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Electrolyte imbalances
Ms Shiwani sah
Water distribution
60% : Total body weight
40%: Intracellular fluid 15%: interstitial
20%: Extracellular 5% intravascular
Major electrolytes:
ECF (Extracellular fluid)
ICF(Intracellular fliuid)
ICF COMPONENTS ECF COMPONENTS
Potassium
Magnesium
phosphate
Sodium
Chloride
Bicarbonate ions
electrolytes
• Electrolytes are positively charged and negatively
charged ions which are in solution in all body fluids.
• Normal cellular functions and survival requires
elctrolytes which are maintained within narrow limits.
Cations (+vely charged) Anions (-vely charged)
Na, Cl,
K, Hco3,
Ca, Po4, So4,
Mg Organic acids
Protein
Normal values:
Electrolye Normal values
Na 135 to 145 mEq/lt
K 3.5 to 5.0 mMol
Ca 8.6 to 10.2 mg/dl
PO4 2.5 to 4.5 mg/dl
Mg 1.7 to2.2 mg/dl
Cl 97 to 107 mEq/l
Sodium imbalances
Hyponatremia:
Low sodium level in the blood
Either deficit of sodium
Excess of water.
Causes of hyponatremia
Causes contd,,
• Severe vomiting or diarrhea
• Retention of water
• Excessive exercise and sweating
• Burns
• Infections or high fever
Clinical manifestation
• Depends on cause
• Usually don’t appear until serum sodium falls below
125 mEq/lt
• Headache
• Confusion
• Neausea and vomiting
• Diarrhea
• Abdominal cramps
• Muscle tremors, weakness
Management
Medical management
• Determine cause and correct it
• If it is due to fluid volume excess, intake of fluids will be restricted fo
regaining balance.
• If falls below 125 mEq/lt Na relacement is indicated.
• Pharmacological management
• For moderate hyponatremia, 0.9%Nacl or Rl may be ordered
• When 115 mEq/lt Or less a concentrated saline dolution such as 3%Na
indicated
Nursing intervention
• Monitor cardiovascular, respiratory, neuromuscular,
cerebral, renal and gi status of the client.
• Administration of NS iv infusion as per prescription to
restore sodium content and fluid volume.
• If accompanied by fluid volume excess, administer osmotic
diuretics to promote the excretion of H2O rather than
sodium.
• Instruct the client to increase oral Na intake and
inform about the foods which are rich in Na.
• Oral salt can be given to patient which is in the form
of 1 or 2 gm.
• If patient is taking lithium (Lithobid), monitor the
level of lithium as it may lead to toxicity.
Hypernatremia
• Usually due to water deficit.
Etiology
• Excess water loss: heat exposure, diabetes
insipidus
• Impaired thirst: primary hypodyspsia,
comatose
• Excessive Na retention
Clinical features
• Excessive thirst, polyuria, nausea
• Muscular weakness
• Neuromuscular irritability
• Altered mental status
• Neurological deficit
• Occassionally coma or seizures.
Management
• Correct water deficit
• Rate of hypernatremia:
• Acute hypernatremia:
1 mEq/lt/hour
• Chronic hypernatremia:
10 mEq/l over 24 hour
Dietary management
•Consume food which is less in salt
diet
•Example pickle, pappad, chips and
salty products.
Hypokalemia
• Serum potassium level of less than 3.5
mEq/l
Causes:
• Overall depletion of K or excessive uptake
by muscle from surrounding fluids.
• Drug induced: diuretics
• Gl losses
• Alcoholism
• Renal disorders
• Cushing’s syndrome
Clinical manifestations
• Alkosis
• Confusion, discomfort
• Weakness, disorentation
• Anorexia, nause, nausea, vomiting
• Muscke cramps, lethargy
• ECG pattern changes
Medical management
• Determining and correcting the cause of
the imbalance.
• Extreme hypokalemia requires
• Cardiac monitoring
Pharmacological management
• Oral replacement therapy
• K binding sachet (15 g) , k is extremely irritating to
gastric mucosa therefore the drug must be takrn with
glass of water or juice or during meals.
• Can be administeref iv for moderate or severe
hypokalemia and must be diluted in Iv fluids.
• Administration of potassium by IV push may result
Cardiac arrest.
• KCl: 10 to 20 mEq/hr diluted in IV fluid if client is on
heart monitor.
• High concentration of potassium is irritating to heart
muscle Thus correcting a potassium deficit may take
several days.
Dietary management
• The administration of foods that are
high in potassium.
• Banana, avocado, potatoes, spinach,
beans, citrus juice, fish
hyperkalamia
• Elevated level over 5.0 mEq/l.
