The document discusses a study that evaluated EVI1 and EVI1s genes as potential therapeutic targets in ovarian cancer. The study found that isoforms of the EVI1 gene were expressed in ovarian cancer tissue samples and cell lines. Knocking down the EVI1 gene inhibited cell growth in ovarian cancer cell lines. While EVI1s did not induce significant growth on its own, EVI1 promoted some proliferation. The expression of these genes was not found to affect chemoresistance to cisplatin. The study concludes that controlling gene transcription could control proliferative processes and that targeting multiple oncogenes in the same locus may provide more specific results.