Presentation insulin resistance modifies the association between obesity and current asthma in adul

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Abstract of my paper collaboration "Insulin Resistance Modifies the Association Between Obesity and Current Asthma in Adults" presented at the annual meeting of the American Academy of Allergy, Asthma and Immunology, Los Angeles, California, USA, 2016.

Health & Medicine

Insulin Resistance Modifies the Association Between Obesity and Current Asthma
in Adults
Saturday, March 5, 2016
South Exhibit Hall H (Convention Center)
Samuel Y Ash, MD, Juan Carlos Cardet, MD, Tope Kusa, MBBS, MPH, Carlos Camargo Jr., MD, DrPH, Elliot Israel, MD FAAAAI
Rationale:
Insulin resistance potentiates the association between obesity and pediatric asthma, but this relationship appears inconsistent in relatively small studies of adults. We
investigated effect modification in adults using a large, nationally-representative database.
Methods:
Data were obtained from the National Health and Nutrition Examination Survey 2003-2012. Insulin resistance and a history of physician-diagnosed current asthma were
obtained from 12,421 adults (ages 18—85 years). Logistic regression was used to determine associations between obesity and current asthma, adjusting for age, sex,
race/ethnicity, poverty index ratio, and smoking status. We evaluated effect modification by insulin resistance of the obesity-asthma association.
Results:
As expected, obesity was positively associated with current asthma, with 97% greater odds of current asthma for obese participants (95% CI 1.64—2.36). Insulin resistance
modified this association, with a stronger obesity-asthma association among those with insulin resistance (P interaction=0.03). Effect modification was also significant with
obesity measured as waist circumference and waist-to-height ratio. Stratifying by tertiles of insulin resistance, analyses of the relationship between obesity and current
asthma showed an ordinal relationship, with OR increasing with greater insulin resistance (1.99, 2.69, 2.95, respectively). Subgroup analysis of participants not on insulin
replacement therapy also showed a significant association (P interaction=0.048). Hypertension and hypertriglyceridemia (components of the metabolic syndrome, along with
obesity and insulin resistance), did not modify the obesity-asthma association (P interaction=0.68 and 0.45, respectively).
Conclusions:
In this large, nationally-representative sample, insulin resistance modified the association between obesity and current asthma in adults. Clinical and basic studies are
needed to better understand this important interaction.
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Austin Journal of Asthma: Open Access is an open access, peer reviewed, scholarly journal dedicated to publish articles covering all areas of Asthma. The journal aims to promote latest information and provide a forum for doctors, researchers, physicians, and healthcare professionals to find most recent advances in the areas of Asthma. Austin Journal of Asthma: Open Access accepts research articles, reviews, mini reviews, case reports and rapid communication covering all aspects of Asthma. Austin Journal of Asthma: Open Access strongly supports the scientific up gradation and fortification in related scientific research community by enhancing access to peer reviewed scientific literary works. Austin Publishing Group also brings universally peer reviewed journals under one roof thereby promoting knowledge sharing.

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These are simplified slides discussing the regulation of cardiac output and venous return. Learning objectives: 1. Comprehend the determinants of cardiac output and factors affecting cardiac output 2. Comprehend the factors affecting stroke volume and heart rate and total peripheral resistance 3. Identify the factors regulating venous return 4. Discuss the causes of high and low output cardiac failure 5. Enlist the functions of veins and recognise the significance of venous reservoirs Study Resources: 1. Chapter 20, Guyton and Hall Textbook of Medical Physiology, 14th edition 2. Chapter 30 and 32, Ganong’s Review of Medical Physiology, 26th edition 3. Chapter 10, Human Physiology by Lauralee Sherwood, 9th edition 4. Physiology, Cardiac Output - StatPearls https://www.ncbi.nlm.nih.gov/books/NBK470455/