Causes:
• Retension of potassium:renal insuffiency,renal
failure,decreased urine output, potassium sparing
diuretics.
• Excessive Iv infusions or oral administration of K
• Excessive release of cellular
K:burns,infections,metabolic acidosis.
Clinical features
• Muscle cramps
• Urine abnormalities
• ECG changes
MEDICAL MANAGEMENT
• When serum 5.0 to 5.5 mEq/l, restrict potassium intake.
• If potassium excess is due to metabolic acidosis, correcting
the acidosis with sodium bicarbonate promotes potassioum
uptake into cells .
• Potassium less dietary intake
Green beans, corn, apples, grapes, pineapple.
Nursing implications
• Prepare to administer potassium excreating diuretics if renal
function is not impaires.
• Initiate a potassium strict diet.
• Cardiac monitoring.
• Discontinue iv potassium and hold oral potassium supplements.
• Administer sodium polysterene sulfonate .
• Prepare the client for dialysis if potassium level is critically
high.
• Hypertonic glucose with regulau insulin to move potassium into
cells.
• Teach to avoid high K rich food and salt susbstitutes or other K
hypocalcemia
• Serum level below 4.5 or 8.5 mg/dl.
Causes:
Malabsorption of fat in intestine
Metabolic alkalosis
Renal failure with hyperphosphotemia.
Acute pancreatitis
Burns
Medications MgSo4
•Clinical features
Medical mangement
• Corrected by oral Ca gluconate ,calcium lactates or Ca Cl-
• Administer Ca supplements 30 minutes before meals for better absorption
and with glass of milk as vit d is necessary for absorption.
• Iv CaCl- or Ca gluconate(10 %) given slowly to avoid hypertension,
bradycardia or other arrhythmias.
• Chronic or mild can be treated by high diet Ca foods
• Red beans, milk, spinach, mushroom, Broccoli, beans curd.
Nursing implications
• Cardiac monitoring.
• When administering Ca Iv, warm the injection
solution to body temperature before administrating
and administer slowly.
• Monitor for hyper calcemia.
• Administer vitD and the medications that aborp Ca
from intestines.
• Seizure precautions
• keep 10 % Ca gluconate for treatment of acute Ca
deficit.
• Encourage for high Ca diet.
hypercalcemia
• Above 5.5m Eq/l.
Etiology:
Metastatic malignancies.
Hyper parathyroidism
Thiazide diuretic hormones.
Prolong immobilization.
Excessive Ca and vita D supplements.
Clinical features
• Anorexia
• Vomiiting
• Constipation
• Weakness
• Extreme lethargy
• Dysrhythmias
•ECG: shortened St segment
•Lengthened QT interval
•ABG: ph< 7.45 serum Ca >5,5 mEq/l
MANAGEMENT
• ACCORDING TO CAUSE
• NS 0.9 % rapidly with furosemide to prevent fluid overload.
• Corticosteriods to decreses Ca levels by competing with VitD
• Reduce Ca rich foods.
• Drug Etidorate di-sodium
• Reduces serum Ca by reducing abnormal bone reabsorption
and secondarily bone formation.
hypomagnesemia
• Plasma Mg < 1.8 mg /dl
Etiology:
Diarrhea,Vomiting
Chronic alcoholism
Prolonged malnutrition
Hyperaldosteronism
Impaired gi absortion, Ng suctioning, Poorly controlled
DM
Clinical manifestations
• Anorexia
• Nausea
• Abdominal distention
• Severe causes: Chvostek”s and
Troussean”s, tetany, convulsion, stroke
• Myocardial irritability
• ECG: shortened QT, broad T
management
• Oral Mgso4 replacement (antacids)
• Parenteral MgSo4
• Increase dietary intake og Mg
Chilli, tofu, wheat gram
Initate safety and seizure precautions, Mg levels,
electrolytes.
hypermagnesemia
• Mg> 3mg/dl
Causes:
Renal insuffiency
Excessive anta-acid use
Adrenal insufficiency
Clinical manifestations
• Related to blocked release of acetylcholine from
myoneromal junction which affects muscle cell activity
• Hypotension.
• Muscle weakness
• Prolonged QT, PR interval.
• Lethargy, drowsiness
• Respiratory paralysis , LOC
MANAGEMENT
• LOW Mg diet
• Chicken , ggs, green peas, white bread, burger
• Decrease MgS04 use
• In severe cases, saline infusion with diuretics to promote
excreation.
• IV Ca (antagonist action)
• Albuterol drug
• If renal failure then dialysis can be done for severe cases.
