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CEREBRAL PALSY
PRESENTER :-
SHILPA JOSHI
• In 1860s, known as “cerebral palsy” or “little’s
disease” after an English surgeon William John Little
wrote the 1st medical description..
• Cerebral word is derived from Latin cerebrum : affected
part of brain and palsy from Greek para – beyond , lysis
– loosening i.e. lack of muscle control.
• Cerebral palsy is a motor function disorder :-
• Caused by permanent, non-progressive brain lesion.
• It can be present at birth or shortly thereafter.
• It is incurable, lifelong condition, damage doesn’t worsens and it may
be congenital or acquired.
• It is a heterogenous group of movement disorders . C.P. is an umbrella
term not a single diagnosis.
• In C.P. muscles are unaffected, brain is unable to send the appropriate
signals necessary to instruct muscles when to contract and relax.
• CAUSES :-
• 1. An insult or injury to the brain –
• Fixed, static lesion(s), in single or multiple areas of the motor centers of the brain.
• Early in CNS deviation
• 2. Developmental malformations –
• The brain fails to develop correctly.
• 3. Neurological damage –
• Can occur before , during or after delivery.
• Rh incompatibility, illness, severe lack of oxygen etc.
• 4. Severe deprivation of oxygen or blood flow to the brain –
• HIE (hypoxic-ischemic encephalopathy or intrapartal asphyxia)
• RISK FACTORS :-
• 1. Prenatal factors –
• Hemorrhage/bleeding –
• Abruptio placetna
• Infections –
• Rubella, cytomegalovirus, toxoplasmosis
• Environmental factors
• Maternal characteristics -
• Age
• Multiple births
• History of fetal deaths/ miscarriages
• Cigarette smoking >30 sticks per day
• Alcoholism and drug addiction
• Social status, mother with MR
• Mother’s medical condition.
• 2.Perinatal factors –
• High or low BP
• Umbilical cord coil
• Breech delivery
• Oversedation of drugs
• Trauma i.e. forceps or vacuum delivery
• Complications of birth
• 3.Postnatal causes –
• Trauma, head injury
• Infections
• Hypoxia
• Stroke in young age
• Tumor, cyst
• TYPES OF CEREBRAL PALSY –
• According to :
• 1.Neurological deficits
• 2.Types of movement involved
• 3.Areas of affected limbs
• 1.According to Neurological deficits –
• Based on the – extent and area of brain damage
• Each type involves the way a person moves.
• 3 main types are :-
• Pyramidal
• Originates from motor areas of the cerebral cortex
• Spastic CP
• Extrapyramidal
• Basal ganglia and cerebellum
• Athethoid CP
• Ataxic CP
• Mixed
• Spastic and athethoid CP
• SPASTIC CP :-
• Increased muscle tone, tense and contracted muscles.
• Have stiff jerky or awkward movements.
• Limbs are usually underdeveloped,
• Increased deep tendon reflexes.
• Most common from 70-80 % of all affected children.
• Types of spastic CP –
• According to affected limbs – plegia or paresis – meaning paralyzed or weak.
• Paraplegia
• Diplegia
• Hemiplegia
• Quadriplegia
• Monoplegia – one limb, extremely rare
• Triplegia – three limbs extremely rare.
• Diplegia / paraplegia –
• Both legs with slight involvement elsewhere both legs. May also have contracture of hips and knees and
talipes equinovarus ( clubfoot).
•
• Hemiplegia –
• Limbs only on one side affected,
• Hip and knee contractures
• Quadriplegia –
• Upper body usually normal or with very minor signs
• Child may develop contractures of ankles and feet.
• When the child walks his arms, head and even his mouth may twist strangely.
• Children with all 4 limbs affected often have such severe brain damage that they never are able to walk.
• Legs and feet turned inward.
• Spastic quadriplegia –
• Characteristic “scissors” position of limbs due to adductor spasm.
• Dyskinesia –
• Dyskinetic movement of mouth
• Grimacing, drooling and dysarthria.
• Adductor spasm.
• Ataxic CP –
• Poor balance and lack of coordination.
• Wide based gait, depth perception usually affected
• Tendency to fall and stumble.
• Inability to walk straight line.
• Least common 5-10% of cases
• Mixed CP –
• A common combination is spastic and athethoid
• Spastic muscle tone and involuntary movements.
