Earth mag fields human h_sr
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This document discusses the links between solar and geomagnetic activity and human health, behaviors, and physiology. It finds that solar and geomagnetic cycles correlate with societal conflicts and mortality from heart disease. Specifically, it reports that the incidence of myocardial infarction increases by 7% in days following geomagnetic storms. It also discusses daily, weekly, yearly, and multi-year rhythms in heart rate, blood pressure, heart rate variability, depression rates, suicide rates, and acts of crime and terrorism that match rhythms in solar and geomagnetic activity. The document suggests that intentional heart coherence may modulate the geomagnetic field and potentially affect others.
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This document discusses the links between solar and geomagnetic activity and human health, behaviors, and physiology. Some key points:
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This document proposes a new hypothesis for some heart diseases under an endogenous toxicological aspect. It suggests that metabolic-catabolic kinetics and gradients over time within the body can create temporary toxic states at the cellular level that result in organ failure. Certain cardiovascular conditions may be caused by endogenous local microenvironment imbalances rather than external factors alone. Considering an endogenous intra-local toxicology perspective that accounts for time-dependent pathological processes could provide new insights into the etiology, diagnosis, and treatment of some heart diseases.NIH-PA Author Manuscript NIH-PA.docx
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Psychosom Med. Author manuscript; available in PMC 2012 January 1.
Published in final edited form as:
Psychosom Med. 2011 January ; 73(1): 67–74. doi:10.1097/PSY.0b013e3182002116.
Modeling Social Influences on Human Health
Kate Karelina, Ph.D.1 and A. Courtney DeVries, Ph.D.1,2
1 Department of Neuroscience, The Ohio State University, Columbus, OH 43210 USA
2 Institute of Behavioral Medicine Research, The Ohio State University, Columbus, OH 43210
USA
Abstract
Social interactions have long-term physiological, psychological and behavioral consequences.
Social isolation is a well recognized but little understood risk factor and prognostic marker of
disease, and can have profoundly detrimental effects on both mental and physical well-being,
particularly during states of compromised health. In contrast, the health benefits associated with
social support (both reduced risk and improved recovery) are evident in a variety of illnesses and
injury states; however, the mechanisms by which social interactions influence disease
pathogenesis remain largely unidentified. The substantial health impact of the psychosocial
environment can occur independently of traditional disease risk factors and is not accounted for
solely by peer-encouraged development of health behaviors. Instead, social interactions are
capable of altering shared pathophysiological mechanisms of multiple disease states in distinct
measurable ways. Converging evidence from animal models of injury and disease recapitulates the
physiological benefits of affiliative social interactions and establishes several endogenous
mechanisms (inflammatory signals, glucocorticoids and oxytocin) by which social interactions
influence health outcomes. Taken together, both clinical and animal research are undoubtedly
necessary in order to develop a complete mechanistic understanding of social influences on health.
Keywords
Social interaction; health; experimental models; oxytocin
Social influences on health
Social interactions shape humans from early development through senescence and have a
strong impact on many aspects of physiology and behavior. Indeed, social interaction is
essential for proper cognitive, affective and behavioral development (1). Among adults, the
social environment remains an important determinant of health and well being; ample
evidence suggests that positive social support accelerates and improves patient recovery
from cancer, cerebrovascular and cardiovascular disease (CVD), atherosclerosis, and other
chronic diseases with an inflammatory component (2–5). This has led to a substantial
interest in the capacity to which the social environment affects physiological systems,
particularly during health challenges. The benefits of a positive social environment are
particularly salient in chronic disease st.
NIH-PA Author Manuscript NIH-PA.docx
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uthor M
anuscript
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IH
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uthor M
anuscript
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IH
-P
A
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uthor M
anuscript
NIH Public Access
Author Manuscript
Psychosom Med. Author manuscript; available in PMC 2012 January 1.
Published in final edited form as:
Psychosom Med. 2011 January ; 73(1): 67–74. doi:10.1097/PSY.0b013e3182002116.
Modeling Social Influences on Human Health
Kate Karelina, Ph.D.1 and A. Courtney DeVries, Ph.D.1,2
1 Department of Neuroscience, The Ohio State University, Columbus, OH 43210 USA
2 Institute of Behavioral Medicine Research, The Ohio State University, Columbus, OH 43210
USA
Abstract
Social interactions have long-term physiological, psychological and behavioral consequences.
