2. DEFINITION
īˇ An impairment of the infants body function
or structure due to adverse influences that
occur at birth
īˇ (National vital statistics report)
3. RISK FACTORS
īˇ Primiparity
īˇ Small maternal stature
īˇ Maternal pelvic anomalies
īˇ Prolonged or unusually rapid labor
īˇ Oligohydramnios
īˇ Malpresentation of the fetus
4. īˇ Use of mid forceps or vaccum
extraction
īˇ Versions and extractions
īˇ Very low birth weight or extreme
prematurity
īˇ Fetal macrosomia or large fetal head
īˇ Fetal anomalies
9. CAPUT SUCCEDANEUM
īˇ A caput succedaneum is a
serosanguinous fluid collection above the
periosteum. It presents as a soft tissue
swelling with purpura and ecchymosis
over the presenting portion of the scalp. It
may extend across the midline and across
suture lines.
10. īˇ The edema disappears within the 1st few
days of life.
âĸ Molding of the head and overriding of
the parietal bones disappear during the
1st weeks of life.
âĸ Rarely, a hemorrhagic caput may result
in shock and require blood transfusion.
11. MANAGEMENT
īˇ No specific treatment is needed
âĸ But if extensive ecchymoses are
present, hyperbilirubinemia may develop
âĸ Shock â Blood transfusion
13. Clinical features
īˇ Swelling, usually over a parietal or
occipital bone
īˇ Swelling does not cross a suture line and
is often not associated with discoloration
of the overlying scalp.
īˇ Limited to the surface of one cranial bone.
15. īˇ If infection is suspected, aspiration of the
mass
īˇ If sepsis, antibiotics
īˇ hyperbilirubinemia â photo therapy
16.
17. SUBGALEAL HEMORRHAGE
īˇ A subgaleal hemorrhage is bleeding
between the galea aponeurosis of the
scalp and the periosteum.
18. FEATURES
īˇ A subgaleal hemorrhage presents as a
firm-to-fluctuant mass that crosses suture
lines.
īˇ The mass is typically noted within 4 hours
of birth.
19. LABORATORY FINDINGS
īˇ serial hemoglobin and hematocrit
monitoring,
īˇ coagulation profile to investigate for the
presence of a coagulopathy.
īˇ Bilirubin levels also need to be monitored
20. TREATMENT
īˇ Supportive
īˇ Transfusions may be required if blood loss
is significant.
âĸ In severe cases, surgery may be
required to cauterize the bleeding vessels.
âĸ These lesions typically resolve over a 2â3
week period
22. LINEAR SKULL FRACTURES
īˇ Usually affect the parietal bones.
īˇ The pathogenesis is related to
compression from the application of
forceps, or from the skull pushing against
the maternal symphysis or ischeal spines.
īˇ Rarely, a linear fracture may be
associated with a dural tear, with
subsequent development of a
leptomeningeal cyst.
23. DEPRESSED SKULL FRACTURES
īˇ Indications for surgery include
īˇ radiographic evidence of bone fragments
in the cerebrum
īˇ presence of neurologic deficits
īˇ signs of increased intracranial pressure
īˇ signs of cerebrospinal fluid beneath the
galea
īˇ failure to respond to closed manipulation.
24. īˇ Indications for nonsurgical management
include
īˇ Depressions less than 2 cm in width and
depressions over a major venous sinus
īˇ Without neurologic symptoms
27. INTRACRANIAL HAEMORRHAGE
īˇ Bleeding can occur
ī External to the brain into the
epidural, subdural or subarachnoid space
ī In to the parenchyma of the cerebrum or
cerebellum
ī Into the ventricles from the subependymal
germinal matrix or choroid plexus
28. RISK FACTORS
īˇ forceps delivery
īˇ vacuum extraction
īˇ precipitous deliver
īˇ prolonged second stage of labor
īˇ macrosomia
30. EPIDURAL HEMORRHAGE
īˇ Epidural hemorrhage primarily arises from
injury to the middle meningeal artery, and
is frequently associated with a
cephalhematoma or skull fracture.
35. īˇ Laceration of the tentorium, with rupture of
the straight sinus, vein of Galen transverse
sinus, or infratentorial veins causing a
posterior fossa clot and brainstem
compression
īˇ Laceration of the falx, with rupture of the
inferior sagittal sinus resulting in a clot in
the longitudinal cerebral fissure
36. īˇ Laceration of the superficial cerebral
vein, causing bleeding over the cerebral
convexity
īˇ Occipital osteodiastasis, with rupture of
the occipital sinus, resulting in a posterior
fossa clot
37. CLINICAL FEATURES
īˇ Respiratory symptoms such as apnea
īˇ Seizures
īˇ Focal neurologic deficits
īˇ Lethargy
īˇ Hypotonia
īˇ Other neurologic symptoms
40. MANIFESTATIONS
īˇ Seizures, often occurring on the second
day of life
īˇ Irritability
īˇ Depressed level of consciousness
īˇ Focal neurologic signs.
41. DIAGNOSIS
īˇ Cranial computed tomography.