• 25% 0f CP cases, fairly common.
• Degree of severity –
• 1.Mild CP -20% of cases
• Not require help for assisting their impaired ambulation capacity.
• 2.Moderate CP -50%
• Requires self help for assisting their impaired ambulation capacity.
• 3.Severe CP –
• Totally incapacitated and bedridden and they always need care from others.
• Sign and symptoms –
• Posture or balance problem’
• Loss of control or coordination
• Abnormal tone
• Abnormal strength
• Abnormal reflexes
• Persistent motor delay
• Cognitive deficit
• Associated handicaps.
• Early signs –
• Infancy (0-3 months ) –
• Stiff or floppy posture
• Excessive lethargy or irritability
• High pitched cry
• Poor head control
• Weak suck/ tongue thrust / tonic bite/ feeding difficulties
• Late infancy –
• Inability to perform motor skills as indicated :
• Control hand grasp by 3 months
• Rolling over by 5 months
• Independent sitting by 7 months
• Abnormal developmental patterns :-
• Hand preference by 12 months
• Excessive arching of back
• Log rolling
• Abnormal or prolonged parachute response.
• Abnormal developmental patterns after 1 year of age :-
• W Sitting – knees flexed, legs extremely rotated.
• Bottom shuffling – scoots along the floor
• Walking on tip toe or hopping
• Behavioural symptoms –
• Poor ability to concentrate
• Unusual tenseness, irritability.
• CP Main problem:-
• Mentation and thought processes are not always affected; child is trapped in their body with their disabilities ,
• Ability to express their intelligence may be limited by difficulty In communicating.
• Associated problems –
• Hearing and visual problems
• Sensory integration problems
• FTT (failure to thrive)
• Feeding problems
• Behavioural / emotional difficulties
• Communication disorders
• Bladder and bowel control problem
• GERD
• Skeletal deformities
• Mental retardation and learning disabilities in some
• Seizures/ epilepsy
• Diagnosis :-
• A useful diagnosis is when the specific type, affected limb , severity, and
cause , if known are identified.
• Physical evaluation, interview
• MRI, CT scan EEG
• Laboratory and radiologic work up
• Assessment tools
• i.e. Peabody development motor skills, Denver test 2 etc.
• the Peabody developmental motor scales :=
• in depth assessment 6 subtests include:
• Reflexes
• Stationary
• Locomotion
• Object manipulation
• Grasping
• Visual-motor integration
• This subtest yields a
• gross motor quotient,
• a fine motor quotient
• and a total motor quotient
• ages covered : from birth through five years of age.
• Denver test 2 :-
• Developmental screening test
• Covers 4 general functions –
• Personal social (smiling)
• Fine motor adaptive (eg. Grasping and drawing)
• Language (eg. Combining words)
• Gross motor (eg. Walking)
• Ages covered from birth to 6 years.
• Denver test 2 :-
• 1. subjective (interview)
• a. history taking :- includes all that may predispose an infant to brain damage or CP
• Risk factors
• Psychosocial factors
• Family adaptation
• b. child’s health history :- often admitted to hospital for corrective
surgeries and other complications.
• Respiratory status
• Motor functions
• Presence of fever
• Feeding and weight loss
• Any changes in physical state
• Medical regimen.
• 2. Objective :-
• Physical examination
• Criteria ---
• P osturing/ poor muscle control and strength
• O ropharyngeal problems
• S trabismus/ squint
• T one (hyper/ hypotonia)
• E volutional maldeveloment
• R eflexes (e.g. increased deep tendon )
• Abnormalities 4/6 strongly point to CP
• Oropharyngeal problems –
• Speech, swallowing, breathing, drooling and poor feeding.
• Appearance of strabismus (impaired side to side movement )
• Evolutional maldevelopment –
• Delay in motor skills , such as rolling over, sitting , crawling and walking.
• Size for age
• Persistence of primitive reflexes or parachute reflex fail to develop
• Asymmetric tonic neck reflex “fencing position” – head to one side, arm and leg on that side extended ,
opposite limbs flexed.
• Test for ataxia – hold a toy or finger in front of the child and ask him to touch it on the first try. The child
with ataxia cannot do it.
• Increased or exaggerated deep tendon reflex
• Treatment :-
• No treatment to cure CP.
