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SUBJECT- MEDICAL SURGICAL NURSING
TOPIC- SHOCK
-MR. MIGRON RUBIN
INTRODUCTION
Cells need two things to function: oxygen and glucose. This allows the
cells to generate energy and do their specific jobs. When cells don’t
receive either of them or both, they stop functioning.
DEFINITION
Shock is defined as a condition where the tissues in the body don't receive
enough oxygen and nutrients to allow the cells to function.
CLASSIFICATION
1. Cardiogenic shock- It occurs due to systolic or diastolic dysfunction.
2. Hypovolemic shock- It occurs due to intravascular fluid volume.
3. Obstructive shock- It occurs when there is physical obstruction in blood flow.
4. Distributive shock- (neurogenic, anaphylactic & septic)
•Neurogenic shock- It occurs from trauma that leads
to spinal cord injuries.
•Anaphylactic shock- It is acute life threatening
hypersensitivity reaction to a sensitizing
substance like drug, chemical, vaccine, food etc.
• Septic shock-Also known as blood poisoning,
is a condition caused by infections that lead to bacteria
entering blood.
ETIOLOGY
• Severe allergic reaction
• Significant blood loss
• Heart failure
• Blood infections
• Dehydration
• Poisoning
• Burns
PATHOPHYSIOLOGY
CARDIOGENIC SHOCK
STRUCTURAL DEFECTS IN HEART, DYSRHYTHMIAS ETC.
SYSTOLIC & DIASTOLIC DYSFUNCTION
DECREASED CARDIAC OUTPUT & INCREASED PULMONARY PRESSURE
PULMONARY EDEMA
DECREASED CELLULAR OXYGEN SUPPLY
DECREASED TISSUE PERFUSION
HYPOVOLEMIC SHOCK
DECREASED BLOOD VOLUME DUE TO ACCIDENT, BURN ETC.
DECREASED VENOUS RETURN
DECREASED CARDIAC OUTPUT
DECREASED TISSUE PERFUSION
DECREASED CELLULAR METABOLISM
NEUROGENIC SHOCK
DISRUPTION OF SYMPATHETIC NERVOUS SYSTEM
VASODILATION
DECREASED BP
DECREASED CARDIAC OUTPUT
DECREASED CELLULAR OXYGEN SUPPLY
DECREASED TISSUE PERFUSION
IMPAIRED CELLULAR METABOLISM
ANAPHYLACTIC SHOCK
ALLERGEN , DRUG ETC.
ANTIGEN ANTIBODY REACTION
VASODILATION
CAPILLARY PERMEABILITY
SEVERE BRONCHO CONSTRICTION
DECREASED OXYGEN SUPPLYAND UTILIZATION
INADEQUATE TISSUE PERFUSION
SEPTIC SHOCK
INFECTION
RELEASE OFTOXIN
PERIPHERALV
ASCULAR EFFECTS MYOCARDIALPROBLEMS
ENDOTHELIALDESTRUCTION DECREASED CONTRACTILITY
INADEQUATE BLOOD FLOW
MICROVASCULAR INSUFFICIENCY
TO TISSUE
INADEQUATE BLOOD FLOW TO THE TISSUE TISSUE HYPOXIA
CELLDEATH
OBSTRUCTIVE SHOCK
PHYSICALOBSTRUCTION IN BLOOD FLOW
DECREASED VENOUS RETURN
DECREASED CARDIAC OUTPUT
DECREASED CELLULAR OXYGEN SUPPLY
DECREASED TISSUE PERFUSION
IMPAIRED CELLULAR METABOLISM
CLINICAL MANIFESTATIONS
• Extremely low blood pressure
• Weakness
• Chest pain
• Weak pulse
• Profuse sweating
• Dizziness
• Moist, clammy skin
• Unconsciousness
• Rapid, shallow breathing
• Feeling anxious, agitated or confused
• Cyanosis
DIAGNOSTIC EVALUATION
• History collection
• Physical examination
• Blood culture & sensitivity test
• CBC- increased WBC & ESR level
• Arterial blood gas analysis- respiratory alkalosis
• ECG-dysarrthmias
• Echocardiogram-to rule out aortic stenosis and pulmonary embolism.
