Dysphagia is the medical term for swallowing difficulties. Some people with dysphagia have problems swallowing certain foods or liquids, while others can't swallow at all.
This document discusses gastrointestinal bleeding, including defining acute upper and lower GI bleeding, assessing the severity of bleeding, determining the location of bleeding, differential diagnoses, and management approaches. It provides details on the signs and symptoms of upper and lower GI bleeding, common causes, and first aid measures. The management of GI bleeding involves assessing the patient's hemodynamic status, localizing the bleeding source, checking blood work, addressing risk factors, and stopping active bleeding while preventing recurrence.
Gastrointestinal bleeding can originate from the upper or lower GI tract. Upper GI bleeding comes from the esophagus, stomach, or duodenum and is often caused by peptic ulcers, gastritis, esophageal varices, or Mallory-Weiss tears. Lower GI bleeding originates farther down the digestive tract and is commonly due to diverticular disease, angiodysplasia, polyps, hemorrhoids, or anal fissures. Symptoms of GI bleeding include vomiting blood, bloody stools, fatigue, and weakness. Medical evaluation and tests like endoscopy are needed to determine the source and severity of bleeding so that appropriate treatment such as medication, surgery, or lifestyle changes can be given.
This document provides an overview of gastrointestinal (GI) bleeding, including its causes, symptoms, diagnosis, and treatment. GI bleeding is classified as upper or lower depending on its location in the GI tract. Common causes of upper GI bleeding include peptic ulcers, gastritis, esophageal varices, and cancers. Lower GI bleeding is often caused by diverticulosis, cancers, inflammatory bowel disease, infections, angiodysplasia, polyps, and hemorrhoids. Symptoms include vomiting blood, black stools, and fatigue. Diagnosis involves medical history, physical exam, endoscopy or colonoscopy, and lab tests. Treatment depends on severity but may include IV fluids, blood transfusions, surgery, or
This document provides information on upper gastrointestinal bleeding (UGIB), including its definition, epidemiology, causes, clinical presentation, diagnostic evaluation, and management. Some key points:
- UGIB is 5 times more common than lower GI bleeding and is most often caused by peptic ulcers (duodenal more than gastric).
- Clinical presentation depends on the rate of bleeding, ranging from chronic anemia to hypovolemic shock.
- Initial management involves resuscitation, blood transfusion, and early endoscopy for diagnosis and treatment.
- Endoscopy allows for diagnosis in 80% of cases and treatment of high-risk stigmata like active bleeding or non-bleeding visible
This document provides an overview of the assessment of digestive and gastrointestinal function. It describes the anatomy and physiology of the mouth, esophagus, stomach, small intestine, large intestine, liver, gallbladder and biliary system. It discusses the enzymes and secretions involved in digestion at each site. It also outlines common gastrointestinal disorders like ulcers, hepatitis, cirrhosis, pancreatitis and approaches to their assessment and management.
Portal hypertension can be caused by conditions that block the portal vein (precirrhotic) or damage the liver (cirrhotic). It leads to complications like ascites, esophageal varices, and bleeding. Symptoms include abdominal swelling and pain, bleeding, and fatigue. Treatment focuses on reducing portal pressure through dietary changes, diuretics, banding or sclerotherapy of varices, transjugular intrahepatic portosystemic shunting (TIPS), or surgery in severe cases.
Acute pancreatitis is inflammation of the pancreas that commonly results from gallstones or alcohol abuse. It requires hospital admission for intravenous fluids and pain management. The condition ranges from mild edema to severe necrosis and affects over 200,000 patients annually in the US. Diagnosis is based on abdominal pain and elevated pancreatic enzymes. Treatment focuses on fluid resuscitation, nutritional support to rest the pancreas, and managing complications.
Upper gastrointestinal hemorrhage is a common medical problem caused by bleeding peptic ulcers, portal hypertension, gastritis, and esophageal varices. Initial assessment and management goals are to assess circulatory status, replace blood loss, determine bleeding rate and amount, and discover the underlying lesion. Specific treatments depend on the cause, and may include endoscopic hemostasis, medical management with H2 antagonists or PPIs, surgical treatment, or management of portal hypertension.
This document discusses gastrointestinal bleeding, including defining acute upper and lower GI bleeding, assessing the severity of bleeding, determining the location of bleeding, differential diagnoses, and management approaches. It provides details on the signs and symptoms of upper and lower GI bleeding, common causes, and first aid measures. The management of GI bleeding involves assessing the patient's hemodynamic status, localizing the bleeding source, checking blood work, addressing risk factors, and stopping active bleeding while preventing recurrence.
