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Risk is the probability that an
individual will get a specific disease
in a given period. The risk of
developing the disease will vary from
individual to individual
 SMOKER ≥100 cigarettes in their lifetime &
currently smoke ( red & orange complex)
 FORMER SMOKER ≥ 100 cigarettes in their
lifetime & do not currently smoke
 NONSMOKER have not smoked 100 cigarettes
in their lifetime & do not currently smoke
 video
 Exaggerated inflammatory response of gingiva
to local factors
 PUBERTY – increased levels of P.intermedia
and capnocytophaga
 MENSURATION - increased gingival bleeding
Increased susceptibility to mechanical irritants
Immune suppression
Dilation of gingival microvasculature, circulatory stasis
Cellular proliferation, permeability of vasculature, pattern of collagen turnover
& metabolic breakdown of folate
Increased progesterone levels
P.intermedia
PREGNANCY TUMOR
•Flat position
may cause
hypotension and
Hypoxia.
•Place a small
pillow under
right hip - left
lateral
displacement.
• Head above
feet. 2
0
SUPINE POSITION AFTER 5TH MONTH
UTERUS COMPRESSES THE INFERIOR
VENACAVA
↑VOL. BLOOD
↓RETURN TO THE HEART
REDUCED PERFUSION OF UTERUS
FETAL HYPOXIA
2
1
Dr. Nitika Jain 8/31/2012
2
2
Dr. Nitika Jain 8/31/2012
2
3
Menopause
• No ovulation.
• No menstrual cycle.
• No estrogen and progestron formed by the ovary.
Changes due to menopause occur in:
• Sex organ
• Endocrine.
• Bone-Osteoporosis
• Metabolic
• CVS
• Lipid profile
• Hypertension, etc
2
4
• Reduction in epithelial keratinization so
thinning of oral mucosa.
• Reduction in salivary glandflow.
• Drying of oral tissue (burningmouth)
• Redness and abnormal palness of
gingivaltissues.
• Bleeding on probing and brushing.
• Gingival recession.
• Altered taste sensation.
• Alveolar bone loss.
• Alveolar ridge resorption.
Clinical findings in periodontaltissues-
• Poor wound Healing.
• Reduced bone mineral content in jaws.
• Increased periodontitis and tooth loss.
Effects of osteoporosis in
periodontal tissues-
• Estrogen deficiency-
• Inc. osteoclastic activity.
• Dec. bone matrix.
• Decreases deposition od bone
calcium and phosphate.
Bone metabolism-
2
5
PROBING DEPTH = GINGIVAL MARGIN TO BASE OF THE SULCUS
CAL = CEJ TO BASE OF THE SULCUS
DEGREE 1 – mobility of crown of 0.2
to 1mm in a horizontal direction
DGREE 2 – mobility of crown of 1 – 2
mm in a horizontal direction.
DEGREE 3 – mobility of croen of >
2mm in horizontal & vertical
direction.( depressible in socket)
 (1) rapid attachment loss and bone destruction in an otherwise clinically healthy
patient
 (2) a familialaggregation.
 3)These patients often present with limited microbial deposits that seem
inconsistent with the severity of tissue destruction.