• X-ray & CT scan
• Cardiac monitoring-Spo2,pulse,temp,BPare monitored continuously.
• Central venous pressure -fluid loss.
COMPLICATIONS
• Loss of consciousness
• Respiratory failure
• Coagulation disorder
• Multi organ damage
• Coma
• Death
MANAGEMENT
I. MEDICALMANAGEMENT
A. PHARMACOLOGICAL MANAGEMENT
• Crystalloids: ringer’s solution and normal saline
• Inotropic agents: like dopamine , dobutamine and epinephrine
• Vasodilators : nitroglycerine
• Diuretics : lasilactone, furosemide
• Antibiotics : ciprofloxacin, amoxicillin and clavulanic acid
• Antihistamines : epinephrine used in anaphylactic shock.
• Corticosteroids : dexamethasone
• Sodium bicarbonate :used to treat metabolic acidosis
• Broncodilators : like atropine , aminophylline etc.
• B. NON- PHARMACOLOGICAL MANAGEMENT
• Modified trendelenberg position
• Assessment of vital signs
• Oxygen administration
• Parenteral nutrition support
II. SURGICAL MANAGEMENT
• Wound debridement- in case of chronic infected wound, burns wound
debridement to be done for fast healing
• Angioplasty-in case of myocardial infarction angioplasty can be
performed
• Tracheostomy
III. NURSING MANAGEMENT
ASSESSMENT
• Continuous monitoring of vital signs should be done.
• AssessAirway, breathing & circulation of the patient.
• Monitor forABG value
• Check for urine output of the client.
•
• NURSING DIAGNOSIS
• Impaired tissue perfusion related to decrease cardiac output, decreased
venous return
• In effective breathing pattern related to hypoxia, bronchospasm
• Fluid volume deficit related to vomiting hemorrhage
• Acute pain related to myocardial infarction
• Imbalanced nutrition less then body requirement related to vomiting,
low intake of food
Medical Surgical Nursing: Types and Treatment of Shock

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Medical Surgical Nursing: Types and Treatment of Shock

  • 1. SUBJECT- MEDICAL SURGICAL NURSING TOPIC- SHOCK -MR. MIGRON RUBIN
  • 2. INTRODUCTION Cells need two things to function: oxygen and glucose. This allows the cells to generate energy and do their specific jobs. When cells don’t receive either of them or both, they stop functioning.
  • 3. DEFINITION Shock is defined as a condition where the tissues in the body don't receive enough oxygen and nutrients to allow the cells to function.
  • 4. CLASSIFICATION 1. Cardiogenic shock- It occurs due to systolic or diastolic dysfunction. 2. Hypovolemic shock- It occurs due to intravascular fluid volume. 3. Obstructive shock- It occurs when there is physical obstruction in blood flow.
  • 5. 4. Distributive shock- (neurogenic, anaphylactic & septic) •Neurogenic shock- It occurs from trauma that leads to spinal cord injuries. •Anaphylactic shock- It is acute life threatening hypersensitivity reaction to a sensitizing substance like drug, chemical, vaccine, food etc. • Septic shock-Also known as blood poisoning, is a condition caused by infections that lead to bacteria entering blood.
