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DIBETIC FOOT ULCER
ETIO-PATHOGENESIS &
MANAGEMENT
DR.ARUN BAL
S.L.RAHEJA HOSPITAL
METHOD OF OFF-LOADING
• BEDREST
• CRUCTHES
• WHEELCHAIR
• SPECIAL FOOTWEAR
• CONTACT CASTING
MECHANISM OF INJURY
• DIRECT PENETRATION OF SKIN
• SMALL AMOUNT OF FORCE
SUSTAINED OVER A PERIOD OF
TIME
• MODERATE AMOUNT OF
REPATATIVE FORCE
INDICATION OF
WORSENING INFECTION
• INCREASED DRAINAGE
• INCREASED ERYTHEMA
• SUDDEN INCREASE IN PAIN
• INCREASED WARMTH
• FOUL ODOUR
• LYMPHANGITIS
INDICATION OF WORSENING
INFECTION(CONT)
• INCREASED BLOOD GLUCOSE LEVEL
• INCREASED WBC LEVEL
• INCREASED E.S.R
• REDUCED QUADRICEPS ACTION
• PERSISTANT ANOREXIA
• HIGH SERUM CREATININE
• OFF LOADING OF
AFFECTED FOOT
WHAT CAUSES HIGH
PLANTAR PRESSURE?
• DISPLACEMENT OF
METATARSAL CUSHION
DISTALLY
• NON ENZYMATIC
GLYCOLISATION
• LIMITATION OF MOVEMENT OF
1ST MTP JOINT
• REDUCED ELASTICITY
WHAT CAUSES HIGH
PLANTAR PRESSURE?
• DECREASED SUBTALAR JOINT
MOVEMENT
• EXCESSIVE PLANTAR
KERATOSIS
• THICKENING OF SESMOID
• ADHESIONS & SCAR TISSUE
HOW DOES FOOT INJURY
OCCUR?
• PEAK PLANTAR PRESSURE:1340kPa
• SYSTOLIC BP 120 mm OF H:15 kPa
• CAPILLARY PRESSURE :6 kPa
• DELAYED/ABSENT RECOVERY FROM
ISCHAEMIA
• DELAYED/ABSENT RECOVERY OF
NORMAL TISSUE OXYGEN CONC.
HOW DOES FOOT INJRY
OCCUR?
• REPATATIVE MODERATE FORCE
• INFLAMMATION
• ERYTHEMA AND WARMTH
• COLLECTION OF EXUDATE
• BLISTER FORMATION
• BREAKDOWN OF SKIN --- ULCER
NEED FOR PROMPT
TREATMENT OF FOOT ULCER
• 85% OF DIABETIC FOOT
AMPUTATIONS ARE DUE TO
INADEQUATELY TREATED FOOT
ULCER
• 30-50% AMPUTEES REQUIRE
CONTRALATERAL AMPUTATION
IN 3 YEARS
NEED FOR PROMPT
TREATMENT IF FOOT ULCER
• 10% MORTALITY IN THREE YEARS IN
AMPUTEES
• ECONOMIC LOSS TO FAMILY AND
SOCIETY
• 22% REQUIRE IPSILATERAL HIGHER
AMPUTATION
FOOT ULCER ASSESSMENT
• PERIWOUND ERYTHEMA
• PERIWOUND ODEMA
• WOUND PURULENCE
• WOUND FIBRIN
• LIMB PITTING ODEMA
• LIMB BRAWNY ODEMA
FOOT ULCER ASSESSMENT
• WOUND GRANULATION
• VASCULAR STATUS
• WOUND MEASUREMENT
• OSTEOMYLITIS &
TENOSYNOVITIS
MECHANISM OF INJURY IN
DIABETIC FOOT
• NORMAL STRESS
• SHEAR STRESS
• FATIGUE
• STRESS CONCENTRATION
• ELATICITY
PRIMARY TREATMENT OF
DIABETIC FOOT ULCER
• EVALUATION
• METABOLIC CONTROL
• DEBRIDEMENT
• BACTERIAL CULTURE
PRIMARY TREATMENT OF
DIABETIC FOOT ULCER
• PARENTERAL ANTIBIOTICS
• OFF LOADING OF AFFECTED
FOOT
• REVASCULARIZATION
• CORRECT FOOTWEAR
OBJECTIVES OF DIABETIC
FOOT WEAR
• REDUCTION OF EXCESSIVE
PLANTAR PRESSURE
• REDUCTION OF SHOCK
• REDUCTION OF SHEAR
• ACCOMODATION OF DEFORMITY
• STABALIZATION OF DEFORMITY
• LIMITATION OF JOINT MOVEMENT
OBJECTIVES OF DIABETIC
FOOTWEAR
• WIDE TOEBOX
• EXTRA DEPTH
• SOFT UPPERS
• MCR/PLASTAZOAT INSOLE
• INSOLE WING PAD
• ORTHOWDGE CORRECTION
• WELL FITTING SOCKS
SURGERY FOR DIABETIC
FOOT ULCER
• PROPHYLACTIC
• THERAPEUTIC
PRPHYLACTIC SURGERY FOR
DIABETIC FOOT ULCER
• METATARSAL OSTEOTOMY
• METATARSAL HEAD RESECTION
• SESMOIDECTOMY
• DIGITAL ARTHROPLASTY
• BUNIONECTOMY
• LOCAL FLAPS

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1362465156 diabetic foot ulcer etiopathogenesis & management