THANK YOU

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Electrolyte imbalances for nurses .pptx

  • 2. Water distribution 60% : Total body weight 40%: Intracellular fluid 15%: interstitial 20%: Extracellular 5% intravascular Major electrolytes: ECF (Extracellular fluid) ICF(Intracellular fliuid)
  • 3.
  • 4.
  • 5.
  • 6. ICF COMPONENTS ECF COMPONENTS Potassium Magnesium phosphate Sodium Chloride Bicarbonate ions
  • 7. electrolytes • Electrolytes are positively charged and negatively charged ions which are in solution in all body fluids. • Normal cellular functions and survival requires elctrolytes which are maintained within narrow limits.
  • 8. Cations (+vely charged) Anions (-vely charged) Na, Cl, K, Hco3, Ca, Po4, So4, Mg Organic acids Protein
  • 10. Electrolye Normal values Na 135 to 145 mEq/lt K 3.5 to 5.0 mMol Ca 8.6 to 10.2 mg/dl PO4 2.5 to 4.5 mg/dl Mg 1.7 to2.2 mg/dl Cl 97 to 107 mEq/l
  • 11.
  • 12. Sodium imbalances Hyponatremia: Low sodium level in the blood Either deficit of sodium Excess of water.
  • 14. Causes contd,, • Severe vomiting or diarrhea • Retention of water • Excessive exercise and sweating • Burns • Infections or high fever
  • 15. Clinical manifestation • Depends on cause • Usually don’t appear until serum sodium falls below 125 mEq/lt • Headache • Confusion • Neausea and vomiting • Diarrhea • Abdominal cramps • Muscle tremors, weakness
  • 16. Management Medical management • Determine cause and correct it • If it is due to fluid volume excess, intake of fluids will be restricted fo regaining balance. • If falls below 125 mEq/lt Na relacement is indicated. • Pharmacological management • For moderate hyponatremia, 0.9%Nacl or Rl may be ordered • When 115 mEq/lt Or less a concentrated saline dolution such as 3%Na indicated
  • 17. Nursing intervention • Monitor cardiovascular, respiratory, neuromuscular, cerebral, renal and gi status of the client. • Administration of NS iv infusion as per prescription to restore sodium content and fluid volume. • If accompanied by fluid volume excess, administer osmotic diuretics to promote the excretion of H2O rather than sodium.
  • 18. • Instruct the client to increase oral Na intake and inform about the foods which are rich in Na. • Oral salt can be given to patient which is in the form of 1 or 2 gm. • If patient is taking lithium (Lithobid), monitor the level of lithium as it may lead to toxicity.
  • 19. Hypernatremia • Usually due to water deficit. Etiology • Excess water loss: heat exposure, diabetes insipidus • Impaired thirst: primary hypodyspsia, comatose • Excessive Na retention
  • 20. Clinical features • Excessive thirst, polyuria, nausea • Muscular weakness • Neuromuscular irritability • Altered mental status • Neurological deficit • Occassionally coma or seizures.
  • 21. Management • Correct water deficit • Rate of hypernatremia: • Acute hypernatremia: 1 mEq/lt/hour • Chronic hypernatremia: 10 mEq/l over 24 hour
  • 22. Dietary management •Consume food which is less in salt diet •Example pickle, pappad, chips and salty products.
  • 23.
  • 24. Hypokalemia • Serum potassium level of less than 3.5 mEq/l
  • 25. Causes: • Overall depletion of K or excessive uptake by muscle from surrounding fluids. • Drug induced: diuretics • Gl losses • Alcoholism • Renal disorders • Cushing’s syndrome
  • 26. Clinical manifestations • Alkosis • Confusion, discomfort • Weakness, disorentation • Anorexia, nause, nausea, vomiting • Muscke cramps, lethargy • ECG pattern changes
  • 27.
  • 28. Medical management • Determining and correcting the cause of the imbalance. • Extreme hypokalemia requires • Cardiac monitoring
  • 29. Pharmacological management • Oral replacement therapy • K binding sachet (15 g) , k is extremely irritating to gastric mucosa therefore the drug must be takrn with glass of water or juice or during meals. • Can be administeref iv for moderate or severe hypokalemia and must be diluted in Iv fluids.
  • 30. • Administration of potassium by IV push may result Cardiac arrest. • KCl: 10 to 20 mEq/hr diluted in IV fluid if client is on heart monitor. • High concentration of potassium is irritating to heart muscle Thus correcting a potassium deficit may take several days.
