEVEL OF CONSCIOUSNESS.
  • 19.
    Reported after severehypertension , chemotherapy , immune suppression , renal disease , vasculitis and eclampsia , lumbar spine fusion , hysterectomy and video assisted thoracoscopic surgery . Treatment is symptomatic.
  • 20.
    ASA PRACTICE ADVISORY Inform patients undergoing spine surgery of long duration and expected excessive blood lossabout small and unpredictable risk of POVL.  Systemic BP to be monitored in high risk patients. Deliberate hypotension should be practiced on case by case basis.  Central venous pressure must be monitored in high risk patients.
  • 21.
    To maintain hematocritof above 28% Use of vasopressors on case by case basis. During positioning , direct compression on eyes must be avoided and head should be maitained in neutral position at the level or higher than the level of heart.
  • 22.
    Staging of surgicalprocedure must be given consideration in high risk patients. Vision must be tested of high risk patients for POVL immediately after they are awake and ophthalmology opinion must be taken urgently if there is any concern. Optimization of Hb level , hemodynamic status and oxygenation may be addressed
  • 23.