Social isolation is a well recognized but little understood risk factor and prognostic marker of
disease, and can have profoundly detrimental effects on both mental and physical well-being,
particularly during states of compromised health. In contrast, the health benefits associated with
social support (both reduced risk and improved recovery) are evident in a variety of illnesses and
injury states; however, the mechanisms by which social interactions influence disease
pathogenesis remain largely unidentified. The substantial health impact of the psychosocial
environment can occur independently of traditional disease risk factors and is not accounted for
solely by peer-encouraged development of health behaviors. Instead, social interactions are
capable of altering shared pathophysiological mechanisms of multiple disease states in distinct
measurable ways. Converging evidence from animal models of injury and disease recapitulates the
physiological benefits of affiliative social interactions and establishes several endogenous
mechanisms (inflammatory signals, glucocorticoids and oxytocin) by which social interactions
influence health outcomes. Taken together, both clinical and animal research are undoubtedly
necessary in order to develop a complete mechanistic understanding of social influences on health.
Keywords
Social interaction; health; experimental models; oxytocin
Social influences on health
Social interactions shape humans from early development through senescence and have a
strong impact on many aspects of physiology and behavior. Indeed, social interaction is
essential for proper cognitive, affective and behavioral development (1). Among adults, the
social environment remains an important determinant of health and well being; ample
evidence suggests that positive social support accelerates and improves patient recovery
from cancer, cerebrovascular and cardiovascular disease (CVD), atherosclerosis, and other
chronic diseases with an inflammatory component (2–5). This has led to a substantial
interest in the capacity to which the social environment affects physiological systems,
particularly during health challenges. The benefits of a positive social environment are
particularly salient in chronic disease st.
NIH-PA Author Manuscript NIH-PA.docx
N
IH
-P
A
A
uthor M
anuscript
N
IH
-P
A
A
uthor M
anuscript
N
IH
-P
A
A
uthor M
anuscript
NIH Public Access
Author Manuscript
Psychosom Med. Author manuscript; available in PMC 2012 January 1.
Published in final edited form as:
Psychosom Med. 2011 January ; 73(1): 67–74. doi:10.1097/PSY.0b013e3182002116.
Modeling Social Influences on Human Health
Kate Karelina, Ph.D.1 and A. Courtney DeVries, Ph.D.1,2
1 Department of Neuroscience, The Ohio State University, Columbus, OH 43210 USA
2 Institute of Behavioral Medicine Research, The Ohio State University, Columbus, OH 43210
USA
Abstract
Social interactions have long-term physiological, psychological and behavioral consequences.
Social isolation is a well recognized but little understood risk factor and prognostic marker of
disease, and can have profoundly detrimental effects on both mental and physical well-being,
particularly during states of compromised health. In contrast, the health benefits associated with
social support (both reduced risk and improved recovery) are evident in a variety of illnesses and
injury states; however, the mechanisms by which social interactions influence disease
pathogenesis remain largely unidentified. The substantial health impact of the psychosocial
environment can occur independently of traditional disease risk factors and is not accounted for
solely by peer-encouraged development of health behaviors. Instead, social interactions are
capable of altering shared pathophysiological mechanisms of multiple disease states in distinct
measurable ways. Converging evidence from animal models of injury and disease recapitulates the
physiological benefits of affiliative social interactions and establishes several endogenous
mechanisms (inflammatory signals, glucocorticoids and oxytocin) by which social interactions
influence health outcomes. Taken together, both clinical and animal research are undoubtedly
necessary in order to develop a complete mechanistic understanding of social influences on health.
Keywords
Social interaction; health; experimental models; oxytocin
Social influences on health
Social interactions shape humans from early development through senescence and have a
strong impact on many aspects of physiology and behavior. Indeed, social interaction is
essential for proper cognitive, affective and behavioral development (1). Among adults, the
social environment remains an important determinant of health and well being; ample
evidence suggests that positive social support accelerates and improves patient recovery
from cancer, cerebrovascular and cardiovascular disease (CVD), atherosclerosis, and other
chronic diseases with an inflammatory component (2–5). This has led to a substantial
interest in the capacity to which the social environment affects physiological systems,
particularly during health challenges. The benefits of a positive social environment are
particularly salient in chronic disease st.
