This document discusses various methods used to monitor fetal well-being during labor, including fetal heart rate monitoring, fetal blood sampling, fetal pulse oximetry, fetal electrocardiography, and ultrasound. It provides details on interpreting fetal heart rate patterns, the results of fetal blood sampling, and evidence regarding electronic fetal monitoring versus intermittent auscultation. While electronic fetal monitoring has increased cesarean rates compared to intermittent auscultation, studies have not found it reduces hypoxic ischemic encephalopathy or cerebral palsy rates.
3. Fetal heart rate (FHR) monitoring
ī Fetal parasympathetic nervous system Matures
by Term.
ī Sympathetic nervous system development is still
ongoing even after delivery.
ī Fetus is conditioned to thrive in a mildly hypoxic and
acidic environment.
ī Change in fetal heart rate, caused by the impact of
hypoxia and acidosis which forms the premise of FHR
it is based on the assumption that fetal heart rate is a
reflection of fetal oxygenation status.
4. ī Electronic fetal monitoring (EFM)> Doppler> Auscultation.
ī Electronic fetal monitoring (EFM)-Is a paper record of FHR
pattern plotted simultaneously in relation to uterine activity.
ī So-
i. Intermittent auscultation alone for low-risk pregnancies
ii. Continuous EFM for pregnancies with higher risk.
5.
6.
7.
8. FHR
surface transducers
on the maternal
abdomen
Not feasible in morbid
obesity.
small electrode into
the skin of the fetal
presenting part
Requires dilatation of the
cervix and rupture of the fetal
membranes
Mild infection at the
electrode site.(4%)
11. Image of CTG monitor and routinely recorded parameters, including fetal heart
rate, maternal heart rate, and strength and frequency of uterine contractions
12. ī Fetal heart rate is assessed for four features-
i. Baseline rate
ii. Baseline variability
iii. Presence or absence of decelerations
iv. Presence of accelerations
13. Baseline heart rate
īBetween 110 and 160 bpm
īFetal bradycardia, defined as an FHR <110 bpm.
īFetal tachycardia, defined as an FHR >160 bpm.
īFetal dysrhythmias are typically associated with FHR
>200 bpm.
īThe base line is judged by looking at the mean heart
rate over 10min.
14. FETAL BRADYCARDIA FETAL TACHYCARDIA
ī congenital heart block
i. congenital heart
malformation
ii. Maternal systemic lupus
erythematosus
iii. Maternal medications(β-
antagonistsâLabetalol)
iv. Fetal acidosis.
i. Maternal fever
ii. Infection
iii. Stimulant medications
(atropine)
iv. Drugs(β2-agonists)
v. Hyperthyroidism
15. Baseline variability
ī Between 5 to 25 bpm.
ī Measured from the peak to a trough of a recording during 1
min.
ī Reduced baseline variability-
1. Depression of the fetal CNS(fetal immaturity, hypoxia)
2. Fetal sleep,
3. Maternal medications such as narcotics, sedatives, β-
blockers, corticosteroids, intravenous magnesium
sulfate.
ī Marked variability-
1. Thumb sucking.
2. Fetal Hypoxia
16.
17. īPersistent absence of variability is considered a pre-
terminal feature and carries with it high probability of
a hypoxic fetus.
īSinusoidal variability is an oscillating pattern of three
to five cycles per minute-
1. fetal anemia.
2. Severe fetal hypoxia.
18.
19. Accelerations
īAccelerations of the FHR in response to movements
(To mechanical stimulation of the fetal Scalp/
vibroacoustic stimulation) are reassuring.
ī>15 beats min for at least 15 sec
22. Early decelerations Late decelerations Variable decelerations
Symmetric in shape
Benign in nature.
Variable in their shape
Mirror uterine
contractions in time of
onset, nadir, duration,
and termination
âlateâ in relation
to uterine contractions.
No specific relationship
with contractions
Seen in active labor when
the fetal head is
compressed
in the pelvis
Result of uteroplacental
insufficiency
Result from fetal
umbilical cord
compression.
