7. ETIOLOGY
• Build up of fatty deposits {plaques}
• Older age [older than 45 for men and >55 for women]
• High blood pressure
• High blood cholesterol
• Cigarette smoking
• Lack of physical activity
• Unhealthy diet
• Obesity or over weight
• Diabetes
• Family history of heart disease, chest pain or stroke.
11. MYOCARDIAL INFRACTION
•Decreased coronary blood circulations and oxygen
circulations due to myocardial tissue necrosis due
to atherosclerosis or occlusion of coronary artery
circulation.
12.
13.
14. PATHOPHYSIOLOGY
• Due to etiological factors
• Deposition of lipids
• Inflammation
• Secrets T lymphocytes and B monocytes
• Infiltrate the area to ingest lipids and then die
• Smooth muscle proliferation
• Forms fibrous cap over the dead fatty core[atheroma]
• Protrude into the lumen of vessels.
• Obstruct the blood flow
15. Obstruct the blood flow
• Atheroma is thick
• Resist blood flow
• Atheroma is thin
• Lipid core grow and rupture
• Haemorrhage
• Thrombus
• Death of heart tissue[ necrosis, ischemia occurs]
• Therefore sudden cardiac death or acute MI.
19. NURSING MANAGEMENT
• Bed rest in a semi fowlers position
• Oxygen administration
• Observe signs of respiratory distress
• Monitor ECG and vital signs
• Music therapy
• Medication
• Monitor ABG and heart sound
• Monitor intake and output chart
• Avoid risk factors.
20. NURSING DIAGNOSIS
• Acute pain related to tissue ischemia as evidenced by reports of chest pain
• Fear and anxiety related to threat or change in health status as evidenced by
fearful attitude.
• Deficient knowledge related to lack of information as evidenced by questions.
• Risk for decreased cardiac output related to changes in rate and rhythm
• Risk for ineffective tissue perfusion related to vasoconstriction.