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ADULT MEDICAL EMERGENCIES
Dr. Swarnalingam Thangavelu M.D
Assistant Professor
Department of Anaesthesiology
TMCH
OBJECTIVES
 To identify, evaluate and treat the emergency medical conditions in a pre
hospital scenario
 The role of Emergency Medical Services (EMS) in prompt & timely referral
services since,
“Time lost is lives lost”
COMMONPRESENTATION-PRE HOSPITAL CARE
Dyspn
ea/
Respir
atory
distres
s
Cardia
c /
Circula
tory
emerge
ncies
Altered
level of
conscio
usness
1.Dyspnea/Respiratory distress
 Most common presenting symptom
 Significant mortality
Common causes: Acute shortness of breath(Non-traumatic)
 Exacerbated COPD
 Asthma
 Cardiogenic pulmonary edema(CCF)
 Non cardiogenic pulmonary Edema (Inhalational, sepsis related)
 Pneumonia
 Pulmonary embolism
 Foreign body obstruction
 Aspiration
 On the scene-identifying the correct diagnosis
 Not always straight forward
 Since the etiology of dyspnea is multifactorial
 Patient with severe COPD & CCF ( who develops pneumonia)
Patient Evaluation
 To identify the condition
 Life threatening with imminent respiratory failure and requires immediate
treatment
Signs and symptoms indicating life threatening condition
 Cyanosis
 Bradypnea RR <6/min or Tachypnea >30 breaths/min
 Tachycardia
 Inability to speak in full sentences < 5-6 words
 Use of accessory respiratory muscles
Pulse Oximetry
 Use of pulse oximetry in diagnosing hypoxemia (Spo2< 92-94% in room air)
 Objective measurement of the severity of respiratory distress
Pre hospital management
A. Bronchospasm: COPD and asthma
 High flow O2 through non-rebreathing mask (irrespective of cause)
 Patient’s medical history
 Currrent medications (inhaled beta2 agonists + corticosteroids in severe case)
 In addition to dyspnea
Wheezing & coughing-classic symptom of bronchospasm
 In extreme bronchospasm
Silent Chest
TREATMENT
OXYGEN
 Inhaled Beta 2 agonists (Nebulised aerosol or MDI)
Albuterol 2.5-5mg every 20 minutes x 3 doses
 + Anti-cholinergics
Ipratropium bromide 0.5mg every 20 minutes x 3
doses
 Iv bolus of Methyl prednisolone 60-125 mg (severe
exacerbation)
 Subcutaneous beta agonists- Adrenaline 0.3-0.5mg
every 20 minutes x 3 doses OR
 Subcutaneous terbutaline 0.25mg every 20 minutesx 3
Impending or Acute respiratory arrest
 Endotracheal Intubation
 Rapid sequence induction with ketamine (bronchodilator)
Vigilant Monitoring
 Look and monitor for additional conditions (that add to the severity of
hypoxemia)
Pneumonia
Pneumothorax
Anaphylaxis
Pulmonary embolism
b. Pulmonary edema
 In addition to standard monitoring
 12 lead ECG is essential to R/O acute MI
 Usage of point of care BNP testing on the scene to determine the cause of
dyspnea (>100pg/ml is diagnostic of cardiogenic cause)
Treatment goals
 To stabilize the patient
 Improve oxygenation and perfusion
 Reduce dyspnea
 NIPPV such as CPAP – standard therapy
 Subsequent therapy should be guided by the patient’s systolic BP
>140mmHg
NIPPV+ Nitrates
NTG spray or
continuous NTG
infusion
5-20 mcg/min
Systolic BP TREATMENT OPTIONS
100-140mmHg
<100mmHg
NIPPV+
Nitrates+
Diuretics ( if
signs of
systemic fluid
retention)
Predominant
signs
Of Cardiogenic
shock
-Inotropes
Dopamine+
Dobutamine +
fluid challenge
 Pulmonary edema with signs of acute coronary syndrome-
Treat like specific ACS – in addition to NIPPV + Nitrates
2.Cardiac & Circulatory emergencies
CHEST PAIN
Circulatory problems
( Syncope, Hypertension)
 Much emphasis
 Role of EMS- in the management of STEMI
 Our Focus
Chest pain
Hypertensive Emergencies
PATIENT EVALUATION-ACS
 Goal: To R/o life threatening condition such as ACS or PE
 Common complaint: Chest pain
 Identifying high risk group among all patients with
Back spasm
Esophageal acid reflux
GI disorders
 History & Physical examination to guide the diagnosis
 12 lead ECG
 Assessing the type of pain- stabbing versus burning
 Quality of chest pain
 Radiation, severity and provocation factors
 To determine the differential diagnosis
Atypical pain symptoms
 Epigastric discomfort
