4. MAIN CLINICAL QUESTIONS
• What is the overall cardiac wall motion?
• Is there a pericardial effusion?
5. CARDIAC PROBE SELECTION
Small round footprint
for scan between ribs
2.5 MHz: above
average sized patient
3.5 MHz: average sized
patient
5.0 MHz: below
average sized patient
or child
8. ORIENTATION
Subcostal or subxiphoid view
Best all around imaging window
Good for identification of:
Circumferential pericardial effusion
Overall wall motion
Easy to obtain – liver is the acoustic window
13. SUBCOSTAL VIEW
Angle probe right to
see IVC
Response of IVC to
sniff indicates central
venous pressure
No collapse
Tamponade
CHF
PE
Pneumothorax
14. PARASTERNAL VIEWS
• Next best imaging window
• Good for imaging LV
• Comparing chamber sizes
• Localized effusions
• Differentiating pericardial from pleural effusions
15. PARASTERNAL LONG AXIS
Near sternum
3rd or 4th left intercostal space
Marker pointed to patient’s right shoulder (or left hip if
screen is not reversed for cardiac imaging)
Rotate enough to elongate cardiac chambers
18. PARASTERNAL SHORT AXIS
Obtained by 90° clockwise rotation of the probe towards
the left shoulder (or right hip)
Sweep the beam from the base of the heart to the apex
for different cross sectional views
29. CASE PRESENTATION
45 year old male presents with SOB and dizziness for 2 days. He has a long
smoking history, and has complained of a non-productive cough for
“weeks”
Initial VS are BP 88/palp, HR 140
PE: Neck veins are distended
Chest: Clear, muffled heart sounds
Bedside sonography was performed
30.
31. ECHO FREE SPACE AROUND THE
HEART
Pericardial effusion
Pleural effusion
Epicardial fat (posterior and/or anterior)
Less common causes:
Aortic aneurysm
Pericardial cyst
Dilated pulmonary artery
32. SIZE OF THE PERICARDIAL EFFUSION
Not Precise
Small: confined to posterior space, < 0.5cm
Moderate: anterior and posterior, 0.5-2cm (diastole)
Large: > 2cm
34. CLINICAL FEATURES OF PERICARDIAL
EFFUSION
Pericardial fluid accumulation may be clinically silent
Symptoms are due to:
mechanical compression of adjacent structures
Increased intrapericardial pressure
35. PERICARDIAL EFFUSION:ASYMPTOMATIC
Up to 40% of pregnant women
Chronic hemodialysis patients
one study showed 11% incidence of pericardial effusion
AIDS
CHF
Hypoproteinemic states
36. SYMPTOMS OF PERICARDIAL EFFUSION
Chest discomfort (most common)
Large effusions:
Dyspnea
Cough
Fatigue
Hiccups
Hoarseness
Nausea and abdominal fullness
45. MASSIVE PE OR RV INFARCT
Dilated Right
ventricle
RV hypokinesis
Normal Left
ventricle function
Stiff IVC
46. CASE PRESENTATION ? OVERDOSE
27 yo f brought in with “passing out” after night of heavy
drinking.
Complaining of inability to breathe!
PE: Obese f BP 88/60
HR 123 Ox 78%
Chest: clear
Ext: No edema
Bedside sonography was performed
47.
48.
49. CHEST PAIN THEN CODE
55 yo male suffered witnessed Vfib arrest in the ED
ALS protocol - restoration of perfusing rhythm
Persistant hypotension
ED ECHO was performed
54. DIRECT VISUALIZATION
Is there effective myocardial contractility?
Asystole
Myocardial “twitch”
Hypokinesis
Normal
Is there a pericardial effusion?
55. ECHO IN PEA
Perform ECHO during “quick look” and in pulse checks
Change management based on “positive” findings
Pericardial tamponade
Pericardiocentesis
Hyperdynamic cardiac wall motion
Volume resuscitate
56. ECHO IN PEA
RV dilatation
Hypoxic?? – Likely PE
ECG – IMI with RV infarct?