īˇ Cranial ultrasonography
īˇ Lumbar puncture shows an increased
number of red blood cells
43. INTRAPARENCHYMAL
HAEMORRHAGE
īˇ TYPES
īˇ Intra cerebral
Causes:
īˇ rupture of an av malformation or aneurysm
īˇ coagulation disturbances
īˇ extracorporeal membrane oxygenation
therapy
īˇ secondary to a large ICH in any other
compartment
44. īˇ Intracerebellar :
more common in preterm than the
term babies. May be a primary
haemorrhage or may result from venous
hemorrhagic infarction or from extension
of GMH/ IVH
45. CLINICAL FEATURES
īˇ In the preterm infant
ī IPH is often clinically silent in either
intracranial fossa , unless the hemorrhage is
quite large
īˇ In the term infant, manifestations are
ī Seizures
ī Hemiparesis
ī Gaze preference
ī Irritability
ī Depressed level of consciousness
49. FACTORS IN THE PATHOGENESIS
īˇ Intra vascular factors
ī Ischemia / reperfusion
ī Fluctuating cerebral blood flow
ī Increase in CBF
ī Increase in cerebral venous pressure
ī Platelet dysfunction
ī Coagulation disturbances
50. īˇ Vascular factors
ī Tenuous involuting capillaries with large
diameter lumen
īˇ Extra vascular factors
ī Deficient vascular support
ī Excessive fibrinolytic activity
51. CLINICAL FEATURES
In the preterm newborn
īˇ Usually clinically silent
īˇ Decreased levels of consciousness and
spontaneous movement
īˇ Hypotonia
īˇ Abnormal eye movement
īˇ Skew deviation
52. In term newborns
īˇ Seizures
īˇ Irritability
īˇ Apnea
īˇ Lethargy
īˇ Vomiting with dehydration
īˇ Full fontanels
58. ERB-DUCHENNE PARALYSIS
īˇ 5th and 6th cervical nerves injury
īˇ The infant loses the power to abduct the
arm from the shoulder, rotate the arm
externally, and supinate the forearm
īˇ Erbâs palsy may also be associated with
injury to the phrenic nerve,
which is innervated with fibers from
C3âC5
59. īˇ Adduction and internal rotation of the arm
with pronation of the forearm.
īˇ Biceps reflex is absent
īˇ Moro reflex is absent on the affected side.
īˇ The involved arm is held in the ââwaiterâs
tipââ position, with adduction and internal
rotation of the shoulder, extension of the
elbow, pronation of the forearm, and
flexion of the wrist and fingers.
60. KLUMPKEâSPALSY
īˇ Involves the C8 and T1 nerves, resulting in
weakness of the intrinsic hand muscles
and long flexors of the wrist and fingers
61. īˇ The grasp reflex is absent but the biceps
reflex is present.
īˇ Flaccid extremity with absent reflexes.
62. ASSOCIATED LESIONS
īˇ Hematomas of the sternocleidomastoid
muscle, and fractures of the clavicle and
humerus.
īˇ Ipsilateral Hornerâs syndrome
(ptosis, miosis, and anhydrosis) when
there is accompanying injury to the
sympathetic fibers of T1.
63. TYPES
īˇ Neuropraxia with temporary conduction
block
īˇ Axonotmesis with a severed axon, but with
intact surrounding neuronal elements
īˇ Neurotmesis with complete postganglionic
disruption of the nerve
īˇ Avulsion with preganglionic disconnection
from the spinal cord
65. MANAGEMENT
īˇ Initial treatment is conservative.
īˇ The arm is immobilized across the upper
abdomen during the first week
īˇ Physical therapy with passive range-of-
motion exercises at the shoulder, elbow
and wrist should begin after the first week.
īˇ Infants without recovery by 3 to 6 months
of age may be considered for surgical
exploration
67. Clinical manifestations
īˇ weakness of both upper and lower facial
muscles.
īˇ At rest, the nasolabial fold is flattened and
the eye remains persistently open on the
affected side.
īˇ During crying, there is inability to
wrinkle the forehead or close the eye on
the ipsilateral side, and the mouth is
drawn awayfrom the affected side.
69. TREATMENT
īˇ protection of the involved eye by
application of artificial tears and taping to
prevent corneal injury.
īˇ neurosurgical repair of the nerve should
be considered only after lack of resolution
during 1 year of observation
70. PHRENIC NERVE INJURY
īˇ The phrenic nerve arises from the third
through fifth cervical nerve roots.
īˇ Injury to the phrenic nerve leads to
paralysis of the ipsilateral diaphragm.
71. CLINICAL MANIFESTATIONS
īˇ respiratory distress, with diminished breath
sounds on the affected side.
īˇ Chest radiographs show elevation of the
affected diaphragm, with mediastinal shift
to the contralateral side.
īˇ Ultrasonography or fluoroscopy can
confirm the diagnosis by showing
paradoxical diaphragmatic movement
during inspiration
72. TREATMENT
īˇ Initial treatment is supportive
īˇ Oxygen
īˇ Respiratory failure may be treated with
continuous positive airway pressure or
mechanical ventilation.
īˇ Gavage feedings.