Gastrointestinal bleeding can originate from the upper or lower GI tract. Upper GI bleeding comes from the esophagus, stomach, or duodenum and is often caused by peptic ulcers, gastritis, esophageal varices, or Mallory-Weiss tears. Lower GI bleeding originates farther down the digestive tract and is commonly due to diverticular disease, angiodysplasia, polyps, hemorrhoids, or anal fissures. Symptoms of GI bleeding include vomiting blood, bloody stools, fatigue, and weakness. Medical evaluation and tests like endoscopy are needed to determine the source and severity of bleeding so that appropriate treatment such as medication, surgery, or lifestyle changes can be given.
This document provides an overview of gastrointestinal (GI) bleeding, including its causes, symptoms, diagnosis, and treatment. GI bleeding is classified as upper or lower depending on its location in the GI tract. Common causes of upper GI bleeding include peptic ulcers, gastritis, esophageal varices, and cancers. Lower GI bleeding is often caused by diverticulosis, cancers, inflammatory bowel disease, infections, angiodysplasia, polyps, and hemorrhoids. Symptoms include vomiting blood, black stools, and fatigue. Diagnosis involves medical history, physical exam, endoscopy or colonoscopy, and lab tests. Treatment depends on severity but may include IV fluids, blood transfusions, surgery, or
This document provides information on upper gastrointestinal bleeding (UGIB), including its definition, epidemiology, causes, clinical presentation, diagnostic evaluation, and management. Some key points:
- UGIB is 5 times more common than lower GI bleeding and is most often caused by peptic ulcers (duodenal more than gastric).
- Clinical presentation depends on the rate of bleeding, ranging from chronic anemia to hypovolemic shock.
- Initial management involves resuscitation, blood transfusion, and early endoscopy for diagnosis and treatment.
- Endoscopy allows for diagnosis in 80% of cases and treatment of high-risk stigmata like active bleeding or non-bleeding visible
This document provides an overview of the assessment of digestive and gastrointestinal function. It describes the anatomy and physiology of the mouth, esophagus, stomach, small intestine, large intestine, liver, gallbladder and biliary system. It discusses the enzymes and secretions involved in digestion at each site. It also outlines common gastrointestinal disorders like ulcers, hepatitis, cirrhosis, pancreatitis and approaches to their assessment and management.
Portal hypertension can be caused by conditions that block the portal vein (precirrhotic) or damage the liver (cirrhotic). It leads to complications like ascites, esophageal varices, and bleeding. Symptoms include abdominal swelling and pain, bleeding, and fatigue. Treatment focuses on reducing portal pressure through dietary changes, diuretics, banding or sclerotherapy of varices, transjugular intrahepatic portosystemic shunting (TIPS), or surgery in severe cases.
Acute pancreatitis is inflammation of the pancreas that commonly results from gallstones or alcohol abuse. It requires hospital admission for intravenous fluids and pain management. The condition ranges from mild edema to severe necrosis and affects over 200,000 patients annually in the US. Diagnosis is based on abdominal pain and elevated pancreatic enzymes. Treatment focuses on fluid resuscitation, nutritional support to rest the pancreas, and managing complications.
Upper gastrointestinal hemorrhage is a common medical problem caused by bleeding peptic ulcers, portal hypertension, gastritis, and esophageal varices. Initial assessment and management goals are to assess circulatory status, replace blood loss, determine bleeding rate and amount, and discover the underlying lesion. Specific treatments depend on the cause, and may include endoscopic hemostasis, medical management with H2 antagonists or PPIs, surgical treatment, or management of portal hypertension.
This document discusses bleeding, types of wounds, and shock. It describes arterial, venous, and capillary bleeding. Severe bleeding requires a pressure dressing and elevation. Shock can be hypovolemic, cardiogenic, septic, anaphylactic, or neurogenic. Hypovolemic shock treatment includes fluid replacement and blood transfusion. Anaphylactic shock is treated with epinephrine injection and other supportive measures.
