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Clinical for 5th Grade
students
Ahmed Osama Hashem
FRCS ophth
PhD,MD Ain Shams university
Associate Professor of ophthalmology
Head Of Vitreoretinal surgery unit KFS
Snellen chart- illiterate E –OR Landlot broken
C
Pinhole
Trial
• Trial frame ,, trial lenses
Phoropter
Test macula function (Amsler grid)
Age related Macular degeneration-Wet type
(CNV)with hemorrhage and scar
Test contrast sensitivity
Test color vision
Test VA in children
• Picture test
Test VA in children
• preferential looking
Test VA in children
• hundreds and thousand test sweet test
Anterior blepharitis
Squamous blepharitis (seborrheic)
• Greasy dandruff scales
• Dandruf like material –as sleeves around lashes
• Associated with dandruff of the scalp
Staphylococcus blepharitis
scales (hard and brittle)around base of lashes
collarettes (bleads on crust removal-ulcerative
lesion under the crust)
hyperemia and telangectasia of anterior lid margin
rosettes
Angular blepharitis
Stye
chalezion
Chalezion excesion
Chalezion excesion
Trichiatic lash trchiatic lash
Bilateral ptosis blepharophemosis
Levator function test
Lagophthalmos
Symblephron
Epicanthus
Complications of blepharitis(Madarosis –poliosis-
tylosis ……. Ectropion
Ectropion
Tylosis
• Thickening of the tarsal border of the eyelid
Loss of lashes
Poliosis
Eye lid edema
Eyelid edema is the medical term for swelling around the eyes
due to fluid retention. It can be caused by allergy,
inflammation, infection, or injury.
. Eyelid edema is usually a sign, not a condition
Dermoid cyst eyelid
Dermoid cyst eye lid
DD –lacrimal gland swelling i.e Dacyoadenitis
Eye lid coloboma
Dacryoadenitis
5. Parasitic blepharitis
• Pubic louse& its ova(nits).
• Typically affects children with poor hyg.conditions.
• Itching.
Treatment - removal, destruction and delousing
Upper lid stye
• .
Madarosis ,Tylosis
chalezion
chalezion
stye
chalezion
Herpes simplex blepharitis vesicles
Lower lid ectropion
Marcus Gunn jaw-winking syndrome
• Accounts for about 5% of all cases of congenital ptosis
• Retraction or ‘wink’ of ptotic lid in conjunction with
stimulation of ipsilateral pterygoid muscles
Opening of mouth Contralateral movement of jaw
Blepharophimosis syndrome (Ptosis -….
Pseudomembranous
Pseudomembranes consist of coagulated
exudate adherent to the inflamed
conjunctival
epithelium. Characteristically, they can be
easily peeled off leaving the epithelium
intact.
Causes(1) severe adenoviral infection, (2)
Bacterial conjunctivitis and (3) autoimmune
conjunctivitis.
Follicular reaction
• Sub epithelial foci of hyperplastic of
lymphoid tissue with in stroma.
• More prominent in fornices.
• Multiple, discrete, slightly elevated,
lesions encircled by a tiny blood
vessel—small grains of rice.
• Size from 0.5 to 5 mm.
1. Viral.
2. Chlamydial.
3. Parinaud oculoglandular syndrome.
4. Hypersensitivity to topical
medications.
Papillae
bacterial conjunctivitis,
allergic conjunctivitis,
chronic blepharitis,
contact lens wear,
superior limbic keratoconjunctivitis floppy
eyelid syndrome.
elevated red dots as a result of
the central vascular channel,
Papillary reaction
• Hyperplastic conjunctival epithelium.
• Can develop in palpebral conjunctiva (firmly attached)
and limbus.
• Papilla may mask follicles.
• Giant papilla (confluence)
• Non-specific; (less diagnostic)
1. Chronic blephritis.
2. Allergic conjunctivitis.
3. Bacterial conjunctivitis.
4. Contact lens wears.
5. Superior limbic keratoconjunctivitis.
6. Floppy eyelid syndrome.
True membranes
True membranes form when the
inflammatory exudate permeates the
superficial layers
of the conjunctival epithelium. Attempts to
remove the membrane may be accompanied
by
tearing of the epithelium and bleeding.
