2. OBJECTIVES
To identify, evaluate and treat the emergency medical conditions in a pre
hospital scenario
The role of Emergency Medical Services (EMS) in prompt & timely referral
services since,
“Time lost is lives lost”
5. Common causes: Acute shortness of breath(Non-traumatic)
Exacerbated COPD
Asthma
Cardiogenic pulmonary edema(CCF)
Non cardiogenic pulmonary Edema (Inhalational, sepsis related)
Pneumonia
Pulmonary embolism
Foreign body obstruction
Aspiration
6. On the scene-identifying the correct diagnosis
Not always straight forward
Since the etiology of dyspnea is multifactorial
Patient with severe COPD & CCF ( who develops pneumonia)
7. Patient Evaluation
To identify the condition
Life threatening with imminent respiratory failure and requires immediate
treatment
8. Signs and symptoms indicating life threatening condition
Cyanosis
Bradypnea RR <6/min or Tachypnea >30 breaths/min
Tachycardia
Inability to speak in full sentences < 5-6 words
Use of accessory respiratory muscles
9. Pulse Oximetry
Use of pulse oximetry in diagnosing hypoxemia (Spo2< 92-94% in room air)
Objective measurement of the severity of respiratory distress
10. Pre hospital management
A. Bronchospasm: COPD and asthma
High flow O2 through non-rebreathing mask (irrespective of cause)
Patient’s medical history
Currrent medications (inhaled beta2 agonists + corticosteroids in severe case)
In addition to dyspnea
Wheezing & coughing-classic symptom of bronchospasm
In extreme bronchospasm
13. Inhaled Beta 2 agonists (Nebulised aerosol or MDI)
Albuterol 2.5-5mg every 20 minutes x 3 doses
+ Anti-cholinergics
Ipratropium bromide 0.5mg every 20 minutes x 3
doses
Iv bolus of Methyl prednisolone 60-125 mg (severe
exacerbation)
Subcutaneous beta agonists- Adrenaline 0.3-0.5mg
every 20 minutes x 3 doses OR
Subcutaneous terbutaline 0.25mg every 20 minutesx 3
14. Impending or Acute respiratory arrest
Endotracheal Intubation
Rapid sequence induction with ketamine (bronchodilator)
15. Vigilant Monitoring
Look and monitor for additional conditions (that add to the severity of
hypoxemia)
Pneumonia
Pneumothorax
Anaphylaxis
Pulmonary embolism
16. b. Pulmonary edema
In addition to standard monitoring
12 lead ECG is essential to R/O acute MI
Usage of point of care BNP testing on the scene to determine the cause of
dyspnea (>100pg/ml is diagnostic of cardiogenic cause)
17. Treatment goals
To stabilize the patient
Improve oxygenation and perfusion
Reduce dyspnea
NIPPV such as CPAP – standard therapy
Subsequent therapy should be guided by the patient’s systolic BP
18. >140mmHg
NIPPV+ Nitrates
NTG spray or
continuous NTG
infusion
5-20 mcg/min
Systolic BP TREATMENT OPTIONS
100-140mmHg
<100mmHg
NIPPV+
Nitrates+
Diuretics ( if
signs of
systemic fluid
retention)
Predominant
signs
Of Cardiogenic
shock
-Inotropes
Dopamine+
Dobutamine +
fluid challenge
19. Pulmonary edema with signs of acute coronary syndrome-
Treat like specific ACS – in addition to NIPPV + Nitrates
21. Much emphasis
Role of EMS- in the management of STEMI
Our Focus
Chest pain
Hypertensive Emergencies
22. PATIENT EVALUATION-ACS
Goal: To R/o life threatening condition such as ACS or PE
Common complaint: Chest pain
Identifying high risk group among all patients with
Back spasm
Esophageal acid reflux
GI disorders
23. History & Physical examination to guide the diagnosis
12 lead ECG
24. Assessing the type of pain- stabbing versus burning
Quality of chest pain
Radiation, severity and provocation factors
To determine the differential diagnosis
25. Atypical pain symptoms
Epigastric discomfort
Jaw pain
No pain ( silent MI in diabetic patients)
26. Pre hospital management-ACS
Continuous ECG monitoring
BP & Spo2 monitoring
