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Introduction
An inadequate supply of essential
nutrients (as vitamins and minerals)
in the diet resulting in malnutrition or
disease.
In India many children are suffering
with nutritional disorders. 2 million
children affected by nutritional
deficiency in every year.
4. FACTORS AFFECTING NUTRITIONAL
STATUS OF CHILD
Nutritional intake of the child
Health status
Child feeding practice
Food demand
Family income/ Occupation
Education of Mother etc.
6. 1.9 billion adults are overweight or obese,
while 462 million are underweight.
47 million children under 5 years of age are
wasted, 14.3 million are severely wasted and
144 million are stunted, while 38.3 million are
overweight or obese.
Around 45% of deaths among children under 5
years of age are linked to undernutrition
WORLD SCENARIO
7. INDIAN SCENARIO
Malnutrition caused 69 per cent of deaths of
children below the age of five in India,
according to a UNICEF
The State of the World's Children 2019,
UNICEF said that every second child in that
age group is affected by some form of
malnutrition.
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Protein Energy Malnutrition
Range of pathological conditions
arising from lack, in varying
proportions , of protein and calories.
• Marasmus: weight for age < 60%
expected
• Kwashiorkor: weight for age < 80% +
edema
• Marasmic kwashiorkor: wt/age <60%
+ edema
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• Only wt for age taken into account
• No comment about height
• All cases of edema in 3rd degree irrespective of wt for
age
Gomez Classification
Nutritional status Wt for Age(% Expected)
Normal > 90
1st Degree PEM 75-90
2nd Degree PEM 60-75
3rd Degree PEM < 60
10.
11. RISK FACTORS
LBW
Multiple birth
Closely spaced birth
Early stoppage of breast
feeding
Too early or late weaning
Recurrent infections
Illiteracy, poverty
Secondary due to
malabsorption
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It refers as combination of
• Edema
• Lethargy
• Growth failure.
KWASHIORKOR
13. ETIOLOGY
Non availability of suitable protein -rich
foods.
Faulty feeding habits.
Prolonged breast feeding.
Infection and infestation
Sudden loss of protein
Seasonal incidence-July - august.
Size of the family.
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CLINICAL FEATURES
Actually develops over a period of week or month.
Growth retardation
Apathy
Diarrhea
Edema.
Hair changes
Mental changes
Wasting
Anaemia
Diarrhoea
Dermatosis (flaky-paint)
15. Skin Changes:- Dry & scaly skin,
Dermatosis
Deep fissures- region of elbows, groins,
knees.
Pinkish area over the legs.
Hair Changes:-Scanty, Lusterless,
Brownish
Hepatomegaly.
Moon face
Anemia
Psycho motor changes
Cardio vascular changes.
16. Bio chemical changes:
• Reduced serum protein (less than 1.5g)
• Blood cholesterol reduced, low serum iron
and copper.
• Decreased water and electrolytes
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Serum electrolytes
Blood Urea Nitrogen
Glucose, and
Possibly levels of Ca, Mg, Phosphate,
Na should be measured.
Urine culture,
Blood cultures,
Tuberculin testing, and
A chest X-ray
DIAGNOSIS
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MARASMUS
There is loss of weight of more
than 50% or the expected
weight for given age.
Causes:
• Primary
• Secondary
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CLASSIFICATION
Grade 1: Wasting starts in axilla & groin
Grade 2: Wasting extended to thigh and buttocks
Grade 3: Chest and abdomen
Grade 4 : Wasting of buccal pad of fat also
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CLINICAL FEATURES
Remarkable wasting of both muscles and
subcutaneous.
Irritable
In early stage Hungry and craving for food.
In later stages apathetic, refusing to take anything
Hair changes
Face has prematurely aged look.
Wrinkle skin with loss of elasticity
Scaphoid abdomen.
Visible intestinal peristalsis.
22. Biochemical changes:
Plasma protein reduced
Increases basal metabolic
rate.
Pathological changes:
Decreased weight of many
organ.
Reduced function of organ.
23. Enter titleDIAGNOSIS
Care dietetic history of
the infant.
Physical examination
Lab investigation
X-ray
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TREATMENT
Principles:
Liberal protein must be offered.
Carbohydrates and fat must also be supplied.
Take care of their total calorie needs
Treat underlying causes.