Causes are infections from ß-haemolytic
streptococci and diphtheria.
Trachomatus Pannus
Limbal vernal
Trantas dots
Mucoid nodule
Phlycten
Limbal phlycten
Pinguecula
Pterygium
Pterygium
Limbal VKC
Trachomatous scar
PTCs Trachoma
Pterygium
lagophthalmos
• Definition: inability to close the palpebral fissure
Herpes zoster ophthalmicus
Lt lacrimal sac massage
Probing 1st punctum dilator
probing
Syringing
DCR
Rose Bengal staining of mucus, debris and
devitalized epithelial cells is observed in the
inferior cornea and bulbar conjunctiva
FL break up time test
Schirmer’s test
Schirmer’s test
Nebula
Leukoma
Desmatocele
Leukoma Adherant
Leukoma Adherant- Irregular pupil
Phlyectunular keratitis
Marginal corneal ulcer
Mooren’s corneal ulcer (Autoimmune)
Megalocornea
Microcornea
Posterior embryotoxin
White limbal girdle of vogt
Keratoglobus
Fleischer’s ring
Kayser Fleisher ring (Copper deposition
,Wilson disease)
Stocker line
Hudson stahli line
Band keratopathy
Granular corneal dystrophy
Arcus senilis
Intracorneal ring
Keratoconus
Cornea Plana
Penetrating keratoplasty
Anterior chamber flare
What you can see at the bottom of the cornea ?
What is the treatment ?
Mutton Fat keratic percepitates
Anterior uveitis if not treated probably may
lead to sever complications, what are they ?
Festooned pupil posterior synechia
What is your main finding?
Mention 3 abnormal findings ?
Mention the most common cause?
Rt Leukocoria
Cortical cataract (nearly complete cortical)
Nuclear cataract
Nuclear cataract
Increase in
refractive index
N=1.42
Red reflex
appears as a ring
around a nuclear
opacity
Nuclear cataract Vs Cortical cataract
Nuclear Brown Cataract
Hypermature cortical cataract
• A)Typical Hypermature.
• B)Morgagnian Type.
Morgagnian cataract
Focal blue dot Cataract
Extremely common and insignificant
Subluxated lens
Congenital or Developmental cataract
Morphological types
• Nuclear,,,, central,,,,+- Microcornea,Microphthalmia..
• Lamellar-Zonular
Opacity between clear Nucleus & Cortex
Sutural Cataract
• Follow Y sutures
Coronary cataract
• Opacity in deep cortex forming a crown like around the nucleus.
• Polar cataract:
-Anterior Polar:pyramidal projecting into AC,,,,
May accompany persistent pupillary membrane
Doesnot affect vision,,,,away from nodal point,,,
• Focal blue dot,,
Extremely common and insignificant
Subluxated lens
Intraocular Artificial lens
Complication of surgery
• PCO
• Posterior capsular opacification
IOL dislocated
Anterior Uveitis Keratic percepitate
Systemic diseases
• DM
-Presenile cataract ,,,senile in earlier stage
Good control of blood sugar,,ophthalm. Exam,surgery
-True diabetic cataract – relatively younger age-snow flake cataract.Ant
& Post flake like opacities.
• Hereditary. Retinitis pigmentosa,
Papilledema(optic disc edema)
1- Intracranial causes:
A- Neoplasms 70 % are caused by brain tumors ,, Always
bilateral unless one nerve is atrophic,, (Foster kennedy
syndrome)
Frontal lobe
tumor
Optic disc edema on
one side and
primary optic
atrophy on the
other side
• Central retinal vein occlusion
Rt optic disc edema
Congested,tortous
retinal veins,,
Flame shaped He
Venous pulsation
disappear
Congested,tortous
retinal veins,,
Lt normal optic disc
• Post papilledemic optic atrophy
Glaucomatous optic atrophy
Glaucomatous optic atrophy
unilateral congenial severe ptosis
Rt leukocoria
Apparent squint
Cover uncover test
Synaptophore
Worth’s four dot test
five dots are seen, namely two red and three green in cases of
heterophoria.