23.
24. Late decelerations
ī A fall in the heart rate of 15 bpm below baseline (even if still within the
range of 110 to 160 bpm) and lasting for >15 seconds is significant.
ī As the uteroplacental insufficiency/hypoxia worsens-
(i) baseline variability will be reduced and then lost,
(ii) decelerations will last longer
(iii) they will begin sooner following the onset of a contraction
(iv) they will take longer to return to baseline
(v) the rate to which the fetal heart slows will be lower.
ī Repetitive late decelerations demand action.
25.
26. Variable decelerations
ī âConcerning characteristicsâ include â
i. Deceleration lasting more than 60 seconds
ii. Reduced baseline variability within the deceleration
iii. Failure to return to the baseline
iv. Biphasic (W) shape
v. Absence of shouldering (transient increase in the
heart rate before the start of deceleration).
31. EVIDENCE
ī A 2009 systematic review of 12 randomized trails
including > 37,000 women(both low and high risk)
comparing continuous electronic FHR monitoring and
intermittent auscultation showed-
i. Intrapartum fetal death rate approximately 0.5/1000
births with either approach
ii. Use of electronic FHR monitoring instead of
intermittent auscultation did not result in a
significant reduction in overall risk of perinatal
death(RR 0.85,95% CI 0.59-1.23)
32. iii. APGAR scores and NICU admission rates were
similar for both modalities
iv. Electronic FHR monitoring instead of intermittent
auscultation did not-reduce the risk of hypoxic
ischemic encephalopathy, long âterm neurologic
impairment , or cerebral palsy.
v. Frequency of neonatal seizure was reduced in the
electronically monitored group(RR 0.50, 95% CI
0.31-0.80)
vi. Use of electronic FHR monitoring instead of
intermittent auscultation increased the frequency of
operative delivery (cesarean delivery RR 1.66, 95% CI
1.30-2.13; instrumental vaginal delivery RR 1.16, 95%
CI 1.01-1.32)
33. Fetal blood sampling (FBS)
ī Done when cesarean section is being planned for fetal
distress.
ī Rationale- Acidosis or rise in lactate is considered as a
marker of fetal hypoxia.
ī Falsely reassuring in the presence of chorioamnionitis or
thick meconium
ī Interpretation of FBS:
i. pH-
ī Normal: âĨ7.25
ī Borderline: 7.21 to 7.24
ī Abnormal: â¤7.20
ii. Lactate
ī Normal: â¤4.1 mmol/L
ī Borderline: 4.2 to 4.8 mmol/L
ī Abnormal: 4.9 âĨmmol/L
34. FETAL BLOOD
SAMPLE
Normal
No accelerations
in response to
fetal scalp
stimulation
second fetal
blood sample no
more than 1 hour
later
Borderline
No accelerations
in response to
fetal scalp
stimulation
fetal blood
sample no more
than 30 minutes
later
still abnormal
Assisted
NVD/LSCS
35. Fetal scalp electrode
īThe fetal heart rate is derived from the R-R interval of
the fetal electrocardiographic (fECG) trace
īCarries the risk of infection
īMay allow more reliable recording of fetal heart rate
36. Fetal pulse oximetry
ī The literature suggests that fetal saturations 30% are
reassuring(usually associated with PH> 7.13), whereas
<30% should prompt consideration of intervention.
ī A Cochrane review of fetal pulse oximetry (FPO) to
supplement CTG concluded that its use did not appear
to enhance clinical practice.
37. Fetal electrocardiograpy
ī Rationale- Hypoxaemia alters characteristics of the f
ECG (P-R interval, T:Q.RS ratio and ST segment).
ī Automated ST analysis (STAN) software which looks at
the CTG and fECG concurrently. The analysis of the
fECG using STAN requires a scalp electrode to be
placed.
ī The current NICE guideline does not make a
recommendation on the use of STAN.
38. Ultrasound
ī Trans abdominal ultrasound scanning for fetal distress
in labour.
ī Has ability to visualize fetal cardiac activity rapidly.