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Anorectal Pain & discharge
DR. Saeed Ahmad Al-Shoaib
Surgical Consultan
Surgery MCS General
(Ph-D) Surgical Oncology
College of Medicine
University of Hadramout
(HUCOM)
The rectum is 12 – 16 cm long.
It extends from the rectosigmoid junction,
marked by the fusion of the tenia, to the
anal canal, marked by the passage into the
pelvic floor musculature.
The anal canal is an invagination of
ectodermal tissue.
The Anorectum develops from fusion of
the rectum and the anal canal.
Of anal canal
Anatomy
•The anatomical canal is about 3 cm long
and it extends from the anal verge to the
dentate line
•The surgical anal canal is about 4 cm long
and it extends from the anal verge to the
anorectal junction
• Pecten (anoderm) is a hairless part of the
external anal canal that exposed by traction on
the rim of the anus ( lined by keratinized
stratified squamous epithelium )
•The junction between the Pecten and the large
mucosa is known as dentate line .
Of anal canal
Anatomy
Anatomy of the anal canal
•The rectum is lined by columnar epithelium
•The transitional zone is lined with cuboidal
epithelium that lines the anal canal from the
columns of morgagni to the dentate line.
•Below the dentate line the anal canal is lined
by squamous epithelium .
1- Internal sphincter (involuntary )
2-External sphincter (voluntary)
Anal Sphincters :
Differences between the upper and the lower halves of the anal
canal according to the dentate line
Below the dentate line
Above the dentate line
Ectodermal in origin
Endodermal in origin
Lined by squamous epithelium
Lined by columnar epithelium
Under control of the somatic
innervation (sensitive to pain, touch
, temperature)
Under control of the autonomic
nervous sys. (sensitive to stretch )
Drains its venous blood via inferoir
rectal vein into the systemic venous
Drains its venous blood via the sup.
rectal vein to the portal venous
Drains its lymph to the inguinal
lymph nodes.
drains its lymph to the internal iliac
lymph nodes.
Surgical Anatomy
•
The blood supply of upper ½ of the anal canal is from the
superior rectal vessels. Where as that of the lower ½ is supply
of the surrounding anal skin the inferior rectal vessels which
derives from the internal pudendal ultimately from the
internal iliac vessels.
Surgical Anatomy
•
The lower ½ is lined by squamous epithelium
and the upper ½ by columnar epithelium so
carcinoma of the upper ½ is adenocarcinoma.
Where as that arising from the lower part is
squamous tumour.
Surgical Anatomy
•
The lymphatic above the mucocutaneous
junction drain along the superior rectal vessels
to the lumbar lymph nodes, where as below
this line drainage is to the inguinal lymph
nodes.
Surgical Anatomy
•
The nerve supply to the upper ½ via
autonomic plexus and the lower ½ is supplied
by the somatic inferior rectal nerves terminal
branch of the pudendal nerve. So the lower ½
is sensitive to the prick needle.
Normal Function of the Anorectun
The normal function of the anorectum is storage
and release of intestinal waste products . The
rectum functions mainly as a capacitance
storage vessel.
Dysfunction of the Anorectum
•Incontinence:
Continence is maintained through rectal
compliance, ano –rectal sensation, anorectal
reflexes, and anal sphincter function . The nature
and quantity of stool and colonic transit are
important as well . The incidence of fecal
incontinence is difficult to determine because of
underreporting and lack of a standard definition
of the term .
Common symptoms
•Anal bleeding
•Anal pain and discomfort
•Perianal itching and irritation
•Something coming down
•perianal discharge
Common Anal Conditions
•
Haemorrhoids
•
Pruritus ani
•
Perianal abscess
•
Anal fissure
•
Anal fistula
•
Rectal prolapse
•
Anal in continence
•
Non malignant strictures
•
Anal neoplasms
Examination of Anus
This requires careful attention to circumstances (couch, light,
gloves). The Sims (left lateral position) is satisfactory. The
examination proceed by;
–
inspection
–
digital examination with index finger
–
proctoscopy
–
sigmoidoscopy
Hemorrhoids
Definition : they are engorgement of venous plexi
“cushions” that sit in the anorectal junction.
