PRESENTED BY:
MS.HELAN MARIA,
M.Sc.(N)1ST
YEAR,
CON-SRIPMS,
CBE.
CARDIAC ARRHYTHMIA
Introduction:
Without a regular rate & rhythm, the heart may not
perform efficiently as a pump to circulate oxygenated
blood & other life sustaining nutrition to all of the
body’s tissues & organs with an irregular or rhythm,
the heart is considered to be dysrhythmic( arrhythmic)
 Dysrhythmia - bad rhythm
 Arrhythmia – without rhythm
Definition:
 An arrhythmia (also called dysrhythmia) is an
abnormal heartbeat. Arrhythmias can start in
different parts of your heart and they can be too
fast, too slow or just irregular.
- WHO
 It is an disorder of the formation or conduction (or
both) of the electrical impulse within the heart.
These disorders can cause disturbance of the heart
rate, heart rhythm or both.
- Brunner & Suddarth’s textbook of
medical surgical nursing.
INCIDENCE
 The prevalence of arrhythmias is expected to be 1.5% to 5%
in the general population, with atrial fibrillation being the
most common.
 Arrhythmias may or may not produce any symptoms and
can be paroxysmal, leading to difficulty in estimating true
prevalence.
 The overall presence of arrhythmia is associated with higher
morbidity and mortality.
ETIOLOGY
There are a number of factors that can cause a heart
arrhythmia, including:
 Heart attack or scarring of the heart tissue from a
heart attack;
 Coronary heart disease
 High blood pressure
 Smoking
 Genetics: Changes to the heart’s structure e.g.
from cardiomyopathy; Diabetes; Sleep apnea
 Alcohol abuse
 Excessive caffeine consumption
 Overactive thyroid gland (hyperthyroidism)
 Underactive thyroid gland (hypothyroidism)
 Drug abuse
 Stress
NORMAL SINUS RHYTHM
 A normal sinus rhythm (NSR) is the regular
rhythm of a healthy heart. It's caused by electrical
impulses from the heart's natural pacemaker, the
sinoatrial (SA) node.
CHARACTERISTICS
 ventricular & atrial rate: - 60- 100bpm
 ventricular & atrial rhythm : regular
 QRS shape & duration : normal
 P – wave: normal & always in front of the QRS
 PR interval : consistent interval 0.12 -0.20 sec
 P:QRST ratio: 1:1
CLASSIFICATION:
Sinus node dysrhythmia.
Atrial dysrhythmias.
Junctional dysrhythmias.
Ventricular dysrhythmias.
Conduction abnormalities.
SINUS NODE DYSRHYTHMIA
Sinus bradycardia.
Sinus tachycardia.
Sinus arrhythmia.
ATRIAL DYSRHYTHMIAS.
 Premature atrial complex.
 Atrial fibrillation
 Atrial flutter
JUNCTIONAL DYSRHYTHMIAS.
 Premature junctional complex
 Junctional rhythm
 Non paroxysmal junctional tachycardia
 Atrioventricular nodal reentry tachycardia
VENTRICULAR
DYSRHYTHMIAS.
 Premature ventricular complex.
 Ventricular fibrillation.
 Idioventricular rhythm.
 Ventricular asystole.
CONDUCTION
ABNORMALITIES.
First –degree AV block
 Delayed conduction from the atrium to the
ventricle (defined as a prolonged pr interval of
>200 ms) without interruption in atrial to
ventricular conduction.
Second-degree AV block
 Second-degree atrioventricular (AV) block is a
heart condition that occurs when some atrial
impulses are blocked from reaching the
ventricles. It's a type of "incomplete" heart block.
Symptoms lightheadedness and syncope.
Types
 Mobitz type I: The site of the block is usually
within the AV node.
 Mobitz type II: The site of the block is almost
always below the AV node.
Third-degree atrioventricular (AV) block, also
known as complete heart block (CHB),
 It is a condition that prevents electrical signals
from traveling between the heart's atria and
ventricles. This results in the atria and ventricles
beating independently, which can lead to
insufficient blood flow and life-threatening
complications.
SINUS BRADYCARDIA
 Sinus bradycardia is a heart rhythm where the
heart beats slower than normal, but in a regular
pattern. It's defined as a heart rate of less than 60
beats per minute (bpm) in adults.
