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Gastroenteritis
• Inflammation of stomach and intestines
– Fluid, electrolyte loss
– Dehydration
• May be acute or chronic
• Nausea, vomiting, diarrhea
Dehydration
• Occurs more readily in infants and young children than it
does in adults.
• The risk is increased in infants and young children because
they have an increased extracellular fluid percentage and a
relative increase in body water compared to adults.
• Increased basal metabolic rate, increased body surface area,
immature renal function & increased insensible fluid loss
through temperature elevation also contribute to the increased
risk for dehydration in infants & young children as compared
to adults.
Dehydration
• Dehydration left unchecked leads to shock, so early
recognition and treatment of dehydration is critical to
prevent progression to hypovolemic shock.
• The goals of therapeutic management of dehydration
are to restore appropriate fluid balance and to prevent
complications
Dehydration
Nursing goals for the infant or child with dehydration are aimed at
restoring fluid volume & preventing progression to hypovolemia.
1. Provide oral rehydration to children for mild to moderate states
of dehydration.
2. Children with severe dehydration should receive intravenous
fluids.
3. Initially, administer 20 mL/kg of normal saline or lactated
Ringer, and then reassess the hydration status.
4. Once initial fluid balance is restored, the physician or nurse
practitioner may order intravenous fluids at the maintenance rate
or as much as 1.5 times maintenance.
Dehydration- Nursing
Management
Dehydration- Nursing
Management
Dehydration- Nursing
Management
Calculate Deficit
– Mild Dehydration: 3-5% deficit (50 ml/kg deficit, 30
ml/kg if >10 kg)
– Moderate Dehydration: 6-10% deficit (100 ml/kg
deficit, 60 ml/kg if >10 kg)
– Severe Dehydration: >10% deficit (120 ml/kg deficit)
Replacement over 24 hours
•First 8 hours: 50% Deficit + Maintenance
•Next 16 hours: 50% Deficit + Maintenance
Ongoing Fluid
• Ongoing fluid losses= 0.9% Sodium
Chloride +/- potassium chloride to replace
ongoing losses.
Vomiting
• Vomiting is the forceful expulsion of gastric
contents through the mouth.
• Vomiting in infants and children has many
different causes and is considered to be a
symptom of some other condition.
• Therapeutic management of vomiting most often
involves slow oral rehydration and at times may
require administration of antiemetics
• Nursing management focuses on promoting fluid and
electrolyte balance.
• Oral rehydration is accomplished successfully in most
outpatient cases of simple vomiting.
• Teach the primary caregiver about oral rehydration
• In the child with mild to moderate dehydration resulting
from vomiting, withhold oral feeding for 1 to 2 hours after
emesis, after which time oral rehydration can begin.
• Give the infant or child 0.5 to 2 ounces of oral rehydration
solution Q 15 minutes, depending on the child’s age & size.
(ounce= 2 table spoon)
Vomiting- Nursing Management
Nursing Management
• Most infants and children can retain this small amount of fluid if
fed the restricted amount every 15 minutes.
• As the child improves, larger amounts will be tolerated
• If oral rehydration is not possible due to continued nausea and
vomiting, intravenous fluids will likely be ordered.
• In some cases, antiemetics may be used to help control the
vomiting. Some antiemetics can cause drowsiness or other side
effects and should not be used until a definitive diagnosis is made
or severe pathologic processes can be excluded.
• Educate the family regarding the prevention of vomiting and use
of antiemetic therapy
Vomiting- Nursing Management
Diarrhea
• Is either an increase in the frequency or a
decrease in the consistency of stool. Diarrhea in
children can either be acute or chronic.
• Acute infectious diarrhea (gastroenteritis) remains
the leading cause of death for children worldwide.
Pathophysiology
• Acute diarrhea in children is most commonly caused by
viruses, but it may also be related to bacterial or parasitic
enteropathogens.
• Acute diarrhea may be bloody or nonbloody.
• Diarrhea may also occur in relation to antibiotic use.
• Though most cases of diarrhea in children are of acute
origin, diarrhea may also occur chronically.
• Chronic diarrhea is diarrhea that lasts for more than 2
weeks.
• This type of diarrhea is not usually caused by serious illnesses.
Diarrhea- Pathophysiology
• Tissue damage and inflammation lead to the release of
exotoxins that cause a secretion of electrolytes and water
into the intestine; as a result, diarrhea & fluid loss occur.