Could be
External: below the dentate line
Internal: above the dentate line
Commonly situated at :
3 o
’
clock “left lateral”
7 o
’
clock “right posterior”
11 o
’
clock “right anterior”
Classification of internal
hemorrhoids
First degree: painless bleeding usually
associated with defecation
Second degree: protrude during defecation
but spontaneously reduce
Third degree: protrude during defecation
and must be manually reduced
Forth degree: permanently prolapsed
Etiology
•
Primary Causes:
–
Hereditary factors e.g, structural weakness of the vein.
–
Anatomical factors.
–
Partial congestion.
–
Chronic constipation.
–
Sphincteric relaxation.
Etiology
•
Secondary Causes:
•
pregnancy
•
venous obstruction
•
straining on micturation
•
venous congestion
•
carcinoma of the rectum
Clinical features
•
Bleeding at defecation
•
Prolapse
•
Discharge with pruritus ani
•
Pain
•
Thrombosed piles
Signs and symptoms
Painless bright red rectal bleeding
Prolapsing lump
Itching
Mucus discharge
N.B.
sever pain is not typically associated with internal hemorrhoids
but is commonly seen with thrombosed external
hemorrhoids
but if pain is present with absence of strangulation look out for
other conditions “fissure , hematoma, abcess”
Assessment and Diagnosis
•
Careful history
Age, type of bleeding, bowel habit
Examination:
•
Abdominal Examination
•
Anorectal Examination
Investigation:
Protoscopy , rigid sigmoidoscopy, colonscopy,Barium enema.
Complications
Profuse hemorhage
Ulcerations
Fibrosis
Strangulation
Gangrene
thrombosis
Complications
Profuse hemorhage
Ulcerations
Fibrosis
Strangulation
Gangrene
thrombosis
Cont. treatment
The definite treatment varies according to
the degree of hemorroids:
1st degree : sclerotherapy
infrared photocoagulation
cryothrapy
2nd degree : rubber band ligation
3rd and 4th degree : hemorrhoidectomy
Cont. treatment
Thrombosed external hemorrhoids should be
excised if seen within 48 hr, beyond this time,
conservative therapy with analgesics and sitz
baths is appropriate.
Anal Fissure
•
Defined as longitudinal tear in the mucosa and skin of the
anal canal.
•
Commonly posterior midline more common in female than
male. ( tear in squamous epithelium ).
It is the commonest cause of sever anal pain
Anal Fissure
•
The acute anal fissure if not treated
becomes chronic
anal fissures. As result
secondary pathological changes
may
occurs:
–
Chronicity
–
A “sentinel” pile
–
Hypertrophied anal papilla
–
Contracture of the anus
–
Suppuration
Anal Fissure
•
The acute anal fissure if not treated
becomes chronic
anal fissures. As result
secondary pathological changes
may
occurs:
–
Chronicity
–
A “sentinel” pile
–
Hypertrophied anal papilla
–
Contracture of the anus
–
Suppuration
Epidemiology
Acute or chronic tear in the anal canal
usually in posterior midline ( M=F in young
adult)
Anterior fissure is common in Females.
Lateral or multiple fissures should raise
suspicion of trauma, inflammatory bowel
disease ( Chron
’
s disease), lymphoma,
neoplasm or infection.
Causes
Constipation
Inflammatory bowel disease
Sexual transmitted disease
Post anal surgery
Signs and symptoms
burning pain is associated with defecation.
Bleeding : the blood is usually bright red
and associated with acute fissures.
Physical exam
Acute fissure showing just tear in the anal
mucosa.
Chronic fissure may reveal a sentinel tag or
hypertrophied papilla.
Treatment
Conservative ( acute fissure )
- fiber supplement
- bulk laxatives
- stool softner
- sitz baths
- topical nitroglycerine ointment
- Botox
Cont. Treatment
Surgical ( with chronic fissure to reduce the
internal sphincter spasm )
- sphincter dilation
- Lateral internal sphincterotomy
External Hemorrhoids
•
Perianal Hematoma
•
Due to rupture of dilated anal vein as result of sever straining.
•
Sudden onset of painful lump at the anus.