CHARACTERISTIC
 Ventricular & atrial rate: < 60bpm
 Ventricular & atrial rhythm : regular
 QRS shape & duration : normal
 P – wave: normal & always in front of the QRS
 PR interval : consistent interval 0.12 -0.20 sec
 P:QRST ratio: 1:1
MANAGEMENT
 Atropine- 0.5mg IV bolus.
 After 3-5 mins, max dosage of 3mg will be
administer
 Dopamine /epinephrine
SINUS TACHYCARDIA
Sinus tachycardia is a regular heart rhythm where the heart
beats faster than normal. It's often a normal response to exercise
or stress, but it can also be a sign of an underlying condition.
Symptoms of sinus tachycardia
 Palpitations,
 Fainting, Chest pain,
 Difficulty breathing, and Dizziness.
CHARACTERISTICS
 ventricular & atrial rate:> 100bpm <120bpm
 ventricular & atrial rhythm : regular
 QRS shape & duration : normal
 P – wave: normal & always in front of the QRS
 PR interval : consistent interval 0.12 -0.20 sec
 P:QRST ratio: 1:1
MANAGEMENT
 Beta-blockers: metoprolol, nadolol, and propranolol, these
can help with tachycardia, especially during exercise
 Calcium channel blockers: verapamil can help lower your
heart rate
 Anti-arrhythmic medications: potassium or sodium
channel blockers, adenosine, digoxin, and magnesium
sulfate
SINUS ARRHYTHMIA
Sinus arrhythmia is a variation in the heart's rhythm that's
usually normal and indicates good heart health. It's a type of
arrhythmia, which is an abnormal heart rhythm.
 When you breathe in, your heart rate increases and the time
between heartbeats shortens.
 When you breathe out, your heart rate decreases and the time
between heartbeats lengthens.
CHARACTERISTIC
 Ventricular & atrial rate:60-100bpm
 Ventricular & atrial rhythm : irregular
 QRS shape & duration : normal
 P – wave: normal & always in front of the QRS
 PR interval : consistent interval 0.12 -0.20 sec
 P:QRST ratio: 1:1
MANAGEMENT
It does not cause any significant hemodynamic
effects & therefore is not typically treated.
PREMATURE ATRIAL
COMPLEX
 PAC is a single ECG complex that occurs when an
electrical impulse starts in the atrium before the
next normal impulse of the sinus node.
characteristics
 Ventricular & atrial rate: depends on the underlying
rhythm
 Ventricular & atrial rhythm : irregular due to early p
wave
 QRS shape & duration : due do early p wave is
abnormal
 P – wave: seen or be hidden
 PR interval : p wave is shorter than normal PR
interval 0.12 -0.20 sec
 P:QRST ratio: 1:1
MANAGEMENT
Treatment is not necessary but, they focus on
underlying causes e.g.: Intake of caffeine, correction
of hypokalemia
ATRIAL FIBRILLATION
Atrial fibrillation(afib or AF) is an irregular heartbeat that occurs
when the upper chambers of the heart beat too fast and out of
rhythm. It's the most common type of arrhythmia that lasts more
than a few seconds.
Risk factors:
 Post operative cardiac surgery
 Hypertension
 Obesity
 VHD
 Heart failure
CHARACTERISTIC
 Ventricular & atrial rate: atrial rate 300-600bpm ventricular
rate 120-200bpm in untreated AF
 Ventricular & atrial rhythm :highly irregular
 QRS shape & duration :usually normal maybe abnormal
 P – wave: no discernible P wave
 PR interval : cannot be measured
 P:QRST ratio: many:1
Management
 Beta blockers: Slow the heart rate
 Calcium channel blockers: Control the heart rate
 Digoxin: Controls the heart rate at rest
 Blood thinners: Prevent blood clots and reduce the risk of
stroke
 Anti-arrhythmics: Restore normal heart rhythm
ATRIAL FLUTTER
Atrial flutter is a heart rhythm abnormality that causes
the upper chambers of the heart to beat too fast.
CHARACTERISTICS
 Ventricular & atrial rate: -atrial rate 250-400bpm
ventricular rate 75-150bpm
 Ventricular & atrial rhythm : atrial rhythm is regular
but ventricular maybe irregular
 QRS shape & duration : usually normal, maybe
abnormal or may be absent.