• Medications such as antibiotics, laxatives
• Toxic agents: Ingestion of metals "lead, mercury“
• Functional causes: Irritable bowel syndrome
• Associated with: URTI., UTI, OM
• Enzymes deficiencies
Diarrhea- Etiology
Risk factors
•Include recent ingestion of undercooked meats,
foreign travel, day care attendance & well water use
Dietary causes
• Over feeding
• Introduction of new foods
• Reinstituting milk too soon after diarrheal episodes
• Osmotic diarrhea from excess sugar in formula
• Excessive ingestion of sorbitol or fructose
Diarrhea- Risk Factors
• Diarrhea, mild to severe
• Anorexia, N&V
• Excessive vomiting may lead to metabolic alkalosis due
to loss of hydrochloric acid from the stomach
• Malaise
• Abdominal distention, pain, cramps
• Electrolyte imbalances such as hypokalemia,
hyponatremia
• Fever
• Headache
Diarrhea- Signs & Symptoms
• Stool culture: may indicate presence of bacteria
• Stool for ova and parasites (O&P): may indicate the
presence of parasites
• Stool viral panel or culture: to determine presence of
rotavirus or other viruses
• Stool for occult blood: may be positive if inflammation
or ulceration is present in the GI tract (hardened
immobile bulk of stool); air–fluid levels may indicate
intestinal obstruction
Diarrhea- Laboratory & Diagnostic Tests
Laboratory and Diagnostic Tests
• Stool for leukocytes: may be positive in cases of
inflammation or infection
• Stool pH/reducing substances: to see if the diarrhea is
caused by carbohydrate intolerance
• Electrolyte panel: may indicate dehydration
• Abdominal radiographs (KUB): presence of stool in
colon may indicate constipation or fecal impaction
Diarrhea- Laboratory & Diagnostic
Tests
• Acute gastroenteritis resolves quickly; if client is
severely ill, medications are prescribed, antibiotics are
used if cause is bacterial (obtain a stool culture first),
• Occasionally antidiarrheals used to increase comfort and
decrease fluid loss or cathartics may be used to remove
toxins from the bowel
• Antiemetics for severe vomiting
• Supportive measures as bed rest, nutritional supplements,
increasing fluids & a bland diet; electrolytes are
replaced; IV fluids are administered if necessary, as
glucose in normal saline or lactated ringers solution
Diarrhea- Treatment
• Restoring Fluid and Electrolyte Balance
Continue the child’s regular diet if the child is not
dehydrated.
Initial nursing management of the dehydrated child
with diarrhea is focused on fluid and electrolyte
balance restoration.
Diarrhea- Nursing Management
 Teach the parents the importance of oral rehydration therapy
 The physician may order medication therapy.
 In such instances, teach the importance of finishing all prescribed
antibiotic therapy.
 After the cause of the diarrhea is known, teach the child and
family how to prevent further occurrences.
 As most cases of acute diarrhea are infectious, provide education
about proper hand-washing techniques and transmission route.
 Chronic diarrhea is often a result of excessive intake of formula,
water, fruit juice, so teach the parents about appropriate fluid
intake.
Diarrhea- Providing Family Education
Appendicitis
• Function of appendix: The entire digestive tract helps with our immune
system, may be a place for our body to store certain healthy types of gut bacteria
that otherwise could be altered or changed during an intestinal illness or with
overuse of antibiotics
• Inflammation of vermiform appendix
• Occurs most often in adolescent boys – can occur in any age
• Pathophysiology: Obstruction of appendiceal lumen –
may perforate or rupture as pressure increases
• Obstruction of the lumen of the appendix by hardened
fecal materials, foreign bodies, microorganisms or
parasite, lymphoid hyperplasia, fibrous stenosis from
earlier inflammation & tumor
Pyloric Stenosis
• Pyloric stenosis is spasms of the pylorus muscle
in the pyloric sphincter that connects the stomach
to the duodenum, causing the pyloric sphincter to
become inflamed and swell, thus preventing the
stomach from emptying into the duodenum.
• The cause is unknown and is seen in children
between 1 and 6 months of age.
• Hypertrophic obstruction of the pyloric sphincter
• Occurs in boys more than girls (4:1 ratio)
• Hypertrophy of pyloric sphincter results in
– Obstruction of sphincter
– Inability to move food from stomach to duodenum
– Vomiting and dehydration
Pyloric Stenosis
Pyloric Stenosis- Pathophysiology
It develops within the first few weeks of life projectile
vomiting, dehydration, metabolic alkalosis, & failure to thrive
• Projectile nonbilious vomiting caused by reverse
peristaltic & gastritis
• No nausea
• Blood-stained vomit caused by gastritis
• Olive-shaped bulge below the right costal margin
• Malnutrition
• Poor weight gain
• Normal appetite
• Eats after vomiting
Pyloric Stenosis- Signs &
Symptoms
• Abdominal ultrasound: Increased size of pyloric
sphincter
• Endoscopy: Increased size of pyloric sphincter
• Upper GI series: Delayed gastric emptying
• Blood chemistry: Decreased calcium, sodium,
potassium
• Blood gases: Metabolic alkalosis
Pyloric Stenosis- Lab
Results
• Insert NG tube to decompress the GI tract.
• Administer IV fluids and electrolytes to maintain
fluid and electrolytes balance.
• Surgery: Pyloromyotomy to repair the pyloric
sphincter.
Pyloric Stenosis-
Treatment
• Nothing by mouth.
• Strict intake and output to assess for dehydration.
• Daily weight.
• Assess vomitus for character and frequency.
• Monitor vital signs.
Pyloric Stenosis- Nursing
Interventions
• Postoperative care:
– Monitor intake and output.
– Elevate the child’s head when feeding.
– Burp frequently.
– Small frequent feedings until the pyloric
sphincter function returns to normal.
– Explain the disorder & treatment to the family.
Pyloric Stenosis- Nursing
Interventions
Intussusception
• One portion of the intestine prolapses and then
invaginates (or, “telescopes”) into itself
• Results in
– Obstructed blood flow
– Ischemia and necrosis
– Hemorrhage, perforation if untreated
• Frequent cause of intestinal obstruction
• Most cases occur in boys <2 years of age
Signs and Symptoms
• Colicky
• Knees drawn to chest
• Grunting
• Fecal material in vomit
• Sweating
• Red currant jelly-like stool
• Dehydration
• Distended abdomen
• Fever
• Shallow respiration
• Sausage-shaped abdominal mass
Test Results
• Abdominal radiograph: Shows mass.
• Barium enema: Shows intussusception.
• Complete blood count—elevated WBC
(leukocytosis)
Treatment
• Insert an NG tube to reduce vomiting and decompress
the GI tract.
