2. DEFINITION
• Forward propulsion of body by the lower
limbs in a systematic,coordinated semi
rotatory movements of the trunk,arm and
head
• One gait cycle constitutes period from heel
strike of a leg to its next heel strike
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4. STANCE PHASE
• It begins at the instant that one extremity contacts the ground &
continues only as long as some portion of the foot is in contact
with the ground.
• It is approx 60% of normal gait duration.
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5. SWING PHASE
• It begins as soon as the toe of one extremity leaves the ground &
ceases just before heel strike or contact of the same extremity.
• It makes up 40% of normal gait cycle.
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6. DOUBLE SUPPORT
• During double support both the lower limbs are
in contact with the ground at the same time
• Lower limb of one side of body is beginning its
stance phase & the opposite side is ending its
stance phase.
• It accounts approx 11% of gait cycle.
• This phase is absent in running
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7. SUBDIVISION OF PHASES
Stance phase –
1) Heel strike
2) Foot flat
3) Mid-stance
4) Heel off
5) Toe off
Swing phase –
1) Acceleration
2) Mid-swing
3) Deceleration
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10. STANCE PHASE
• Heel strike phase:
• Begins with initial contact & ends with
foot flat
• It is beginning of the stance phase when
the heel contacts the ground.
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11. STANCE PHASE
• Foot flat:
• It occurs immediately following heel strike
• It is the point at which the foot fully
contacts the floor.
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12. STANCE PHASE
• Mid stance:
• It is the point at which the body passes
directly over the supporting extremity.
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13. STANCE PHASE
• Heel off:
• the point following midstance at which
time the heel of the reference extremity
leaves the ground.
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14. STANCE PHASE
• Toe off:
• The point following heel off when only the toe
of the reference extremity is in contact with
the ground.
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15. SWING PHASE
• Acceleration phase:
• It begins once the toe leaves the ground &
continues until mid-swing, or the point at which
the swinging extremity is directly under the body.
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16. • Mid-swing:
• It occurs approx when the extremity passes
directly beneath the body, or from the end of
acceleration to the beginning of deceleration.
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17. SWING PHASE
• Deceleration:
• It occurs after mid-swing when limb is
decelerating in preparation for heel
strike.
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20. VARIABLES OF GAIT
• There are two basic variables which provide a
basic description of human gait.
• Time/ Temporal variable & Distance variables.
• Provide essential quantitative information
about gait
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21. VARIABLES
• Temporal variable –
• Stance time
• Single-limb & double-
support time,
• Swing time,
• Stride and step time,
• Cadence and
• Speed
• Distance variable –
• Stride length,
• Step length and width
• Degree of toe-out
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22. • Stance time:
• It is the amount of time that elapses during the stance phase of
one extremity in a gait cycle.
• Single-support time:
• It is the amount of time that elapses during the period when only
one extremity is on the supporting surface in a gait cycle.
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23. • Double-support time:
• It is the amount of time spent with both feet on the ground
during one gait cycle.
• The % of time spent increased in elderly persons and in those
with balance disorders.
• The percentage of time spent decreases as the speed of walking
increases.
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24. • Stride length:
• It is the linear distance from the heel strike of
one lower limb to the next heel strike of the
same limb.
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25. • Step length:
• It is the linear distance from the heel strike of
one lower limb to the next heel strike of opposite
limb.
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26. • Stride duration:
• It refers to amount of time taken to accomplish one stride.
• Stride duration and gait cycle duration are synonymous.
• One stride, for a normal adult, lasts approx 1 sec
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27. • Step duration:
• It refers to the amount of time spent during a single step.
• Measurement usually is expressed as sec/step.
• When weakness or pain in limb, step duration may be decreased
on the affected side and increased on the unaffected side.
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28. • Cadence:
• It is the no of steps taken by a person per unit of time.
• It is measured as the no of steps / sec or per minute.
Cadence = Number of steps / Time
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29. • Walking velocity:
• It is the rate of linear forward motion of the
body, which can be measured in meters or
cm/second, meters/minute, or miles/hour.
Walking velocity (meters/sec)=Distance walked
(meters)/time (sec)
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30. • Speed of gait:
• It is referred to as slow, free, and fast.
• Free speed of gait refers to a person’s normal walking speed
• Slow & fast speeds of gait refer to speeds slower or faster than the
person’s normal comfortable walking speed, designated in a variety
of ways.