 P – wave: saw-toothed shape
 PR interval :multiple f wave cannot be measure
 P:QRST ratio: 2:1,3:1or4:1
Management
 Adenosine IV followed by 20ml saline flush
 Elevation of arm with IV line to promote rapid
circulation of medication.
 Antiarrhythmics - amiodarone
 Beta blockers- adrenaline
 Calcium channel blockers
 Anticoagulants- prevent blood clots, such as
heparin, warfarin,
PREMATURE JUNCTIONAL
COMPLEX
A premature junctional complex (PJC) is an extra
heartbeat that originates in the atrioventricular (AV)
junction of the heart.
Causes:
 Heart failure
 CAD
CHARACTERISTICS
 P wave: may be inverted, absent, or occur after the
QRS complex
 PR interval: short or absent
 QRS complex: normal in shape and morphology
JUNCTIONAL RHYTHM
Junctional or idionodal rhythm occurs when the AV node
, instead of the sinus node, become the pacemaker of the
heart.
CHARACTERISTIC
 ventricular & atrial rate: 40-60bpm
 ventricular & atrial rhythm : regular
 QRS shape & duration : normal/abnormal
 P – wave: maybe absent after or before QRS
complex
 PR interval : PR interval is < 0.12 sec
 P:QRST ratio: 1:1 or 0:1
MANAGEMENT
Pacemaker therapy
NON PAROXYSMAL
JUNCTIONAL TACHYCARDIA
NPJT is a form of SPV originating from AV junction
with a gradual onset & termination
 it is similar to junctional rhythm
ATRIOVENTRICULAR NODAL
REENTRY TACHYCARDIA
It is a type of abnormal, rapid heart rhythm where
electrical signals within the atrioventricular (AV) node of
the heart loop back on themselves, creating a "reentry
circuit" that causes the heart to beat very fast, often with
sudden onset and termination
CHARACTERISTICS
 Ventricular & atrial rate: - 60- 100bpm
 Ventricular & atrial rhythm : regular
 QRS shape & duration : normal
 P – wave: normal & always in front of the QRS
 PR interval : consistent interval 0.12 -0.20 sec
 P:QRST ratio: 1:1
MANAGEMENT
 Vagal maneuvers: simple actions like coughing,
holding your breath, or applying cold water to the
face can sometimes stop an AVNRT episode.
 Adenosine: this is the first-line medication
 Calcium channel blockers: verapamil can be used.
 Beta-blockers
PREMATURE VENTRICULAR CONTRACTION
A ventricular premature complex (VPC), also known as
a premature ventricular contraction (PVC), is an extra
heartbeat that originates in the heart's lower
chambers. Pvc are common and usually harmless.
CHARACTERS
 Ventricular & atrial rate: depends on the underlying
rhythm
 Ventricular & atrial rhythm : irregular
 P – wave: visible P wave
 PR interval : consistent interval 0.12 sec
 P:QRST ratio: 0:1 ,1:1
MEDICAL MANAGEMENT
 Beta blocker : to lower blood pressure and reduce pvcs
 Calcium channel blockers: to lower blood pressure and
reduce pvc
 Antiarrhythmic: to control irregular heart rhythms
VENTRICULAR TACHYCARDIA
Ventricular tachycardia (VT) is a condition in which the
heart's lower chambers beat abnormally fast. It can be life-
threatening and may require immediate medical care.
Symptoms
•Chest pain
•Lightheadedness or dizziness
•Fainting
•Shortness of breath
•Palpitations
•Anxiety
•Tightness in the neck
•Tiredness
CAUSES
•Heart attack
•Anti-arrhythmic drugs
•Congenital heart disease
•Ischemic heart disease
•Structural heart disease
•Electrolyte imbalances
CHARACTERISTICS
 ventricular & atrial rate: - 100-200bpm
 ventricular & atrial rhythm : regular
 QRS shape & duration : abnormal
 P – wave: very defect to detect
 PR interval : irregular
 P:QRST ratio: difficult to determine
MEDICAL MANAGEMENT
Defibrillation to restore normal heart rhythm and Anti-
arrhythmic medication injections.