Approach to Management of Upper Gastrointestinal (GI) BleedingArun Vasireddy
Upper gastrointestinal bleeding is gastrointestinal bleeding in the upper gastrointestinal tract, commonly defined as bleeding arising from the esophagus, stomach, or duodenum. Blood may be observed in vomit (hematemesis) or in altered form in the stool (melena). Depending on the severity of the blood loss, there may be symptoms of insufficient circulating blood volume and shock. As a result, upper gastrointestinal bleeding is considered a medical emergency and typically requires hospital care for urgent diagnosis and treatment. Upper gastrointestinal bleeding can be caused by peptic ulcers, gastric erosions, esophageal varices, and some rarer causes such as gastric cancer.
The initial assessment includes measurement of the blood pressure and heart rate, as well as blood tests to determine hemoglobin concentration. In significant bleeding, fluid replacement is often required, as well as blood transfusion, before the source of bleeding can be determined by endoscopy of the upper digestive tract with an esophagogastroduodenoscopy. Depending on the source, endoscopic therapy can be applied to reduce rebleeding risk. Specific medical treatments (such as proton pump inhibitors for peptic ulcer disease) or procedures (such as TIPS for variceal hemorrhage) may be used. Recurrent or refractory bleeding may lead to need for surgery, although this has become uncommon as a result of improved endoscopic and medical treatment.
This document defines and describes lower gastrointestinal bleeding. Key points:
- Lower GI bleeding occurs distal to the ligament of Treitz and is defined as abnormal bleeding into the bowel lumen. Significant bleeding is over 10ml/day.
- The lower GI tract includes the small intestine (duodenum, jejunum, ileum), large intestine (cecum, colon, rectum), and anus.
- Lower GI bleeding typically presents as hematochezia (red or dark blood in stool) or melena (dark stools). Massive bleeding causes hemodynamic instability while moderate bleeding is stable.
- Common causes include vascular abnormalities, inflammatory bowel disease, cancers, polyps, diverticular
This document provides an overview of upper gastrointestinal bleeding, including its causes, types, diagnostic studies, treatment options, and nursing management. The main causes of upper GI bleeding include drug use, esophageal varices, esophagitis, peptic ulcers, and stomach or duodenal cancers. Diagnostic studies involve endoscopy to identify the source of bleeding. Treatment may involve endoscopic therapies, drugs to reduce bleeding, or surgery in severe cases. Nursing care focuses on emergency stabilization and monitoring for signs of ongoing bleeding or complications.
This document discusses portal hypertension and its causes, effects, diagnosis, and treatment. It begins by describing portal vein anatomy and how portal pressure is normally measured. The main causes of portal hypertension are then outlined as being pre-hepatic (portal vein obstruction), intra-hepatic (liver disease), or post-hepatic (hepatic vein issues). Key sequelae include portosystemic collaterals, splenomegaly, gastrointestinal congestion, bleeding varices, and ascites. Investigations focus on liver function tests, detecting varices, and assessing severity using the Child-Pugh classification. Treatment involves resuscitation, preventing encephalopathy, and procedures like sclerotherapy, banding, drugs,
This document provides information on portal hypertension, including its definition, causes, presentations, investigations, and management. Some key points:
- Portal hypertension is defined as a sustained elevation of portal pressure above 12 mm Hg. It can be caused by conditions affecting blood flow pre-sinusoidally (e.g. portal vein thrombosis), sinusoidally (e.g. cirrhosis), or post-sinusoidally (e.g. Budd-Chiari syndrome).
- Common presentations include esophageal varices, splenomegaly, ascites, and features of hepatic encephalopathy. Investigations include liver function tests, ultrasound, and endoscopy to identify varices.
-
Portal hypertension occurs when blood pressure within the portal venous system exceeds normal levels. It can be caused by prehepatic issues like portal vein obstruction, or intrinsic liver diseases that increase fibrosis within the liver. Children with portal hypertension may experience bleeding from esophageal varices, splenomegaly, ascites, or other clinical signs. Diagnosis involves medical history, lab tests, imaging like Doppler ultrasound or CT, and often measurement of portal pressure. Treatment depends on severity but may include medication, balloon tamponade, endoscopic procedures to band or sclerose varices, or shunt procedures like TIPS to decompress the portal system.
This document discusses gastrointestinal (GI) bleeding, including:
1) GI bleeding can present as overt (visible bleeding) or occult (hidden bleeding) and can originate from the upper or lower GI tract. Common symptoms include hematemesis, melena, and hematochezia.
2) Etiologies of upper GI bleeding include esophageal varices, peptic ulcers, Mallory-Weiss tears, and Dieulafoy's lesions. Management involves endoscopy, vasoactive drugs, proton pump inhibitors, and blood transfusions.