•
Swelling tense & tender, bluish in colour covered with
smooth shining skin.
Treatment
•
Evacuation if the patient come within 48hours
•
If patient come late conservative treatment.
•
If untreated the haematoma undergoes:
–
resolution
–
ulceration
–
suppuration to forms in abscess
–
fibrosis which give rise to skin tag.
Perianal Abscess
•
The infection usually starts in one of the crypts of Morgagni
and extends along the related anal gland to the
intersphincteric plane where it forms as abscess.
•
Soon it tracks in various directions to produce different types
of abscesses .
Types of Abscess
•
Perianal abscess (60%)
•
Ischiorectal abscess (30%)
•
Sub mucous abscess (5%)
•
Pelvirectal abscess
Perianal Abscess
•
Patient with recurrent anorectal abscess always consider
associated underlying diseases such as Crohn’s, UC, rectal
cancer and active TB.
Perianal Abscess
•
Symptoms
•
Acute pain
•
High fever
•
Signs
•
Swelling
•
Tenderness with induration
•
Treatment
•
Incision and drainage and covered by antibiotics.
Fistula in ano
•
Defined as track lined by granulation tissues, which connects
deeply in the anal canal or rectum and superficially on the
skin around the anus.
•
It usually result from an anorectal abscess.
Fistula in ano
•
Anal fistulas have well recognized association with crohn’s
disease, UC, TB, colloid carcinoma of the rectum and lympho
granuloma venercum.
Types of Anal Fistulas
•
According to whether their natural opening is below or above the
anorectal ring
•
Low level e.g., subcutaneous, low
anal, sub mucous.
•
High level – open into anal canal
at or above the anorectal ring
e.g.,
high anal, pelvirectal
Park’s Classification
•
Inter sphincteric (70%) low level
anal fistula
•
Trans-sphincteric (25%) high
level anal fistula
•
Supra sphincteric fistulae (4%).
•
Extra sphincteric (1%) rare type
include the tract passes outside
all
sphincter muscles to open in
the rectum.
Good Sall's Rule
•
Fistulas with external opening in
relation
to the anterior ½ of the anus
tend to be
direct type.
Clinical features
•
Persistent discharge which irritates the skin and causes
discomfort at the anus may be associated with pain.
•
External opening may be seen with
palpation the tracks
is often palpable
as cord.
Investigations
•
Proctoscopy
•
Radiology
•
Biopsy
•
Always sent track for biopsy.
Anorectal Tumours
•
Benign tumours
–
Epithelial Tumours
•
Anal warts (virus)
•
Juvenile polyp
•
Adenomatous polyps
•
Villous papilloma
•
Familial polyposis
•
Pseudo polyps
•
Endometrioma
Anorectal Tumours
•
Connective Tissue Tumours
–
Fibrous polyp
–
Lipoma
–
Myoma
–
Haemangioma
–
Benign Lymphoma
Malignant Tumours of the Anal Canal
•
The lesion is usually squamous cell carcinoma.
•
Rarely adenocarcinoma, malignant melanoma or basal cell
carcinoma.
Squamous cell carcinoma
•
5% of all anorectal malignancies. Arising from the stratified squamous
epithelium of the lower ½ of the anal canal.
•
It is disease of elderly.
•
Squamous cell carcinoma more common in males.
•
The aetiology of anal carcinoma unknown but chronic irritation or
infection may be predisposing factors.
Clinical Features
•
Localized ulcer or raised growth with
irregular ulcerated
surface.
•
History of bleeding.
•
History of pain with discomfort.
•
Tenesmus with incontinence.
•
Discharge.
Examination
•
On palpation squamous carcinoma feels hard and woody due to invasion
of perianal tissues.
•
P/R examination may prove impossible because of stenosis or discomfort.
•
Inguinal LN are examined carefully
as they receive lymph from the
lower anal canal and perianal region and may be the
site of
metastasis.
Treatment
•
Above the pectinate line
–
Abdomino perineal excision
•
Below the pertinate line
–
local excision.
–
If inguinal LN metastasis present should be removed by block
dissection.
Treatment
•
Late cases
–
Palliative colostomy.
–
Radiotherapy.