 Causative bacteria - Aggregatibacter actinomycetemcomitans

 Affects <30 yrs
 LAP – Localized first molar or incisor disease with proximal attachment
loss on at least two permanent teeth, one of which is a first molar
 GAP- Generalized proximal attachment loss that affects at least three
teeth other than the first molars and incisors
 Gingival inflammation
 Periodontal pocket
 Mobility
 Loss of clinical ttachment
 Gingival recession
 Furcation involvement
 Occasional suppuration
MILLERS
CLASSIFICATION TREATMENT
1. Free gingival autograft
2. Free connective tissue autograft
3. Pedicle autografts
• Laterally (horizontally) positioned pedicle
flap
• Coronally positioned flap; includes
semilunar pedicle(Tarnow)
4. Subepithelial connective tissue graft
(Langer)
5. Guided tissue regeneration (GTR)
6. Pouch and tunnel technique (coronally
advanced tunnel
technique
DISCLOSING AGENTS
MECHANISM OF ACTION
COMPONENT
PASTE &
GEL %
POWDER
%
MATERIALS PURPOSE
ABRASIVE 20-55 90-98
•Ca CARBONATE
•DIBASIC Ca PO4
DIHYDRATE
•HYDRATED ALUMINA
•HYDRATED SILICA
•SODIUM BICAR
•MIXTURES OF ANY
REMOVAL OF
PLAQUE /
STAIN &
POLISHING
DETERGENTS 1-2 1-6 Na LAURYAL SULFATE
DEBRIS
REMOVAL
COLORANTS 1-2 1-2 FOOD COLORANTS APPEARANCE
FLAVORING 1 - 2 1 - 2
• OILS OF SPEARMINT
• PEPPERMINT
• WINTERGREEN
• CINNAMON
FLAVOR
HUMECTANT
20 -
35
0
• SORBITOL
• GLYCERINE
MAINTAINS
MOISTURE CONTENT
WATER
15 -
25
0 DEIONIZED WATER SUSPENSION AGENT
BINDER 3 0 CARRAGEENAN
THICKENER,
PREVENTS LIQUID-
SOLID SEPARATION
FLOURIDE 0-1 0
1. Na MONOFLUROPHOSPHATE
2. Na FLUORIDE
3. STANNOUS FLUORIDE
CARIES
PREVENTION
ANTICALCULUS 0-1 0
• DISODIUM PYROPHOSPHATE
• TETRASODIUM
PYROPHOSPHATE
• TETRAPOTASSIUM
PYROPHOSPHATE
 CALCULUS
FORMATION
DESENSITIZING
AGENTS
0-5 0
POTASSIUM NITRATE
STRONTIUM CHLORIDE
OCCLUSION
OF
DENTINAL
TUBULES
 LOCAL
 SYSTEMIC
 HMT
video
 HAND INSTRUMENTS (video)
 SONIC & ULTRASONIC INSTRUMENTS

 Indications
 • Supragingival debridement of
dental calculus and extrinsic stain

• Subgingival debridement of
calculus, oral biofilm, root surface
constituents, and periodontal
pathogens
 • Removal of orthodontic cement
 • Gingival and periodontal
conditions and diseases
 • Surgical interventions
 • Margination (reduces amalgam
overhangs)
 Precautions
• Unshielded pacemakers

• Infectious diseases: human
immunodeficiency virus, hepatitis,
tuberculosis (active stages)
 • Demineralized tooth surface
 • Exposed dentin (especially associated
with sensitivity)
 • Restorative materials (porcelain,
amalgam, gold, composite)
 • Titanium implant abutments unless
using special insert, e.g.,
Quixonic SofTip Prophy Tips
 • Children (primary teeth)
 • Immunosuppression from disease or
chemotherapy
 • Uncontrolled diabetes mellitus
Contraindications
• Chronic pulmonary disease: asthma,
emphysema, cystic fibrosis,
pneumonia
• Cardiovascular disease with secondary
pulmonary disease
• Swallowing difficulty (dysphagia)
1. Vertical grooving
2. Radicular blending
3. Flattening interproximal bone
4. Gradualizing marginal bone
video
OSTEOPLASTY
OSTECTOMY
Prognosis can be divided into
 Overall prognosis
 Individual tooth prognosis
 BIOLOGIC WIDTH
video
COMPLEMENTARY
TREATMENT STRATEGIES IN
PERIODONTICS
Dental implants
NEVER DO IMPLANTS FOR GROWING CHILDREN
 Ti O2 is essential for the nucleation process that
forms CaPO4 precipitates, leading to
mineralised bone formation.
Osseointegration modifications
•
•Sandblasting with Al2O3
•Etching with H3PO4
•Plasma spraying
risk, diagnosis, prognosis and treatment plan.pptx
risk, diagnosis, prognosis and treatment plan.pptx
risk, diagnosis, prognosis and treatment plan.pptx

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risk, diagnosis, prognosis and treatment plan.pptx

  • 1.
