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Cardiac Ultrasound in
Emergency Medicine
Anthony J. Weekes MD, RDMS
Sarah A. Stahmer MD
For the SAEM US Interest Group
Primary Indications
 Thoraco-abdominal trauma
 Pulseless Electrical Activity
 Unexplained hypotension
 Suspicion of pericardial
effusion/tamponade
Secondary Indications
 Acute Cardiac Ischemia
 Pericardiocentesis
 External pacer capture
 Transvenous pacer placement
Main Clinical Questions
 What is the overall cardiac wall motion?
 Is there a pericardial effusion?
Cardiac probe selection
 Small round footprint
for scan between ribs
 2.5 MHz: above
average sized patient
 3.5 MHz: average
sized patient
 5.0 MHz: below
average sized patient
or child
Main cardiac views
 Parasternal
 Subcostal
 Apical
Wall Motion
 Normal
 Hyperkinetic
 Akinetic
 Dyskinetic: may fail
to contract, bulges
outward at systole
 Hypokinetic
Orientation
 Subcostal or subxiphoid view
 Best all around imaging window
 Good for identification of:
– Circumferential pericardial effusion
– Overall wall motion
 Easy to obtain – liver is the acoustic
window
Subcostal View
 Most practical in
trauma setting
 Away from airway
and neck/chest
procedures
Subcostal View
 Liver as acoustic
window
 Alternative to
apical 4 chamber
view
Subcostal View
Subcostal View
Subcostal View
 Angle probe right to
see IVC
 Response of IVC to
sniff indicates central
venous pressure
 No collapse
– Tamponade
– CHF
– PE
– Pneumothorax
Parasternal Views
 Next best imaging window
 Good for imaging LV
 Comparing chamber sizes
 Localized effusions
 Differentiating pericardial from pleural
effusions
Parasternal Long Axis
 Near sternum
 3rd or 4th left intercostal space
 Marker pointed to patient’s right
shoulder (or left hip if screen is not
reversed for cardiac imaging)
 Rotate enough to elongate cardiac
chambers
Parasternal Long Axis
Parasternal Long Axis View
Parasternal Short Axis
 Obtained by 90° clockwise rotation
of the probe towards the left
shoulder (or right hip)
 Sweep the beam from the base of
the heart to the apex for different
cross sectional views
Parasternal Short Axis View
Parasternal Short Axis
Apical View
 Difficult view to obtain
 Allows comparison of ventricular
chamber size
 Good window to assess septal/wall
motion abnormalities
Apical Views
 Patient in left
lateral decubitus
position
 Probe placed at
PMI
 Probe marker at 6
o’clock (or right
shoulder)
 4 chamber view
Apical 4 chamber view
 Marker pointed to
the floor
 Similar to
parasternal view
but apex well
visualized
 Angle beam
superiorly for 5
chamber view
Apical 4 chamber view
Apical 2 chamber view
 Patient in left
lateral decubitus
position
 Probe placed at
PMI
 Probe marker at 3
o’clock
 2 chamber view
Apical 2 chamber view
 Good look at inferior and anterior walls
Apical 2 chamber view
 From apical 4,
rotate probe 90°
counterclockwise
 Good view for
long view of left
sided chambers
and mitral valve
Abnormal findings
Pericardial Effusion
Case Presentation
 45 year old male presents with SOB
and dizziness for 2 days. He has a long
smoking history, and has complained of
a non-productive cough for “weeks”
 Initial VS are BP 88/palp, HR 140
 PE: Neck veins are distended
 Chest: Clear, muffled heart sounds
 Bedside sonography was performed
Echo free space around the heart
 Pericardial effusion
 Pleural effusion
 Epicardial fat (posterior and/or
anterior)
 Less common causes:
– Aortic aneurysm
– Pericardial cyst
– Dilated pulmonary artery
Size of the Pericardial
Effusion
 Not Precise
 Small: confined to posterior space,
< 0.5cm
 Moderate: anterior and posterior,
0.5-2cm (diastole)
 Large: > 2cm
Pericardial Fluid: Subcostal
Clinical features of
Pericardial effusion
 Pericardial fluid accumulation may
be clinically silent
 Symptoms are due to:
– mechanical compression of adjacent
structures
– Increased intrapericardial pressure
Pericardial
Effusion:Asymptomatic
 Up to 40% of pregnant women
 Chronic hemodialysis patients
– one study showed 11% incidence of
pericardial effusion
 AIDS
 CHF
 Hypoproteinemic states
Symptoms of Pericardial
Effusion
 Chest discomfort (most common)
 Large effusions:
– Dyspnea
– Cough
– Fatigue
– Hiccups
– Hoarseness
– Nausea and abdominal fullness
Cardiac Tamponade
 Increased intracardiac pressures
 Limitation of ventricular diastolic
filling
 Reduction of stroke volume and
cardiac output
Ventricular collapse in
diastole
Tamponade
Hypotension
Abnormal findings
 Is the cause of hypotension cardiac in
etiology?