• Brain damage cannot be corrected.
• Crucial for children with CP:
• Early identification
• Multidisciplinary care and
• Support
• 1. Nonphysical therapy
• A. General management
• - Proper nutrition and personal care
• B. Pharmacologic
• Botox, intrathecal, baclofen
• - Controls muscle spasm and seizures
• - Delivered directly to the spinal fluid to avoid brain effects.
• Glycopyrrolate – control drooling
• Pamidronate – may help with osteoporosis.
• C. Surgery :-
• - To loosen joints,
• - Relieve muscle tightness
• - Straightening of different twists or unusual curvatures of leg muscles
• - Improves the ability to sit , stand and walk.
• Selective posterior rhizotomy :-
• Is used to improve spasticity (muscle stiffness) in cerebral palsy. In some cases
nerves need to be served to decrease muscle tension of inappropriate contraction.
• Intensive rehabilitation is required after the surgery , usually up to 6 weeks, followed
by physical therapy on an ongoing basis.
• D. Physical aids :-
• Orthosis, braces and splints
• - Keeps limbs In correct alignment
• - Prevent deformities
• Positioning devices
• - Enable better postures
• Walkers, special scooters, wheel chairs
• - Makes it easier to move about.
• E. Special education
• - To meet the child’s special needs.
• - Improve learning
• - Vocational training can help prepare young adults for jobs.
• F.Rehabilitation services –
• Speech and occupational therapies may improve the ability to speak, and perform activities of daily living and to do some
suitable works to have their own income.
• G. Family services –
• Professional support helps a patient and family cope with cerebral palsy.
• Counsellors help parents learn how to modify behaviours.
• Caring for a child with CP can be very stressful .
• Some families find support groups help.
• H. Other treatments –
• Therapeutic electrical stimulation ,
• Acupuncture
• Hyperbaric therapy
• Massage therapy might help.
• 2. Physical therapy
• The ultimate long term goal is realistic independence. To get there we have to have some
short term goals . those being a working communication system, education to his potential,
computer skills and , above all, friends.
• A. Sitting
• - Vertical head control and control of head and trunk.
• B. Standing and walking
• - Establish an equal distribution of weight on each foot, train to use steps or inclines.
• C. Prone development
• D. Supine development
• - Head control on supine and positions.

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CP PPT.pptx

  • 2. • In 1860s, known as “cerebral palsy” or “little’s disease” after an English surgeon William John Little wrote the 1st medical description.. • Cerebral word is derived from Latin cerebrum : affected part of brain and palsy from Greek para – beyond , lysis – loosening i.e. lack of muscle control.
  • 3. • Cerebral palsy is a motor function disorder :- • Caused by permanent, non-progressive brain lesion. • It can be present at birth or shortly thereafter. • It is incurable, lifelong condition, damage doesn’t worsens and it may be congenital or acquired. • It is a heterogenous group of movement disorders . C.P. is an umbrella term not a single diagnosis. • In C.P. muscles are unaffected, brain is unable to send the appropriate signals necessary to instruct muscles when to contract and relax.
  • 4.
  • 5.
  • 6. • CAUSES :- • 1. An insult or injury to the brain – • Fixed, static lesion(s), in single or multiple areas of the motor centers of the brain. • Early in CNS deviation • 2. Developmental malformations – • The brain fails to develop correctly. • 3. Neurological damage – • Can occur before , during or after delivery. • Rh incompatibility, illness, severe lack of oxygen etc. • 4. Severe deprivation of oxygen or blood flow to the brain – • HIE (hypoxic-ischemic encephalopathy or intrapartal asphyxia)
  • 7.
  • 8. • RISK FACTORS :- • 1. Prenatal factors – • Hemorrhage/bleeding – • Abruptio placetna • Infections – • Rubella, cytomegalovirus, toxoplasmosis • Environmental factors • Maternal characteristics - • Age • Multiple births • History of fetal deaths/ miscarriages • Cigarette smoking >30 sticks per day • Alcoholism and drug addiction • Social status, mother with MR • Mother’s medical condition.