3) Lower GI bleeding can originate from the small intestine or colon. Causes include vascular ectasias, cancers, diverticulosis, and inflammatory bowel disease
Ascites refers to an accumulation of non-inflammatory fluid in the peritoneal cavity, characterized by a distended lower abdomen. It can be caused by decreased plasma protein levels, increased hydrostatic pressure, or liver or kidney damage. Clinically, affected animals have an enlarged, barrel-shaped abdomen with fluid waves visible on palpation. Diagnosis involves abdominal ultrasound or paracentesis to analyze fluid characteristics. Treatment focuses on resolving underlying causes, increasing plasma proteins, diuretics to promote fluid excretion, and draining excess fluid in severe cases.
This document discusses gastrointestinal bleeding in children. It defines different types of GI bleeding based on the location such as melena, hematochezia, and hematemesis. Common causes of upper and lower GI bleeding are listed for newborns, infants, children, and adolescents. Evaluation involves history, physical exam, lab tests, and imaging. Treatment depends on the severity and includes supportive care, medications, endoscopic procedures, and surgery. Portal hypertension is described as a cause of GI bleeding along with its pathophysiology, clinical manifestations, diagnosis, and management.
Cirrhosis is a chronic disease characterized by the replacement of normal liver tissue with scar tissue. This disrupts liver function and structure. Major complications of cirrhosis include ascites, portal hypertension, hepatic encephalopathy, and esophageal varices. Ascites is the accumulation of fluid in the abdominal cavity caused by portal hypertension and sodium retention. It is managed medically through dietary changes, diuretics, and paracentesis. Hepatic encephalopathy occurs when toxic metabolites accumulate in the bloodstream due to liver damage. Esophageal varices develop due to portal hypertension and can rupture, causing life-threatening bleeding.
lower GIT bleeding: is bleeding from a source distal to the ligament of Treitz (duodenojejunal junction), presented as
Hematochezia is blood passed with stool from the anus,
Melena is black, tarry stool produced by the oxidation of heme by intestinal flora; as little as 50 mL of blood may result in melena, and it may persist for 3 to 5 days following resolution of the bleed.
Maroon-colored stool is associated with rapidly bleeding small bowel lesions in which the transit of blood is too fast for complete oxidation.
Currant-jelly stool is associated with ischemic small bowel or proximal colonic lesions such as may be seen in intussusception.
Upper GIT bleeding: is bleeding from a source proximal to the ligament of Treitz (duodenojejunal junction).
Discussion included the definition of bleeding per rectum, it's types according to child age groups, it's presentation, how to diagnose each type and how to treat.
The pancreas develops from ventral and dorsal buds that fuse during gestation. It has both exocrine and endocrine functions. Acute pancreatitis is commonly caused by gallstones or alcohol and results from premature activation of pancreatic enzymes within the pancreas, leading to autodigestion. It can range from mild to severe, with severe cases involving hemorrhage and necrosis. Treatment is usually initially conservative but surgery may be needed for complications or failure to improve.
This document summarizes portal hypertension, including its causes, pathophysiology, clinical presentation, diagnosis, and treatment. Portal hypertension is defined as increased pressure in the portal vein greater than 10 mmHg. It can be caused by conditions that obstruct portal blood flow within or outside the liver. Increased pressure leads to formation of collateral veins and complications like variceal bleeding, splenomegaly, and ascites. Diagnosis involves identifying the underlying liver disease and assessing its severity. Treatment aims to prevent variceal bleeding through medication, band ligation, shunt procedures, or TIPS.
Hematemesis- vomiting of blood , a brief studymartinshaji
There can be many causes of hematemesis, such as: bleeding ulcers. prolonged and vigorous retching that causes tears in the esophageal mucosa (known as Mallory-Weiss Syndrome) gastric or intestinal varices.Haematemesis is simply defined as “vomiting blood”. It is caused by bleeding from part of the upper portion of the gastrointestinal tract. It has a wide range of possible causes, depending on the site of blood loss and the tissue that is actively bleeding. Hence it is necessary to analyse and treat the condition perfectly , this is brief study about all the aspects hematemesis ,vomiting of blood including etiology, definition,management ,treatment by drugs etc
please comment
thank u
Vomiting blood, also known as hematemesis, refers to vomiting of significant amounts of blood or contents mixed with blood. It can range from minor to very serious and requires determination of the underlying cause. Common causes include ulcers, gastritis, esophageal varices, cancer, and side effects of medications like NSAIDs. Diagnosis involves medical history, physical exam, imaging tests, endoscopy, and biopsy. Treatment depends on the cause but may include medications to stop bleeding, endoscopic procedures, surgery, blood transfusions, and lifestyle changes. Complications can include further bleeding, anemia, shock, and aspiration.
ascites-mu1.ppt case presentation of ascitespankajpatle8
Serum-ascites albumin gradient (SAAG) is a test used to determine the cause of ascites. It is calculated by subtracting the ascitic fluid albumin level from the serum albumin level.
