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PNEUMOCONIOSIS
HISTORY
It is likely that humans have suffered
from occupational lung disease since the
change from hunting to agriculture as a
means of providing food.
In roman times it was recorded that
mining was a dangerous trade, fit only for
convicts and slaves.
The first recorded mention of
breathlessness among handlers of grain
was done by Ramazzini, the father of
occupational medicine, in 1713.
BERNARDINUS
RAMAZZINI
DEFNITION
Pneumoconiosis is a lung disease
due to exposure of lung for
variable period to dust in size
range of 0.5 to 3 microns leading
to lung fibrosis and other
complications.
HAZARDS OF DUST
PARTICLES ON LUNG
 Chemical composition
 Fineness
 Concentration of dust in air
 Health status of the person
exposed
 Period of exposure
VARIOUS DUST RELATED
DISEASES
 Silicosis- from silica dust
 Asbestosis- from asbestos dust
 Coal workers pneumoconiosis- from coal
dust
 Byssinosis- from cotton dust
 Bagassosis- from sugarcane dust
 Farmer’s lung- from hay dust or mold spores
or other agriculture products.
 Berylliosis- from beryllium
TYPES
Pneumoconiosis is usually divided into
three groups:
Major pneumoconiosis
Minor pneumoconiosis
Benign pneumoconiosis
MAJOR
PNEUMOCONIOSIS
 Inhalation of some dusts results in “major
fibrosis” of the lungs, Which results in
interference of lung architecture or lung
function tests.
 Examples are:
Silica- silicosis
Asbestos- asbestosis
Coal- anthracosis
Healthy lung Silicotic lung
MINOR
PNEUMOCONIOSIS
 Inhalation of some dusts results in
“minor fibrosis” of the lungs.
 There is minimal fibrosis of the lungs
without interference of lung architecture or
lung function tests.
 These dusts include:
Mica pneumoconiosis
Kaolin(china clay) pneumoconiosis
BENIGN
PNEUMOCONIOSIS
 There isn’t any reaction in the lungs, but dust
deposition casts a shadow in X-ray of the lung.
There is no fibrosis and no disturbance of lung
functions.
 It results from inhalation of:
Iron dust- siderosis
Tin dust- stannosis
Calcium dust- chalcosis
 These deposits in the lung disappear when
exposure is discontinued.
SYMPTOMS
Shortness of breath
Cough
Lots of phlegm
PREVENTIVE MEASURES
Prohibit dry cutting Promote wet cutting
PREVENTIVE MEASURES
Personal protective equipments:
DIAGNOSIS
Personal history of work
exposure
Physical examination
Chest X-ray
CT scan of the chest
Pulmonary function
studies
biopsy
There are no specific treatments or
medications for pneumoconiosis, and
there is no cure.
Most treatments for patients with
pneumoconiosis are aimed at limiting
further damage to the lung, decreasing
symptoms and improving quality of life.
TREATMENT
Thank you

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Austin Journal of Lung Cancer Research

  • 2. HISTORY It is likely that humans have suffered from occupational lung disease since the change from hunting to agriculture as a means of providing food. In roman times it was recorded that mining was a dangerous trade, fit only for convicts and slaves. The first recorded mention of breathlessness among handlers of grain was done by Ramazzini, the father of occupational medicine, in 1713. BERNARDINUS RAMAZZINI
  • 3. DEFNITION Pneumoconiosis is a lung disease due to exposure of lung for variable period to dust in size range of 0.5 to 3 microns leading to lung fibrosis and other complications.
  • 4. HAZARDS OF DUST PARTICLES ON LUNG  Chemical composition  Fineness  Concentration of dust in air  Health status of the person exposed  Period of exposure
  • 5. VARIOUS DUST RELATED DISEASES  Silicosis- from silica dust  Asbestosis- from asbestos dust  Coal workers pneumoconiosis- from coal dust  Byssinosis- from cotton dust  Bagassosis- from sugarcane dust  Farmer’s lung- from hay dust or mold spores or other agriculture products.  Berylliosis- from beryllium
  • 6. TYPES Pneumoconiosis is usually divided into three groups: Major pneumoconiosis Minor pneumoconiosis Benign pneumoconiosis
  • 7. MAJOR PNEUMOCONIOSIS  Inhalation of some dusts results in “major fibrosis” of the lungs, Which results in interference of lung architecture or lung function tests.  Examples are: Silica- silicosis Asbestos- asbestosis Coal- anthracosis Healthy lung Silicotic lung
  • 8. MINOR PNEUMOCONIOSIS  Inhalation of some dusts results in “minor fibrosis” of the lungs.  There is minimal fibrosis of the lungs without interference of lung architecture or lung function tests.  These dusts include: Mica pneumoconiosis Kaolin(china clay) pneumoconiosis
  • 9. BENIGN PNEUMOCONIOSIS  There isn’t any reaction in the lungs, but dust deposition casts a shadow in X-ray of the lung. There is no fibrosis and no disturbance of lung functions.  It results from inhalation of: Iron dust- siderosis Tin dust- stannosis Calcium dust- chalcosis  These deposits in the lung disappear when exposure is discontinued.
  • 11. PREVENTIVE MEASURES Prohibit dry cutting Promote wet cutting
  • 13. DIAGNOSIS Personal history of work exposure Physical examination Chest X-ray CT scan of the chest Pulmonary function studies biopsy
  • 14. There are no specific treatments or medications for pneumoconiosis, and there is no cure. Most treatments for patients with pneumoconiosis are aimed at limiting further damage to the lung, decreasing symptoms and improving quality of life. TREATMENT