 Is it due to a pericardial effusion?
 Is is due to pump failure?
Unexplained Hypotension
 Cardiogenic shock
– Poor LV contractility
 Hypovolemia
– Hyperdynamic ventricules
 Right ventricular infarct/large
pulmonary embolism
– Marked RV dilitation/hypokinesis
 Tamponade
– RV diastolic collapse
Cardiogenic shock
 Dilated left
ventricle
 Hypocontractile
walls
Hypovolemia
 Small chamber filling size
 Aggressive wall motion
 Flat IVC or exaggerated collapse
with deep inspiration
Massive PE or RV infarct
 Dilated Right
ventricle
 RV hypokinesis
 Normal Left
ventricle function
 Stiff IVC
Case presentation ? overdose
 27 yo f brought in with “passing out”
after night of heavy drinking.
 Complaining of inability to breathe!
 PE: Obese f BP 88/60 HR 123 Ox
78%
 Chest: clear
 Ext: No edema
 Bedside sonography was performed
Chest pain then code
 55 yo male suffered witnessed Vfib
arrest in the ED
 ALS protocol - restoration of perfusing
rhythm
 Persistant hypotension
 ED ECHO was performed
R sided leads
Non Traumatic
Resuscitation
Direct Visualization
 Is there effective myocardial
contractility?
– Asystole
– Myocardial “twitch”
– Hypokinesis
– Normal
 Is there a pericardial effusion?
ECHO in PEA
 Perform ECHO during “quick look”
and in pulse checks
 Change management based on
“positive” findings
 Pericardial tamponade
– Pericardiocentesis
 Hyperdynamic cardiac wall motion
– Volume resuscitate
ECHO in PEA
 RV dilatation
– Hypoxic?? – Likely PE
– ECG – IMI with RV infarct?
 Profound hypokinesis
– Inotropic support
 Asystole
– Follow ACLS protocols (for now)
– Early data suggesting poor prognosis
ECHO in PEA
 False positive cardiac motion
– Transthoracic pacemaker
– Positive pressure ventilation
Case presentation
 Morbidly obese female with severe asthma
 Intubated for respiratory failure
 Subcutaneous emphysema developed
 Bilateral chest tubes placed
 Persistent hypotension at 90/palp
 Dependent mottling noted
 ECHO was performed
Ineffective cardiac
contractions
Optimizing Performance
 Assessing capture by transthoracic
pacemaker
 Pericardiocentesis
 Transvenous pacemaker placement
Optimizing Performance
 Assessment of capture by transthoracic
pacemaker
 Ettin D et al: Using ultrasound to
determine external pacer capture JEM
1999
Case Presentation
70 yo f collapsed in lobby. She was brought into
the ED apneic, hypotensive. She was quickly
intubated and volume resuscitation begun.
VS: BP 80/50 HR 50 Afebrile
Physical exam : Thin, minimally responsive f.
Clear lungs, nl heart sounds, abdomen slightly
distended with decreased bowel sounds. No
HSM, ? Pelvic mass
ECG: SB, LVH, no active ischemia
Clinical questions?
 Why is she hypotensive?
 Volume loss
?Ruptured AAA
 Pump failure
 Bedside sonography was performed
while we were waiting for the “labs”
Increase HR with PM “on”
What did this tell us?