 Jaw pain
 No pain ( silent MI in diabetic patients)
Pre hospital management-ACS
 Continuous ECG monitoring
 BP & Spo2 monitoring
 Rapid access to defibrillation
 Goal:
To reduce myocardial O2 demand while improving O2 supply
MONA- Acroynm
 Morphine-pain control Dose: 2-4 mg iv bolus
 Oxygen-high flow O2
 Nitrates- if patient is not hypotensive (caution in RV infarction)
 Aspirin Dose: 162-325 mg (non-enteric coated chewable form)
 Beta blockers- if tachycardic and/or hypertensive
Metoprolol 5 mg iv every 5 minutes x 3 doses
PRE -HOSPITAL ECG
 Vital component of pre hospital care
 Only tool –reliably identify the patients who will benefit from prehospital
reperfusion therapy
 Goal: limit myocardial ischemia time <120 minutes (ideally <60 minutes)
Early out of hospital fibrinolysis
 Reduces mortality in patients with STEMI
 New onset LBBB
 The recommended standard for fibrinolysis –capable EMS systems
Fibrinolytic agents
 Streptokinase
 Recombinant tissue plasminogen activator (rt-PA or t-PA)
 Modified forms of t-PA
Reteplase Dose: 10 U iv bolus and repeat 10 U iv after 30 minutes
Tenecteplase
Contraindications
 Risk of ICH ---Major disability or death in 1-2% of patients
 If fibrinolysis is not possible-
 Transport to a facility with PCI with goal of EMS arrival to balloon time <90
minutes since
“Time is muscle”
B.PULMONARY EMBOLISM
 Definitive diagnosis –difficult to establish on the scene
 Since the signs & symptoms are non specific
 No rapid objective measures-12 lead ECG in NSTEMI
 Medical history
Immobilization
Signs of pre existing DVT (unilateral, tender swelling in one extremity)
 Severity of PE:
Related to the size of clots
Ranges from mild symptom( dyspnea & coughing) to life threatening
states & cardiogenic shock due to acute right sided heart failure
Classic symptoms of Pulmonary Embolism
 Distended neck veins
 Hypoxemia
 Tachycardia
 Tachypnea
 Chest pain
Non specific findings
 More objective
 New onset RBBB
 Symmetric T inversion in anterior leads V1-V4
 S1 QIII pattern in 12 lead ECG- more useful to R/O MI rather than PE
Management
 Largely supportive –absence of objective diagnosis
 High flow O2
 IV line
 Pain control with opioid analgesics
 Hypotension(severe PE) –inotropic agents for circulatory support
 No universal recommendation for pre-hospital usage of heparin
 Determined on a case to case basis
 Successfully used in extremis
c.Hypertensive emergencies
 Classificaton of hypertensive episodes
Very useful in determining the urgency of pre hospital intervention
 Acute end organ damage in conjunction with hypertensive episodes
Hypertensive encephalopathy/stroke
Pulmonary edema/MI/Aortic dissection
Acute renal failure
Pre-eclampsia
Classification of acute hypertensive episodes (JNC Seventh Report-
2003)
Hypertensive Emergency Also termed hypertensive crisis
Hypertension with acute EOD
Hypertensive Urgency Hypertensive episode with high risk of
imminent EOD that has not yet occurred
Acute hypertensive episode SBP >180mmHg or DBP >110mmHg
Without signs of evolving or imminent
EOD
Transient hypertension Related to anxiety or primary complaint
Management
 Must be immediately treated & progression must be stopped
 Goal: To lower BP by 20-25% within 30-60 minutes
 In setting of cerebral infarction
Great care in reducing BP
Since the ischemic territory –critically dependent on collateral
perfusion pressure
 Evidence for benefits of anti-HT treatment is scant
 Some – only excessively high SBP or DBP should be treated
 Patients with aortic dissection-require swift and aggressive treatment with a
target BP
SBP <120mmHg or DBP< 80 mmHg
Treatment
 Choice of anti-hypertensive – underlying pathophysiology
 Hypertension + ACS + Tachycardia- Beta blockers & NTG
 ICH – Labetalol –preferred agent Dose: 20mg iv over 2 minutes & repeat every
10 minutes @ dosage of 20-40mg
 European EMS system- Urapidil ( alpha – adrenoreceptor antagonist)
THANK YOU

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Adult medical emergencies

  • 1. ADULT MEDICAL EMERGENCIES Dr. Swarnalingam Thangavelu M.D Assistant Professor Department of Anaesthesiology TMCH