• Hydrostatic reduction of bowel: The bowel is moved
back into position using a barium solution, water-soluble
contrast solution, or air pressure.
• Surgery: Pulling the intussusceptions back into position
or resectioning the bowel if other treatments fail to
resolve the intussusceptions.
Nursing Intervention
• Monitor vital signs.
• Insert the NG tube as ordered and measure output.
• Administer the enema as ordered.
• Strict intake and output.
• Postoperative care:
Administer antibiotics
Monitor signs of infection
Monitor bowel sounds
Explain the disorder and treatment to the family.
Cleft Lip and Palate
• A cleft palate and cleft lip is a birth defect caused
by chromosomal abnormalities “chromosome
8” or exposure to alcohol, anticonvulsant
medication, and other teratogens resulting in the
upper jaw and palate bone and tissue not fusing
together properly in the second month of
pregnancy.
• Occur singly or in combination
• Maxillary processes fail to fuse
• Multifactorial cause suspected
– Environment
– Genetics
– Folate
Unilateral
Cleft Lip
Bilateral
Cleft Lip
Signs and Symptoms
• Distended abdominal due to swallowing air
• Abnormal formation of the lip
• Abnormal formation of the palate
Treatment
• Cheiloplasty to surgically repair the cleft lip
within the first 3 months after birth to provide
adequate sucking.
• Palatoplasty to surgically repair the cleft palate
between 12 months and 18 months of age before
the child begins speaking.
Nursing Intervention
Cleft lip:
• Elevate the infant when feeding.
• Use an oversized nipple.
• Stimulate the sucking reflex because this helps with speech.
• Give the infant time to swallow to prevent choking.
• Feed slowly giving the infant a rest period after swallowing.
• Burp frequently to expel swallowed air and prevent vomiting.
• Provide small, frequent feedings to prevent the infant from getting
tired.
• Give water following feedings to flush food from the mouth and
thereby preventing the growth of microorganisms
Nursing Intervention
• Cleft palate:
– Feed using a cleft palate nipple.
– Assess for abdominal
distention.
– Strict intake and output.
– Assess the ability to suck.
– Monitor the vital signs
Nursing Intervention
Nursing Intervention
• Cleft lip Postoperative care:
– Maintain airway.
– Monitor for respiratory distress.
– Restrain the infant from accessing the site.
– Clean the site after feeding.
– Place the infant on the right side after feedings
to prevent aspiration.
Nursing Intervention
Nursing Intervention
• Cleft palate postoperative:
• Don’t use a pacifier.
• Use a cup rather than a nipple for feeding.
• Don’t brush teeth for 2 weeks following surgery.
• Don’t use pointed objects near the infant’s mouth.
• Explain the disorder and treatment to the family.
Hirschsprung Disease
• Congenital aganglionic megacolon
• Congenital anomaly; genetic basis (RET
protooncogene)
• Portion of large intestine lacks parasympathetic
innervation
• Results in
– Inadequate motility
– Obstruction of the intestine
Signs and Symptoms
• Failure to pass meconium within the first 48 hours following birth
• Failure to pass stool within the first 48 hours following birth
• Abdominal distention
• Abdominal mass
• Ribbon-like or liquid stool
• Sunken eyes
• Pallor
• Dehydration
• Irritable
• Weight loss
• Lethargic
Test Results
• Abdominal radiograph: Shows distended colon
• Rectal biopsy: Absence of ganglion cells in the
colon
• Full-thickness surgical biopsy: Absence of
ganglion cells in the colon
• Suction aspiration of rectum: Absence of
ganglion cells in the colon
Treatment
• Surgery: After 9 months of age, the affected
portion of the colon is removed.
Nursing Intervention
• Preoperative care:
– Nothing by mouth.
– Administer IV fluids as ordered to prevent maintain
fluid and electrolyte balance.
– Insert an nasogastric (NG) tube to decompress the
upper GI tract.
– Administer normal saline or mineral oil enemas to
clean the bowel.
– Administer antibiotics as ordered.
Nursing Intervention
• Postoperative care:
– Strict input and output.
– Provide care for the colostomy or ileostomy, if necessary.
– Monitor bowel sounds.
– Begin feeding by mouth when bowel sounds are present.
– Nothing should be placed in the rectum.
– Monitor for constipation.
– Explain the disorder and treatment to the family and instruct
them on proper care for the wound and how to care for the
colostomy or ileostomy, if necessary. Tell the family to call the
health-care provider at the first signs of constipation,
dehydration, fever, vomiting, and diarrhea.
CELIAC DISEASE
• Celiac disease occurs when enzymes in the
intestinal mucosal cells are damaged when they
are in contact with gluten, resulting in decreased
absorption by the small intestines.
• Gluten is a protein found in wheat, rye, oats
“ ‫الشوفان‬
» ”, & barley “ »
‫الشعير‬
Âť . Celiac disease is
detected in infants when they are introduced to
gluten-containing foods.
• Diarrhea
• Fatty, foul-smelling stool
• Distention
• Vomiting
• Irritability
• Abdominal pain
CELIAC DISEASE- Signs &
Symptoms
• Serum: Decreased level of albumin, potassium,
cholesterol, calcium, and sodium
• Blood count: Decreased platelets, white blood
cells (WBC), and hematocrit
• Stool: High fat content
• Biopsy: Positive for celiac disease
CELIAC DISEASE- Lab Results
Treatment
• Diet free from wheat, rye, oats, and barley.
• Increase dietary protein, calories.
• Decrease dietary fat.