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Step into the captivating realm of cardiovascular physiology as Dr. Faiza, an esteemed Assistant Professor of Physiology, unravels the intricacies of recording an electrocardiogram (ECG). In this illuminating lecture, students are guided through the fundamental principles of ECG interpretation, from lead systems and electrode placement to the analysis of waves, segments, and intervals. Beginning with an exploration of the twelve-lead system, Dr. Faiza elucidates the significance of bipolar limb leads (I, II, III), unipolar limb leads (aVR, aVL, aVF), and unipolar chest leads (V1-V6). Through mnemonic aids and clinical anecdotes, students gain a comprehensive understanding of lead placement techniques, enabling them to navigate the complexities of ECG recording with confidence and precision. As the lecture unfolds, Dr. Faiza delves into the nuances of ECG interpretation, empowering students to correlate ECG findings with electrophysiological events in the cardiac cycle. With a focus on waveform morphology and rhythm analysis, students learn to determine heart rate, rhythm, and axis deviation, deciphering the electrocardiographic manifestations of various cardiac pathologies and physiological states. Moreover, the lecture explores the normal waves, segments, and intervals of an ECG, providing students with a detailed roadmap for interpreting these essential components. From the P wave to the T wave, students gain insight into the voltage, duration, and clinical significance of each waveform, equipping them with the skills to identify aberrations and anomalies in ECG tracings. Enriched with clinical pearls and practical tips, this lecture serves as a cornerstone for students embarking on their journey into cardiovascular medicine. By demystifying the art of ECG interpretation, Dr. Faiza empowers students to harness the power of this diagnostic tool in clinical practice, fostering a deeper appreciation for the intricate interplay between electrical activity and cardiac function.

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Presentation insulin resistance modifies the association between obesity and current asthma in adul

1. Insulin Resistance Modifies the Association Between Obesity and Current Asthma in Adults Saturday, March 5, 2016 South Exhibit Hall H (Convention Center) Samuel Y Ash, MD, Juan Carlos Cardet, MD, Tope Kusa, MBBS, MPH, Carlos Camargo Jr., MD, DrPH, Elliot Israel, MD FAAAAI Rationale: Insulin resistance potentiates the association between obesity and pediatric asthma, but this relationship appears inconsistent in relatively small studies of adults. We investigated effect modification in adults using a large, nationally-representative database. Methods: Data were obtained from the National Health and Nutrition Examination Survey 2003-2012. Insulin resistance and a history of physician-diagnosed current asthma were obtained from 12,421 adults (ages 18—85 years). Logistic regression was used to determine associations between obesity and current asthma, adjusting for age, sex, race/ethnicity, poverty index ratio, and smoking status. We evaluated effect modification by insulin resistance of the obesity-asthma association. Results: As expected, obesity was positively associated with current asthma, with 97% greater odds of current asthma for obese participants (95% CI 1.64—2.36). Insulin resistance modified this association, with a stronger obesity-asthma association among those with insulin resistance (P interaction=0.03). Effect modification was also significant with obesity measured as waist circumference and waist-to-height ratio. Stratifying by tertiles of insulin resistance, analyses of the relationship between obesity and current asthma showed an ordinal relationship, with OR increasing with greater insulin resistance (1.99, 2.69, 2.95, respectively). Subgroup analysis of participants not on insulin replacement therapy also showed a significant association (P interaction=0.048). Hypertension and hypertriglyceridemia (components of the metabolic syndrome, along with obesity and insulin resistance), did not modify the obesity-asthma association (P interaction=0.68 and 0.45, respectively). Conclusions: In this large, nationally-representative sample, insulin resistance modified the association between obesity and current asthma in adults. Clinical and basic studies are needed to better understand this important interaction. See more of: Asthma Epidemiology See more of: Abstracts << Previous Abstract | Next Abstract >> © 1996-2015 All Rights Reserved · American Academy of Allergy, Asthma & Immunology · Disclaimers and Contact Information Back to Meeting Home Print Page 30 Program & Planner Home Browse by Day Author Index Type Index Topic Index Personal Planner

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