A SAAG value helps differentiate between:
- Portal hypertension-related ascites (SAAG >1.1 g/dL) which is most commonly seen in liver cirrhosis.
- Non-portal hypertension related ascites (SAAG <1.1 g/dL) which can be caused by things like peritoneal carcinomatosis, tuberculosis, pancreatitis, nephrotic syndrome etc.
In simple terms, a SAAG value helps determine if the ascites is caused by portal
The document summarizes the key parts and functions of the digestive system. It describes the small intestine absorbing most nutrients and transporting waste to the large intestine. It then details the liver processing nutrients and filtering blood, before waste is expelled by the large intestine and rectum.
The document summarizes the key parts and functions of the digestive system. It describes the small intestine absorbing most nutrients and transporting waste to the large intestine. It then details the liver which filters blood and produces bile, and the gallbladder which stores and concentrates bile. Diseases like cirrhosis and conditions like jaundice are also mentioned.
How to Add Chatter in the odoo 17 ERP ModuleCeline George
In Odoo, the chatter is like a chat tool that helps you work together on records. You can leave notes and track things, making it easier to talk with your team and partners. Inside chatter, all communication history, activity, and changes will be displayed.
How to Manage Your Lost Opportunities in Odoo 17 CRMCeline George
Odoo 17 CRM allows us to track why we lose sales opportunities with "Lost Reasons." This helps analyze our sales process and identify areas for improvement. Here's how to configure lost reasons in Odoo 17 CRM
This document discusses bleeding, types of wounds, and shock. It describes arterial, venous, and capillary bleeding. Severe bleeding requires a pressure dressing and elevation. Shock can be hypovolemic, cardiogenic, septic, anaphylactic, or neurogenic. Hypovolemic shock treatment includes fluid replacement and blood transfusion. Anaphylactic shock is treated with epinephrine injection and other supportive measures.
Approach to Management of Upper Gastrointestinal (GI) BleedingArun Vasireddy
Upper gastrointestinal bleeding is gastrointestinal bleeding in the upper gastrointestinal tract, commonly defined as bleeding arising from the esophagus, stomach, or duodenum. Blood may be observed in vomit (hematemesis) or in altered form in the stool (melena). Depending on the severity of the blood loss, there may be symptoms of insufficient circulating blood volume and shock. As a result, upper gastrointestinal bleeding is considered a medical emergency and typically requires hospital care for urgent diagnosis and treatment. Upper gastrointestinal bleeding can be caused by peptic ulcers, gastric erosions, esophageal varices, and some rarer causes such as gastric cancer.
The initial assessment includes measurement of the blood pressure and heart rate, as well as blood tests to determine hemoglobin concentration. In significant bleeding, fluid replacement is often required, as well as blood transfusion, before the source of bleeding can be determined by endoscopy of the upper digestive tract with an esophagogastroduodenoscopy. Depending on the source, endoscopic therapy can be applied to reduce rebleeding risk. Specific medical treatments (such as proton pump inhibitors for peptic ulcer disease) or procedures (such as TIPS for variceal hemorrhage) may be used. Recurrent or refractory bleeding may lead to need for surgery, although this has become uncommon as a result of improved endoscopic and medical treatment.
This document defines and describes lower gastrointestinal bleeding. Key points:
- Lower GI bleeding occurs distal to the ligament of Treitz and is defined as abnormal bleeding into the bowel lumen. Significant bleeding is over 10ml/day.
- The lower GI tract includes the small intestine (duodenum, jejunum, ileum), large intestine (cecum, colon, rectum), and anus.
- Lower GI bleeding typically presents as hematochezia (red or dark blood in stool) or melena (dark stools). Massive bleeding causes hemodynamic instability while moderate bleeding is stable.