VENTRICULAR FIBRILLATION
Ventricular fibrillation (v-fib) is a life-threatening heart rhythm
disorder that causes the heart's ventricles to quiver instead of
pumping blood. It's the most common cause of sudden cardiac
death.
SYMPTOMS
•Chest pain, fullness, discomfort, or pressure
•Dizziness or lightheadedness
•Nausea
•Racing or erratic pulse
•Heart palpitations
•Shortness of breath
CAUSES
•Heart attack
•Heart disease
•Heart surgery
•Sudden blow to the chest
•Medicines
•Potassium levels in the blood
MANAGEMENT
 Treatment cardiopulmonary resuscitation (CPR),
 Shocks to the heart with an automated external defibrillator
(AED),
 Medications,
 Implanted devices and surgery.
IDIOVENTRICULAR RHYTHM
Idioventricular rhythm is a heart rhythm where the lower
chambers of the heart beat more slowly than normal. It's also
known as a "slow ventricular tachycardia”
CHARACTERISTICS
 A rate of less than 50 beats per minute
 No conducted P waves, andA widened QRS complex.
MANAGEMENT
 Atropine may be used to increase heart rate, and
verapamil,
 Antiarrhythmic drugs
VENTRICULAR ASYSTOLE
Ventricular asystole is a complete absence of the heart's
electrical and mechanical activity, also known as flat line.
MANAGEMENT
 High –quality CPR
 Assess for possible causes
Hs
 Hypoxia
 Hypovolemia
 Hydrogen ion (acid- base imbalance)
 Hypo/hyperglycemia
 Hypo/hyperkalemia
 hyperthermia
Ts
 Trauma
 Toxins
 Tamponade
 Tension pneumothorax
 Thrombus
CPR>INTUBATION>IV MEDICATION(emergency
med)
SYMPTOMS
 Tightness
 Chest pain
 Palpitation
 Sob
 Dizziness
 Fainting
DIAGNOSTIC EVALUATION
 History Collection
 Physical Examination
 ECG
 Echo- cardiogram
 Blood test
-electrolyte
-cardiac biomarker
 Genetic testing(if needed)
COMPLICATIONS
 Stroke
 Heart failure
 Sudden cardiac arrest
 Cardiogenic shock
 Chronic fatigue
 MI
 End- organ damage
SURGICAL MANAGEMENT
 Maze procedure
 Valve replacement
 Pacemaker & implantable cardioverter defibrillator(ICD)
 CABG
 Heart transplantation
NURSING DIAGNOSIS
 Decreased Cardiac Output Related to Altered heart rate
and rhythmAs evidenced by Irregular pulse
 Risk for Decreased CardiacTissue Perfusion Related to
Inadequate blood flow due to abnormal heart rhythms
 IneffectiveTissue Perfusion Related to Inadequate cardiac
output from arrhythmia as evidenced by weak pulses
 Activity Intolerance Related to Insufficient oxygenation due
to abnormal heart rhythms as evidenced by Fatigue
 Anxiety Related to Fear of life-threatening arrhythmia
episodes as evidenced by Restlessness
HEALTH EDUCATION
Lifestyle Changes
 Eat a healthy diet (low salt, low cholesterol, more
vegetables).
 Exercise regularly (as recommended by your doctor).
 Quit smoking and limit alcohol and caffeine.
 Reduce stress with relaxation techniques like yoga or
meditation .
 Monitor your blood pressure, cholesterol, and blood sugar.
Seek Emergency Help
 Call emergency services immediately if you experience:
 Severe chest pain or pressure.
 Sudden shortness of breath.
 Fainting or losing consciousness.
 A very fast or very slow heart rate that doesn’t stop.
Patient Self-Care & Monitoring
 Check your pulse regularly and report irregularities to your
doctor.
 Take medications as prescribed and attend all follow-up
visits.
 Wear a medical alert bracelet if you have a serious
arrhythmia.
 Educate family members on CPR in case of emergencies.
CONCLUSION
In conclusion ,arrhythmias are abnormal heart rhythms
that can range from harm- less to life-threating .however,
ongoing research is essential to enhance prevention, early
intervention & treatment strategies to reduce the risk of
complications like stoke and sudden cardiac arrest.
Cardiac Arrhythmias for paramedical students.

Cardiac Arrhythmias for paramedical students.

