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By
MS. KAMINI SAO
 At the end of the seminar the group will
be able to get the knowledge about the
oxygen insufficiency, develop a positive
attitude towards it and practice this
knowledge in teaching and clinical areas.
At the end of the class the student will be able
to :
 Define oxygen insufficiency.
 Enlist the etiology of oxygen insufficiency.
 Enumerate factors affecting oxygenation
 Know the types of oxygen insufficiency.
 Discuss the pathophysiology of oxygen
insufficiency.
 Enlist sign and symptoms of oxygen
insufficiency.
 Explain different diagnostic evaluation of
oxygen insufficiency.
 Describe the management of oxygen
insufficiency.
 Identify the nurses role in the management of
oxygen insufficiency.
 Explain about oxygen therapy.
 Discuss about nurses responsibility while
administering oxygen.
 Oxygen insufficiency means “ deficient in
oxygen”.
 The normal range of oxygen in the external
blood should be 80-100 mm of Hg.
 For treating Oxygen insufficiency effectively,
early diagnosis and correct cause should be
ruled out.
 The only management for Oxygen
insufficiency is Oxygen administration.
 Oxygen insufficiency is a condition in
which the body as a whole or a region is
deprived of adequate oxygen supply.
 Oxygen insufficiency is a failure to
provide adequate oxygen to cells of the
body and to remove excess carbon
dioxide from them.
1. Decreased hemoglobin 2. High altitude
3. Inability of the tissue to
extract oxygen from the
blood
4. Decreased diffusion
of oxygen from the
alveoli to the blood
5. Poor tissue perfusion with
oxygenated blood
6. Impaired ventilation
 DEVELOPMENTAL FACTORS
 PHYSIOLOGICAL FACTORS
 BEHAVIOURAL FACTORS
 LIFE STYLE FACTORS
 ENVIRONMENTAL FACTORS
 MEDICATIONS
TYPES
OF
HYPOXIA
Due to reduced oxygen tension in
arterial blood (supply problem)
Causes:-
Low o2 tension in the inhaled air.
Leaking mask, inadequate o2 regulator function
Impaired gas exchange in the lungs e.g. CHRONIC
BRONCHITIS & EMPHYSEMA
Gross ventilation/perfusion mismatch, as occur in
high G forces
 DUE TO DECREASED OXYGEN O2 CARRYING CAPACITY OF THE
BLOOD (Transport Problem)
CO poisoning chemicals/ drugs
hemorrhage/ hemolysis Anemia
OCCURS WHEN BLOOD CIRCULATION THROUGH
TISSUE IS REDUCED (Distribution Problem)
Causes:-
 High G forces
 Syncope (fainting)
 Heart failure
 Shock
DUE TO INABILITY OF THE TISSUES TO MAKE
USE OF THE OXYGEN SUPPLIED TO THEM
(Utilization Problem)
EXAMPLE:
 CYANIDE POISONING
 ALCOHOL & BARBITURATE
 OXYGEN TOXICITY
PATHOPHYSIOLOGY
ANXIETYAND
TIRED
HEADACHE,
DIZZINESS,
IRRITABILITY AND
MEMORY LOSS
NAUSEA
VOMITTING
OLIGURIA /
ANURIA
VISUAL
IMPAIREMENT
 Clubbing of finger
 Impairment in
judgement
 Shortness of breath
1. History Collection 2. Physical
Examination
3. Pulmonary
function test
4. Arterial blood
gas analysis
5. SPUTUM STUDIES 6. CHEST X- RAY
AND CT- SCAN
7. BRONCHOSCOPY
8. Thoracentesis 9. Spirometry
10. Pulse Oxymetry 11. Pulmonary
Angiography
1. POSITIONING
2. BREATHING EXERCISES
3. NEBULIZATION
4. CHEST PHYSIOTHERAPY
5. SUCTIONING
6. OXYGEN THERAPY
 Nasal canula
 Face mask
 Non breather mask
 Venture mask
 Face tent
 Transtracheal oxygenation
 Check the identification data of
the patient.
 Confirm diagnosis and the need
of oxygenation.
 Assess the patient for any sign
of clinical anoxia.
 Monitor for result of ABG.
 Oxygen should be monitored
for toxicity.
 Check that oxygen is properly
humidified.
 Precaution to be taken to
prevent infection.
 Discontinue oxygen therapy
gradually.
 Place a calling bell near patient
for emergency.
 Since oxygen supports
combustion, fire precautions to
be taken during oxygen therapy.
 Do proper documentation
including rate of flow of oxygen.
 Infection
 Combustion
 Drying of mucous membrane
of respiratory tract.
 Oxygen toxicity
 Atelectasis
 Oxygen induced apnea
 Ulceration, edema and
visual impairment
 Asphyxia
 It is a positive pressure or
negative pressure breathing
device that can maintain
ventilation and oxygen
delivery for prolonged period.