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31. • Step width or width of the walking base:
• It is the measure of linear distance between
the midpoint of the heel of one foot and the
same point on the other foot
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32. • Degree of toe-out (DTO):
• It represents the angle of foot formed by each
foot’s line of progression and a line intersecting
the centre of the heel and the second toe.
• The angle is about 70 from the line of
progression of each foot at free speed walking.
• The DTO decreases as the speed of walking
increases
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34. FACTORS AFFECTING
VARIABLES
• Age,
• Gender,
• Height,
• Size & shape of bony
components,
• Distribution of mass in body
segments,
• Joint mobility,
• Muscle strength,
• Type of clothing & footwear,
• Habit,
• Psychological status.
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35. PATH OF COG
• Point in a body at which entire mass
of body is assumed to be concentrated
• Center of Gravity (COG):
• Midway between the hips
• Few cm in front of s2
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36. • Vertical displacement:
• Rhythmic up & down movement
• Highest point: midstance
• Lowest point: double support
• Average displacement: 5cm
• Path: extremely smooth sinusoidal curve
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38. • Overall displacement:
• Sum of vertical & horizontal
displacement
• Figure ‘8’ movement of COG as
seen from AP view
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Horizontal
plane
Vertical
plane
39. SAUNDERS’ DETERMINANTS OF
GAIT
• Gait “determinants” was first described by “Saunders &
Coworkers” in 1953.
• Six optimizations used to minimize excursion of COG in
vertical & horizontal planes.
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40. • The “determinants” represent adjustments made by the pelvis,
hips, knees, and ankles that help to keep movt of the body’s
COG to a minimum.
• By decreasing the vertical & lateral excursions of the body’s
COG it was thought that energy expenditure would be less &
gait more efficient.
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41. PELVIC ROTATION:
Forward rotation of the pelvis in the
horizontal plane is approx. 8o on the swing-
phase side.
It reduces the angle of hip flexion & extension
It enables a slightly longer step-length
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42. PELVIC TILT:
5o dip of the swinging side (i.e.
hip abd)
In standing, this dip is a +ve
Trendelenberg sign
It reduces the height of the
apex of the curve of COG
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43. KNEE FLEXION IN STANCE
PHASE
Approx. 20o dip
It shortens the leg in the middle of stance phase
It reduces the height of the apex of COG curve
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44. ANKLE MECHANISM
It lengthens the leg at heel contact
It smoothens the curve of COG
It reduces the lowering of COG
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45. FOOT MECHANISM:
Lengthens the leg at toe-off as ankle
moves from dorsiflexion to plantarflexion
Smoothens the curve of COG
Reduces the lowering of COG
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46. LATERAL DISPLACEMENT
OF BODY
• Physiologic valgus of the knee reduce side-to-
side movement of the COG in frontal plane.
• The normally narrow width of the walking base
minimizes the lateral displacement of CG
Reduced muscular energy consumption due to
reduced lateral acceleration & deceleration
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48. MUSCLE ACTIVITY DURING
GAIT
• ECCENTRIC CONTRACTION
• Lengthening with contraction
• Commonest
• Antagonistic muscles dampen the action of agonistic muscles
• Act as shock absorbers
• Examples quadriceps and tibialis anterior at initial contact
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49. • CONCENTRIC CONTRACTION
• Muscle shortening with contraction
• Enables muscle to advance joint through space
• Examples gastrosoleus and iliopsoas which are primary
accelerators of gait
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50. • ISOCENTRIC CONTRACTION
• Muscle length remains same
• Hip abductors like gluteus medius which act as postural
stabilisers
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52. GAIT ANALYSIS
• Systematic study of a persons gait through visual observation
or with quantitative measurments
• 3 primary components measured in gait lab
• 1.Kinetics-analysis of forces
• 2.Dynamic EMG-determines muscle activity
• 3.Kinematics-analysis of movements
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53. PATHOLOGICAL GAITS
• Abnormality in gait may be caused
by –
• Pain
• Joint muscle range-of-motion (ROM) limitation
• Muscular weakness/paralysis
• Neurological involvement (UMNL/ LMNL)
• Leg length discrepancy
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54. TYPES OF PATHOLOGICAL
GAIT
• Due to pain –
• Antalgic or limping gait – (Psoatic Gait)
• Due to neurological disturbance –
• Muscular paralysis – both
• Spastic (Circumductory Gait, Scissoring Gait, Dragging
or Paralytic Gait, Robotic Gait[Quadriplegic]) and
• Flaccid (Lurching Gait, Waddling Gait, Gluteus Maximus
Gait, Quadriceps Gait, Foot Drop or High Stepping Gait)
• Cerebellar dysfunction (Ataxic Gait)
• Loss of kinesthetic sensation (Stamping Gait)
• Basal ganglia dysfunction (Festinant Gait)
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55. TYPES OF PATHOLOGICAL
GAIT
• Due to deformities –
• Equinus gait
• Equinovarus gait
• Calcaneal gait
• Knock & bow knee gait
• Genu recurvatum gait
• Due to Leg Length Discrepancy (LLD) –
• Equinus gait
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56. ANTALGIC GAIT
• This is a compensatory gait pattern adopted
in order to remove or diminish the
discomfort caused by pain in the LL or
pelvis.