  • 9. • 2.Perinatal factors – • High or low BP • Umbilical cord coil • Breech delivery • Oversedation of drugs • Trauma i.e. forceps or vacuum delivery • Complications of birth • 3.Postnatal causes – • Trauma, head injury • Infections • Hypoxia • Stroke in young age • Tumor, cyst
  • 10. • TYPES OF CEREBRAL PALSY – • According to : • 1.Neurological deficits • 2.Types of movement involved • 3.Areas of affected limbs • 1.According to Neurological deficits – • Based on the – extent and area of brain damage • Each type involves the way a person moves. • 3 main types are :- • Pyramidal • Originates from motor areas of the cerebral cortex • Spastic CP
  • 11.
  • 12. • Extrapyramidal • Basal ganglia and cerebellum • Athethoid CP • Ataxic CP • Mixed • Spastic and athethoid CP
  • 13. • SPASTIC CP :- • Increased muscle tone, tense and contracted muscles. • Have stiff jerky or awkward movements. • Limbs are usually underdeveloped, • Increased deep tendon reflexes. • Most common from 70-80 % of all affected children. • Types of spastic CP – • According to affected limbs – plegia or paresis – meaning paralyzed or weak. • Paraplegia • Diplegia • Hemiplegia • Quadriplegia • Monoplegia – one limb, extremely rare • Triplegia – three limbs extremely rare.
  • 14. • Diplegia / paraplegia – • Both legs with slight involvement elsewhere both legs. May also have contracture of hips and knees and talipes equinovarus ( clubfoot). • • Hemiplegia – • Limbs only on one side affected, • Hip and knee contractures • Quadriplegia – • Upper body usually normal or with very minor signs • Child may develop contractures of ankles and feet. • When the child walks his arms, head and even his mouth may twist strangely. • Children with all 4 limbs affected often have such severe brain damage that they never are able to walk. • Legs and feet turned inward.
  • 15.
  • 16. • Spastic quadriplegia – • Characteristic “scissors” position of limbs due to adductor spasm. • Dyskinesia – • Dyskinetic movement of mouth • Grimacing, drooling and dysarthria. • Adductor spasm. • Ataxic CP – • Poor balance and lack of coordination. • Wide based gait, depth perception usually affected • Tendency to fall and stumble. • Inability to walk straight line. • Least common 5-10% of cases
  • 17.
  • 18.
  • 19. • Mixed CP – • A common combination is spastic and athethoid • Spastic muscle tone and involuntary movements. • 25% 0f CP cases, fairly common. • Degree of severity – • 1.Mild CP -20% of cases • Not require help for assisting their impaired ambulation capacity. • 2.Moderate CP -50% • Requires self help for assisting their impaired ambulation capacity. • 3.Severe CP – • Totally incapacitated and bedridden and they always need care from others.
  • 20. • Sign and symptoms – • Posture or balance problem’ • Loss of control or coordination • Abnormal tone • Abnormal strength • Abnormal reflexes • Persistent motor delay • Cognitive deficit • Associated handicaps.
  • 21.
  • 22. • Early signs – • Infancy (0-3 months ) – • Stiff or floppy posture • Excessive lethargy or irritability • High pitched cry • Poor head control • Weak suck/ tongue thrust / tonic bite/ feeding difficulties • Late infancy – • Inability to perform motor skills as indicated : • Control hand grasp by 3 months • Rolling over by 5 months • Independent sitting by 7 months
  • 23. • Abnormal developmental patterns :- • Hand preference by 12 months • Excessive arching of back • Log rolling • Abnormal or prolonged parachute response. • Abnormal developmental patterns after 1 year of age :- • W Sitting – knees flexed, legs extremely rotated. • Bottom shuffling – scoots along the floor • Walking on tip toe or hopping • Behavioural symptoms – • Poor ability to concentrate • Unusual tenseness, irritability.
  • 24. • CP Main problem:- • Mentation and thought processes are not always affected; child is trapped in their body with their disabilities , • Ability to express their intelligence may be limited by difficulty In communicating. • Associated problems – • Hearing and visual problems • Sensory integration problems • FTT (failure to thrive) • Feeding problems • Behavioural / emotional difficulties • Communication disorders • Bladder and bowel control problem • GERD • Skeletal deformities • Mental retardation and learning disabilities in some • Seizures/ epilepsy
  • 25. • Diagnosis :- • A useful diagnosis is when the specific type, affected limb , severity, and cause , if known are identified. • Physical evaluation, interview • MRI, CT scan EEG • Laboratory and radiologic work up • Assessment tools • i.e. Peabody development motor skills, Denver test 2 etc.