 Positive pressure ventilation
 Negative pressure
ventilation
MODES OF
VENTILATOR
 Continuous mandatory volume (CMV)
 Assist control ventilation (ACV)
 Pressure support ventilation (PSV)
 Intermittent mandatory ventilation (IMV)
 Continuous positive airway pressure
(CPAP)
 Synchronized intermittent mandatory
ventilation (SIMV)
 Positive end expiratory pressure (PEEP)
MODES OF VENTILATOR
Continuous mandatory volume is a mode
of mechanical ventilation where breaths
are delivered based on set variables and
makes no effort to sense patient effort
When the patient triggers the ventilator,
he/she receives a breaths of identical
duration and magnitude as the
mandatory breath.
The ventilator only provides support of
each breath to a preset amount of pressure,
thus the volume breathed can differ from
breath to breath taken.
It refers to any mode where a regular series of
breaths are scheduled but the ventilator
senses patient effort and reschedules
mandatory breaths based on the calculated
need of the patient.
 The ventilator adjunct is used with only
spontaneous ventilation; the patient breaths
spontaneously through the ventilator at an
elevated baseline pressure through the
breathing cycle.
 It facilitate the liberation from mechanical
ventilation.
 A demand valve is placed in it, so that
patient could take spontaneous breaths
without taking breath through apparatus of
the ventilator.
 PEEP is the alveolar pressure above
atmospheric pressure that exist at the end of
expiration
These are of two types
 Extrinsic PEEP
 Intrinsic PEEP
1) Impaired gas exchange related
to broncho- construction and
inflammation of airways.
2) Ineffective airway
clearance related to
increased mucous production
due to upper respiratory
infection and asthma.
3) Activity intolerance
related to dyspnoea and
hypoxia manifested by
fatigue.
4) Anxiety related to
difficulty in breathing as
manifested by asking more
doubts.
Abstract
It was recently established that supplemental oxygen
administration significantly enhances memory formation in healthy
young adults. In the present study, a double-blind, placebo-
controlled design was employed to assess the cognitive and
physiological effects of subjects' inspiration of oxygen or air
(control) prior to undergoing simple memory and reaction-time
tasks. Arterial blood oxygen saturation and heart rate were
monitored during each of six phases of the experiment,
corresponding to baseline, gas inhalation, word presentation,
reaction time, distractor and word recall, respectively.
The results confirm that oxygen administration significantly
enhances cognitive performance above that seen in the air
inhalation condition. Subjects who received oxygen recalled more
words and had faster reaction times.
Oxygen insufficiency
Oxygen insufficiency

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Oxygen insufficiency

  • 2.  At the end of the seminar the group will be able to get the knowledge about the oxygen insufficiency, develop a positive attitude towards it and practice this knowledge in teaching and clinical areas.
  • 3. At the end of the class the student will be able to :  Define oxygen insufficiency.  Enlist the etiology of oxygen insufficiency.  Enumerate factors affecting oxygenation  Know the types of oxygen insufficiency.  Discuss the pathophysiology of oxygen insufficiency.  Enlist sign and symptoms of oxygen insufficiency.
  • 4.  Explain different diagnostic evaluation of oxygen insufficiency.  Describe the management of oxygen insufficiency.  Identify the nurses role in the management of oxygen insufficiency.  Explain about oxygen therapy.  Discuss about nurses responsibility while administering oxygen.
  • 5.  Oxygen insufficiency means “ deficient in oxygen”.  The normal range of oxygen in the external blood should be 80-100 mm of Hg.  For treating Oxygen insufficiency effectively, early diagnosis and correct cause should be ruled out.  The only management for Oxygen insufficiency is Oxygen administration.
  • 6.  Oxygen insufficiency is a condition in which the body as a whole or a region is deprived of adequate oxygen supply.  Oxygen insufficiency is a failure to provide adequate oxygen to cells of the body and to remove excess carbon dioxide from them.