• Administer water-soluble vitamins A and D.
• Administer iron supplements.
• Administer folic acid.
• Nursing Intervention
• Teach the family about the disorder and treatment and inform them
that dietary changes are for the child’s entire life.
• If untreated, it may result in cancers such as intestinal lymphoma,
and a slightly increased risk of early death
CELIAC DISEASE- Treatment
Esophageal Atresia and
Tracheoesophageal Fistula (TEF)
• A tracheoesophageal fistula is a congenital anomaly in
which the trachea and the esophagus are connected.
• A child with a tracheoesophageal fistula is likely to have
an esophageal atresia where the esophagus ends in a
blind pouch preventing food from entering the stomach.
• These disorders occur approximately at 5 weeks of
gestation when the foregut normally develops into the
trachea and esophagus.
• Esophagus and trachea do not develop as
parallel tracts
• Esophagus ends as either
– Blind pouch
– Connected to trachea by a fistula
Esophageal Atresia and
Tracheoesophageal Fistula (TEF)
Signs and Symptoms
• Frothy saliva
• Coughing due to excessive secretions
• Gagging when feeding
• Feedings exiting the mouth and nose
• Aspiration
• Difficulty breathing
• Distended stomach
Test Results
• Radiopaque catheter radiograph: A radiograph is
taken after the radiopaque catheter is inserted into
the esophagus to identify the fistula or blind
pouch.
• Bronchoscopy: Shows the fistula.
• Chest radiograph: Shows pneumonia and air in the
esophageal pouch.
• Suction the contents of the blind pouch.
• Insert NG tube to decompress the stomach.
• Surgery: Repair the fistula &/or blind pouch.
Treament
• Nothing by mouth
• Insert the NG tube as ordered.
• Suction the contents of the blind pouch as
ordered.
• No pacifier because this increases saliva
production.
• Elevate the child’s head.
Nursing Interventions
• Postoperative care:
– Monitor vital signs.
– Place NG tube to low suction per order.
– Monitor output of NG tube every 4 hours.
– Provide gastrostomy feeding until esophagus returns to
normal.
– Monitor chest tube drain, if necessary.
– Use pacifier.
– Start feeding by mouth with sterile water and then advance
diet as tolerated.
– Explain the disorder and treatment to the family.
Nursing Interventions
Abdominal Wall Defects
• Omphalocele
– Congenital malformation
– Intra-abdominal contents
herniate through umbilical
cord
– Covered with peritoneal
membrane
– Often associated with other
anomalies
Abdominal Wall Defects
• Gastroschisis
– Abdominal organs herniate through abdominal
wall
– Not covered with peritoneal membrane
Abdominal Wall Defects
Anorectal Malformations
• Common congenital defects
• Minor to complex
• Occur in isolation or with other defects –
VACTERL “vertebral defects, anal atresia, cardiac defects,
tracheo-esophageal fistula, renal anomalies & limb abnormalities”.
• Types
– Anal stenosis
– Anal atresia
– Imperforate anus
• Malformation occurring during fetal development;
rectum can end and not connect to the anus or
may connect to wrong location as the vagina,
urethra, or bladder; anal agenesis in which rectal
pouch ends blindly above the perineum, can be an
anal fistula formation or anal stenosis; no
prevention is known.
Etiology
Predisposing factors
• Due to an abnormal congenital defect
formed during fetal development;
associated with other congenital defects
as Down’s syndrome, cardiac defects,
esophageal atresia, or renal anomalies.
Predisposing Factors
Signs and symptoms:
• Without an anal opening, or misplaced anal
opening
• No passage of stool 24–48 hours after birth
• Passage of meconium from vagina or urinary
meatus
• Abdominal distention
Diagnosis
• Made by examination of the perineum,
abdominal X-ray, ultrasound of rectal and
vaginal areas to evaluate for malformations
Treatment
• Prophylactic antibiotics
• Excision of membrane
• Daily anal dilation by nurse & at home by parents
• Surgical reconstruction, called an anoplasty, to
repair malformations
• Postoperative pain medications
• Temporary colostomy may be performed if
classified as high
Failure To Thrive (FTT)
• Failure to thrive (FTT): growth failure.
• It is the inability to utilize or obtain enough food,
resulting in poor growth patterns.
• It represents a continual deceleration on the growth curve.
• Etiology: Organic FTT caused by the presence of a major
disease, inorganic FTT caused by many psychosocial
factors. Mixed FTT may also occur
• Weight &/or height falls below the 5th percentile for
child’s age
• Developmental milestones are not achieved
• Slow social, lingual, and motor development
• Irregular sleep patterns
• Weakness
• Limited smiling
• Absence of appropriate stranger anxiety
Failure To Thrive (FTT)-Signs &
Symptoms
• Health and dietary history; physical examination
and diagnostic tests to rule out major illnesses;
developmental screening; evaluation of growth
patterns; family assessment; evidence of growth
retardation
Failure To Thrive (FTT)- Diagnosis
• Reversal of malnutrition
• Adequate nutrition to increase calorie intake
• Treat psychosocial problems with a
multidisciplinary team
• Multivitamins & minerals
Failure To Thrive (FTT)-
Treatment
• Assess parent & child behavior during feeding: how
child is held, eye-to-eye contact, sensitivity to child’s
needs
• Provide feedings by consistent nursing staff, to learn
child’s feeding behavior
• Take initial height and weight and weigh daily
• Evaluate when hungry and when hunger is met
• Record food intake
Failure To Thrive (FTT)- Nursing
Management
• Use a structured feeding schedule
• Provide quiet none stimulating surroundings
• Offer appropriate developmental stimulation
• Arrange home care visits, contact social agencies for
financial assistance to family
Failure To Thrive (FTT)- Nursing
Management

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-11-The child with alterations in gastrointestinal functions.ppt

  • 1.