 Normal wall motion
 No pericardial/pleural effusion
 Good capture with the transthoracic PM
Asystole w/ Transthoracic PM
Optimizing performance
 Pericardiocentesis
– Standard of care by cardiology/CT surgery
to use ECHO to guide aspiration
US Guided-
Pericardiocentesis
 Subcostal approach
– Traditional approach
– Blind
– Increased risk of injury to liver, heart
 Echo guided
– Left parasternal preferred for needle entry
or…
– Largest area of fluid collection adjacent to
the chest wall
Large pericardial effusion
Technique
Optimizing performance
 Placement of transvenous pacemaker
 Aguilera P et al: Emergency
transvenous cardiac pacing placement
using ultrasound guidance. Ann Emerg
Med 2000
Untimely end
 30 yo brought in after he “fell out”
 Ashen m with no spontaneous
respirations
 VS: No pulse, agonal rhythm on monitor
 Intubated/CPR
 Transvenous pacemaker placed, no
capture.
 ECHO showed
Penetrating Chest Trauma
Penetrating Cardiac Trauma
 Physician’s ability to determine whether there is
a hemodynamically significant effusion is poor
 Beck’s Triad
– Dependent on patient cardiovascular status
– Findings are often late
 Determinants of hemodynamic compromise
– Size of the effusion
– Rate of formation
Penetrating Cardiac Injury
 Emergency department
echocardiography improves outcome in
penetrating cardiac injury.
Plummer D et al. Ann Emerg Med. 1992
 28 had ED echo c/w 21 without ED echo
 Survival: 100% in echo, 57.1% in nonecho
 Time to Dx: 15 min echo, 42 min nonecho
Penetrating Cardiac Injury
The role of ultrasound in patients with possible
penetrating cardiac wounds: a prospective
multicenter study.
Rozycki GS: J Trauma. 1999
 Pericardial scans performed in 261 patients
 Sensitivity 100%, specificity 96.9%
 PPV: 81% NPV:100%
 Time interval BUS to OR: 12.1 +/- 5.9 min
Emergency Department Echocardiography
Improves Outcome in Penetrating Cardiac
Injury
Plummer D, et al. Ann Emerg Med 21:709-712, 1992.
“Since the introduction of immediate ED two-
dimensional echocardiography, the time to
diagnosis of penetrating cardiac injury has
decreased and both the survival rate and
neurologic outcome of survivors has improved.”
Penetrating Cardiac Trauma
Stab wound to the chest
 Echocardiographic signs of rising
intrapericardial pressure
– Collapse of RV free walls
– Dilated IVC and hepatic veins
 Goal: Early detection of pericardial effusion
– Develops suddenly or discretely
– May exist before clinical signs develop
 Salvage rates better if detected before
hypotension develops
Penetrating Cardiac Trauma
Technical Problems
 Subcutaneous air
 Pneumopericardium
 Mechanical ventilation
 Scanning limited by:
– Pain/tenderness
– Spinal immobilization
– Ongoing procedures
Technical Problems
 Narrow intercostal spaces
 Obesity
 Muscular chest
 COPD
 Calcified rib cartilages
 Abdominal distention
Sonographic Pitfalls
 Pericardial versus pleural fluid
 Pericardial clot
 Pericardial fat
Pericardial or Pleural Fluid
 Left parasternal long axis:
– Pericardial fluid does not extend posterior
to descending aorta or left atrium
 Subcostal:
– No pleural reflection between liver and R
sided chambers
– A pleural effusion will not extend between
to RV free wall and the liver
Pleural and Pericardial fluid
Pleural effusion
Blunt Cardiac Trauma
 Cardiac contusion
 Cardiac rupture
 Valvular disruption
 Aortic disruption/dissection
Blunt Cardiac Trauma
 Pericardial effusion
 Assess for wall motion abnormality
– RV dyskinesis (takes the first hit)
 Assess thoracic aorta:
– Hematoma
– Intimal flap
– Abnormal contour
 Valvular dysfunction or septal rupture
Cardiac Contusion
 Akinetic anterior RV wall
 Small pericardial effusion
 Diminished ejection fraction
RV Contusion
Blunt Cardiac Trauma
 Assess thoracic aorta
– Hematoma
– Intimal flap
– Abnormal contour
– Requires TEE and expertise!