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ABSTRACT:Heart disease is a condition that affects the human heart and blood vessels. Heart disease affects
about half of American adults, and it also played a role in the high death rate in the rest of the world. The data
extracted from National Center for Health Statistics (NCHS) span from December 2019 to December 2021. The
only goal of this study is to look at the risk factors that affect the incidence of heart disease. After that, it will
estimate a Youden index to find the best cut-off point and measure how well the multivariate logistic regression
model's diagnostic test performed, adding to the body of knowledge. The application of logistic regression
yielded the finding that socioeconomic and health risk variables strongly influence the incidence of heart
disease. According to the Youden index, the ideal cutoff value is around 52%. Consequently, it is crucial for
American adults to monitor their lifestyle, have their BMI, blood pressure, diabetes, and other risk factors for
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socioeconomic factors.
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2) Certain cardiac events like sudden death in untrained individuals may be due to time-related intra-toxicity of metabolic, immune, or electrical cellular processes. Conditions like coronary artery spasm may also be linked to local microenvironmental toxicology that changes over time.
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This document summarizes recent epidemiological research on various rheumatic diseases such as rheumatoid arthritis, juvenile rheumatoid arthritis, and osteoarthritis. Some key findings over the past decade include:
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2) Mortality is increased in patients with rheumatic diseases compared to the general population. The mortality gap between rheumatic disease patients and others has widened.
3) Comorbid conditions are more common in rheumatoid arthritis patients and outcomes tend to be poorer when comorbidities are present.
4) Estimates of the prevalence and incidence of juvenile rheumatoid
Health Impacts Of Global Warming1
The document discusses several ways that global warming may negatively impact human health, including increased deaths from heat waves, worsening air pollution and spread of diseases. Higher temperatures are projected to increase heat-related deaths in cities across the United States. Flooding from stronger storms can spread infectious illnesses if sanitation is compromised. Rising seas may also force coastal population displacement.
Cardiology and vascular research heart disease new hypotesys under endogenous...
Cardiology and vascular research heart disease new hypotesys under endogenous...M. Luisetto Pharm.D.Spec. Pharmacology
This document proposes a new hypothesis for some heart diseases under an endogenous toxicological aspect. It suggests that metabolic-catabolic kinetics and gradients over time within the body can create temporary toxic states at the cellular level that result in organ failure. Certain cardiovascular conditions may be caused by endogenous local microenvironment imbalances rather than external factors alone. Considering an endogenous intra-local toxicology perspective that accounts for time-dependent pathological processes could provide new insights into the etiology, diagnosis, and treatment of some heart diseases.NIH-PA Author Manuscript NIH-PA.docx
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Psychosom Med. Author manuscript; available in PMC 2012 January 1.
Published in final edited form as:
Psychosom Med. 2011 January ; 73(1): 67–74. doi:10.1097/PSY.0b013e3182002116.
Modeling Social Influences on Human Health
Kate Karelina, Ph.D.1 and A. Courtney DeVries, Ph.D.1,2
1 Department of Neuroscience, The Ohio State University, Columbus, OH 43210 USA
2 Institute of Behavioral Medicine Research, The Ohio State University, Columbus, OH 43210
USA
Abstract
Social interactions have long-term physiological, psychological and behavioral consequences.
Social isolation is a well recognized but little understood risk factor and prognostic marker of
disease, and can have profoundly detrimental effects on both mental and physical well-being,
particularly during states of compromised health. In contrast, the health benefits associated with
social support (both reduced risk and improved recovery) are evident in a variety of illnesses and
injury states; however, the mechanisms by which social interactions influence disease
pathogenesis remain largely unidentified. The substantial health impact of the psychosocial
environment can occur independently of traditional disease risk factors and is not accounted for
solely by peer-encouraged development of health behaviors. Instead, social interactions are
capable of altering shared pathophysiological mechanisms of multiple disease states in distinct
measurable ways. Converging evidence from animal models of injury and disease recapitulates the
physiological benefits of affiliative social interactions and establishes several endogenous
mechanisms (inflammatory signals, glucocorticoids and oxytocin) by which social interactions
influence health outcomes. Taken together, both clinical and animal research are undoubtedly
necessary in order to develop a complete mechanistic understanding of social influences on health.
