This document provides an overview of medical emergencies that can occur in dental practice and how to manage them. It begins with defining what constitutes a medical emergency and discussing prevention through thorough patient history and risk assessment. Potential emergencies from the respiratory, cardiovascular, and central nervous systems are then reviewed along with their signs, symptoms and management. Specific conditions covered include asthma, hyperventilation, emphysema, seizures, strokes, and syncope. The document emphasizes maintaining an open airway, administering oxygen, and recognizing signs of distress. It provides guidelines for treating acute episodes through the use of inhalers, epinephrine, benzodiazepines and other medications. Overall prevention and swift management of emerg
MEDICAL EMERGENCIES IN DENTAL CLINIC.pptxBhargabeeDas2
Dentists must be prepared to manage medical emergencies which may arise in practice.
Medical emergencies were most likely to occur during and after local anesthesia, primarily during tooth extraction and endodontics. Over 60% of the emergencies were syncope, with hyperventilation the next most frequent at 7%.
The extent of treatment by the dentist requires preparation, prevention and then management, as necessary. Prevention is accomplished by conducting a thorough medical history with appropriate alterations to dental treatment as required. The most important aspect of nearly all medical emergencies in the dental office is to prevent, or correct, insufficient oxygenation of the brain and heart. Therefore, the management of all medical emergencies should include ensuring that oxygenated blood is being delivered to these critical organs. This is consistent with basic cardiopulmonary resuscitation, with which the dentist must be competent.
MEDICAL EMERGENCIES IN DENTAL CLINIC.pptxBhargabeeDas2
Dentists must be prepared to manage medical emergencies which may arise in practice.
Medical emergencies were most likely to occur during and after local anesthesia, primarily during tooth extraction and endodontics. Over 60% of the emergencies were syncope, with hyperventilation the next most frequent at 7%.
The extent of treatment by the dentist requires preparation, prevention and then management, as necessary. Prevention is accomplished by conducting a thorough medical history with appropriate alterations to dental treatment as required. The most important aspect of nearly all medical emergencies in the dental office is to prevent, or correct, insufficient oxygenation of the brain and heart. Therefore, the management of all medical emergencies should include ensuring that oxygenated blood is being delivered to these critical organs. This is consistent with basic cardiopulmonary resuscitation, with which the dentist must be competent.
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Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
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5. Recognition of
patients at RISK
A thorough medical history and drug history
(medical history questionnaires )
A detailed physical examination should be
carried out; this includes evaluation of vital
signs such as pulse, blood pressure,
respiration and temperature.
8. Manifestations of Acute Asthmatic Attack
Mild to Moderate
• Wheezing (high-pitched whistling)
• Dyspnea
• Chest tightness
• Tachycardia
• Coughing
• Anxiety
Severe
• Intense dyspnea, flaring of nostrils & use
of accessory muscles for breathing
• Cyanosis – mucous membrane and nail
bed
• Flushing of face
• Extreme anxiety
• Mental confusion
• Perspiration
9. Asthma
50% cases of Asthma due to –
External antigens
Expirations >>>> Inspirations
(Wheezing sound)
Precipitated by –
• Exposure to specific allergens
• Unusual excitement
• Emotional stress
• Infections
10. Management of
acute severe asthma
Oxygen – high conc. (humidified, if possible)
to maintain oxygen saturation above 92% in
adults
High doses of inhaled bronchodilators –
Short-acting beta-2 agonists. (Salbutamol)
Metered-dose inhaler via a space device.
Systemic corticosteroid – to reduce
inflammation; Prednisolone is given orally,
Hydrocortisone IV.
11. Pt position – erect or semierect position
O2 administration
When aerosol therapy is ineffective – Epinephrine
(0.3 ml of a 1:1,000 dilution IM or SC)
Start IV line and drip of crystalloid solution
(30mL/min)
If pt fails to improve –
IV Mg – to provide additional bronchodilation
Aminophylline/Theophylline 250 mg IV given over
10 mins + Cortione 100 mg IV
13. Manifestations of Hyperventilation syndrome
Neurogenic
Dizziness
Tingling or
numbness – lips,
toes, fingers
Syncope
01 02
Cardiac
Palpitations
Tachycardia
05
Psychologic
Extreme anxiety
04
Musculoskeletal
Myalgia
Muscle spasm
Tremor
Tetany
03
Respiratory
ed rate and depth
of breaths
Feeling of
shortness of
breath
Chest pain
Xerostomia
14. Management of Hyperventilation
01
Terminate all dental treatment and remove
foreign bodies from mouth
02
Pt position – fully upright
03
Verbally calm the patient
04 Pt breath – CO2 enriched air (paper bag)
15. Management of Hyperventilation
05
If symptoms persist - Diazepam 10 mg IM or
titrate slowly IV until anxiety is relieved
06
OR Administer – Midazolam 5 mg IM or titrate
slowly IV until anxiety is relieved
07
Monitor vital signs
08 Perform surgery using Anxiety- reducing
protocols
16. Acute or chronic characterized by abnormal
dilatation of the alveoli and distal bronchioles
Emphysema
19. Management
1
Pt. is asked to cough
3
In adults –
Heimlich maneuver
2
In pediatric patients – turn
the pt.’s head downwards +
5 back blows followed by
5 chest thrusts
4
Otherwise, direct
laryngoscopy to
detect the cause of
obstruction
+
Magill forcep
20. Heimlich manoeuvre
•Reach around the person's waist.