  • 2. Gastroenteritis • Inflammation of stomach and intestines – Fluid, electrolyte loss – Dehydration • May be acute or chronic • Nausea, vomiting, diarrhea
  • 3. Dehydration • Occurs more readily in infants and young children than it does in adults. • The risk is increased in infants and young children because they have an increased extracellular fluid percentage and a relative increase in body water compared to adults. • Increased basal metabolic rate, increased body surface area, immature renal function & increased insensible fluid loss through temperature elevation also contribute to the increased risk for dehydration in infants & young children as compared to adults.
  • 4. Dehydration • Dehydration left unchecked leads to shock, so early recognition and treatment of dehydration is critical to prevent progression to hypovolemic shock. • The goals of therapeutic management of dehydration are to restore appropriate fluid balance and to prevent complications Dehydration
  • 5.
  • 6. Nursing goals for the infant or child with dehydration are aimed at restoring fluid volume & preventing progression to hypovolemia. 1. Provide oral rehydration to children for mild to moderate states of dehydration. 2. Children with severe dehydration should receive intravenous fluids. 3. Initially, administer 20 mL/kg of normal saline or lactated Ringer, and then reassess the hydration status. 4. Once initial fluid balance is restored, the physician or nurse practitioner may order intravenous fluids at the maintenance rate or as much as 1.5 times maintenance. Dehydration- Nursing Management
  • 9. Calculate Deficit – Mild Dehydration: 3-5% deficit (50 ml/kg deficit, 30 ml/kg if >10 kg) – Moderate Dehydration: 6-10% deficit (100 ml/kg deficit, 60 ml/kg if >10 kg) – Severe Dehydration: >10% deficit (120 ml/kg deficit) Replacement over 24 hours •First 8 hours: 50% Deficit + Maintenance •Next 16 hours: 50% Deficit + Maintenance
  • 10. Ongoing Fluid • Ongoing fluid losses= 0.9% Sodium Chloride +/- potassium chloride to replace ongoing losses.
  • 11. Vomiting • Vomiting is the forceful expulsion of gastric contents through the mouth. • Vomiting in infants and children has many different causes and is considered to be a symptom of some other condition. • Therapeutic management of vomiting most often involves slow oral rehydration and at times may require administration of antiemetics
  • 12. • Nursing management focuses on promoting fluid and electrolyte balance. • Oral rehydration is accomplished successfully in most outpatient cases of simple vomiting. • Teach the primary caregiver about oral rehydration • In the child with mild to moderate dehydration resulting from vomiting, withhold oral feeding for 1 to 2 hours after emesis, after which time oral rehydration can begin. • Give the infant or child 0.5 to 2 ounces of oral rehydration solution Q 15 minutes, depending on the child’s age & size. (ounce= 2 table spoon) Vomiting- Nursing Management
  • 13. Nursing Management • Most infants and children can retain this small amount of fluid if fed the restricted amount every 15 minutes. • As the child improves, larger amounts will be tolerated • If oral rehydration is not possible due to continued nausea and vomiting, intravenous fluids will likely be ordered. • In some cases, antiemetics may be used to help control the vomiting. Some antiemetics can cause drowsiness or other side effects and should not be used until a definitive diagnosis is made or severe pathologic processes can be excluded. • Educate the family regarding the prevention of vomiting and use of antiemetic therapy Vomiting- Nursing Management
  • 14. Diarrhea • Is either an increase in the frequency or a decrease in the consistency of stool. Diarrhea in children can either be acute or chronic. • Acute infectious diarrhea (gastroenteritis) remains the leading cause of death for children worldwide.
  • 15. Pathophysiology • Acute diarrhea in children is most commonly caused by viruses, but it may also be related to bacterial or parasitic enteropathogens. • Acute diarrhea may be bloody or nonbloody. • Diarrhea may also occur in relation to antibiotic use. • Though most cases of diarrhea in children are of acute origin, diarrhea may also occur chronically. • Chronic diarrhea is diarrhea that lasts for more than 2 weeks. • This type of diarrhea is not usually caused by serious illnesses. Diarrhea- Pathophysiology
  • 16. • Tissue damage and inflammation lead to the release of exotoxins that cause a secretion of electrolytes and water into the intestine; as a result, diarrhea & fluid loss occur. • Medications such as antibiotics, laxatives • Toxic agents: Ingestion of metals "lead, mercury“ • Functional causes: Irritable bowel syndrome • Associated with: URTI., UTI, OM • Enzymes deficiencies Diarrhea- Etiology
  • 17. Risk factors •Include recent ingestion of undercooked meats, foreign travel, day care attendance & well water use Dietary causes • Over feeding • Introduction of new foods • Reinstituting milk too soon after diarrheal episodes • Osmotic diarrhea from excess sugar in formula • Excessive ingestion of sorbitol or fructose Diarrhea- Risk Factors
  • 18. • Diarrhea, mild to severe • Anorexia, N&V • Excessive vomiting may lead to metabolic alkalosis due to loss of hydrochloric acid from the stomach • Malaise • Abdominal distention, pain, cramps • Electrolyte imbalances such as hypokalemia, hyponatremia • Fever • Headache Diarrhea- Signs & Symptoms
  • 19. • Stool culture: may indicate presence of bacteria • Stool for ova and parasites (O&P): may indicate the presence of parasites • Stool viral panel or culture: to determine presence of rotavirus or other viruses • Stool for occult blood: may be positive if inflammation or ulceration is present in the GI tract (hardened immobile bulk of stool); air–fluid levels may indicate intestinal obstruction Diarrhea- Laboratory & Diagnostic Tests