Keywords
Social interaction; health; experimental models; oxytocin
Social influences on health
Social interactions shape humans from early development through senescence and have a
strong impact on many aspects of physiology and behavior. Indeed, social interaction is
essential for proper cognitive, affective and behavioral development (1). Among adults, the
social environment remains an important determinant of health and well being; ample
evidence suggests that positive social support accelerates and improves patient recovery
from cancer, cerebrovascular and cardiovascular disease (CVD), atherosclerosis, and other
chronic diseases with an inflammatory component (2–5). This has led to a substantial
interest in the capacity to which the social environment affects physiological systems,
particularly during health challenges. The benefits of a positive social environment are
particularly salient in chronic disease st.
NIH-PA Author Manuscript NIH-PA.docx
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Author Manuscript
Psychosom Med. Author manuscript; available in PMC 2012 January 1.
Published in final edited form as:
Psychosom Med. 2011 January ; 73(1): 67–74. doi:10.1097/PSY.0b013e3182002116.
Modeling Social Influences on Human Health
Kate Karelina, Ph.D.1 and A. Courtney DeVries, Ph.D.1,2
1 Department of Neuroscience, The Ohio State University, Columbus, OH 43210 USA
2 Institute of Behavioral Medicine Research, The Ohio State University, Columbus, OH 43210
USA
Abstract
Social interactions have long-term physiological, psychological and behavioral consequences.
Social isolation is a well recognized but little understood risk factor and prognostic marker of
disease, and can have profoundly detrimental effects on both mental and physical well-being,
particularly during states of compromised health. In contrast, the health benefits associated with
social support (both reduced risk and improved recovery) are evident in a variety of illnesses and
injury states; however, the mechanisms by which social interactions influence disease
pathogenesis remain largely unidentified. The substantial health impact of the psychosocial
environment can occur independently of traditional disease risk factors and is not accounted for
solely by peer-encouraged development of health behaviors. Instead, social interactions are
capable of altering shared pathophysiological mechanisms of multiple disease states in distinct
measurable ways. Converging evidence from animal models of injury and disease recapitulates the
physiological benefits of affiliative social interactions and establishes several endogenous
mechanisms (inflammatory signals, glucocorticoids and oxytocin) by which social interactions
influence health outcomes. Taken together, both clinical and animal research are undoubtedly
necessary in order to develop a complete mechanistic understanding of social influences on health.
Keywords
Social interaction; health; experimental models; oxytocin
Social influences on health
Social interactions shape humans from early development through senescence and have a
strong impact on many aspects of physiology and behavior. Indeed, social interaction is
essential for proper cognitive, affective and behavioral development (1). Among adults, the
social environment remains an important determinant of health and well being; ample
evidence suggests that positive social support accelerates and improves patient recovery
from cancer, cerebrovascular and cardiovascular disease (CVD), atherosclerosis, and other
chronic diseases with an inflammatory component (2–5). This has led to a substantial
interest in the capacity to which the social environment affects physiological systems,
particularly during health challenges. The benefits of a positive social environment are
particularly salient in chronic disease st.
NIH-PA Author Manuscript NIH-PA.docx
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anuscript
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IH
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uthor M
anuscript
NIH Public Access
Author Manuscript
Psychosom Med. Author manuscript; available in PMC 2012 January 1.
Published in final edited form as:
Psychosom Med. 2011 January ; 73(1): 67–74. doi:10.1097/PSY.0b013e3182002116.
Modeling Social Influences on Human Health
Kate Karelina, Ph.D.1 and A. Courtney DeVries, Ph.D.1,2
1 Department of Neuroscience, The Ohio State University, Columbus, OH 43210 USA
2 Institute of Behavioral Medicine Research, The Ohio State University, Columbus, OH 43210
USA
Abstract
Social interactions have long-term physiological, psychological and behavioral consequences.