Maddox rod and tangent scale test
The Maddox wing test
Cover test
Deviation of one eye Lt Exotropia( XT)
Rt 3rd N palsy
Lt 6th N palsy
Blow-out fractures of the orbit
• 1-(Periocular signs) include Black eye, edema and subcutaneous
emphysema.
• 2- Injury to the infraorbital nerve. This can cause hypoesthesia of the facial
skin of lower lid (Cheek anaesthesia).
• 3-Impingement of the inferior rectus can result in (Diplopia), The diplopia
typically occurs in both up-gaze and down-gaze (Vertical diplopia)
(Diplopia worse in upward gaze).
• 4- (Enophthalmos) is usually absent at the beginning because of associated
orbital edema or hemorrhage and, Enophthalmos may develop after 10
days as the edema subsides.
5- uncommon, Intraocular ocular damage (e.g. hyphema, angle recession,
retinal detachment) should be excluded
What is the diagnosis?
Mention 2 useful investigations?
Rt fracture orbital floor
What is the diagnosis?
The most common cause is ………
This CT scan shows fracture of the orbital floor and
opacification of the maxillary sinus.
Such a patient usually complain of …………
Eye lid hematoma
(black eye)
Eye lid laceration
Eye lid laceration
Ectropion
Lagophthalmos
coloboma
Vertical mattress suture for full thickness
eyelid injury repair
Why this injury needs urgent repair?
Lacrimal canalicular injury with nasolacrimal tube repair (Eagle Tube)
An injury like this should be done immediately
otherwise the patient will suffer from………..
Subconjunctival Hemorrhage
• Fragile vessels rupture from trauma,
Valsalva pressure spikes (sneezing,
coughing, retching),
hypertension,blood diseases, or
without obvious cause.
Subconjunctival Hemorrhage
• Cornea not involved.
• Resolves within 2 weeks.
But take care
You should exclude (Rupture globe) Scleral wound
And you should exclude fracture skull base
DD Local ocular trauma Fracture base of skull
onset immediate Delayed
Trauma To eye ,no proptosis To head + proptosis
consciousness Normal Loss of consciousness
site Temporal Fornices
shape Triangular,base to the cornea Tringular,,apex to cornea
color Bright red Dark red
Posterior limit definite Is not seen
Corneal Abrasion
• Corneal abrasions often worsened by
rubbing and scratching.
• Foreign body sensation common.
This photo took after an injury to the right Eye
Tell me the complications
Rupture globe with iris prolapse Corneal Limbal wound
Describe the finding in this picture
Angle recession glaucoma occurs after Blunt trauma
What is the diagnosis ?
Mention the most common underlying cause ?
What are the complications may follow this
trauma ?
Complication of hyphema
1-Secondary glaucoma (Open OR Close)
2-Blood stained cornea (5%) (need increase of IOP).
3-Decrease vision (due to cross pupil).
4-Recurrent hyphema (20%)(Secondary hyphema) within 3-5 days,
more common in Black people
How can you manage a case like this?
What are your findings?
Iridodyalysis D shaped pupil
Adie’s pupil,,ciliary ganglia lesion
Ultra sound
biomicroscopy
Voissous Ring
Characteristic stellate (star shape)
appearance of traumatic cataract
Lens subluxation (Sunset)
Rosette shape cataract
Commotio retinae is a complication of ……….
Retina Dyalisis from its Insertion
Choroidal rupture
Subretinal Hemorrhage-Suprachoroidal
hemorrhage
RT OPTIC DISC EDEMA AFTER TRAUAM
LT EYE COMPLETE OPTIC ATROPHY
This patient sustained trauma after ECCE,
describe the findings?
Perforating Injury
• Eye-threatening emergency requiring
emergency ophthalmologic surgical
intervention.