īˇ Plication of the diaphragm
75. Treatment
īˇ Small frequent feedings may be required
to decrease the risk of aspiration.
īˇ Intubation
īˇ Tracheostomy
īˇ Bilateral paralysis tends to produce more
severe distress, and therefore requires
intubation and tracheostomy placement
more frequently
76. SPINAL CORD INJURY
īˇ Clinical findings
īˇ decreased or absent spontaneous
movement
īˇ absent deep tendon reflexes
īˇ absent or periodic breathing
īˇ lack of response to painful stimuli below
the level of the lesion.
77. īˇ Lesions above C4 are almost always
associated with apnea
īˇ Lesions between C4 and T4 may have
respiratory distress secondary to varying
degrees of involvement of the phrenic
nerve and innervation to the intercostal
muscles
78. MANAGEMENT
īˇ If cord injury is suspected in the delivery
room, the head, neck, and spine should be
immobilized.
īˇ Therapy is supportive.
80. NASAL SEPTAL
DISLOCATION
īˇ Nasal septal dislocation involves
dislocation of the triangular cartilaginous
portion of the septum from the vomerine
groove
81. CLINICAL FEATURES
īˇ airway obstruction.
īˇ deviation of the nose to one side
īˇ The nares are asymmetric, with flattening
of the side of the dislocation (Metzenbaum
sign).
âĸ Application of pressure on the tip of the
nose (Jeppesen and Windfeld test) causes
collapse of the nostrils, and the deviated
septum becomes more apparent.
82. MANAGEMENT
īˇ Definitive diagnosis can be made by
rhinoscopy
īˇ manual reduction performed by an
otolaryngologist using a nasal elevator.
īˇ Reduction should be performed by 3 days
of age
83. OCULAR INJURIES
īˇ Rupture of Descemetâs membrane of the
cornea
īˇ lid lacerations
īˇ hyphema (blood in anterior chamber)
īˇ vitreous hemorrhage
īˇ Purtscherâs retinopathy
īˇ corneal edema,
īˇ corneal abrasion
84. CONGENITAL MUSCULAR
TORTICOLLIS
īˇ atrophic muscle fibers surrounded by
collagen and fibroblasts.
īˇ tearing of the muscle fibers or fascial
sheath with hematoma formation and
subsequent fibrosis.
85. CLINICAL FEATURES
īˇ The head is tilted toward the side of the
lesion and rotated to the contralateral side,
īˇ chin is slightly elevated.
īˇ If a mass is present, it is firm, spindle-
shaped, immobile, and located in the
midportion of the sternocleidomastoid
muscle, without accompanying
discoloration or inflammation.
86. DIAGNOSIS
īˇ physical examination
īˇ Radiographs should be obtained to rule
out abnormalities of the cervical spine.
īˇ Ultrasonography may be useful both
diagnostically and prognostically.
90. Risk factors
īˇ higher birth weight
īˇ prolonged second stage of labor
īˇ shoulder dystocia
īˇ instrumented deliveries
91. MANAGEMENT
īˇ Asymptomatic incomplete fractures require
no treatment.
âĸ Complete fractures are treated with
immobilization of the arm for 7 to 10 days
92. LONG BONE FRACTURES
Risk factors
īˇ breech presentation
īˇ cesarean delivery
īˇ low birthweight
95. TREATMENT
īˇ immobilization and splinting
īˇ Closed reduction and casting are required
only when the bones are displaced.
īˇ Proximal femoral fractures may require a
spica cast or use of a Pavlik harness
96. INTRA-ABDOMINAL INJURY
Liver injury is the most common
īˇ Three potential mechanisms lead to intra-
abdominal injury:
īˇ (1) direct trauma,
īˇ (2) compression of the chest against the
surface of the spleen or liver
īˇ (3) chest compression leading to tearing of
the ligamentaous insertions of the liver or
spleen
97. CLINICAL MANIFESTATIONS
īˇ With hepatic or splenic rupture, patients
develop sudden pallor, hemorrhagic
shock, abdominal distention, and
abdominal discoloration.
âĸ Presentation of a liver rupture with
scrotal swelling and discoloration has
been described.
98. īˇ Subcapsular hematomas may present
more insidiously, with anemia, poor
feeding, tachypnea, and tachycardia.
īˇ Adrenal hemorrhage may present as a
flank mass
99. DIAGNOSIS
īˇ abdominal ultrasound
īˇ Computed tomography
īˇ Abdominal radiographs may show
nonspecific intraperitoneal fluid or
hepatomegaly.
īˇ Abdominal paracentesis is diagnostic if a
hemoperitoneum is present
100. TREATMENT
īˇ volume replacement and correction of any
coagulopathy.
īˇ If the infant is hemodynamically stable,
conservative management is indicated.
īˇ With rupture or hemodynamic instability, a
laparotomy is required to control the
bleeding.
īˇ Patients with adrenal hemorrhage may
require hormone replacement therapy.
101. SOFT TISSUE INJURIES
īˇ Petechiae and ecchymoses
âĸ Lacerations and abrasions
âĸ Subcutaneous fat necrosis