• Characteristic feature:
• Decreased in duration of stance phase of the affected limb
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57. PSOATIC GAIT
• Psoas bursa may be inflamed & edematous,
which cause limitation of movement due to pain
& produce a atypical gait.
• Hip externally rotated
• Hip adducted
• Knee in slight flexion
• This process seems to relieve tension of the
muscle & hence relieve the inflamed structures.
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58. GLUTEUS MAXIMUS GAIT
• The gluteus maximus act as a
restraint for forward progression.
• In gluteus maximus paralysis
trunk quickly shifts posteriorly at
heel strike (initial contact).
• This will shift the body’s COG
posteriorly over the gluteus
maximus, moving the line of force
posterior to the hip joints.
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59. • This shifting is referred to as a
“Rocking Horse Gait” because of
the extreme backward-forward
movement of the trunk.
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60. GLUTEUS MEDIUS GAIT
• It is also known as “Trendelenberg
gait” or “Lurching Gait” when one
side affected.
• The individual shifts the trunk over
the affected side during stance phase.
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61. • Bilateral paralysis, waddling or duck gait.
• The patient lurch to both sides while
walking.
• The body sways from side to side on a wide
base with excessive shoulder swing.
• E.g. Muscular dystrophy,Bilateral CDH
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62. QUADRICEPS GAIT
• Quadriceps action is needed during heel
strike & foot flat when there is a flexion
movement acting at the knee.
• Quadriceps weakness/ paralysis will lead to
buckling of the knee during gait & thus loss
of balance.
• Patient can compensate this if he has normal
hip extensor & plantar flexors. 62
63. • Compensation:
• With quadriceps weakness, the individual may lean
forward over the quadriceps at the early part of stance
phase, as weight is being shifted on to the stance leg.
• Normally, the line of force falls behind the knee,
requiring quadriceps action to keep the knee from
buckling.
• By leaning forward at the hip, the COG is shifted
forward & the line of force now falls in front of the
knee.
• This will force the knee backward into extension. 63
64. • In addition, the person may
physically push on the anterior
thigh during stance phase,
holding the knee in extension
• Also known as hand to knee gait
or five finger quadriceps
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65. GENU RECURVATUM GAIT
• Hamstrings are weak, 2 things may
happen
• During stance phase, the knee will go into
excessive hyperextension, referred to as
“genu recurvatum” gait.
• During the deceleration (terminal swing)
part of swing phase, without the hamstrings
to slow down the swing forward of the lower
leg, the knee will snap into extension.
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66. HEMIPLEGIC GAIT
• With spastic pattern of hemiplegic
leg
• Hip into extension, adduction & medial
rotation
• Knee in extension, though often
unstable
• Ankle in drop foot with ankle plantar
flexion and inversion (equinovarus),
which is present during both stance and
swing phases.
• In order to clear the foot from the
ground the hip & knee should flex.
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67. • But the spastic muscles won’t allow the hip & knee
to flex for the floor clearance.
• So the patient hikes hip & bring the affected leg by
making a half circle i.e. circumducting the leg.
• Hence the gait is known as “Circumductory Gait”.
• Usually, there will be no reciprocal arm swing.
• Step length tends to be lengthened on the involved
side & shortened on the uninvolved side.
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68. SCISSORING GAIT
• It results from spasticity of bilateral adductor
muscle of hip.
• One leg crosses directly over the other with each
step like crossing the blades of a scissor.
• E.g. Cerebral Palsy
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69. DRAGGING OR PARAPLEGIC
GAIT
• There is spasticity of both hip & knee extensors & ankle
plantar flexors.
