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Adipose tissue as an endocrine organ: Adipose tissue has been recognized as the quantitatively most important energy store of the human body for many years, in addition to its functions as mechanical and thermal insulator. During the last 10 years, adipose tissue has come into focus as an endocrine organ important for development of many diseases related to obesity including insulin resistance, type 2 diabetes, dyslipidemia, hypertension and cardiovascular disease. Adipose tissue secretes a variety of bioactive peptides that play important roles in insulin action, energy homeostasis, inflammation, and cell growth. These secretory proteins from the adipose organ are named adipokines and have many physiological effects on different organs including the brain, bone, reproductive organs, liver, skeletal muscles, immune cells and blood vessels. Adipokines may locally regulate fat mass by modulating adipocyte size/number or angiogenesis and inversely increased fat mass leads to dysregulation of adipocyte functions.
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Altered metabolism is one of the hallmarks of cancer cells. Cell cycling and protein synthesis are both key physiological tasks for cancer cells. In recent years, interest has been renewed as clear that many of the signaling pathways that are affected by genetic mutations and the tumor microenvironment have a profound effect on core metabolism of cancer cells. Metabolic alterations in cancer cells are numerous and include aerobic glycolysis, reduced oxidative phosphorylation and the increased generation of biosynthetic intermediates needed for cell growth and proliferation. Furthermore, accelerated protein turnover seen in many cancer patients and whole body protein turnover is increased with advancing stage of disease. Cancer cells alter their consumption and the way they process sugars, fats, amino acids and other energy sources to satisfy the demands of continuous proliferation. The possible effects of specific amino acid, methionine, asparagine, arginine, tyrosine and glutamine, etc. on protein cancer metabolism are discussed. Evidences confirm a contribution of proteins in all cancer stages and describe metabolism of protein in cancer and how amino acids can be targeted to management or initially prevent different types of cancer. Several studies suggest that people who eat more red meat have higher risk for developing colorectal cancer than those who eat less red meat, but avoiding processed meats is even more important for cancer prevention. In this review we summarize the role of proteins in cancer etiology, metabolism, its complication, prevention and treatments.
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Adipose tissue as an endocrine organ: Adipose tissue has been recognized as the quantitatively most important energy store of the human body for many years, in addition to its functions as mechanical and thermal insulator. During the last 10 years, adipose tissue has come into focus as an endocrine organ important for development of many diseases related to obesity including insulin resistance, type 2 diabetes, dyslipidemia, hypertension and cardiovascular disease. Adipose tissue secretes a variety of bioactive peptides that play important roles in insulin action, energy homeostasis, inflammation, and cell growth. These secretory proteins from the adipose organ are named adipokines and have many physiological effects on different organs including the brain, bone, reproductive organs, liver, skeletal muscles, immune cells and blood vessels. Adipokines may locally regulate fat mass by modulating adipocyte size/number or angiogenesis and inversely increased fat mass leads to dysregulation of adipocyte functions.
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Altered metabolism is one of the hallmarks of cancer cells. Cell cycling and protein synthesis are both key physiological tasks for cancer cells. In recent years, interest has been renewed as clear that many of the signaling pathways that are affected by genetic mutations and the tumor microenvironment have a profound effect on core metabolism of cancer cells. Metabolic alterations in cancer cells are numerous and include aerobic glycolysis, reduced oxidative phosphorylation and the increased generation of biosynthetic intermediates needed for cell growth and proliferation. Furthermore, accelerated protein turnover seen in many cancer patients and whole body protein turnover is increased with advancing stage of disease. Cancer cells alter their consumption and the way they process sugars, fats, amino acids and other energy sources to satisfy the demands of continuous proliferation. The possible effects of specific amino acid, methionine, asparagine, arginine, tyrosine and glutamine, etc. on protein cancer metabolism are discussed. Evidences confirm a contribution of proteins in all cancer stages and describe metabolism of protein in cancer and how amino acids can be targeted to management or initially prevent different types of cancer. Several studies suggest that people who eat more red meat have higher risk for developing colorectal cancer than those who eat less red meat, but avoiding processed meats is even more important for cancer prevention. In this review we summarize the role of proteins in cancer etiology, metabolism, its complication, prevention and treatments.
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Obesity
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OBESITY . DISEASE
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