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risk, diagnosis, prognosis and treatment plan.pptx
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2. Risk is the probability that an
individual will get a specific disease
in a given period. The risk of
developing the disease will vary from
individual to individual
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10. SMOKER ≥100 cigarettes in their lifetime &
currently smoke ( red & orange complex)
FORMER SMOKER ≥ 100 cigarettes in their
lifetime & do not currently smoke
NONSMOKER have not smoked 100 cigarettes
in their lifetime & do not currently smoke
17. Exaggerated inflammatory response of gingiva
to local factors
PUBERTY – increased levels of P.intermedia
and capnocytophaga
MENSURATION - increased gingival bleeding
18. Increased susceptibility to mechanical irritants
Immune suppression
Dilation of gingival microvasculature, circulatory stasis
Cellular proliferation, permeability of vasculature, pattern of collagen turnover
& metabolic breakdown of folate
Increased progesterone levels
P.intermedia
21. SUPINE POSITION AFTER 5TH MONTH
UTERUS COMPRESSES THE INFERIOR
VENACAVA
↑VOL. BLOOD
↓RETURN TO THE HEART
REDUCED PERFUSION OF UTERUS
FETAL HYPOXIA
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24. Menopause
• No ovulation.
• No menstrual cycle.
• No estrogen and progestron formed by the ovary.
Changes due to menopause occur in:
• Sex organ
• Endocrine.
• Bone-Osteoporosis
• Metabolic
• CVS
• Lipid profile
• Hypertension, etc
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25. • Reduction in epithelial keratinization so
thinning of oral mucosa.
• Reduction in salivary glandflow.
• Drying of oral tissue (burningmouth)
• Redness and abnormal palness of
gingivaltissues.
• Bleeding on probing and brushing.
• Gingival recession.
• Altered taste sensation.
• Alveolar bone loss.
• Alveolar ridge resorption.
Clinical findings in periodontaltissues-
• Poor wound Healing.
• Reduced bone mineral content in jaws.
• Increased periodontitis and tooth loss.
Effects of osteoporosis in
periodontal tissues-
• Estrogen deficiency-
• Inc. osteoclastic activity.
• Dec. bone matrix.
• Decreases deposition od bone
calcium and phosphate.
Bone metabolism-
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29. PROBING DEPTH = GINGIVAL MARGIN TO BASE OF THE SULCUS
CAL = CEJ TO BASE OF THE SULCUS
30. DEGREE 1 – mobility of crown of 0.2
to 1mm in a horizontal direction
DGREE 2 – mobility of crown of 1 – 2
mm in a horizontal direction.
DEGREE 3 – mobility of croen of >
2mm in horizontal & vertical
direction.( depressible in socket)
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36. (1) rapid attachment loss and bone destruction in an otherwise clinically healthy
patient
(2) a familialaggregation.
3)These patients often present with limited microbial deposits that seem
inconsistent with the severity of tissue destruction.
Causative bacteria - Aggregatibacter actinomycetemcomitans
Affects <30 yrs
LAP – Localized first molar or incisor disease with proximal attachment
loss on at least two permanent teeth, one of which is a first molar
GAP- Generalized proximal attachment loss that affects at least three
teeth other than the first molars and incisors
76. Ti O2 is essential for the nucleation process that
forms CaPO4 precipitates, leading to
mineralised bone formation.
Osseointegration modifications
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•Sandblasting with Al2O3
•Etching with H3PO4
•Plasma spraying