LT CORNEAL WOUND BY A KNIFE WITH
IRIS PROLAPSE
CORNEAL WOUND BY A KNIFE WITH
IRIS PROLAPSE
MY DEAR students please
notice the difference
between penetrating injury
and blunt one
Perforating injury
Corneal perforation repair
What are the complications?
Metallic FB
penetration
Corneal Foreign bodyy
What is your diagnosis ?
What is the treatment ?
Corneal FB over the limbus
Fb in sulcus subtarsalis –corneal abrasion by Fluoresceine
Rust ring after FB removal
225
What is this test ?
What does it indicate ?
River sign
Penetratin FB
injury
What is your diagnosis ?
FB in the vitrous cavity
One of the
complications of
perforating eye injury
is endophthalmitis
with hypopyon
What is the diagnosis?
What is the treatment?
Lt FB intraocular by
Xray
Physical keratoconjunctivitis-phototoxicity-UV
photophthalmia
keratitis
+ve FL cobalt blue filter
True exfoliation :Infrared effect in ovens and
glassblowers
Pseudo exfoliation occur as a part of systemic
disease and lead to open angle glaucoma
Pseudoexf
oliation
Infrared (Glassblower Cataract)
Notice the effect of light energy over the photoreceptors in OCT
Vascularized cornel opacity
Vascularized cornel opacity
Cicatricial ectropion
Symblephron
What is the medical treatment of sever
chemical injury ?
Alkali burn more dangerous than acid burn.why?
Necrosis over
the limbus
Corneal
necrosis
after
chemica
burn
In chemical injury ,the first step is to irrigate the eye.
What next ?
Lid speculum
Autograft
Amniotic membrane graft
Lysis of symblepharon
Penetrating keratoplasty
Boston Keratoprothesis
Keratoconus
Myopic fundus with temporal crescent
•
Chorioretinal atrophy Fuch’s spot
CNV hemorrhage in myopic fundus
Giant retinal tear
Fuch’s spot in myopic fundus
Periperal retinal break surrounded by Argon
laser
Mucormycosis
Fungal infection
over lt orbit and lt
maxillary sinus
Cavernous hemangioma vs capillary
(Age)Commenest benign MCQ..
Commonest Adult tumor ,,(Orbital lymphoma,, ,,
Salmon patch---Lscrimal gland) Malignant
Rt Proptosis
Rt non axial proptosis
Bilateral lid retraction DD bilateral proptosis
Lt lid retraction Vs Lt proptosis
Lt Non-axial proptosis
RTl axial proptosis with exposure keratopathy
Preseptal cellulitis
Orbital cellulitis
Exophthalmometry(Hertel)
Preseptal vs Orbital cellulitis CT
Lt congenital Horner’s Syndrome with iris
heterochromia
Lt Horner’s syndrome (Ptosis –miosis -
Attenuation of arteries
Waxy disc pallor
Normal margin
Normal cup
Glaucomatous optic atrophy
Peripapillary atrophy in glaucoma.
Slit lamp + Gonio lens
90 Diopter auxillary lens
Slit lamp + applanation tonometry
Applanation + FL
Digital method for IOP measurment
Tono pen
Confrontation test for the Lt eye
Visual field by projection
Rt diabetic retinopathy maculopathy
Rt diabetic retinopathy maculopathy
Lt diabetic maculopathy
Vitrous pre retinal hemorrhage
Lt Hypertensive retinopathy
What is your diagnosis ?
Mention the ttt briefly ?
Mention 3 abnormal findings ?
Mention the most common cause?
Band shaped keratopathy
Festooned pupil posterior synechia anterior uveitis
Iris naevus
Anterior uveitis if not treated probably may
lead to sever complications, what are they ?
- Rubeosis Iridis
Iridotomy
Iridectomy
Subluxated
lens
Subluxated Lens or Aphake
:Tremolous Iris
RT CRVO
Rt inferior temporal vein occlusion
Lt superior temporal vein occlusion
Retinal tear surrounded by Argon laser
Giant retinal break
Rt upper temporal artery occlusion
Lt retinal artery occlusion with macula sparing
(cilioretinal artery sparing
Lt cherry red spot DD ……
Thank You

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