  • 2. OBJECTIVES  To identify, evaluate and treat the emergency medical conditions in a pre hospital scenario  The role of Emergency Medical Services (EMS) in prompt & timely referral services since, “Time lost is lives lost”
  • 3. COMMONPRESENTATION-PRE HOSPITAL CARE Dyspn ea/ Respir atory distres s Cardia c / Circula tory emerge ncies Altered level of conscio usness
  • 4. 1.Dyspnea/Respiratory distress  Most common presenting symptom  Significant mortality
  • 5. Common causes: Acute shortness of breath(Non-traumatic)  Exacerbated COPD  Asthma  Cardiogenic pulmonary edema(CCF)  Non cardiogenic pulmonary Edema (Inhalational, sepsis related)  Pneumonia  Pulmonary embolism  Foreign body obstruction  Aspiration
  • 6.  On the scene-identifying the correct diagnosis  Not always straight forward  Since the etiology of dyspnea is multifactorial  Patient with severe COPD & CCF ( who develops pneumonia)
  • 7. Patient Evaluation  To identify the condition  Life threatening with imminent respiratory failure and requires immediate treatment
  • 8. Signs and symptoms indicating life threatening condition  Cyanosis  Bradypnea RR <6/min or Tachypnea >30 breaths/min  Tachycardia  Inability to speak in full sentences < 5-6 words  Use of accessory respiratory muscles
  • 9. Pulse Oximetry  Use of pulse oximetry in diagnosing hypoxemia (Spo2< 92-94% in room air)  Objective measurement of the severity of respiratory distress
  • 10. Pre hospital management A. Bronchospasm: COPD and asthma  High flow O2 through non-rebreathing mask (irrespective of cause)  Patient’s medical history  Currrent medications (inhaled beta2 agonists + corticosteroids in severe case)  In addition to dyspnea Wheezing & coughing-classic symptom of bronchospasm  In extreme bronchospasm
  • 13.  Inhaled Beta 2 agonists (Nebulised aerosol or MDI) Albuterol 2.5-5mg every 20 minutes x 3 doses  + Anti-cholinergics Ipratropium bromide 0.5mg every 20 minutes x 3 doses  Iv bolus of Methyl prednisolone 60-125 mg (severe exacerbation)  Subcutaneous beta agonists- Adrenaline 0.3-0.5mg every 20 minutes x 3 doses OR  Subcutaneous terbutaline 0.25mg every 20 minutesx 3
  • 14. Impending or Acute respiratory arrest  Endotracheal Intubation  Rapid sequence induction with ketamine (bronchodilator)
  • 15. Vigilant Monitoring  Look and monitor for additional conditions (that add to the severity of hypoxemia) Pneumonia Pneumothorax Anaphylaxis Pulmonary embolism
  • 16. b. Pulmonary edema  In addition to standard monitoring  12 lead ECG is essential to R/O acute MI  Usage of point of care BNP testing on the scene to determine the cause of dyspnea (>100pg/ml is diagnostic of cardiogenic cause)
  • 17. Treatment goals  To stabilize the patient  Improve oxygenation and perfusion  Reduce dyspnea  NIPPV such as CPAP – standard therapy  Subsequent therapy should be guided by the patient’s systolic BP
  • 18. >140mmHg NIPPV+ Nitrates NTG spray or continuous NTG infusion 5-20 mcg/min Systolic BP TREATMENT OPTIONS 100-140mmHg <100mmHg NIPPV+ Nitrates+ Diuretics ( if signs of systemic fluid retention) Predominant signs Of Cardiogenic shock -Inotropes Dopamine+ Dobutamine + fluid challenge
  • 19.  Pulmonary edema with signs of acute coronary syndrome- Treat like specific ACS – in addition to NIPPV + Nitrates
  • 20. 2.Cardiac & Circulatory emergencies CHEST PAIN Circulatory problems ( Syncope, Hypertension)
  • 21.  Much emphasis  Role of EMS- in the management of STEMI  Our Focus Chest pain Hypertensive Emergencies
  • 22. PATIENT EVALUATION-ACS  Goal: To R/o life threatening condition such as ACS or PE  Common complaint: Chest pain  Identifying high risk group among all patients with Back spasm Esophageal acid reflux GI disorders
  • 23.  History & Physical examination to guide the diagnosis  12 lead ECG
  • 24.  Assessing the type of pain- stabbing versus burning  Quality of chest pain  Radiation, severity and provocation factors  To determine the differential diagnosis