• In order to clear the ground the patient has to drag his both
lower limbs swings them & places them forward.
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70. CEREBELLAR ATAXIC OR
DRUNKARD’S GAIT
• Abnormal function of cerebellum result in a
disturbance of normal mechanism controlling
balance & therefore patient walks with wider
base.
• The wider base creates a larger side to side
deviation of COG.
• This result in irregularly swinging sideways to a
tendency to fall with each steps.
• Hence it is known as “Reeling Gait”.
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71. SENSORY ATAXIC GAIT
• This is a typical gait pattern seen in patients
affected by tabes dorsalis.
• It is a degenerative disease affecting the
posterior horn cells & posterior column of the
spinal cord.
• Because of lesion, the proprioceptive impulse
won’t reach the cerebellum.
• The patient will loss his joint sense & position
for his limb on space.
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72. • Because of loss of joint sense, the patient
abnormally raises his leg (high step) jerks it
forward to strike the ground with a stamp.
• So it is also called as “Stamping Gait”.
• The patient compensated this loss of joint
position sense by vision.
• So his head will be down while he is
walking.
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73. SHORT SHUFFLING OR
FESTINANT GAIT
• Normal function at basal ganglia are:
• Control of muscle tone
• Planning & programming of normal
movements.
• Control of associated movements like
reciprocal arm swing.
• Typical example for basal ganglia leision is
parkinsonism.
• Because of rigidity, all the joint will go for a
flexion position with spine stooping forward.
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74. • This posture displaces the COG anteriorly.
• So in order to keep the COG within the base of support,
the patient will take no of small shuffling steps.
• Due to loss of voluntary control over the movement, they
loses balance & walks faster as if he is chasing the COG.
• So it is called as “Festinant Gait”.
• Since his shuffling steps, it is otherwise called as
“Shuffling Gait”.
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75. FOOT DROP OR SLAPPING GAIT
• This is due to dorsiflexor weakness caused by paralysis of
common peroneal nerve.
• There won’t be normal heel strike, instead the foot comes in
contact with ground as a whole with a slapping sound.
• So it is also known as “Slapping gait”.
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76. • Due to plantarflexion of the ankle, there will be relatively
lengthening at the leading extremity.
• So to clear the ground the patient lift the limb too high.
• Hence the gait get s its another name i.e. “High Stepping
Gait”
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77. EQUINUS GAIT
• Equinus = Horse
• Because of paralysis of dorsiflexors which
results in plantar flexors contracture.
• The patients will walk on his toes (toe
walking).
• Other cause may be compensation by
plantar flexor for a short leg.
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78. UNEQUAL LEG LENGTH
• Leg length discrepancy (LLD) are divided into
–
• Minimal leg length discrepancy
• Moderate leg length discrepancy
• Severe leg length discrepancy
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79. MINIMAL LLD
• Shortening less than 1.5cms
• Compensation occurs by dropping the pelvis on the affected
side
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80. MODERATE LLD
• If shortening upto 5cms dropping the pelvis on the affected
side will no longer be effective.
• A longer leg is needed, so the person usually walks on the ball
of the foot on the involved (shorter) side.
• This is called an “Equinus Gait”.
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81. SEVERE LLD
• It is usually discrepancy of more than 5 cms.
• The person may compensate in a variety of
ways.
• Dropping the pelvis and walking in an
equinnus gait plus flexing the knee on the
uninvolved side is often used.
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82. EQUINOVARUS GAIT
• There will be ankle plantar flexion & subtalar inversion.
• So the patient will be walking on the outer border of the foot.
• E.g. CTEV
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83. CALCANEAL GAIT
• Result from paralysis plantar flexors causing
dorsiflexor contracture.
• The patient will be walking on his heel (heel
walking)
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84. KNOCK KNEE GAIT
• It is also known as genu valgum gait.
• Due to increased physiological valgus of
knee.
• Both the knee face each other widening the
base of support.
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85. BOW LEG GAIT
• It is also known as genu varum gait.
• Knee face outwards.
• Due to decreased physiological valgus of knee.
• The legs will be in a bowed position.
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86. IN-TOEING GAIT
• Results due to metatarsus adductus,tibial bowing with tibial
torsion or persistent femoral anteversion
• OUT-TOEING GAIT
• Normal out-toeing range is 0 to 30 degrees which get resolves
spontaneously
• When seen in association with lateral tibial torsion becomes
worse with growth
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