  • 7. 1. Decreased hemoglobin 2. High altitude
  • 8. 3. Inability of the tissue to extract oxygen from the blood 4. Decreased diffusion of oxygen from the alveoli to the blood
  • 9. 5. Poor tissue perfusion with oxygenated blood 6. Impaired ventilation
  • 10.  DEVELOPMENTAL FACTORS  PHYSIOLOGICAL FACTORS  BEHAVIOURAL FACTORS  LIFE STYLE FACTORS  ENVIRONMENTAL FACTORS  MEDICATIONS
  • 12. Due to reduced oxygen tension in arterial blood (supply problem) Causes:- Low o2 tension in the inhaled air. Leaking mask, inadequate o2 regulator function Impaired gas exchange in the lungs e.g. CHRONIC BRONCHITIS & EMPHYSEMA Gross ventilation/perfusion mismatch, as occur in high G forces
  • 13.  DUE TO DECREASED OXYGEN O2 CARRYING CAPACITY OF THE BLOOD (Transport Problem) CO poisoning chemicals/ drugs hemorrhage/ hemolysis Anemia
  • 14. OCCURS WHEN BLOOD CIRCULATION THROUGH TISSUE IS REDUCED (Distribution Problem) Causes:-  High G forces  Syncope (fainting)  Heart failure  Shock
  • 15. DUE TO INABILITY OF THE TISSUES TO MAKE USE OF THE OXYGEN SUPPLIED TO THEM (Utilization Problem) EXAMPLE:  CYANIDE POISONING  ALCOHOL & BARBITURATE  OXYGEN TOXICITY
  • 19.  Clubbing of finger  Impairment in judgement  Shortness of breath
  • 20. 1. History Collection 2. Physical Examination
  • 21. 3. Pulmonary function test 4. Arterial blood gas analysis
  • 22. 5. SPUTUM STUDIES 6. CHEST X- RAY AND CT- SCAN 7. BRONCHOSCOPY
  • 23. 8. Thoracentesis 9. Spirometry 10. Pulse Oxymetry 11. Pulmonary Angiography
  • 25. 3. NEBULIZATION 4. CHEST PHYSIOTHERAPY
  • 27.  Nasal canula  Face mask  Non breather mask  Venture mask  Face tent  Transtracheal oxygenation
  • 28.  Check the identification data of the patient.  Confirm diagnosis and the need of oxygenation.  Assess the patient for any sign of clinical anoxia.
  • 29.  Monitor for result of ABG.  Oxygen should be monitored for toxicity.  Check that oxygen is properly humidified.  Precaution to be taken to prevent infection.
  • 30.  Discontinue oxygen therapy gradually.  Place a calling bell near patient for emergency.  Since oxygen supports combustion, fire precautions to be taken during oxygen therapy.  Do proper documentation including rate of flow of oxygen.
  • 31.  Infection  Combustion  Drying of mucous membrane of respiratory tract.  Oxygen toxicity
  • 32.  Atelectasis  Oxygen induced apnea  Ulceration, edema and visual impairment  Asphyxia
  • 33.  It is a positive pressure or negative pressure breathing device that can maintain ventilation and oxygen delivery for prolonged period.
  • 34.  Positive pressure ventilation  Negative pressure ventilation
  • 36.  Continuous mandatory volume (CMV)  Assist control ventilation (ACV)  Pressure support ventilation (PSV)  Intermittent mandatory ventilation (IMV)  Continuous positive airway pressure (CPAP)  Synchronized intermittent mandatory ventilation (SIMV)  Positive end expiratory pressure (PEEP) MODES OF VENTILATOR
  • 37. Continuous mandatory volume is a mode of mechanical ventilation where breaths are delivered based on set variables and makes no effort to sense patient effort
  • 38. When the patient triggers the ventilator, he/she receives a breaths of identical duration and magnitude as the mandatory breath.
  • 39. The ventilator only provides support of each breath to a preset amount of pressure, thus the volume breathed can differ from breath to breath taken.
  • 40. It refers to any mode where a regular series of breaths are scheduled but the ventilator senses patient effort and reschedules mandatory breaths based on the calculated need of the patient.
  • 41.  The ventilator adjunct is used with only spontaneous ventilation; the patient breaths spontaneously through the ventilator at an elevated baseline pressure through the breathing cycle.
  • 42.  It facilitate the liberation from mechanical ventilation.  A demand valve is placed in it, so that patient could take spontaneous breaths without taking breath through apparatus of the ventilator.
  • 43.  PEEP is the alveolar pressure above atmospheric pressure that exist at the end of expiration These are of two types  Extrinsic PEEP  Intrinsic PEEP
  • 44.
  • 45. 1) Impaired gas exchange related to broncho- construction and inflammation of airways.
  • 46. 2) Ineffective airway clearance related to increased mucous production due to upper respiratory infection and asthma.
  • 47. 3) Activity intolerance related to dyspnoea and hypoxia manifested by fatigue.
  • 48. 4) Anxiety related to difficulty in breathing as manifested by asking more doubts.
  • 49. Abstract It was recently established that supplemental oxygen administration significantly enhances memory formation in healthy young adults. In the present study, a double-blind, placebo- controlled design was employed to assess the cognitive and physiological effects of subjects' inspiration of oxygen or air (control) prior to undergoing simple memory and reaction-time tasks. Arterial blood oxygen saturation and heart rate were monitored during each of six phases of the experiment, corresponding to baseline, gas inhalation, word presentation, reaction time, distractor and word recall, respectively. The results confirm that oxygen administration significantly enhances cognitive performance above that seen in the air inhalation condition. Subjects who received oxygen recalled more words and had faster reaction times.