  • 1.
  • 3.
    Introduction: Without a regularrate & rhythm, the heart may not perform efficiently as a pump to circulate oxygenated blood & other life sustaining nutrition to all of the body’s tissues & organs with an irregular or rhythm, the heart is considered to be dysrhythmic( arrhythmic)  Dysrhythmia - bad rhythm  Arrhythmia – without rhythm
  • 4.
    Definition:  An arrhythmia(also called dysrhythmia) is an abnormal heartbeat. Arrhythmias can start in different parts of your heart and they can be too fast, too slow or just irregular. - WHO  It is an disorder of the formation or conduction (or both) of the electrical impulse within the heart. These disorders can cause disturbance of the heart rate, heart rhythm or both. - Brunner & Suddarth’s textbook of medical surgical nursing.
  • 5.
    INCIDENCE  The prevalenceof arrhythmias is expected to be 1.5% to 5% in the general population, with atrial fibrillation being the most common.  Arrhythmias may or may not produce any symptoms and can be paroxysmal, leading to difficulty in estimating true prevalence.  The overall presence of arrhythmia is associated with higher morbidity and mortality.
  • 6.
    ETIOLOGY There are anumber of factors that can cause a heart arrhythmia, including:  Heart attack or scarring of the heart tissue from a heart attack;  Coronary heart disease  High blood pressure  Smoking  Genetics: Changes to the heart’s structure e.g. from cardiomyopathy; Diabetes; Sleep apnea
  • 7.
     Alcohol abuse Excessive caffeine consumption  Overactive thyroid gland (hyperthyroidism)  Underactive thyroid gland (hypothyroidism)  Drug abuse  Stress
  • 8.
    NORMAL SINUS RHYTHM A normal sinus rhythm (NSR) is the regular rhythm of a healthy heart. It's caused by electrical impulses from the heart's natural pacemaker, the sinoatrial (SA) node.
  • 9.
    CHARACTERISTICS  ventricular &atrial rate: - 60- 100bpm  ventricular & atrial rhythm : regular  QRS shape & duration : normal  P – wave: normal & always in front of the QRS  PR interval : consistent interval 0.12 -0.20 sec  P:QRST ratio: 1:1
  • 10.
    CLASSIFICATION: Sinus node dysrhythmia. Atrialdysrhythmias. Junctional dysrhythmias. Ventricular dysrhythmias. Conduction abnormalities.
  • 11.
    SINUS NODE DYSRHYTHMIA Sinusbradycardia. Sinus tachycardia. Sinus arrhythmia.
  • 12.
    ATRIAL DYSRHYTHMIAS.  Prematureatrial complex.  Atrial fibrillation  Atrial flutter
  • 13.
    JUNCTIONAL DYSRHYTHMIAS.  Prematurejunctional complex  Junctional rhythm  Non paroxysmal junctional tachycardia  Atrioventricular nodal reentry tachycardia
  • 14.
    VENTRICULAR DYSRHYTHMIAS.  Premature ventricularcomplex.  Ventricular fibrillation.  Idioventricular rhythm.  Ventricular asystole.
  • 15.
    CONDUCTION ABNORMALITIES. First –degree AVblock  Delayed conduction from the atrium to the ventricle (defined as a prolonged pr interval of >200 ms) without interruption in atrial to ventricular conduction.
  • 16.
    Second-degree AV block Second-degree atrioventricular (AV) block is a heart condition that occurs when some atrial impulses are blocked from reaching the ventricles. It's a type of "incomplete" heart block.
  • 17.
    Symptoms lightheadedness andsyncope. Types  Mobitz type I: The site of the block is usually within the AV node.  Mobitz type II: The site of the block is almost always below the AV node.
  • 18.
    Third-degree atrioventricular (AV)block, also known as complete heart block (CHB),  It is a condition that prevents electrical signals from traveling between the heart's atria and ventricles. This results in the atria and ventricles beating independently, which can lead to insufficient blood flow and life-threatening complications.
  • 19.
    SINUS BRADYCARDIA  Sinusbradycardia is a heart rhythm where the heart beats slower than normal, but in a regular pattern. It's defined as a heart rate of less than 60 beats per minute (bpm) in adults.
  • 20.