Rapid access to defibrillation
Goal:
To reduce myocardial O2 demand while improving O2 supply
27. MONA- Acroynm
Morphine-pain control Dose: 2-4 mg iv bolus
Oxygen-high flow O2
Nitrates- if patient is not hypotensive (caution in RV infarction)
Aspirin Dose: 162-325 mg (non-enteric coated chewable form)
Beta blockers- if tachycardic and/or hypertensive
Metoprolol 5 mg iv every 5 minutes x 3 doses
28. PRE -HOSPITAL ECG
Vital component of pre hospital care
Only tool –reliably identify the patients who will benefit from prehospital
reperfusion therapy
Goal: limit myocardial ischemia time <120 minutes (ideally <60 minutes)
29. Early out of hospital fibrinolysis
Reduces mortality in patients with STEMI
New onset LBBB
The recommended standard for fibrinolysis –capable EMS systems
30. Fibrinolytic agents
Streptokinase
Recombinant tissue plasminogen activator (rt-PA or t-PA)
Modified forms of t-PA
Reteplase Dose: 10 U iv bolus and repeat 10 U iv after 30 minutes
Tenecteplase
31. Contraindications
Risk of ICH ---Major disability or death in 1-2% of patients
If fibrinolysis is not possible-
Transport to a facility with PCI with goal of EMS arrival to balloon time <90
minutes since
“Time is muscle”
32.
33. B.PULMONARY EMBOLISM
Definitive diagnosis –difficult to establish on the scene
Since the signs & symptoms are non specific
No rapid objective measures-12 lead ECG in NSTEMI
34. Medical history
Immobilization
Signs of pre existing DVT (unilateral, tender swelling in one extremity)
Severity of PE:
Related to the size of clots
Ranges from mild symptom( dyspnea & coughing) to life threatening
states & cardiogenic shock due to acute right sided heart failure
36. Non specific findings
More objective
New onset RBBB
Symmetric T inversion in anterior leads V1-V4
S1 QIII pattern in 12 lead ECG- more useful to R/O MI rather than PE
37. Management
Largely supportive –absence of objective diagnosis
High flow O2
IV line
Pain control with opioid analgesics
Hypotension(severe PE) –inotropic agents for circulatory support
No universal recommendation for pre-hospital usage of heparin
Determined on a case to case basis
Successfully used in extremis
38. c.Hypertensive emergencies
Classificaton of hypertensive episodes
Very useful in determining the urgency of pre hospital intervention
Acute end organ damage in conjunction with hypertensive episodes
Hypertensive encephalopathy/stroke
Pulmonary edema/MI/Aortic dissection
Acute renal failure
Pre-eclampsia
39. Classification of acute hypertensive episodes (JNC Seventh Report-
2003)
Hypertensive Emergency Also termed hypertensive crisis
Hypertension with acute EOD
Hypertensive Urgency Hypertensive episode with high risk of
imminent EOD that has not yet occurred
Acute hypertensive episode SBP >180mmHg or DBP >110mmHg
Without signs of evolving or imminent
EOD
Transient hypertension Related to anxiety or primary complaint
40. Management
Must be immediately treated & progression must be stopped
Goal: To lower BP by 20-25% within 30-60 minutes
In setting of cerebral infarction
Great care in reducing BP
Since the ischemic territory –critically dependent on collateral
perfusion pressure
Evidence for benefits of anti-HT treatment is scant
Some – only excessively high SBP or DBP should be treated
Patients with aortic dissection-require swift and aggressive treatment with a
target BP
SBP <120mmHg or DBP< 80 mmHg
41. Treatment
Choice of anti-hypertensive – underlying pathophysiology
Hypertension + ACS + Tachycardia- Beta blockers & NTG
ICH – Labetalol –preferred agent Dose: 20mg iv over 2 minutes & repeat every
10 minutes @ dosage of 20-40mg
European EMS system- Urapidil ( alpha – adrenoreceptor antagonist)