  • 20. Laboratory and Diagnostic Tests • Stool for leukocytes: may be positive in cases of inflammation or infection • Stool pH/reducing substances: to see if the diarrhea is caused by carbohydrate intolerance • Electrolyte panel: may indicate dehydration • Abdominal radiographs (KUB): presence of stool in colon may indicate constipation or fecal impaction Diarrhea- Laboratory & Diagnostic Tests
  • 21. • Acute gastroenteritis resolves quickly; if client is severely ill, medications are prescribed, antibiotics are used if cause is bacterial (obtain a stool culture first), • Occasionally antidiarrheals used to increase comfort and decrease fluid loss or cathartics may be used to remove toxins from the bowel • Antiemetics for severe vomiting • Supportive measures as bed rest, nutritional supplements, increasing fluids & a bland diet; electrolytes are replaced; IV fluids are administered if necessary, as glucose in normal saline or lactated ringers solution Diarrhea- Treatment
  • 22. • Restoring Fluid and Electrolyte Balance Continue the child’s regular diet if the child is not dehydrated. Initial nursing management of the dehydrated child with diarrhea is focused on fluid and electrolyte balance restoration. Diarrhea- Nursing Management
  • 23.  Teach the parents the importance of oral rehydration therapy  The physician may order medication therapy.  In such instances, teach the importance of finishing all prescribed antibiotic therapy.  After the cause of the diarrhea is known, teach the child and family how to prevent further occurrences.  As most cases of acute diarrhea are infectious, provide education about proper hand-washing techniques and transmission route.  Chronic diarrhea is often a result of excessive intake of formula, water, fruit juice, so teach the parents about appropriate fluid intake. Diarrhea- Providing Family Education
  • 24. Appendicitis • Function of appendix: The entire digestive tract helps with our immune system, may be a place for our body to store certain healthy types of gut bacteria that otherwise could be altered or changed during an intestinal illness or with overuse of antibiotics • Inflammation of vermiform appendix • Occurs most often in adolescent boys – can occur in any age • Pathophysiology: Obstruction of appendiceal lumen – may perforate or rupture as pressure increases • Obstruction of the lumen of the appendix by hardened fecal materials, foreign bodies, microorganisms or parasite, lymphoid hyperplasia, fibrous stenosis from earlier inflammation & tumor
  • 25. Pyloric Stenosis • Pyloric stenosis is spasms of the pylorus muscle in the pyloric sphincter that connects the stomach to the duodenum, causing the pyloric sphincter to become inflamed and swell, thus preventing the stomach from emptying into the duodenum. • The cause is unknown and is seen in children between 1 and 6 months of age.
  • 26. • Hypertrophic obstruction of the pyloric sphincter • Occurs in boys more than girls (4:1 ratio) • Hypertrophy of pyloric sphincter results in – Obstruction of sphincter – Inability to move food from stomach to duodenum – Vomiting and dehydration Pyloric Stenosis
  • 27. Pyloric Stenosis- Pathophysiology It develops within the first few weeks of life projectile vomiting, dehydration, metabolic alkalosis, & failure to thrive
  • 28. • Projectile nonbilious vomiting caused by reverse peristaltic & gastritis • No nausea • Blood-stained vomit caused by gastritis • Olive-shaped bulge below the right costal margin • Malnutrition • Poor weight gain • Normal appetite • Eats after vomiting Pyloric Stenosis- Signs & Symptoms
  • 29. • Abdominal ultrasound: Increased size of pyloric sphincter • Endoscopy: Increased size of pyloric sphincter • Upper GI series: Delayed gastric emptying • Blood chemistry: Decreased calcium, sodium, potassium • Blood gases: Metabolic alkalosis Pyloric Stenosis- Lab Results
  • 30. • Insert NG tube to decompress the GI tract. • Administer IV fluids and electrolytes to maintain fluid and electrolytes balance. • Surgery: Pyloromyotomy to repair the pyloric sphincter. Pyloric Stenosis- Treatment
  • 31. • Nothing by mouth. • Strict intake and output to assess for dehydration. • Daily weight. • Assess vomitus for character and frequency. • Monitor vital signs. Pyloric Stenosis- Nursing Interventions
  • 32. • Postoperative care: – Monitor intake and output. – Elevate the child’s head when feeding. – Burp frequently. – Small frequent feedings until the pyloric sphincter function returns to normal. – Explain the disorder & treatment to the family. Pyloric Stenosis- Nursing Interventions
  • 33. Intussusception • One portion of the intestine prolapses and then invaginates (or, “telescopes”) into itself • Results in – Obstructed blood flow – Ischemia and necrosis – Hemorrhage, perforation if untreated • Frequent cause of intestinal obstruction • Most cases occur in boys <2 years of age
  • 34. Signs and Symptoms • Colicky • Knees drawn to chest • Grunting • Fecal material in vomit • Sweating • Red currant jelly-like stool • Dehydration • Distended abdomen • Fever • Shallow respiration • Sausage-shaped abdominal mass
  • 35. Test Results • Abdominal radiograph: Shows mass. • Barium enema: Shows intussusception. • Complete blood count—elevated WBC (leukocytosis)
  • 36. Treatment • Insert an NG tube to reduce vomiting and decompress the GI tract. • Hydrostatic reduction of bowel: The bowel is moved back into position using a barium solution, water-soluble contrast solution, or air pressure. • Surgery: Pulling the intussusceptions back into position or resectioning the bowel if other treatments fail to resolve the intussusceptions.