  • 6. ETIOLOGY • Severe allergic reaction • Significant blood loss • Heart failure • Blood infections • Dehydration • Poisoning • Burns
  • 7. PATHOPHYSIOLOGY CARDIOGENIC SHOCK STRUCTURAL DEFECTS IN HEART, DYSRHYTHMIAS ETC. SYSTOLIC & DIASTOLIC DYSFUNCTION DECREASED CARDIAC OUTPUT & INCREASED PULMONARY PRESSURE PULMONARY EDEMA DECREASED CELLULAR OXYGEN SUPPLY DECREASED TISSUE PERFUSION
  • 8. HYPOVOLEMIC SHOCK DECREASED BLOOD VOLUME DUE TO ACCIDENT, BURN ETC. DECREASED VENOUS RETURN DECREASED CARDIAC OUTPUT DECREASED TISSUE PERFUSION DECREASED CELLULAR METABOLISM
  • 9. NEUROGENIC SHOCK DISRUPTION OF SYMPATHETIC NERVOUS SYSTEM VASODILATION DECREASED BP DECREASED CARDIAC OUTPUT DECREASED CELLULAR OXYGEN SUPPLY DECREASED TISSUE PERFUSION IMPAIRED CELLULAR METABOLISM
  • 10. ANAPHYLACTIC SHOCK ALLERGEN , DRUG ETC. ANTIGEN ANTIBODY REACTION VASODILATION CAPILLARY PERMEABILITY SEVERE BRONCHO CONSTRICTION DECREASED OXYGEN SUPPLYAND UTILIZATION INADEQUATE TISSUE PERFUSION
  • 11. SEPTIC SHOCK INFECTION RELEASE OFTOXIN PERIPHERALV ASCULAR EFFECTS MYOCARDIALPROBLEMS ENDOTHELIALDESTRUCTION DECREASED CONTRACTILITY INADEQUATE BLOOD FLOW MICROVASCULAR INSUFFICIENCY TO TISSUE INADEQUATE BLOOD FLOW TO THE TISSUE TISSUE HYPOXIA CELLDEATH
  • 12. OBSTRUCTIVE SHOCK PHYSICALOBSTRUCTION IN BLOOD FLOW DECREASED VENOUS RETURN DECREASED CARDIAC OUTPUT DECREASED CELLULAR OXYGEN SUPPLY DECREASED TISSUE PERFUSION IMPAIRED CELLULAR METABOLISM
  • 13. CLINICAL MANIFESTATIONS • Extremely low blood pressure • Weakness • Chest pain • Weak pulse • Profuse sweating • Dizziness
  • 14. • Moist, clammy skin • Unconsciousness • Rapid, shallow breathing • Feeling anxious, agitated or confused • Cyanosis
  • 15. DIAGNOSTIC EVALUATION • History collection • Physical examination • Blood culture & sensitivity test • CBC- increased WBC & ESR level • Arterial blood gas analysis- respiratory alkalosis
  • 16. • ECG-dysarrthmias • Echocardiogram-to rule out aortic stenosis and pulmonary embolism. • X-ray & CT scan • Cardiac monitoring-Spo2,pulse,temp,BPare monitored continuously. • Central venous pressure -fluid loss.
  • 17. COMPLICATIONS • Loss of consciousness • Respiratory failure • Coagulation disorder • Multi organ damage • Coma • Death
  • 18. MANAGEMENT I. MEDICALMANAGEMENT A. PHARMACOLOGICAL MANAGEMENT • Crystalloids: ringer’s solution and normal saline • Inotropic agents: like dopamine , dobutamine and epinephrine • Vasodilators : nitroglycerine • Diuretics : lasilactone, furosemide • Antibiotics : ciprofloxacin, amoxicillin and clavulanic acid • Antihistamines : epinephrine used in anaphylactic shock. • Corticosteroids : dexamethasone • Sodium bicarbonate :used to treat metabolic acidosis • Broncodilators : like atropine , aminophylline etc.
  • 19. • B. NON- PHARMACOLOGICAL MANAGEMENT • Modified trendelenberg position • Assessment of vital signs • Oxygen administration • Parenteral nutrition support
  • 20. II. SURGICAL MANAGEMENT • Wound debridement- in case of chronic infected wound, burns wound debridement to be done for fast healing • Angioplasty-in case of myocardial infarction angioplasty can be performed • Tracheostomy
  • 21. III. NURSING MANAGEMENT ASSESSMENT • Continuous monitoring of vital signs should be done. • AssessAirway, breathing & circulation of the patient. • Monitor forABG value • Check for urine output of the client. •
  • 22. • NURSING DIAGNOSIS • Impaired tissue perfusion related to decrease cardiac output, decreased venous return • In effective breathing pattern related to hypoxia, bronchospasm • Fluid volume deficit related to vomiting hemorrhage • Acute pain related to myocardial infarction • Imbalanced nutrition less then body requirement related to vomiting, low intake of food