Allopathic Non-surgical HairGrowth Treatment - Affordable for Everyone.

An awareness campaign by “Hair Crown” on various hair problems
and their solutions .
Dispelling false notions & concepts surrounding hair problems.
“SAVE YOUR HAIR CROWN”
Consult Dr. K.Nikhil 9225020002

Structure of Hair

Blood supply as only source of nutrition to the hair

Hair oil application stops hair fall Hairfall is result of hormonal causes
genetics & vitamins or iron deficiency which is the function of blood
supply. Hair oil has no role since external application does not
penetrate the blood.
Common misconceptions

Androgenic alopecia is a result of
Genetics
DHT
Stress
Hence No Herbal application can stop Alopecia.
Common misconceptions
Baldness or androgenic alopecia can be reversed by onion
juice/ Herbal application

grey hair is the result of failure of melanin synthesis –the black pigment
which gives colour to hair & skin.
Misconception
Grey hair is the result of vitamin deficiency

DHT & Androgenetic alopecia

HAIR TYPES
Fetal hair -
Lanugo hair : soft, fine, lightly pigmented hairs.
Adult hair -
Vellus hair : fine hairs cover most of the body
of youngsters and adults.
Terminal hair: long, coarse, pigmented hairs with
larger diameters.

NUMBER OF HAIRS
Scalp : about 1,00,000 hairs.
Face : about 600 hairs /cm2.
Rest of the body : about 60 hairs/cm2
.

ANAGEN (GROWING PHASE)
 Last for about 1000 days.
 Follicular cells grow, divide and become keratinized
to form growing phase.
 A darkly pigmented portion is evident just above the
hair bulb.

TELOGEN (RESTING PHASE)
Lasts for about 100 days.
Club-shaped proximal end shed from the
follicle during telogen or subsequent anagen.
Growth of a new anagen hair leads to
shedding of any remaining telogen hair.
But new hair does not “push out” the hair from
the previous cycle.

CATAGEN (INVOLUTING PHASE)
 Lasts for about 10 days.
 Scalp hairs show a gradual thinning and decrease of the
pigment.
 Melanocytes cease producing melanin.
 Matrix keratinocytes abruptly cease proliferating so that
lower follicle involutes and regresses.

EXOGEN (HAIR SHEDDING PHASE)
Recently added phase.
The term describes relationship between hair
shaft and base of telogen follicle.
Hairs can be retained for more than one
cycle.
Shedding phase is most likely independent of
anagen and telogen.

Hair Growth Cycle

Hair Loss
Natural shedding of hair accounts for normal daily
hair loss. Recent measurements indicate that the
average rate of hair loss is closer to 35 to 40 hairs
per day

Emotional impact
• How perceived by others
– Recent study showed that compared to men
who had hair, bald men were PERCEIVED as
having a number of characteristics
• Less physically attractive – by both sexes
• Less assertive
• Less successful
• Less personably likable
• Older
– By about 5 years

Emotional impact
• Men with severe hair loss
– Negative social and emotional effects
– More preoccupation with baldness
– Make effort to conceal or compensate for hair loss
• Women with hair loss
– Devastating- women try to hide it from everyone
including their doctor
– Causes anxiety – women feel helpless and less
attractive

Diffuse Hair Loss 
A. Abnormality of cycling –
i. Alopecia areata.
ii. Telogen effluvium.
iii. Anagen effluvium.
iv. Loose anagen syndrome.
B. Hair shaft abnormality-
i. Hair breakage.
ii. Unruly hair.

TELOGEN EFFLUVIUM
It is a reaction pattern to a variety of
physical and mental stressors represents
a precipitous shift of a percentage of
anagen hairs to telogen.

Causes of Telogen Effluvium
• Endocrine
- Hypo- or hyperthyroidism.
- Postpartum.
- Peri- or postmenopausal state.
• Nutritional
- Biotin deficiency.
- Caloric deprivation.
- Essential fatty acid deficiency.
- Iron deficiency.
- Protein deprivation.
- Zinc deficiency.

 Drugs
- Angiotensin-converting enzyme inhibitors.
- Anticoagulants.
- Antimitotic agents.
- Benzimidazoles.
- Beta blockers.
- Interferon
- Lithium

- Oral contraceptives.
- Retinoids.
- Vitamin A excess.
Physical stress
- Anemia
- Surgery.
- Systemic illness.
Psychological stress

Alopecia
• Abnormal hair loss
• Most common types
– Androgenic alopecia
– Alopecia areata
– Postpartum alopecia
– Over 63 million people in our country suffer
from abnormal hair loss

Androgenetic Alopecia
• Can begin as early as teens
• Frequently seen by age 40
• By age 35, almost 40 % of men & women
show some degree of loss
• Gene can be inherited from either side of
family

ANDROGENETIC ALOPECIA
Age : Twenties or early thirties.
sites : Chiefly vertex and frontotemporal regions.
Etiopathogenesis:
• Exact mechanism is still unknown.
• Hereditary (Probably autosomal dominant) &
• Androgen (specifically dihydrotestesterone)

Alopecia Areata
• Sudden loss of hair in round or irregular patches;
may occur on scalp or anywhere else on body. It’s
highly unpredictable.
• Immune system attacks hair follicles
• Begins with one or more small, bald patches.

Alopecia Areata
• Occurs in males and females of all ages and
races
– Can begin in childhood
• Scalp shows no sign of inflammation
• No obvious signs of skin disorder or disease

Postpartum Alopecia
• Temporary hair loss at conclusion of
pregnancy
• Growth cycle generally returns to normal
within one year after the baby is delivered

Androgenetic alopecia in women
Etiology :
i. Genetic Predisposition,
ii. Androgen excess,
Ovarian cause-
- Polycystic ovarian syndrome,
- Other ovarian tumor

ETIOPATHOGENESIS
EFFECTS
- Shortening of anagen and
lengthening of telogen
- Follicle become short and sclerosis of dermis
- and miniaturization or reduction of hair present.

CLINICAL FEATURE
Other evidence of androgen excess:
• Acne.
• Hirsutism.
• Menstrual irregularities.
Majority of women with pattern hair loss have
• No increased serum androgen,
• No other sign symptom of
androgen hypersensitivity.

ANDROGENETIC ALOPECIA IN WOMEN

PIGMENTATION OF HAIR
 Hair color is determined by melanocytes.
 Melanocytes are present in the bulb.
 Melanocytes feed melanosomes mainly to
the medulla and cortex.
 Melanocytic follicles produce melanin-
. eumelanin (dominant in brown-black hairs)
. phaeomelanin (dominant in red-blond hairs)

 Greying of hair – due to decreased
number and activities of melanocytes.
 Vitiligo – due to destruction of
melanocytes.
 Albinism – due to inactivity of
melanocytes.
PIGMENTATION OF HAIR

•Hair &Diagnostic tests recommended to as certain
exact cause of problems
•Treatment modality chalked out according to
the cause of hai problem
•Treatment includes Topical &subdermal medicinal application.
Oral medicines.
• scalp examined by Dermoscope
Approach to treatment

•Complete stoppage of all types of hairfall in 15 day
•Complete coverage of visible scalp without hair transplant
(non-surgical approach).
What to expect from treatment ?

Allopathic Non-surgical HairGrowth Treatment - Affordable for Everyone.

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