 Valvular dysfunction or septal rupture
– Requires expertise beyond our scope
Summary
 Bedside ECHO can help assess:
– Overall cardiac wall motion
– Identify clinically significant pericardial effusions
 Useful in the assessment of the patient with:
– Unexplained hypotension
– Dyspnea
– Thoracic trauma

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26 USCardio (1).ppt

  • 1. Cardiac Ultrasound in Emergency Medicine Anthony J. Weekes MD, RDMS Sarah A. Stahmer MD For the SAEM US Interest Group
  • 2. Primary Indications  Thoraco-abdominal trauma  Pulseless Electrical Activity  Unexplained hypotension  Suspicion of pericardial effusion/tamponade
  • 3. Secondary Indications  Acute Cardiac Ischemia  Pericardiocentesis  External pacer capture  Transvenous pacer placement
  • 4. Main Clinical Questions  What is the overall cardiac wall motion?  Is there a pericardial effusion?
  • 5. Cardiac probe selection  Small round footprint for scan between ribs  2.5 MHz: above average sized patient  3.5 MHz: average sized patient  5.0 MHz: below average sized patient or child
  • 6. Main cardiac views  Parasternal  Subcostal  Apical
  • 7. Wall Motion  Normal  Hyperkinetic  Akinetic  Dyskinetic: may fail to contract, bulges outward at systole  Hypokinetic
  • 8. Orientation  Subcostal or subxiphoid view  Best all around imaging window  Good for identification of: – Circumferential pericardial effusion – Overall wall motion  Easy to obtain – liver is the acoustic window
  • 9. Subcostal View  Most practical in trauma setting  Away from airway and neck/chest procedures
  • 10. Subcostal View  Liver as acoustic window  Alternative to apical 4 chamber view
  • 13. Subcostal View  Angle probe right to see IVC  Response of IVC to sniff indicates central venous pressure  No collapse – Tamponade – CHF – PE – Pneumothorax
  • 14. Parasternal Views  Next best imaging window  Good for imaging LV  Comparing chamber sizes  Localized effusions  Differentiating pericardial from pleural effusions
  • 15. Parasternal Long Axis  Near sternum  3rd or 4th left intercostal space  Marker pointed to patient’s right shoulder (or left hip if screen is not reversed for cardiac imaging)  Rotate enough to elongate cardiac chambers
  • 18. Parasternal Short Axis  Obtained by 90° clockwise rotation of the probe towards the left shoulder (or right hip)  Sweep the beam from the base of the heart to the apex for different cross sectional views
  • 21. Apical View  Difficult view to obtain  Allows comparison of ventricular chamber size  Good window to assess septal/wall motion abnormalities
  • 22. Apical Views  Patient in left lateral decubitus position  Probe placed at PMI  Probe marker at 6 o’clock (or right shoulder)  4 chamber view
  • 23. Apical 4 chamber view  Marker pointed to the floor  Similar to parasternal view but apex well visualized  Angle beam superiorly for 5 chamber view
  • 25. Apical 2 chamber view  Patient in left lateral decubitus position  Probe placed at PMI  Probe marker at 3 o’clock  2 chamber view
  • 26. Apical 2 chamber view  Good look at inferior and anterior walls
  • 27. Apical 2 chamber view  From apical 4, rotate probe 90° counterclockwise  Good view for long view of left sided chambers and mitral valve
  • 29. Case Presentation  45 year old male presents with SOB and dizziness for 2 days. He has a long smoking history, and has complained of a non-productive cough for “weeks”  Initial VS are BP 88/palp, HR 140  PE: Neck veins are distended  Chest: Clear, muffled heart sounds  Bedside sonography was performed
  • 30.