    CHARACTERISTIC  Ventricular &atrial rate: < 60bpm  Ventricular & atrial rhythm : regular  QRS shape & duration : normal  P – wave: normal & always in front of the QRS  PR interval : consistent interval 0.12 -0.20 sec  P:QRST ratio: 1:1
  • 21.
    MANAGEMENT  Atropine- 0.5mgIV bolus.  After 3-5 mins, max dosage of 3mg will be administer  Dopamine /epinephrine
  • 22.
    SINUS TACHYCARDIA Sinus tachycardiais a regular heart rhythm where the heart beats faster than normal. It's often a normal response to exercise or stress, but it can also be a sign of an underlying condition. Symptoms of sinus tachycardia  Palpitations,  Fainting, Chest pain,  Difficulty breathing, and Dizziness.
  • 23.
    CHARACTERISTICS  ventricular &atrial rate:> 100bpm <120bpm  ventricular & atrial rhythm : regular  QRS shape & duration : normal  P – wave: normal & always in front of the QRS  PR interval : consistent interval 0.12 -0.20 sec  P:QRST ratio: 1:1
  • 24.
    MANAGEMENT  Beta-blockers: metoprolol,nadolol, and propranolol, these can help with tachycardia, especially during exercise  Calcium channel blockers: verapamil can help lower your heart rate  Anti-arrhythmic medications: potassium or sodium channel blockers, adenosine, digoxin, and magnesium sulfate
  • 25.
    SINUS ARRHYTHMIA Sinus arrhythmiais a variation in the heart's rhythm that's usually normal and indicates good heart health. It's a type of arrhythmia, which is an abnormal heart rhythm.  When you breathe in, your heart rate increases and the time between heartbeats shortens.  When you breathe out, your heart rate decreases and the time between heartbeats lengthens.
  • 26.
    CHARACTERISTIC  Ventricular &atrial rate:60-100bpm  Ventricular & atrial rhythm : irregular  QRS shape & duration : normal  P – wave: normal & always in front of the QRS  PR interval : consistent interval 0.12 -0.20 sec  P:QRST ratio: 1:1
  • 27.
    MANAGEMENT It does notcause any significant hemodynamic effects & therefore is not typically treated.
  • 28.
    PREMATURE ATRIAL COMPLEX  PACis a single ECG complex that occurs when an electrical impulse starts in the atrium before the next normal impulse of the sinus node.
  • 29.
    characteristics  Ventricular &atrial rate: depends on the underlying rhythm  Ventricular & atrial rhythm : irregular due to early p wave  QRS shape & duration : due do early p wave is abnormal  P – wave: seen or be hidden  PR interval : p wave is shorter than normal PR interval 0.12 -0.20 sec  P:QRST ratio: 1:1
  • 30.
    MANAGEMENT Treatment is notnecessary but, they focus on underlying causes e.g.: Intake of caffeine, correction of hypokalemia
  • 31.
    ATRIAL FIBRILLATION Atrial fibrillation(afibor AF) is an irregular heartbeat that occurs when the upper chambers of the heart beat too fast and out of rhythm. It's the most common type of arrhythmia that lasts more than a few seconds. Risk factors:  Post operative cardiac surgery  Hypertension  Obesity  VHD  Heart failure
  • 32.
    CHARACTERISTIC  Ventricular &atrial rate: atrial rate 300-600bpm ventricular rate 120-200bpm in untreated AF  Ventricular & atrial rhythm :highly irregular  QRS shape & duration :usually normal maybe abnormal  P – wave: no discernible P wave  PR interval : cannot be measured  P:QRST ratio: many:1
  • 33.
    Management  Beta blockers:Slow the heart rate  Calcium channel blockers: Control the heart rate  Digoxin: Controls the heart rate at rest  Blood thinners: Prevent blood clots and reduce the risk of stroke  Anti-arrhythmics: Restore normal heart rhythm
  • 34.
    ATRIAL FLUTTER Atrial flutteris a heart rhythm abnormality that causes the upper chambers of the heart to beat too fast.
  • 35.
    CHARACTERISTICS  Ventricular &atrial rate: -atrial rate 250-400bpm ventricular rate 75-150bpm  Ventricular & atrial rhythm : atrial rhythm is regular but ventricular maybe irregular  QRS shape & duration : usually normal, maybe abnormal or may be absent.  P – wave: saw-toothed shape  PR interval :multiple f wave cannot be measure  P:QRST ratio: 2:1,3:1or4:1
  • 36.