Rare Malignant Anal Tumor's
•
Adenocarcinoma
•
Basal cell carcinoma
•
Malignant melanoma
Benign strictures
•
Stricture of the anus and rectum may be:
–
Congenital
–
Postoperative
–
Inflammatory
Clinical features
•
Progressive difficulty in defecation
•
In cases of inflammatory strictures
–
Bleeding
–
Discharge
–
Tenesmus
•
Late cases subacute intestinal obstruction
Diagnosis
•
Rectal examination reveals
the location type and
degree
of the stenosis.
•
Proctoscopy
•
Biopsy
Treatment
•
Dilation
•
Superficial external
proctotomy
Thanks

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TAIQ.pptx

  • 1. Anorectal Pain & discharge DR. Saeed Ahmad Al-Shoaib Surgical Consultan Surgery MCS General (Ph-D) Surgical Oncology College of Medicine University of Hadramout (HUCOM)
  • 2. The rectum is 12 – 16 cm long. It extends from the rectosigmoid junction, marked by the fusion of the tenia, to the anal canal, marked by the passage into the pelvic floor musculature.
  • 3. The anal canal is an invagination of ectodermal tissue. The Anorectum develops from fusion of the rectum and the anal canal.
  • 4. Of anal canal Anatomy •The anatomical canal is about 3 cm long and it extends from the anal verge to the dentate line •The surgical anal canal is about 4 cm long and it extends from the anal verge to the anorectal junction
  • 5. • Pecten (anoderm) is a hairless part of the external anal canal that exposed by traction on the rim of the anus ( lined by keratinized stratified squamous epithelium ) •The junction between the Pecten and the large mucosa is known as dentate line . Of anal canal Anatomy
  • 6. Anatomy of the anal canal •The rectum is lined by columnar epithelium •The transitional zone is lined with cuboidal epithelium that lines the anal canal from the columns of morgagni to the dentate line. •Below the dentate line the anal canal is lined by squamous epithelium .
  • 7. 1- Internal sphincter (involuntary ) 2-External sphincter (voluntary) Anal Sphincters :
  • 8. Differences between the upper and the lower halves of the anal canal according to the dentate line Below the dentate line Above the dentate line Ectodermal in origin Endodermal in origin Lined by squamous epithelium Lined by columnar epithelium Under control of the somatic innervation (sensitive to pain, touch , temperature) Under control of the autonomic nervous sys. (sensitive to stretch ) Drains its venous blood via inferoir rectal vein into the systemic venous Drains its venous blood via the sup. rectal vein to the portal venous Drains its lymph to the inguinal lymph nodes. drains its lymph to the internal iliac lymph nodes.
  • 9. Surgical Anatomy • The blood supply of upper ½ of the anal canal is from the superior rectal vessels. Where as that of the lower ½ is supply of the surrounding anal skin the inferior rectal vessels which derives from the internal pudendal ultimately from the internal iliac vessels.
  • 10. Surgical Anatomy • The lower ½ is lined by squamous epithelium and the upper ½ by columnar epithelium so carcinoma of the upper ½ is adenocarcinoma. Where as that arising from the lower part is squamous tumour.
  • 11. Surgical Anatomy • The lymphatic above the mucocutaneous junction drain along the superior rectal vessels to the lumbar lymph nodes, where as below this line drainage is to the inguinal lymph nodes.
  • 12. Surgical Anatomy • The nerve supply to the upper ½ via autonomic plexus and the lower ½ is supplied by the somatic inferior rectal nerves terminal branch of the pudendal nerve. So the lower ½ is sensitive to the prick needle.
  • 13. Normal Function of the Anorectun The normal function of the anorectum is storage and release of intestinal waste products . The rectum functions mainly as a capacitance storage vessel.
  • 14. Dysfunction of the Anorectum •Incontinence: Continence is maintained through rectal compliance, ano –rectal sensation, anorectal reflexes, and anal sphincter function . The nature and quantity of stool and colonic transit are important as well . The incidence of fecal incontinence is difficult to determine because of underreporting and lack of a standard definition of the term .