  • 31. Dietary management • The administration of foods that are high in potassium. • Banana, avocado, potatoes, spinach, beans, citrus juice, fish
  • 32. hyperkalamia • Elevated level over 5.0 mEq/l. Causes: • Retension of potassium:renal insuffiency,renal failure,decreased urine output, potassium sparing diuretics. • Excessive Iv infusions or oral administration of K • Excessive release of cellular K:burns,infections,metabolic acidosis.
  • 33. Clinical features • Muscle cramps • Urine abnormalities • ECG changes
  • 34.
  • 35. MEDICAL MANAGEMENT • When serum 5.0 to 5.5 mEq/l, restrict potassium intake. • If potassium excess is due to metabolic acidosis, correcting the acidosis with sodium bicarbonate promotes potassioum uptake into cells . • Potassium less dietary intake Green beans, corn, apples, grapes, pineapple.
  • 36. Nursing implications • Prepare to administer potassium excreating diuretics if renal function is not impaires. • Initiate a potassium strict diet. • Cardiac monitoring. • Discontinue iv potassium and hold oral potassium supplements. • Administer sodium polysterene sulfonate . • Prepare the client for dialysis if potassium level is critically high. • Hypertonic glucose with regulau insulin to move potassium into cells. • Teach to avoid high K rich food and salt susbstitutes or other K
  • 37. hypocalcemia • Serum level below 4.5 or 8.5 mg/dl. Causes: Malabsorption of fat in intestine Metabolic alkalosis Renal failure with hyperphosphotemia. Acute pancreatitis Burns Medications MgSo4
  • 39. Medical mangement • Corrected by oral Ca gluconate ,calcium lactates or Ca Cl- • Administer Ca supplements 30 minutes before meals for better absorption and with glass of milk as vit d is necessary for absorption. • Iv CaCl- or Ca gluconate(10 %) given slowly to avoid hypertension, bradycardia or other arrhythmias. • Chronic or mild can be treated by high diet Ca foods • Red beans, milk, spinach, mushroom, Broccoli, beans curd.
  • 40. Nursing implications • Cardiac monitoring. • When administering Ca Iv, warm the injection solution to body temperature before administrating and administer slowly. • Monitor for hyper calcemia. • Administer vitD and the medications that aborp Ca from intestines.
  • 41. • Seizure precautions • keep 10 % Ca gluconate for treatment of acute Ca deficit. • Encourage for high Ca diet.
  • 42. hypercalcemia • Above 5.5m Eq/l. Etiology: Metastatic malignancies. Hyper parathyroidism Thiazide diuretic hormones. Prolong immobilization. Excessive Ca and vita D supplements.
  • 43. Clinical features • Anorexia • Vomiiting • Constipation • Weakness • Extreme lethargy • Dysrhythmias
  • 44. •ECG: shortened St segment •Lengthened QT interval •ABG: ph< 7.45 serum Ca >5,5 mEq/l
  • 45. MANAGEMENT • ACCORDING TO CAUSE • NS 0.9 % rapidly with furosemide to prevent fluid overload. • Corticosteriods to decreses Ca levels by competing with VitD • Reduce Ca rich foods. • Drug Etidorate di-sodium • Reduces serum Ca by reducing abnormal bone reabsorption and secondarily bone formation.
  • 46. hypomagnesemia • Plasma Mg < 1.8 mg /dl Etiology: Diarrhea,Vomiting Chronic alcoholism Prolonged malnutrition Hyperaldosteronism Impaired gi absortion, Ng suctioning, Poorly controlled DM
  • 47. Clinical manifestations • Anorexia • Nausea • Abdominal distention • Severe causes: Chvostek”s and Troussean”s, tetany, convulsion, stroke • Myocardial irritability • ECG: shortened QT, broad T
  • 48. management • Oral Mgso4 replacement (antacids) • Parenteral MgSo4 • Increase dietary intake og Mg Chilli, tofu, wheat gram Initate safety and seizure precautions, Mg levels, electrolytes.
  • 49. hypermagnesemia • Mg> 3mg/dl Causes: Renal insuffiency Excessive anta-acid use Adrenal insufficiency
  • 50. Clinical manifestations • Related to blocked release of acetylcholine from myoneromal junction which affects muscle cell activity • Hypotension. • Muscle weakness • Prolonged QT, PR interval. • Lethargy, drowsiness • Respiratory paralysis , LOC
  • 51. MANAGEMENT • LOW Mg diet • Chicken , ggs, green peas, white bread, burger • Decrease MgS04 use • In severe cases, saline infusion with diuretics to promote excreation. • IV Ca (antagonist action) • Albuterol drug • If renal failure then dialysis can be done for severe cases.