    Management  Adenosine IVfollowed by 20ml saline flush  Elevation of arm with IV line to promote rapid circulation of medication.  Antiarrhythmics - amiodarone  Beta blockers- adrenaline  Calcium channel blockers  Anticoagulants- prevent blood clots, such as heparin, warfarin,
  • 37.
    PREMATURE JUNCTIONAL COMPLEX A prematurejunctional complex (PJC) is an extra heartbeat that originates in the atrioventricular (AV) junction of the heart. Causes:  Heart failure  CAD
  • 38.
    CHARACTERISTICS  P wave:may be inverted, absent, or occur after the QRS complex  PR interval: short or absent  QRS complex: normal in shape and morphology
  • 39.
    JUNCTIONAL RHYTHM Junctional oridionodal rhythm occurs when the AV node , instead of the sinus node, become the pacemaker of the heart.
  • 40.
    CHARACTERISTIC  ventricular &atrial rate: 40-60bpm  ventricular & atrial rhythm : regular  QRS shape & duration : normal/abnormal  P – wave: maybe absent after or before QRS complex  PR interval : PR interval is < 0.12 sec  P:QRST ratio: 1:1 or 0:1
  • 41.
  • 42.
    NON PAROXYSMAL JUNCTIONAL TACHYCARDIA NPJTis a form of SPV originating from AV junction with a gradual onset & termination  it is similar to junctional rhythm
  • 43.
    ATRIOVENTRICULAR NODAL REENTRY TACHYCARDIA Itis a type of abnormal, rapid heart rhythm where electrical signals within the atrioventricular (AV) node of the heart loop back on themselves, creating a "reentry circuit" that causes the heart to beat very fast, often with sudden onset and termination
  • 44.
    CHARACTERISTICS  Ventricular &atrial rate: - 60- 100bpm  Ventricular & atrial rhythm : regular  QRS shape & duration : normal  P – wave: normal & always in front of the QRS  PR interval : consistent interval 0.12 -0.20 sec  P:QRST ratio: 1:1
  • 45.
    MANAGEMENT  Vagal maneuvers:simple actions like coughing, holding your breath, or applying cold water to the face can sometimes stop an AVNRT episode.  Adenosine: this is the first-line medication  Calcium channel blockers: verapamil can be used.  Beta-blockers
  • 46.
    PREMATURE VENTRICULAR CONTRACTION Aventricular premature complex (VPC), also known as a premature ventricular contraction (PVC), is an extra heartbeat that originates in the heart's lower chambers. Pvc are common and usually harmless.
  • 47.
    CHARACTERS  Ventricular &atrial rate: depends on the underlying rhythm  Ventricular & atrial rhythm : irregular  P – wave: visible P wave  PR interval : consistent interval 0.12 sec  P:QRST ratio: 0:1 ,1:1
  • 48.
    MEDICAL MANAGEMENT  Betablocker : to lower blood pressure and reduce pvcs  Calcium channel blockers: to lower blood pressure and reduce pvc  Antiarrhythmic: to control irregular heart rhythms
  • 49.
    VENTRICULAR TACHYCARDIA Ventricular tachycardia(VT) is a condition in which the heart's lower chambers beat abnormally fast. It can be life- threatening and may require immediate medical care.
  • 50.
    Symptoms •Chest pain •Lightheadedness ordizziness •Fainting •Shortness of breath •Palpitations •Anxiety •Tightness in the neck •Tiredness
  • 51.
    CAUSES •Heart attack •Anti-arrhythmic drugs •Congenitalheart disease •Ischemic heart disease •Structural heart disease •Electrolyte imbalances
  • 52.
    CHARACTERISTICS  ventricular &atrial rate: - 100-200bpm  ventricular & atrial rhythm : regular  QRS shape & duration : abnormal  P – wave: very defect to detect  PR interval : irregular  P:QRST ratio: difficult to determine
  • 53.
    MEDICAL MANAGEMENT Defibrillation torestore normal heart rhythm and Anti- arrhythmic medication injections.
  • 54.