  • 25. Atypical pain symptoms  Epigastric discomfort  Jaw pain  No pain ( silent MI in diabetic patients)
  • 26. Pre hospital management-ACS  Continuous ECG monitoring  BP & Spo2 monitoring  Rapid access to defibrillation  Goal: To reduce myocardial O2 demand while improving O2 supply
  • 27. MONA- Acroynm  Morphine-pain control Dose: 2-4 mg iv bolus  Oxygen-high flow O2  Nitrates- if patient is not hypotensive (caution in RV infarction)  Aspirin Dose: 162-325 mg (non-enteric coated chewable form)  Beta blockers- if tachycardic and/or hypertensive Metoprolol 5 mg iv every 5 minutes x 3 doses
  • 28. PRE -HOSPITAL ECG  Vital component of pre hospital care  Only tool –reliably identify the patients who will benefit from prehospital reperfusion therapy  Goal: limit myocardial ischemia time <120 minutes (ideally <60 minutes)
  • 29. Early out of hospital fibrinolysis  Reduces mortality in patients with STEMI  New onset LBBB  The recommended standard for fibrinolysis –capable EMS systems
  • 30. Fibrinolytic agents  Streptokinase  Recombinant tissue plasminogen activator (rt-PA or t-PA)  Modified forms of t-PA Reteplase Dose: 10 U iv bolus and repeat 10 U iv after 30 minutes Tenecteplase
  • 31. Contraindications  Risk of ICH ---Major disability or death in 1-2% of patients  If fibrinolysis is not possible-  Transport to a facility with PCI with goal of EMS arrival to balloon time <90 minutes since “Time is muscle”
  • 32.
  • 33. B.PULMONARY EMBOLISM  Definitive diagnosis –difficult to establish on the scene  Since the signs & symptoms are non specific  No rapid objective measures-12 lead ECG in NSTEMI
  • 34.  Medical history Immobilization Signs of pre existing DVT (unilateral, tender swelling in one extremity)  Severity of PE: Related to the size of clots Ranges from mild symptom( dyspnea & coughing) to life threatening states & cardiogenic shock due to acute right sided heart failure
  • 35. Classic symptoms of Pulmonary Embolism  Distended neck veins  Hypoxemia  Tachycardia  Tachypnea  Chest pain
  • 36. Non specific findings  More objective  New onset RBBB  Symmetric T inversion in anterior leads V1-V4  S1 QIII pattern in 12 lead ECG- more useful to R/O MI rather than PE
  • 37. Management  Largely supportive –absence of objective diagnosis  High flow O2  IV line  Pain control with opioid analgesics  Hypotension(severe PE) –inotropic agents for circulatory support  No universal recommendation for pre-hospital usage of heparin  Determined on a case to case basis  Successfully used in extremis
  • 38. c.Hypertensive emergencies  Classificaton of hypertensive episodes Very useful in determining the urgency of pre hospital intervention  Acute end organ damage in conjunction with hypertensive episodes Hypertensive encephalopathy/stroke Pulmonary edema/MI/Aortic dissection Acute renal failure Pre-eclampsia
  • 39. Classification of acute hypertensive episodes (JNC Seventh Report- 2003) Hypertensive Emergency Also termed hypertensive crisis Hypertension with acute EOD Hypertensive Urgency Hypertensive episode with high risk of imminent EOD that has not yet occurred Acute hypertensive episode SBP >180mmHg or DBP >110mmHg Without signs of evolving or imminent EOD Transient hypertension Related to anxiety or primary complaint
  • 40. Management  Must be immediately treated & progression must be stopped  Goal: To lower BP by 20-25% within 30-60 minutes  In setting of cerebral infarction Great care in reducing BP Since the ischemic territory –critically dependent on collateral perfusion pressure  Evidence for benefits of anti-HT treatment is scant  Some – only excessively high SBP or DBP should be treated  Patients with aortic dissection-require swift and aggressive treatment with a target BP SBP <120mmHg or DBP< 80 mmHg
  • 41. Treatment  Choice of anti-hypertensive – underlying pathophysiology  Hypertension + ACS + Tachycardia- Beta blockers & NTG  ICH – Labetalol –preferred agent Dose: 20mg iv over 2 minutes & repeat every 10 minutes @ dosage of 20-40mg  European EMS system- Urapidil ( alpha – adrenoreceptor antagonist)
  • 42.