Social isolation is a well recognized but little understood risk factor and prognostic marker of
disease, and can have profoundly detrimental effects on both mental and physical well-being,
particularly during states of compromised health. In contrast, the health benefits associated with
social support (both reduced risk and improved recovery) are evident in a variety of illnesses and
injury states; however, the mechanisms by which social interactions influence disease
pathogenesis remain largely unidentified. The substantial health impact of the psychosocial
environment can occur independently of traditional disease risk factors and is not accounted for
solely by peer-encouraged development of health behaviors. Instead, social interactions are
capable of altering shared pathophysiological mechanisms of multiple disease states in distinct
measurable ways. Converging evidence from animal models of injury and disease recapitulates the
physiological benefits of affiliative social interactions and establishes several endogenous
mechanisms (inflammatory signals, glucocorticoids and oxytocin) by which social interactions
influence health outcomes. Taken together, both clinical and animal research are undoubtedly
necessary in order to develop a complete mechanistic understanding of social influences on health.
Keywords
Social interaction; health; experimental models; oxytocin
Social influences on health
Social interactions shape humans from early development through senescence and have a
strong impact on many aspects of physiology and behavior. Indeed, social interaction is
essential for proper cognitive, affective and behavioral development (1). Among adults, the
social environment remains an important determinant of health and well being; ample
evidence suggests that positive social support accelerates and improves patient recovery
from cancer, cerebrovascular and cardiovascular disease (CVD), atherosclerosis, and other
chronic diseases with an inflammatory component (2–5). This has led to a substantial
interest in the capacity to which the social environment affects physiological systems,
particularly during health challenges. The benefits of a positive social environment are
particularly salient in chronic disease st.
ENVIRONMENT AND CARDIOVASCULAR DISEASE.pptx
Non-communicable diseases like cardiovascular disease account for the majority of global mortality. Growing evidence shows that environmental stressors such as air pollution, noise pollution, and climate change significantly contribute to NCD burden. Noise pollution, in particular from road traffic, has been associated with increased risks of cardiovascular outcomes like ischemic heart disease, stroke, and hypertension. The physiological effects of noise include disrupted sleep, increased stress hormone levels, inflammation, and impaired vascular function - all of which can promote cardiovascular disease over time. Mitigation strategies include building insulation, reducing vehicle noise, implementing noise barriers, and modifying land use planning.
Quantifying Cardiovascular and Behavioral Correlates of Fear in Mice: Implica...
This document summarizes research on quantifying cardiovascular and behavioral correlates of fear in mice, with implications for PTSD and cardiovascular disease risk. The research pairs behavioral and cardiovascular responses to cued fear learning in mice. It demonstrates that fear-based disorders like PTSD can contribute to increased cardiovascular disease risk by causing physiological hyperarousal and impaired fear processing. The researchers developed methods to remotely trigger conditioned stimuli based on a mouse's cardiovascular state, allowing measurement of cardiovascular responses to learned fear in the home cage environment. This integrated approach provides insights into mechanisms linking PTSD and cardiovascular dysfunction.
Danho_2019_IOP_Conf._Ser. _Mater._Sci._Eng._564_012081.pdf
Innovative technical implementation of the
Schumann resonances and its influence on
organisms and biological cells
Prognosis and treatment of cardiogenic shock complicating acute myocardial in...
This document discusses the prognosis and treatment of cardiogenic shock complicating acute myocardial infarction. It notes that while the mortality rate for cardiogenic shock used to be 80-90%, studies now report in-hospital mortality rates between 42-74%. Several factors predict higher mortality, such as increasing age, prior heart attack, and low blood pressure/cardiac output. The document recommends general measures like aspirin, heparin, and vasopressors to treat cardiogenic shock. It advises against beta blockers and favors early revascularization when possible to improve outcomes for patients experiencing this complication of a heart attack.
Effects of emotions on heart rate asymmetry
Heart rate asymmetry (HRA) is an index that accounts for an uneven contribution of decelerations and accelerations to the heart rate variability (HRV). Clinical studies indicated that HRA measures have additive clinical value over the more frequently used HRV indexes. Despite the abundance of studies on psychological influences on HRV, little is known whether psychological factors influence HRA. Based on previous research regarding HRA and stress, we expected that negative emotions compared to positive emotions would decrease the contribution of decelerations to HRV. Thirty female participants watched three clips that produced negative emotions, positive emotions, and neutral affect. Besides electrocardiogram, we measured several physiological and behavioral responses to ascertain the affective impact of the clips. Using the RR interval time-series, we calculated HRV and HRA indexes. We found that HRA differentiated between positive emotions and negative emotions reactivity. Positive emotions produced a higher number of decelerations in short-term variability to the total short-term variability (C1d) compared to negative emotions. Moreover, C1d correlated with subjective ratings of affect. In sum, the results of this study indicated that HRA is sensitive to psychological influences. HRA indexes are likely to contribute to a more nuanced physiological differentiation between emotions.