  • 1. DIBETIC FOOT ULCER ETIO-PATHOGENESIS & MANAGEMENT DR.ARUN BAL S.L.RAHEJA HOSPITAL
  • 2. METHOD OF OFF-LOADING • BEDREST • CRUCTHES • WHEELCHAIR • SPECIAL FOOTWEAR • CONTACT CASTING
  • 3. MECHANISM OF INJURY • DIRECT PENETRATION OF SKIN • SMALL AMOUNT OF FORCE SUSTAINED OVER A PERIOD OF TIME • MODERATE AMOUNT OF REPATATIVE FORCE
  • 4. INDICATION OF WORSENING INFECTION • INCREASED DRAINAGE • INCREASED ERYTHEMA • SUDDEN INCREASE IN PAIN • INCREASED WARMTH • FOUL ODOUR • LYMPHANGITIS
  • 5. INDICATION OF WORSENING INFECTION(CONT) • INCREASED BLOOD GLUCOSE LEVEL • INCREASED WBC LEVEL • INCREASED E.S.R • REDUCED QUADRICEPS ACTION • PERSISTANT ANOREXIA • HIGH SERUM CREATININE
  • 6. • OFF LOADING OF AFFECTED FOOT
  • 7. WHAT CAUSES HIGH PLANTAR PRESSURE? • DISPLACEMENT OF METATARSAL CUSHION DISTALLY • NON ENZYMATIC GLYCOLISATION • LIMITATION OF MOVEMENT OF 1ST MTP JOINT • REDUCED ELASTICITY
  • 8. WHAT CAUSES HIGH PLANTAR PRESSURE? • DECREASED SUBTALAR JOINT MOVEMENT • EXCESSIVE PLANTAR KERATOSIS • THICKENING OF SESMOID • ADHESIONS & SCAR TISSUE
  • 9. HOW DOES FOOT INJURY OCCUR? • PEAK PLANTAR PRESSURE:1340kPa • SYSTOLIC BP 120 mm OF H:15 kPa • CAPILLARY PRESSURE :6 kPa • DELAYED/ABSENT RECOVERY FROM ISCHAEMIA • DELAYED/ABSENT RECOVERY OF NORMAL TISSUE OXYGEN CONC.
  • 10. HOW DOES FOOT INJRY OCCUR? • REPATATIVE MODERATE FORCE • INFLAMMATION • ERYTHEMA AND WARMTH • COLLECTION OF EXUDATE • BLISTER FORMATION • BREAKDOWN OF SKIN --- ULCER
  • 11. NEED FOR PROMPT TREATMENT OF FOOT ULCER • 85% OF DIABETIC FOOT AMPUTATIONS ARE DUE TO INADEQUATELY TREATED FOOT ULCER • 30-50% AMPUTEES REQUIRE CONTRALATERAL AMPUTATION IN 3 YEARS
  • 12. NEED FOR PROMPT TREATMENT IF FOOT ULCER • 10% MORTALITY IN THREE YEARS IN AMPUTEES • ECONOMIC LOSS TO FAMILY AND SOCIETY • 22% REQUIRE IPSILATERAL HIGHER AMPUTATION
  • 13. FOOT ULCER ASSESSMENT • PERIWOUND ERYTHEMA • PERIWOUND ODEMA • WOUND PURULENCE • WOUND FIBRIN • LIMB PITTING ODEMA • LIMB BRAWNY ODEMA
  • 14. FOOT ULCER ASSESSMENT • WOUND GRANULATION • VASCULAR STATUS • WOUND MEASUREMENT • OSTEOMYLITIS & TENOSYNOVITIS
  • 15. MECHANISM OF INJURY IN DIABETIC FOOT • NORMAL STRESS • SHEAR STRESS • FATIGUE • STRESS CONCENTRATION • ELATICITY
  • 16. PRIMARY TREATMENT OF DIABETIC FOOT ULCER • EVALUATION • METABOLIC CONTROL • DEBRIDEMENT • BACTERIAL CULTURE
  • 17. PRIMARY TREATMENT OF DIABETIC FOOT ULCER • PARENTERAL ANTIBIOTICS • OFF LOADING OF AFFECTED FOOT • REVASCULARIZATION • CORRECT FOOTWEAR
  • 18. OBJECTIVES OF DIABETIC FOOT WEAR • REDUCTION OF EXCESSIVE PLANTAR PRESSURE • REDUCTION OF SHOCK • REDUCTION OF SHEAR • ACCOMODATION OF DEFORMITY • STABALIZATION OF DEFORMITY • LIMITATION OF JOINT MOVEMENT
  • 19. OBJECTIVES OF DIABETIC FOOTWEAR • WIDE TOEBOX • EXTRA DEPTH • SOFT UPPERS • MCR/PLASTAZOAT INSOLE • INSOLE WING PAD • ORTHOWDGE CORRECTION • WELL FITTING SOCKS
  • 20. SURGERY FOR DIABETIC FOOT ULCER • PROPHYLACTIC • THERAPEUTIC
  • 21. PRPHYLACTIC SURGERY FOR DIABETIC FOOT ULCER • METATARSAL OSTEOTOMY • METATARSAL HEAD RESECTION • SESMOIDECTOMY • DIGITAL ARTHROPLASTY • BUNIONECTOMY • LOCAL FLAPS