  • 37. Nursing Intervention • Monitor vital signs. • Insert the NG tube as ordered and measure output. • Administer the enema as ordered. • Strict intake and output. • Postoperative care: Administer antibiotics Monitor signs of infection Monitor bowel sounds Explain the disorder and treatment to the family.
  • 38. Cleft Lip and Palate • A cleft palate and cleft lip is a birth defect caused by chromosomal abnormalities “chromosome 8” or exposure to alcohol, anticonvulsant medication, and other teratogens resulting in the upper jaw and palate bone and tissue not fusing together properly in the second month of pregnancy.
  • 39. • Occur singly or in combination • Maxillary processes fail to fuse • Multifactorial cause suspected – Environment – Genetics – Folate Unilateral Cleft Lip Bilateral Cleft Lip
  • 40. Signs and Symptoms • Distended abdominal due to swallowing air • Abnormal formation of the lip • Abnormal formation of the palate
  • 41. Treatment • Cheiloplasty to surgically repair the cleft lip within the first 3 months after birth to provide adequate sucking. • Palatoplasty to surgically repair the cleft palate between 12 months and 18 months of age before the child begins speaking.
  • 42. Nursing Intervention Cleft lip: • Elevate the infant when feeding. • Use an oversized nipple. • Stimulate the sucking reflex because this helps with speech. • Give the infant time to swallow to prevent choking. • Feed slowly giving the infant a rest period after swallowing. • Burp frequently to expel swallowed air and prevent vomiting. • Provide small, frequent feedings to prevent the infant from getting tired. • Give water following feedings to flush food from the mouth and thereby preventing the growth of microorganisms
  • 43.
  • 44. Nursing Intervention • Cleft palate: – Feed using a cleft palate nipple. – Assess for abdominal distention. – Strict intake and output. – Assess the ability to suck. – Monitor the vital signs Nursing Intervention
  • 45. Nursing Intervention • Cleft lip Postoperative care: – Maintain airway. – Monitor for respiratory distress. – Restrain the infant from accessing the site. – Clean the site after feeding. – Place the infant on the right side after feedings to prevent aspiration. Nursing Intervention
  • 46. Nursing Intervention • Cleft palate postoperative: • Don’t use a pacifier. • Use a cup rather than a nipple for feeding. • Don’t brush teeth for 2 weeks following surgery. • Don’t use pointed objects near the infant’s mouth. • Explain the disorder and treatment to the family.
  • 47. Hirschsprung Disease • Congenital aganglionic megacolon • Congenital anomaly; genetic basis (RET protooncogene) • Portion of large intestine lacks parasympathetic innervation • Results in – Inadequate motility – Obstruction of the intestine
  • 48. Signs and Symptoms • Failure to pass meconium within the first 48 hours following birth • Failure to pass stool within the first 48 hours following birth • Abdominal distention • Abdominal mass • Ribbon-like or liquid stool • Sunken eyes • Pallor • Dehydration • Irritable • Weight loss • Lethargic
  • 49. Test Results • Abdominal radiograph: Shows distended colon • Rectal biopsy: Absence of ganglion cells in the colon • Full-thickness surgical biopsy: Absence of ganglion cells in the colon • Suction aspiration of rectum: Absence of ganglion cells in the colon
  • 50. Treatment • Surgery: After 9 months of age, the affected portion of the colon is removed.
  • 51. Nursing Intervention • Preoperative care: – Nothing by mouth. – Administer IV fluids as ordered to prevent maintain fluid and electrolyte balance. – Insert an nasogastric (NG) tube to decompress the upper GI tract. – Administer normal saline or mineral oil enemas to clean the bowel. – Administer antibiotics as ordered.
  • 52. Nursing Intervention • Postoperative care: – Strict input and output. – Provide care for the colostomy or ileostomy, if necessary. – Monitor bowel sounds. – Begin feeding by mouth when bowel sounds are present. – Nothing should be placed in the rectum. – Monitor for constipation. – Explain the disorder and treatment to the family and instruct them on proper care for the wound and how to care for the colostomy or ileostomy, if necessary. Tell the family to call the health-care provider at the first signs of constipation, dehydration, fever, vomiting, and diarrhea.
  • 53. CELIAC DISEASE • Celiac disease occurs when enzymes in the intestinal mucosal cells are damaged when they are in contact with gluten, resulting in decreased absorption by the small intestines. • Gluten is a protein found in wheat, rye, oats “ ‫الشوفان‬ Âť ”, & barley “ Âť ‫الشعير‬ Âť . Celiac disease is detected in infants when they are introduced to gluten-containing foods.
  • 54. • Diarrhea • Fatty, foul-smelling stool • Distention • Vomiting • Irritability • Abdominal pain CELIAC DISEASE- Signs & Symptoms
  • 55. • Serum: Decreased level of albumin, potassium, cholesterol, calcium, and sodium • Blood count: Decreased platelets, white blood cells (WBC), and hematocrit • Stool: High fat content • Biopsy: Positive for celiac disease CELIAC DISEASE- Lab Results
  • 56. Treatment • Diet free from wheat, rye, oats, and barley. • Increase dietary protein, calories. • Decrease dietary fat. • Administer water-soluble vitamins A and D. • Administer iron supplements. • Administer folic acid. • Nursing Intervention • Teach the family about the disorder and treatment and inform them that dietary changes are for the child’s entire life. • If untreated, it may result in cancers such as intestinal lymphoma, and a slightly increased risk of early death CELIAC DISEASE- Treatment
  • 57. Esophageal Atresia and Tracheoesophageal Fistula (TEF) • A tracheoesophageal fistula is a congenital anomaly in which the trachea and the esophagus are connected. • A child with a tracheoesophageal fistula is likely to have an esophageal atresia where the esophagus ends in a blind pouch preventing food from entering the stomach. • These disorders occur approximately at 5 weeks of gestation when the foregut normally develops into the trachea and esophagus.