  • 2. Risk is the probability that an individual will get a specific disease in a given period. The risk of developing the disease will vary from individual to individual
  • 3.
  • 4.
  • 5.
  • 6.
  • 7.
  • 8.
  • 9.
  • 10.  SMOKER ≥100 cigarettes in their lifetime & currently smoke ( red & orange complex)  FORMER SMOKER ≥ 100 cigarettes in their lifetime & do not currently smoke  NONSMOKER have not smoked 100 cigarettes in their lifetime & do not currently smoke
  • 11.
  • 12.
  • 13.
  • 14.
  • 15.
  • 17.  Exaggerated inflammatory response of gingiva to local factors  PUBERTY – increased levels of P.intermedia and capnocytophaga  MENSURATION - increased gingival bleeding
  • 18. Increased susceptibility to mechanical irritants Immune suppression Dilation of gingival microvasculature, circulatory stasis Cellular proliferation, permeability of vasculature, pattern of collagen turnover & metabolic breakdown of folate Increased progesterone levels P.intermedia
  • 20. •Flat position may cause hypotension and Hypoxia. •Place a small pillow under right hip - left lateral displacement. • Head above feet. 2 0
  • 21. SUPINE POSITION AFTER 5TH MONTH UTERUS COMPRESSES THE INFERIOR VENACAVA ↑VOL. BLOOD ↓RETURN TO THE HEART REDUCED PERFUSION OF UTERUS FETAL HYPOXIA 2 1
  • 22. Dr. Nitika Jain 8/31/2012 2 2
  • 23. Dr. Nitika Jain 8/31/2012 2 3
  • 24. Menopause • No ovulation. • No menstrual cycle. • No estrogen and progestron formed by the ovary. Changes due to menopause occur in: • Sex organ • Endocrine. • Bone-Osteoporosis • Metabolic • CVS • Lipid profile • Hypertension, etc 2 4
  • 25. • Reduction in epithelial keratinization so thinning of oral mucosa. • Reduction in salivary glandflow. • Drying of oral tissue (burningmouth) • Redness and abnormal palness of gingivaltissues. • Bleeding on probing and brushing. • Gingival recession. • Altered taste sensation. • Alveolar bone loss. • Alveolar ridge resorption. Clinical findings in periodontaltissues- • Poor wound Healing. • Reduced bone mineral content in jaws. • Increased periodontitis and tooth loss. Effects of osteoporosis in periodontal tissues- • Estrogen deficiency- • Inc. osteoclastic activity. • Dec. bone matrix. • Decreases deposition od bone calcium and phosphate. Bone metabolism- 2 5
  • 26.
  • 27.
  • 28.
  • 29. PROBING DEPTH = GINGIVAL MARGIN TO BASE OF THE SULCUS CAL = CEJ TO BASE OF THE SULCUS
  • 30. DEGREE 1 – mobility of crown of 0.2 to 1mm in a horizontal direction DGREE 2 – mobility of crown of 1 – 2 mm in a horizontal direction. DEGREE 3 – mobility of croen of > 2mm in horizontal & vertical direction.( depressible in socket)
  • 31.
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  • 35.
  • 36.  (1) rapid attachment loss and bone destruction in an otherwise clinically healthy patient  (2) a familialaggregation.  3)These patients often present with limited microbial deposits that seem inconsistent with the severity of tissue destruction.  Causative bacteria - Aggregatibacter actinomycetemcomitans   Affects <30 yrs  LAP – Localized first molar or incisor disease with proximal attachment loss on at least two permanent teeth, one of which is a first molar  GAP- Generalized proximal attachment loss that affects at least three teeth other than the first molars and incisors
  • 37.  Gingival inflammation  Periodontal pocket  Mobility  Loss of clinical ttachment  Gingival recession  Furcation involvement  Occasional suppuration
  • 38.