  • 31. Echo free space around the heart  Pericardial effusion  Pleural effusion  Epicardial fat (posterior and/or anterior)  Less common causes: – Aortic aneurysm – Pericardial cyst – Dilated pulmonary artery
  • 32. Size of the Pericardial Effusion  Not Precise  Small: confined to posterior space, < 0.5cm  Moderate: anterior and posterior, 0.5-2cm (diastole)  Large: > 2cm
  • 34. Clinical features of Pericardial effusion  Pericardial fluid accumulation may be clinically silent  Symptoms are due to: – mechanical compression of adjacent structures – Increased intrapericardial pressure
  • 35. Pericardial Effusion:Asymptomatic  Up to 40% of pregnant women  Chronic hemodialysis patients – one study showed 11% incidence of pericardial effusion  AIDS  CHF  Hypoproteinemic states
  • 36. Symptoms of Pericardial Effusion  Chest discomfort (most common)  Large effusions: – Dyspnea – Cough – Fatigue – Hiccups – Hoarseness – Nausea and abdominal fullness
  • 37. Cardiac Tamponade  Increased intracardiac pressures  Limitation of ventricular diastolic filling  Reduction of stroke volume and cardiac output
  • 41. Abnormal findings  Is the cause of hypotension cardiac in etiology?  Is it due to a pericardial effusion?  Is is due to pump failure?
  • 42. Unexplained Hypotension  Cardiogenic shock – Poor LV contractility  Hypovolemia – Hyperdynamic ventricules  Right ventricular infarct/large pulmonary embolism – Marked RV dilitation/hypokinesis  Tamponade – RV diastolic collapse
  • 43. Cardiogenic shock  Dilated left ventricle  Hypocontractile walls
  • 44. Hypovolemia  Small chamber filling size  Aggressive wall motion  Flat IVC or exaggerated collapse with deep inspiration
  • 45. Massive PE or RV infarct  Dilated Right ventricle  RV hypokinesis  Normal Left ventricle function  Stiff IVC
  • 46. Case presentation ? overdose  27 yo f brought in with “passing out” after night of heavy drinking.  Complaining of inability to breathe!  PE: Obese f BP 88/60 HR 123 Ox 78%  Chest: clear  Ext: No edema  Bedside sonography was performed
  • 47.
  • 48.
  • 49. Chest pain then code  55 yo male suffered witnessed Vfib arrest in the ED  ALS protocol - restoration of perfusing rhythm  Persistant hypotension  ED ECHO was performed
  • 50.
  • 51.
  • 54. Direct Visualization  Is there effective myocardial contractility? – Asystole – Myocardial “twitch” – Hypokinesis – Normal  Is there a pericardial effusion?
  • 55. ECHO in PEA  Perform ECHO during “quick look” and in pulse checks  Change management based on “positive” findings  Pericardial tamponade – Pericardiocentesis  Hyperdynamic cardiac wall motion – Volume resuscitate
  • 56. ECHO in PEA  RV dilatation – Hypoxic?? – Likely PE – ECG – IMI with RV infarct?  Profound hypokinesis – Inotropic support  Asystole – Follow ACLS protocols (for now) – Early data suggesting poor prognosis
  • 57. ECHO in PEA  False positive cardiac motion – Transthoracic pacemaker – Positive pressure ventilation
  • 58. Case presentation  Morbidly obese female with severe asthma  Intubated for respiratory failure  Subcutaneous emphysema developed  Bilateral chest tubes placed  Persistent hypotension at 90/palp  Dependent mottling noted  ECHO was performed
  • 60. Optimizing Performance  Assessing capture by transthoracic pacemaker  Pericardiocentesis  Transvenous pacemaker placement
  • 61. Optimizing Performance  Assessment of capture by transthoracic pacemaker  Ettin D et al: Using ultrasound to determine external pacer capture JEM 1999
  • 62. Case Presentation 70 yo f collapsed in lobby. She was brought into the ED apneic, hypotensive. She was quickly intubated and volume resuscitation begun. VS: BP 80/50 HR 50 Afebrile Physical exam : Thin, minimally responsive f. Clear lungs, nl heart sounds, abdomen slightly distended with decreased bowel sounds. No HSM, ? Pelvic mass ECG: SB, LVH, no active ischemia
  • 63. Clinical questions?  Why is she hypotensive?  Volume loss ?Ruptured AAA  Pump failure  Bedside sonography was performed while we were waiting for the “labs”
  • 64. Increase HR with PM “on”
  • 65. What did this tell us?  Normal wall motion  No pericardial/pleural effusion  Good capture with the transthoracic PM
  • 67. Optimizing performance  Pericardiocentesis – Standard of care by cardiology/CT surgery to use ECHO to guide aspiration
  • 68. US Guided- Pericardiocentesis  Subcostal approach – Traditional approach – Blind – Increased risk of injury to liver, heart  Echo guided – Left parasternal preferred for needle entry or… – Largest area of fluid collection adjacent to the chest wall
  • 71. Optimizing performance  Placement of transvenous pacemaker  Aguilera P et al: Emergency transvenous cardiac pacing placement using ultrasound guidance. Ann Emerg Med 2000
  • 72. Untimely end  30 yo brought in after he “fell out”  Ashen m with no spontaneous respirations  VS: No pulse, agonal rhythm on monitor  Intubated/CPR  Transvenous pacemaker placed, no capture.  ECHO showed
  • 73.