  • 58. • Esophagus and trachea do not develop as parallel tracts • Esophagus ends as either – Blind pouch – Connected to trachea by a fistula Esophageal Atresia and Tracheoesophageal Fistula (TEF)
  • 59.
  • 60.
  • 61. Signs and Symptoms • Frothy saliva • Coughing due to excessive secretions • Gagging when feeding • Feedings exiting the mouth and nose • Aspiration • Difficulty breathing • Distended stomach
  • 62. Test Results • Radiopaque catheter radiograph: A radiograph is taken after the radiopaque catheter is inserted into the esophagus to identify the fistula or blind pouch. • Bronchoscopy: Shows the fistula. • Chest radiograph: Shows pneumonia and air in the esophageal pouch.
  • 63. • Suction the contents of the blind pouch. • Insert NG tube to decompress the stomach. • Surgery: Repair the fistula &/or blind pouch. Treament
  • 64. • Nothing by mouth • Insert the NG tube as ordered. • Suction the contents of the blind pouch as ordered. • No pacifier because this increases saliva production. • Elevate the child’s head. Nursing Interventions
  • 65. • Postoperative care: – Monitor vital signs. – Place NG tube to low suction per order. – Monitor output of NG tube every 4 hours. – Provide gastrostomy feeding until esophagus returns to normal. – Monitor chest tube drain, if necessary. – Use pacifier. – Start feeding by mouth with sterile water and then advance diet as tolerated. – Explain the disorder and treatment to the family. Nursing Interventions
  • 66. Abdominal Wall Defects • Omphalocele – Congenital malformation – Intra-abdominal contents herniate through umbilical cord – Covered with peritoneal membrane – Often associated with other anomalies
  • 67. Abdominal Wall Defects • Gastroschisis – Abdominal organs herniate through abdominal wall – Not covered with peritoneal membrane Abdominal Wall Defects
  • 68. Anorectal Malformations • Common congenital defects • Minor to complex • Occur in isolation or with other defects – VACTERL “vertebral defects, anal atresia, cardiac defects, tracheo-esophageal fistula, renal anomalies & limb abnormalities”. • Types – Anal stenosis – Anal atresia – Imperforate anus
  • 69. • Malformation occurring during fetal development; rectum can end and not connect to the anus or may connect to wrong location as the vagina, urethra, or bladder; anal agenesis in which rectal pouch ends blindly above the perineum, can be an anal fistula formation or anal stenosis; no prevention is known. Etiology
  • 70. Predisposing factors • Due to an abnormal congenital defect formed during fetal development; associated with other congenital defects as Down’s syndrome, cardiac defects, esophageal atresia, or renal anomalies. Predisposing Factors
  • 71. Signs and symptoms: • Without an anal opening, or misplaced anal opening • No passage of stool 24–48 hours after birth • Passage of meconium from vagina or urinary meatus • Abdominal distention
  • 72. Diagnosis • Made by examination of the perineum, abdominal X-ray, ultrasound of rectal and vaginal areas to evaluate for malformations
  • 73. Treatment • Prophylactic antibiotics • Excision of membrane • Daily anal dilation by nurse & at home by parents • Surgical reconstruction, called an anoplasty, to repair malformations • Postoperative pain medications • Temporary colostomy may be performed if classified as high
  • 74. Failure To Thrive (FTT) • Failure to thrive (FTT): growth failure. • It is the inability to utilize or obtain enough food, resulting in poor growth patterns. • It represents a continual deceleration on the growth curve. • Etiology: Organic FTT caused by the presence of a major disease, inorganic FTT caused by many psychosocial factors. Mixed FTT may also occur
  • 75. • Weight &/or height falls below the 5th percentile for child’s age • Developmental milestones are not achieved • Slow social, lingual, and motor development • Irregular sleep patterns • Weakness • Limited smiling • Absence of appropriate stranger anxiety Failure To Thrive (FTT)-Signs & Symptoms
  • 76. • Health and dietary history; physical examination and diagnostic tests to rule out major illnesses; developmental screening; evaluation of growth patterns; family assessment; evidence of growth retardation Failure To Thrive (FTT)- Diagnosis
  • 77. • Reversal of malnutrition • Adequate nutrition to increase calorie intake • Treat psychosocial problems with a multidisciplinary team • Multivitamins & minerals Failure To Thrive (FTT)- Treatment
  • 78. • Assess parent & child behavior during feeding: how child is held, eye-to-eye contact, sensitivity to child’s needs • Provide feedings by consistent nursing staff, to learn child’s feeding behavior • Take initial height and weight and weigh daily • Evaluate when hungry and when hunger is met • Record food intake Failure To Thrive (FTT)- Nursing Management
  • 79. • Use a structured feeding schedule • Provide quiet none stimulating surroundings • Offer appropriate developmental stimulation • Arrange home care visits, contact social agencies for financial assistance to family Failure To Thrive (FTT)- Nursing Management