- Common causes include vascular abnormalities, inflammatory bowel disease, cancers, polyps, diverticular
This document provides an overview of upper gastrointestinal bleeding, including its causes, types, diagnostic studies, treatment options, and nursing management. The main causes of upper GI bleeding include drug use, esophageal varices, esophagitis, peptic ulcers, and stomach or duodenal cancers. Diagnostic studies involve endoscopy to identify the source of bleeding. Treatment may involve endoscopic therapies, drugs to reduce bleeding, or surgery in severe cases. Nursing care focuses on emergency stabilization and monitoring for signs of ongoing bleeding or complications.
This document discusses portal hypertension and its causes, effects, diagnosis, and treatment. It begins by describing portal vein anatomy and how portal pressure is normally measured. The main causes of portal hypertension are then outlined as being pre-hepatic (portal vein obstruction), intra-hepatic (liver disease), or post-hepatic (hepatic vein issues). Key sequelae include portosystemic collaterals, splenomegaly, gastrointestinal congestion, bleeding varices, and ascites. Investigations focus on liver function tests, detecting varices, and assessing severity using the Child-Pugh classification. Treatment involves resuscitation, preventing encephalopathy, and procedures like sclerotherapy, banding, drugs,
This document provides information on portal hypertension, including its definition, causes, presentations, investigations, and management. Some key points:
- Portal hypertension is defined as a sustained elevation of portal pressure above 12 mm Hg. It can be caused by conditions affecting blood flow pre-sinusoidally (e.g. portal vein thrombosis), sinusoidally (e.g. cirrhosis), or post-sinusoidally (e.g. Budd-Chiari syndrome).
- Common presentations include esophageal varices, splenomegaly, ascites, and features of hepatic encephalopathy. Investigations include liver function tests, ultrasound, and endoscopy to identify varices.
-
Portal hypertension occurs when blood pressure within the portal venous system exceeds normal levels. It can be caused by prehepatic issues like portal vein obstruction, or intrinsic liver diseases that increase fibrosis within the liver. Children with portal hypertension may experience bleeding from esophageal varices, splenomegaly, ascites, or other clinical signs. Diagnosis involves medical history, lab tests, imaging like Doppler ultrasound or CT, and often measurement of portal pressure. Treatment depends on severity but may include medication, balloon tamponade, endoscopic procedures to band or sclerose varices, or shunt procedures like TIPS to decompress the portal system.
This document discusses gastrointestinal (GI) bleeding, including:
1) GI bleeding can present as overt (visible bleeding) or occult (hidden bleeding) and can originate from the upper or lower GI tract. Common symptoms include hematemesis, melena, and hematochezia.
2) Etiologies of upper GI bleeding include esophageal varices, peptic ulcers, Mallory-Weiss tears, and Dieulafoy's lesions. Management involves endoscopy, vasoactive drugs, proton pump inhibitors, and blood transfusions.
3) Lower GI bleeding can originate from the small intestine or colon. Causes include vascular ectasias, cancers, diverticulosis, and inflammatory bowel disease
Ascites refers to an accumulation of non-inflammatory fluid in the peritoneal cavity, characterized by a distended lower abdomen. It can be caused by decreased plasma protein levels, increased hydrostatic pressure, or liver or kidney damage. Clinically, affected animals have an enlarged, barrel-shaped abdomen with fluid waves visible on palpation. Diagnosis involves abdominal ultrasound or paracentesis to analyze fluid characteristics. Treatment focuses on resolving underlying causes, increasing plasma proteins, diuretics to promote fluid excretion, and draining excess fluid in severe cases.
This document discusses gastrointestinal bleeding in children. It defines different types of GI bleeding based on the location such as melena, hematochezia, and hematemesis. Common causes of upper and lower GI bleeding are listed for newborns, infants, children, and adolescents. Evaluation involves history, physical exam, lab tests, and imaging. Treatment depends on the severity and includes supportive care, medications, endoscopic procedures, and surgery. Portal hypertension is described as a cause of GI bleeding along with its pathophysiology, clinical manifestations, diagnosis, and management.
Cirrhosis is a chronic disease characterized by the replacement of normal liver tissue with scar tissue. This disrupts liver function and structure. Major complications of cirrhosis include ascites, portal hypertension, hepatic encephalopathy, and esophageal varices. Ascites is the accumulation of fluid in the abdominal cavity caused by portal hypertension and sodium retention. It is managed medically through dietary changes, diuretics, and paracentesis. Hepatic encephalopathy occurs when toxic metabolites accumulate in the bloodstream due to liver damage. Esophageal varices develop due to portal hypertension and can rupture, causing life-threatening bleeding.
lower GIT bleeding: is bleeding from a source distal to the ligament of Treitz (duodenojejunal junction), presented as
Hematochezia is blood passed with stool from the anus,
Melena is black, tarry stool produced by the oxidation of heme by intestinal flora; as little as 50 mL of blood may result in melena, and it may persist for 3 to 5 days following resolution of the bleed.