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LIBERAL PROTEIN
(MILD & MODERATE DEGREE)
wheat-50gm
Bengal gram-16.5gm
Groundnuts-8.5gm
Dry skimmed milk-8.5grams or
defatted soya-8.5gms
In that total protein is-16.8grams
Calories-370 gms. total calories
requirement is - 120-150 cal/kg/day
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CONTINUED
(FOR SEVERE DEGREE)
Hospitalization
Parenteral therapy
N.G tube feeding (2-3gms/kg)
Control the infection
Correction of metabolic derangement and
deficiencies
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MANAGEMENT
Mild and moderate malnutrition
Mainstay of treatment is to give
adequate amounts of protein and
energy.
At Least 150 kcal/kg/day, protein
intake of 3g/kg/day.
Best measure of efficacy of the
treatment is weight gain.
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WHO Steps Of Recovery
STEP 1
Prevent/Treat Hypoglycemia
STEP 2
Prevent/Treat Hypothermia
STEP 3
Prevent/Treat Dehydration
STEP 4
Correct Electrolyte Imbalance
STEP 5
Prevent/Treat Infection
30. STEP 6
Correct Micronutrient Deficiency
STEP 7
Initiate Re-feeding
STEP 8
Achieve Catch-up Growth
STEP 9
Provide Sensory Stimulation And Emotional
Support
STEP 10
Prepare For Follow Up After Recovery
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VITAMIN C DEFICIENCY
Disease result from severe vitamin
C deficiency:
Scurvy
Cardiovascular Disease
Stroke
Cataract
Lead toxicity
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SCURVY
ETIOLOGY:-
Diet lacking in vitamin C-rich foods
Destruction of vitamin C in foods by
overexposure to air or by overcooking
PEM
Malabsorption
Artificial feeding
Illness and infection
Daily requirement 40 mg/ kg/day
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CLINICAL FEATURES
Infantile scurvy:
Irritability
Excessive crying
Tenderness to touch
Frog position
Hemorrhages (skin and mucus membranes,
gums)
Hemorrhages in gums (result in spongy,
swollen, bluish purple gums)
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CONTINUED
Childhood scurvy:
Minute hemorrhages at the root of the hair
Follicles.
Bleeding to skin leads to petechiae or large
purpuric swelling, gums
35. Enter titleDIAGNOSIS
History Collection
Physical Examination
Ascorbic Acid level in the serum usually less
than 0.1 mg/100 ml
Excretion Ascorbic acid
X ray
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TREATMENT
Administer 500mg of single dose of
vit c Followed by daily dose of 100
to 300 mg for several week.
Vitamin c rich diet
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SPECIAL CONSIDERATION
HOSPITAL
Administer ascorbic acid orally or by slow I.V. infusion, as
ordered.
Avoid moving the child unnecessarily, to avoid irritating
painful joints and muscles.
Encourage child to drink orange juice.
Explain the importance of supplemental ascorbic acid.
Counsel the child and family about good dietary sources of
vitamin C.
Advise against taking too much vitamin C.
Explain that excessive doses of ascorbic acid may cause
nausea, diarrhea, and renal calculi formation and may also
interfere with anticoagulant therapy.
38. PREVENTION
At national level
1. Nutrition supplementation - Fortification,
iodination
2. Nutritional surveillance- define the character
and magnitude of nutritional problems and
strategies to tackle.
3. Nutritional planning- formulation of nutrition
policy, improve food production and supplies,
ensure distribution.
39. At community level
Health and nutritional education
Promotion of education and
literacy in the community
Growth monitoring
Integrated health package
Vigorous promotion of family
planning programs
PREVENTION
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PREVENTION
At family level
Exclusive breast feeding
Complementary feeds at 6
months
Vaccination
Spacing between
pregnancies
41. GOVT INITIATIVE TO PREVENT
MALNUTRITION
National Rural Health Mission Of India
Mid-day meal Scheme
Integrated Child Development Scheme
National Children's Fund
United Nations Children's Fund
42. REFERENCE
S
1. Paul Vinod, Bagaa Arvind. Ghai Essential
Pediatrics: Nutrition.8th Edition.New Delhi:CBS
publishers,2013
2. Datta Parul. Pediatric Nursing: Nutritionl
Deficiency Disorder. 3rd Edition. New
Delhi:Jaypee,2014
3. Healthline. Nutritional Deficiencies: Symptoms
and Treatment[online].2018(cited 2020 May 28).
Available from:
www.healthline.com/health/malnutrition