Profound hypokinesis
Inotropic support
Asystole
Follow ACLS protocols (for now)
Early data suggesting poor prognosis
57. ECHO IN PEA
False positive cardiac motion
Transthoracic pacemaker
Positive pressure ventilation
58. CASE PRESENTATION
Morbidly obese female with severe asthma
Intubated for respiratory failure
Subcutaneous emphysema developed
Bilateral chest tubes placed
Persistent hypotension at 90/palp
Dependent mottling noted
ECHO was performed
61. OPTIMIZING PERFORMANCE
Assessment of capture by transthoracic pacemaker
Ettin D et al: Using ultrasound to determine external pacer capture JEM
1999
62. CASE PRESENTATION
70 yo f collapsed in lobby. She was brought
into the ED apneic, hypotensive. She was
quickly intubated and volume resuscitation
begun.
VS: BP 80/50 HR 50 Afebrile
Physical exam : Thin, minimally responsive f.
Clear lungs, nl heart sounds, abdomen slightly
distended with decreased bowel sounds. No
HSM, ? Pelvic mass
ECG: SB, LVH, no active ischemia
63. CLINICAL QUESTIONS?
Why is she hypotensive?
Volume loss
?Ruptured AAA
Pump failure
Bedside sonography was performed while we were waiting
for the “labs”
68. US GUIDED- PERICARDIOCENTESIS
Subcostal approach
Traditional approach
Blind
Increased risk of injury to liver, heart
Echo guided
Left parasternal preferred for needle entry or…
Largest area of fluid collection adjacent to the
chest wall
71. OPTIMIZING PERFORMANCE
Placement of transvenous pacemaker
Aguilera P et al: Emergency transvenous cardiac pacing
placement using ultrasound guidance. Ann Emerg Med
2000
72. UNTIMELY END
30 yo brought in after he “fell out”
Ashen m with no spontaneous respirations
VS: No pulse, agonal rhythm on monitor
Intubated/CPR
Transvenous pacemaker placed, no capture.
ECHO showed
75. PENETRATING CARDIAC TRAUMA
Physician’s ability to determine whether there
is a hemodynamically significant effusion is
poor
Beck’s Triad
Dependent on patient cardiovascular status
Findings are often late
Determinants of hemodynamic compromise
Size of the effusion
Rate of formation
76. PENETRATING CARDIAC INJURY
Emergency department echocardiography improves outcome in penetrating
cardiac injury.
Plummer D et al. Ann Emerg Med. 1992
28 had ED echo c/w 21 without ED echo
Survival: 100% in echo, 57.1% in nonecho
Time to Dx: 15 min echo, 42 min nonecho
77. PENETRATING CARDIAC INJURY
The role of ultrasound in patients with
possible penetrating cardiac wounds: a
prospective multicenter study.
Rozycki GS: J Trauma. 1999
Pericardial scans performed in 261 patients
Sensitivity 100%, specificity 96.9%
PPV: 81% NPV:100%
Time interval BUS to OR: 12.1 +/- 5.9 min
78. PENETRATING CARDIAC TRAUMA
Emergency Department Echocardiography Improves Outcome in Penetrating
Cardiac Injury
Plummer D, et al. Ann Emerg Med 21:709-712, 1992.
“Since the introduction of immediate ED two-
dimensional echocardiography, the time to
diagnosis of penetrating cardiac injury has
decreased and both the survival rate and
neurologic outcome of survivors has
improved.”
80. PENETRATING CARDIAC TRAUMA
Echocardiographic signs of rising
intrapericardial pressure
Collapse of RV free walls
Dilated IVC and hepatic veins
Goal: Early detection of pericardial effusion
Develops suddenly or discretely
May exist before clinical signs develop
Salvage rates better if detected before
hypotension develops
84. PERICARDIAL OR PLEURAL FLUID
Left parasternal long axis:
Pericardial fluid does not extend posterior to descending aorta or
left atrium
Subcostal:
No pleural reflection between liver and R sided chambers
A pleural effusion will not extend between to RV free wall and the
liver
92. SUMMARY
Bedside ECHO can help assess:
Overall cardiac wall motion
Identify clinically significant pericardial effusions
Useful in the assessment of the patient
with:
Unexplained hypotension
Dyspnea
Thoracic trauma