2006 mac michael-climate-human-health
There is scientific consensus that rising greenhouse gas emissions from human activity will cause global warming and other climate changes. The IPCC projects an increase in average global temperatures by 2100 of 1.4-5.8°C. Climate change will affect human health in many ways, both beneficial and adverse. Research has focused on impacts of extreme heat, infectious diseases, and food security, but climate change may also disrupt societies and economies in ways that indirectly impact health. While some effects are already apparent, such as heat waves increasing mortality, estimating future health impacts involves uncertainty. Evidence of health risks will strengthen arguments for policies to reduce emissions and adapt to climate change impacts.
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Examination of the incidence of heart disease in the US. A multivariate logis...
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Emotional Factor in the Etiology & Pathogeny of Cancer | Crimson Publishers
Emotional Factor in the Etiology & Pathogeny of Cancer | Crimson Publishers
Heart disease new-hypotesys under endogenous-toxicological-aspect.journal of ...
Heart disease new-hypotesys under endogenous-toxicological-aspect.journal of ...
Cardiology and vascular research heart disease new hypotesys under endogenous...
Cardiology and vascular research heart disease new hypotesys under endogenous...
Quantifying Cardiovascular and Behavioral Correlates of Fear in Mice: Implica...
Quantifying Cardiovascular and Behavioral Correlates of Fear in Mice: Implica...
Danho_2019_IOP_Conf._Ser. _Mater._Sci._Eng._564_012081.pdf
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Prognosis and treatment of cardiogenic shock complicating acute myocardial in...
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Earth mag fields human h_sr
- 1. Earth and Ionospheric Magnetic Fields and their Effects on Heart Health and Behaviors Rollin McCraty, Ph.D.
- 2. It has become increasingly clear that invisible magnetic influences emanating from the sun have left genetic footprints in endogenous physiological rhythms and continue to affect life on earth. Solar and geomagnetic cycles correlate with such major societal conflicts as violence, crime, terrorism, and war.
- 3. Numerous interdisciplinary studies suggest a link between human health, (e.g. blood pressure, heart rate and heart rate variability), myocardial infarctions and geomagnetism.
- 15. 30 Second Spectra Single 30-second Spectra (30 seconds) 10 Average 30-second Spectra (5 minutes) 50 Average 30-second Spectra (25 minutes) Frequency (Hz) µV
- 16. The Schuman Resonances 30 Second Average Spectra Over 8-Hour Period
- 22. MESOR-Hypertension Midline-Estimating Statistic of Rhythm Based on 24 hour, 7 day (hourly) ambulatory BP measures
- 23. Circadian Hyper-Amplitude Tension (CHAT) Based on 24 hour, 7 day (hourly) ambulatory BP measures
- 24. In a population of patients with MESOR-hypertension 42.2% had additional Vascular Variability Disorders, that further increase their vascular disease risk from ~10 up to ~100%. These added risks were not considered in any treatment plans (since current practice does not assess these Variability Disorders ) Partial diagnoses
- 30. The heart radiates an electromagnetic field that energetically affects those in our environment, whether we are conscious of it or not. We experience this when we are affected by each others’ moods, attitudes and feelings.
- 33. Random Event Generator and the Lunar Cycle
- 38. As people pray for social or global coherence while in a heart coherent state, their hearts collectively put out an energetic wave that helps to create coherence in the environment, in the home and in society.
- 46. In Islam, the “intelligence of the heart” combines knowing and loving into a single, unified function. Only from the heart can you touch the sky. --Rumi
Editor's Notes
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
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- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
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- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath Second version of the mission
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
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- Narration--the 20th Centuries greatest thinker.
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath
- The Resilient Educator: Trainer’s Notes © 2004 Institute of HeartMath