Maroon-colored stool is associated with rapidly bleeding small bowel lesions in which the transit of blood is too fast for complete oxidation.
Currant-jelly stool is associated with ischemic small bowel or proximal colonic lesions such as may be seen in intussusception.
Upper GIT bleeding: is bleeding from a source proximal to the ligament of Treitz (duodenojejunal junction).
Discussion included the definition of bleeding per rectum, it's types according to child age groups, it's presentation, how to diagnose each type and how to treat.
The pancreas develops from ventral and dorsal buds that fuse during gestation. It has both exocrine and endocrine functions. Acute pancreatitis is commonly caused by gallstones or alcohol and results from premature activation of pancreatic enzymes within the pancreas, leading to autodigestion. It can range from mild to severe, with severe cases involving hemorrhage and necrosis. Treatment is usually initially conservative but surgery may be needed for complications or failure to improve.
This document summarizes portal hypertension, including its causes, pathophysiology, clinical presentation, diagnosis, and treatment. Portal hypertension is defined as increased pressure in the portal vein greater than 10 mmHg. It can be caused by conditions that obstruct portal blood flow within or outside the liver. Increased pressure leads to formation of collateral veins and complications like variceal bleeding, splenomegaly, and ascites. Diagnosis involves identifying the underlying liver disease and assessing its severity. Treatment aims to prevent variceal bleeding through medication, band ligation, shunt procedures, or TIPS.
Hematemesis- vomiting of blood , a brief studymartinshaji
There can be many causes of hematemesis, such as: bleeding ulcers. prolonged and vigorous retching that causes tears in the esophageal mucosa (known as Mallory-Weiss Syndrome) gastric or intestinal varices.Haematemesis is simply defined as “vomiting blood”. It is caused by bleeding from part of the upper portion of the gastrointestinal tract. It has a wide range of possible causes, depending on the site of blood loss and the tissue that is actively bleeding. Hence it is necessary to analyse and treat the condition perfectly , this is brief study about all the aspects hematemesis ,vomiting of blood including etiology, definition,management ,treatment by drugs etc
please comment
thank u
Vomiting blood, also known as hematemesis, refers to vomiting of significant amounts of blood or contents mixed with blood. It can range from minor to very serious and requires determination of the underlying cause. Common causes include ulcers, gastritis, esophageal varices, cancer, and side effects of medications like NSAIDs. Diagnosis involves medical history, physical exam, imaging tests, endoscopy, and biopsy. Treatment depends on the cause but may include medications to stop bleeding, endoscopic procedures, surgery, blood transfusions, and lifestyle changes. Complications can include further bleeding, anemia, shock, and aspiration.
ascites-mu1.ppt case presentation of ascitespankajpatle8
Serum-ascites albumin gradient (SAAG) is a test used to determine the cause of ascites. It is calculated by subtracting the ascitic fluid albumin level from the serum albumin level.
A SAAG value helps differentiate between:
- Portal hypertension-related ascites (SAAG >1.1 g/dL) which is most commonly seen in liver cirrhosis.
- Non-portal hypertension related ascites (SAAG <1.1 g/dL) which can be caused by things like peritoneal carcinomatosis, tuberculosis, pancreatitis, nephrotic syndrome etc.
In simple terms, a SAAG value helps determine if the ascites is caused by portal
The document summarizes the key parts and functions of the digestive system. It describes the small intestine absorbing most nutrients and transporting waste to the large intestine. It then details the liver processing nutrients and filtering blood, before waste is expelled by the large intestine and rectum.
The document summarizes the key parts and functions of the digestive system. It describes the small intestine absorbing most nutrients and transporting waste to the large intestine. It then details the liver which filters blood and produces bile, and the gallbladder which stores and concentrates bile. Diseases like cirrhosis and conditions like jaundice are also mentioned.
How to Add Chatter in the odoo 17 ERP ModuleCeline George
In Odoo, the chatter is like a chat tool that helps you work together on records. You can leave notes and track things, making it easier to talk with your team and partners. Inside chatter, all communication history, activity, and changes will be displayed.