  • 26. • the Peabody developmental motor scales := • in depth assessment 6 subtests include: • Reflexes • Stationary • Locomotion • Object manipulation • Grasping • Visual-motor integration • This subtest yields a • gross motor quotient, • a fine motor quotient • and a total motor quotient • ages covered : from birth through five years of age.
  • 27. • Denver test 2 :- • Developmental screening test • Covers 4 general functions – • Personal social (smiling) • Fine motor adaptive (eg. Grasping and drawing) • Language (eg. Combining words) • Gross motor (eg. Walking) • Ages covered from birth to 6 years. • Denver test 2 :- • 1. subjective (interview) • a. history taking :- includes all that may predispose an infant to brain damage or CP • Risk factors • Psychosocial factors • Family adaptation
  • 28. • b. child’s health history :- often admitted to hospital for corrective surgeries and other complications. • Respiratory status • Motor functions • Presence of fever • Feeding and weight loss • Any changes in physical state • Medical regimen.
  • 29. • 2. Objective :- • Physical examination • Criteria --- • P osturing/ poor muscle control and strength • O ropharyngeal problems • S trabismus/ squint • T one (hyper/ hypotonia) • E volutional maldeveloment • R eflexes (e.g. increased deep tendon ) • Abnormalities 4/6 strongly point to CP
  • 30.
  • 31. • Oropharyngeal problems – • Speech, swallowing, breathing, drooling and poor feeding. • Appearance of strabismus (impaired side to side movement ) • Evolutional maldevelopment – • Delay in motor skills , such as rolling over, sitting , crawling and walking. • Size for age • Persistence of primitive reflexes or parachute reflex fail to develop • Asymmetric tonic neck reflex “fencing position” – head to one side, arm and leg on that side extended , opposite limbs flexed. • Test for ataxia – hold a toy or finger in front of the child and ask him to touch it on the first try. The child with ataxia cannot do it. • Increased or exaggerated deep tendon reflex
  • 32.
  • 33. • Treatment :- • No treatment to cure CP. • Brain damage cannot be corrected. • Crucial for children with CP: • Early identification • Multidisciplinary care and • Support • 1. Nonphysical therapy • A. General management • - Proper nutrition and personal care • B. Pharmacologic • Botox, intrathecal, baclofen • - Controls muscle spasm and seizures • - Delivered directly to the spinal fluid to avoid brain effects. • Glycopyrrolate – control drooling • Pamidronate – may help with osteoporosis.
  • 34. • C. Surgery :- • - To loosen joints, • - Relieve muscle tightness • - Straightening of different twists or unusual curvatures of leg muscles • - Improves the ability to sit , stand and walk. • Selective posterior rhizotomy :- • Is used to improve spasticity (muscle stiffness) in cerebral palsy. In some cases nerves need to be served to decrease muscle tension of inappropriate contraction. • Intensive rehabilitation is required after the surgery , usually up to 6 weeks, followed by physical therapy on an ongoing basis.
  • 35. • D. Physical aids :- • Orthosis, braces and splints • - Keeps limbs In correct alignment • - Prevent deformities • Positioning devices • - Enable better postures • Walkers, special scooters, wheel chairs • - Makes it easier to move about. • E. Special education • - To meet the child’s special needs. • - Improve learning • - Vocational training can help prepare young adults for jobs.
  • 36. • F.Rehabilitation services – • Speech and occupational therapies may improve the ability to speak, and perform activities of daily living and to do some suitable works to have their own income. • G. Family services – • Professional support helps a patient and family cope with cerebral palsy. • Counsellors help parents learn how to modify behaviours. • Caring for a child with CP can be very stressful . • Some families find support groups help. • H. Other treatments – • Therapeutic electrical stimulation , • Acupuncture • Hyperbaric therapy • Massage therapy might help.
  • 37. • 2. Physical therapy • The ultimate long term goal is realistic independence. To get there we have to have some short term goals . those being a working communication system, education to his potential, computer skills and , above all, friends. • A. Sitting • - Vertical head control and control of head and trunk. • B. Standing and walking • - Establish an equal distribution of weight on each foot, train to use steps or inclines. • C. Prone development • D. Supine development • - Head control on supine and positions.