•Position one clenched fist above the navel
and below the rib cage.
•Grasp your fist with your other hand. Pull
the clenched fist sharply and directly
backward and upward under the rib cage 6
to 10 times quickly.
•If the person is obese or in late
pregnancy, give chest compressions.
•Continue uninterrupted until the
obstruction is relieved or advanced life
support is available.
23. Seizure
Seizure or convulsions is an abnormal electrical activity of brain, resulting in attack
involving changes in consciousness, motor activity or altered sensory phenomenon
Other causes –
Hypoglycemia,
hypoxia
and local anesthetic overdose
Most of the patients have epilepsy as a cause for
convulsions.
NEVER try to restrain the
movements or put any object
in the patient’s mouth.
Patients should be turned to their
side to protect airways and
protective padding may be
placed below the head to
prevent injury
Self-limiting – usually lasts 1–2 minutes
24. Grand mal convulsions/ Generalised tonic-clonic seizures
A loss of consciousness, involuntary and excessive muscle movements, and muscle rigidity
Symptoms -
Headache
Vomiting
Muscle soreness
25. Petit mal convulsions/ Typical absence (< 30s)
❏Loss of consciousness
❏Postural tone intact
❏Age – 4-10 years
❏Lasts – few seconds
❏Vacant stare - daydreamer
26. Management of Convulsions
Maintain a patent airway
Prevent body injury
Well- paded tongue blade
In pt. who is able to warn the dentist
of an impending attack –
IV Pentobarbital sodium (Nembutal)
Or
Secobarbital sodium (Seconal)
Severe convulsive episodes –
20-40 mg Succinycholine
chloride IV
27. Diazepam 10 mg can be
administered intravenously
slowly over a 2-minute period
to stop the seizure activity
Management of Status Epilepticus
Diazepam in a dose of
0.2–0.5 mg/kg intravenously
Adults Children
A seizure > 5 minutes, or > 1 seizure within a 5 minutes
period, without returning to a normal level of
consciousness between episodes is called status
epilepticus.
30. Risk Factors
Elderly pt. with –
Uncontrolled hypertension
DM
Atherosclerosis
Symptoms of TIA
episodes of slurred
speech
temporary numbness
paralysis of face arm
or leg.
monocular diplopia,
loss of balance and
coordination.
Symptoms of Stroke -
altered consciousness
headache
nausea
vomiting
inability to speak
facial paralysis
paraesthesia
hemipalsy.
31. Management
❏Maintanance of patent airway
❏Adequate ventilation with oxygen
❏BLS measures - positioning to prevent aspiration,
suctioning to maintain airway, monitoring of vital signs
33. Reflex Syncope (Neurally Mediated Syncope, Vasovagal Syncope,
Vasodepressor Syncope, the Common Faint)
❏ Syncope is defined as
sudden, transient loss of
consciousness due to
global impairment of
cerebral blood flow
❏ Reflex syncope is the
result of a reflex
response to some trigger
Inadequate supply of
blood to brain
Due to
Vagus mediated reflex
bradycardia
With or without
peripheral vasodilatation
34. Syncope
Symptoms -
Pallor
The patient complains of feeling strange
Loss of consciousness – extreme manifestation
Management
Trendelenburg position (head down)
If pt. is conscious – ask the pt. to take a
few deep breaths
Most frequent complication in dental office
Form of Neurogenic shock caused by Cerebral ischemia secondary to vasodilation
or an increase in the peripheral vascular bed, with a corresponding loss in blood
pressure
36. References
● Monheim’s (7th edition). Local Anesthesia and pain control in Dental Practice.
CBS
● Davidson (22nd Edition). Davidson’s Principles and Practice of Medicine. Elsevier
● KD Tripathi (7th Edition). Essentials of Medical Pharmacology. Jaypee
● Neelima Malik (2016). Textbook of Oral and Maxillofacial Surgery. Jaypee
● https://www.nature.com/articles/s41415-020-1789-y.