  • 39.
  • 40.
  • 41.
  • 42.
  • 43. MILLERS CLASSIFICATION TREATMENT 1. Free gingival autograft 2. Free connective tissue autograft 3. Pedicle autografts • Laterally (horizontally) positioned pedicle flap • Coronally positioned flap; includes semilunar pedicle(Tarnow) 4. Subepithelial connective tissue graft (Langer) 5. Guided tissue regeneration (GTR) 6. Pouch and tunnel technique (coronally advanced tunnel technique
  • 44.
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  • 53.
  • 54. COMPONENT PASTE & GEL % POWDER % MATERIALS PURPOSE ABRASIVE 20-55 90-98 •Ca CARBONATE •DIBASIC Ca PO4 DIHYDRATE •HYDRATED ALUMINA •HYDRATED SILICA •SODIUM BICAR •MIXTURES OF ANY REMOVAL OF PLAQUE / STAIN & POLISHING DETERGENTS 1-2 1-6 Na LAURYAL SULFATE DEBRIS REMOVAL COLORANTS 1-2 1-2 FOOD COLORANTS APPEARANCE
  • 55. FLAVORING 1 - 2 1 - 2 • OILS OF SPEARMINT • PEPPERMINT • WINTERGREEN • CINNAMON FLAVOR HUMECTANT 20 - 35 0 • SORBITOL • GLYCERINE MAINTAINS MOISTURE CONTENT WATER 15 - 25 0 DEIONIZED WATER SUSPENSION AGENT BINDER 3 0 CARRAGEENAN THICKENER, PREVENTS LIQUID- SOLID SEPARATION
  • 56. FLOURIDE 0-1 0 1. Na MONOFLUROPHOSPHATE 2. Na FLUORIDE 3. STANNOUS FLUORIDE CARIES PREVENTION ANTICALCULUS 0-1 0 • DISODIUM PYROPHOSPHATE • TETRASODIUM PYROPHOSPHATE • TETRAPOTASSIUM PYROPHOSPHATE  CALCULUS FORMATION DESENSITIZING AGENTS 0-5 0 POTASSIUM NITRATE STRONTIUM CHLORIDE OCCLUSION OF DENTINAL TUBULES
  • 58.  HAND INSTRUMENTS (video)  SONIC & ULTRASONIC INSTRUMENTS 
  • 59.  Indications  • Supragingival debridement of dental calculus and extrinsic stain  • Subgingival debridement of calculus, oral biofilm, root surface constituents, and periodontal pathogens  • Removal of orthodontic cement  • Gingival and periodontal conditions and diseases  • Surgical interventions  • Margination (reduces amalgam overhangs)  Precautions • Unshielded pacemakers  • Infectious diseases: human immunodeficiency virus, hepatitis, tuberculosis (active stages)  • Demineralized tooth surface  • Exposed dentin (especially associated with sensitivity)  • Restorative materials (porcelain, amalgam, gold, composite)  • Titanium implant abutments unless using special insert, e.g., Quixonic SofTip Prophy Tips  • Children (primary teeth)  • Immunosuppression from disease or chemotherapy  • Uncontrolled diabetes mellitus Contraindications • Chronic pulmonary disease: asthma, emphysema, cystic fibrosis, pneumonia • Cardiovascular disease with secondary pulmonary disease • Swallowing difficulty (dysphagia)
  • 60.
  • 61.
  • 62. 1. Vertical grooving 2. Radicular blending 3. Flattening interproximal bone 4. Gradualizing marginal bone video OSTEOPLASTY OSTECTOMY
  • 63.
  • 64. Prognosis can be divided into  Overall prognosis  Individual tooth prognosis
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  • 75. NEVER DO IMPLANTS FOR GROWING CHILDREN
  • 76.  Ti O2 is essential for the nucleation process that forms CaPO4 precipitates, leading to mineralised bone formation. Osseointegration modifications • •Sandblasting with Al2O3 •Etching with H3PO4 •Plasma spraying