    VENTRICULAR FIBRILLATION Ventricular fibrillation(v-fib) is a life-threatening heart rhythm disorder that causes the heart's ventricles to quiver instead of pumping blood. It's the most common cause of sudden cardiac death.
  • 55.
    SYMPTOMS •Chest pain, fullness,discomfort, or pressure •Dizziness or lightheadedness •Nausea •Racing or erratic pulse •Heart palpitations •Shortness of breath
  • 56.
    CAUSES •Heart attack •Heart disease •Heartsurgery •Sudden blow to the chest •Medicines •Potassium levels in the blood
  • 57.
    MANAGEMENT  Treatment cardiopulmonaryresuscitation (CPR),  Shocks to the heart with an automated external defibrillator (AED),  Medications,  Implanted devices and surgery.
  • 58.
    IDIOVENTRICULAR RHYTHM Idioventricular rhythmis a heart rhythm where the lower chambers of the heart beat more slowly than normal. It's also known as a "slow ventricular tachycardia”
  • 59.
    CHARACTERISTICS  A rateof less than 50 beats per minute  No conducted P waves, andA widened QRS complex.
  • 60.
    MANAGEMENT  Atropine maybe used to increase heart rate, and verapamil,  Antiarrhythmic drugs
  • 61.
    VENTRICULAR ASYSTOLE Ventricular asystoleis a complete absence of the heart's electrical and mechanical activity, also known as flat line.
  • 62.
    MANAGEMENT  High –qualityCPR  Assess for possible causes Hs  Hypoxia  Hypovolemia  Hydrogen ion (acid- base imbalance)  Hypo/hyperglycemia  Hypo/hyperkalemia  hyperthermia
  • 63.
    Ts  Trauma  Toxins Tamponade  Tension pneumothorax  Thrombus CPR>INTUBATION>IV MEDICATION(emergency med)
  • 64.
    SYMPTOMS  Tightness  Chestpain  Palpitation  Sob  Dizziness  Fainting
  • 65.
    DIAGNOSTIC EVALUATION  HistoryCollection  Physical Examination  ECG  Echo- cardiogram  Blood test -electrolyte -cardiac biomarker  Genetic testing(if needed)
  • 66.
    COMPLICATIONS  Stroke  Heartfailure  Sudden cardiac arrest  Cardiogenic shock  Chronic fatigue  MI  End- organ damage
  • 67.
    SURGICAL MANAGEMENT  Mazeprocedure  Valve replacement  Pacemaker & implantable cardioverter defibrillator(ICD)  CABG  Heart transplantation
  • 68.
    NURSING DIAGNOSIS  DecreasedCardiac Output Related to Altered heart rate and rhythmAs evidenced by Irregular pulse  Risk for Decreased CardiacTissue Perfusion Related to Inadequate blood flow due to abnormal heart rhythms  IneffectiveTissue Perfusion Related to Inadequate cardiac output from arrhythmia as evidenced by weak pulses  Activity Intolerance Related to Insufficient oxygenation due to abnormal heart rhythms as evidenced by Fatigue  Anxiety Related to Fear of life-threatening arrhythmia episodes as evidenced by Restlessness
  • 69.
    HEALTH EDUCATION Lifestyle Changes Eat a healthy diet (low salt, low cholesterol, more vegetables).  Exercise regularly (as recommended by your doctor).  Quit smoking and limit alcohol and caffeine.  Reduce stress with relaxation techniques like yoga or meditation .  Monitor your blood pressure, cholesterol, and blood sugar.
  • 70.
    Seek Emergency Help Call emergency services immediately if you experience:  Severe chest pain or pressure.  Sudden shortness of breath.  Fainting or losing consciousness.  A very fast or very slow heart rate that doesn’t stop.
  • 71.
    Patient Self-Care &Monitoring  Check your pulse regularly and report irregularities to your doctor.  Take medications as prescribed and attend all follow-up visits.  Wear a medical alert bracelet if you have a serious arrhythmia.  Educate family members on CPR in case of emergencies.
  • 72.
    CONCLUSION In conclusion ,arrhythmiasare abnormal heart rhythms that can range from harm- less to life-threating .however, ongoing research is essential to enhance prevention, early intervention & treatment strategies to reduce the risk of complications like stoke and sudden cardiac arrest.