How to Manage Your Lost Opportunities in Odoo 17 CRMCeline George
Odoo 17 CRM allows us to track why we lose sales opportunities with "Lost Reasons." This helps analyze our sales process and identify areas for improvement. Here's how to configure lost reasons in Odoo 17 CRM
বাংলাদেশের অর্থনৈতিক সমীক্ষা ২০২৪ [Bangladesh Economic Review 2024 Bangla.pdf] কম্পিউটার , ট্যাব ও স্মার্ট ফোন ভার্সন সহ সম্পূর্ণ বাংলা ই-বুক বা pdf বই " সুচিপত্র ...বুকমার্ক মেনু 🔖 ও হাইপার লিংক মেনু 📝👆 যুক্ত ..
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তাই একজন নাগরিক হিসাবে এই তথ্য গুলো আপনার জানা প্রয়োজন ...।
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Macroeconomics- Movie Location
This will be used as part of your Personal Professional Portfolio once graded.
Objective:
Prepare a presentation or a paper using research, basic comparative analysis, data organization and application of economic information. You will make an informed assessment of an economic climate outside of the United States to accomplish an entertainment industry objective.
A review of the growth of the Israel Genealogy Research Association Database Collection for the last 12 months. Our collection is now passed the 3 million mark and still growing. See which archives have contributed the most. See the different types of records we have, and which years have had records added. You can also see what we have for the future.
ISO/IEC 27001, ISO/IEC 42001, and GDPR: Best Practices for Implementation and...PECB
Denis is a dynamic and results-driven Chief Information Officer (CIO) with a distinguished career spanning information systems analysis and technical project management. With a proven track record of spearheading the design and delivery of cutting-edge Information Management solutions, he has consistently elevated business operations, streamlined reporting functions, and maximized process efficiency.
Certified as an ISO/IEC 27001: Information Security Management Systems (ISMS) Lead Implementer, Data Protection Officer, and Cyber Risks Analyst, Denis brings a heightened focus on data security, privacy, and cyber resilience to every endeavor.
His expertise extends across a diverse spectrum of reporting, database, and web development applications, underpinned by an exceptional grasp of data storage and virtualization technologies. His proficiency in application testing, database administration, and data cleansing ensures seamless execution of complex projects.
What sets Denis apart is his comprehensive understanding of Business and Systems Analysis technologies, honed through involvement in all phases of the Software Development Lifecycle (SDLC). From meticulous requirements gathering to precise analysis, innovative design, rigorous development, thorough testing, and successful implementation, he has consistently delivered exceptional results.
Throughout his career, he has taken on multifaceted roles, from leading technical project management teams to owning solutions that drive operational excellence. His conscientious and proactive approach is unwavering, whether he is working independently or collaboratively within a team. His ability to connect with colleagues on a personal level underscores his commitment to fostering a harmonious and productive workplace environment.
Date: May 29, 2024
Tags: Information Security, ISO/IEC 27001, ISO/IEC 42001, Artificial Intelligence, GDPR
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Strategies for Effective Upskilling is a presentation by Chinwendu Peace in a Your Skill Boost Masterclass organisation by the Excellence Foundation for South Sudan on 08th and 09th June 2024 from 1 PM to 3 PM on each day.
This slide is special for master students (MIBS & MIFB) in UUM. Also useful for readers who are interested in the topic of contemporary Islamic banking.
25. The esophageal bleeding
The esophageal bleeding shows up the spontaneous throwing out of mouth of
scarlet blood. At her small volume it is accompanied by a belch, and the troop
landing of high-cube of blood is vomiting. The esophageal bleeding begins at night
or after the abundant eating, because the inflow of blood rises to the organs of
digestive tract.
26. Cause of esophageal bleeding
Peptic ulcers
inflammations of mucous membrane
one or a few diverticulum's (thrusting out of wall of gastric intestinal
highway)
Trauma
Varicose veins of the esophagus with portal hypertension
27. Symptoms of esophageal bleeding
This is a sudden vomiting, the presence of fresh
blood in the vomit masses of scarlet or maroon
color. After a while, the blood is released with
fecal masses, giving them a black color.
28. The first emergency medical assistance
in case of esophageal bleeding.
The victim should be laid on his side so that his legs are bent at the knees,
put a cold compress or an ice pack on the underlying area of the abdomen,
provide rest and do not allow to drink and eat.
As a first aid, and aminocaproic acid-5 % solution of 100 ml intravenously.
The patient must be urgently taken to the surgical Department of the
hospital.