  • 75. Penetrating Cardiac Trauma  Physician’s ability to determine whether there is a hemodynamically significant effusion is poor  Beck’s Triad – Dependent on patient cardiovascular status – Findings are often late  Determinants of hemodynamic compromise – Size of the effusion – Rate of formation
  • 76. Penetrating Cardiac Injury  Emergency department echocardiography improves outcome in penetrating cardiac injury. Plummer D et al. Ann Emerg Med. 1992  28 had ED echo c/w 21 without ED echo  Survival: 100% in echo, 57.1% in nonecho  Time to Dx: 15 min echo, 42 min nonecho
  • 77. Penetrating Cardiac Injury The role of ultrasound in patients with possible penetrating cardiac wounds: a prospective multicenter study. Rozycki GS: J Trauma. 1999  Pericardial scans performed in 261 patients  Sensitivity 100%, specificity 96.9%  PPV: 81% NPV:100%  Time interval BUS to OR: 12.1 +/- 5.9 min
  • 78. Emergency Department Echocardiography Improves Outcome in Penetrating Cardiac Injury Plummer D, et al. Ann Emerg Med 21:709-712, 1992. “Since the introduction of immediate ED two- dimensional echocardiography, the time to diagnosis of penetrating cardiac injury has decreased and both the survival rate and neurologic outcome of survivors has improved.” Penetrating Cardiac Trauma
  • 79. Stab wound to the chest
  • 80.  Echocardiographic signs of rising intrapericardial pressure – Collapse of RV free walls – Dilated IVC and hepatic veins  Goal: Early detection of pericardial effusion – Develops suddenly or discretely – May exist before clinical signs develop  Salvage rates better if detected before hypotension develops Penetrating Cardiac Trauma
  • 81. Technical Problems  Subcutaneous air  Pneumopericardium  Mechanical ventilation  Scanning limited by: – Pain/tenderness – Spinal immobilization – Ongoing procedures
  • 82. Technical Problems  Narrow intercostal spaces  Obesity  Muscular chest  COPD  Calcified rib cartilages  Abdominal distention
  • 83. Sonographic Pitfalls  Pericardial versus pleural fluid  Pericardial clot  Pericardial fat
  • 84. Pericardial or Pleural Fluid  Left parasternal long axis: – Pericardial fluid does not extend posterior to descending aorta or left atrium  Subcostal: – No pleural reflection between liver and R sided chambers – A pleural effusion will not extend between to RV free wall and the liver
  • 87. Blunt Cardiac Trauma  Cardiac contusion  Cardiac rupture  Valvular disruption  Aortic disruption/dissection
  • 88. Blunt Cardiac Trauma  Pericardial effusion  Assess for wall motion abnormality – RV dyskinesis (takes the first hit)  Assess thoracic aorta: – Hematoma – Intimal flap – Abnormal contour  Valvular dysfunction or septal rupture
  • 89. Cardiac Contusion  Akinetic anterior RV wall  Small pericardial effusion  Diminished ejection fraction
  • 91. Blunt Cardiac Trauma  Assess thoracic aorta – Hematoma – Intimal flap – Abnormal contour – Requires TEE and expertise!  Valvular dysfunction or septal rupture – Requires expertise beyond our scope
  • 92. Summary  Bedside ECHO can help assess: – Overall cardiac wall motion – Identify clinically significant pericardial effusions  Useful in the assessment of the patient with: – Unexplained hypotension – Dyspnea – Thoracic trauma