  • 15. Common symptoms •Anal bleeding •Anal pain and discomfort •Perianal itching and irritation •Something coming down •perianal discharge
  • 16. Common Anal Conditions • Haemorrhoids • Pruritus ani • Perianal abscess • Anal fissure • Anal fistula • Rectal prolapse • Anal in continence • Non malignant strictures • Anal neoplasms
  • 17. Examination of Anus This requires careful attention to circumstances (couch, light, gloves). The Sims (left lateral position) is satisfactory. The examination proceed by; – inspection – digital examination with index finger – proctoscopy – sigmoidoscopy
  • 18. Hemorrhoids Definition : they are engorgement of venous plexi “cushions” that sit in the anorectal junction. Could be External: below the dentate line Internal: above the dentate line Commonly situated at : 3 o ’ clock “left lateral” 7 o ’ clock “right posterior” 11 o ’ clock “right anterior”
  • 19. Classification of internal hemorrhoids First degree: painless bleeding usually associated with defecation Second degree: protrude during defecation but spontaneously reduce Third degree: protrude during defecation and must be manually reduced Forth degree: permanently prolapsed
  • 20. Etiology • Primary Causes: – Hereditary factors e.g, structural weakness of the vein. – Anatomical factors. – Partial congestion. – Chronic constipation. – Sphincteric relaxation.
  • 21. Etiology • Secondary Causes: • pregnancy • venous obstruction • straining on micturation • venous congestion • carcinoma of the rectum
  • 22. Clinical features • Bleeding at defecation • Prolapse • Discharge with pruritus ani • Pain • Thrombosed piles
  • 23. Signs and symptoms Painless bright red rectal bleeding Prolapsing lump Itching Mucus discharge N.B. sever pain is not typically associated with internal hemorrhoids but is commonly seen with thrombosed external hemorrhoids but if pain is present with absence of strangulation look out for other conditions “fissure , hematoma, abcess”
  • 24. Assessment and Diagnosis • Careful history Age, type of bleeding, bowel habit Examination: • Abdominal Examination • Anorectal Examination Investigation: Protoscopy , rigid sigmoidoscopy, colonscopy,Barium enema.
  • 27. Cont. treatment The definite treatment varies according to the degree of hemorroids: 1st degree : sclerotherapy infrared photocoagulation cryothrapy 2nd degree : rubber band ligation 3rd and 4th degree : hemorrhoidectomy
  • 28. Cont. treatment Thrombosed external hemorrhoids should be excised if seen within 48 hr, beyond this time, conservative therapy with analgesics and sitz baths is appropriate.
  • 29. Anal Fissure • Defined as longitudinal tear in the mucosa and skin of the anal canal. • Commonly posterior midline more common in female than male. ( tear in squamous epithelium ). It is the commonest cause of sever anal pain
  • 30. Anal Fissure • The acute anal fissure if not treated becomes chronic anal fissures. As result secondary pathological changes may occurs: – Chronicity – A “sentinel” pile – Hypertrophied anal papilla – Contracture of the anus – Suppuration
  • 31. Anal Fissure • The acute anal fissure if not treated becomes chronic anal fissures. As result secondary pathological changes may occurs: – Chronicity – A “sentinel” pile – Hypertrophied anal papilla – Contracture of the anus – Suppuration
  • 32. Epidemiology Acute or chronic tear in the anal canal usually in posterior midline ( M=F in young adult) Anterior fissure is common in Females. Lateral or multiple fissures should raise suspicion of trauma, inflammatory bowel disease ( Chron ’ s disease), lymphoma, neoplasm or infection.
  • 33. Causes Constipation Inflammatory bowel disease Sexual transmitted disease Post anal surgery
  • 34. Signs and symptoms burning pain is associated with defecation. Bleeding : the blood is usually bright red and associated with acute fissures. Physical exam Acute fissure showing just tear in the anal mucosa. Chronic fissure may reveal a sentinel tag or hypertrophied papilla.
  • 35. Treatment Conservative ( acute fissure ) - fiber supplement - bulk laxatives - stool softner - sitz baths - topical nitroglycerine ointment - Botox
  • 36. Cont. Treatment Surgical ( with chronic fissure to reduce the internal sphincter spasm ) - sphincter dilation - Lateral internal sphincterotomy
  • 37. External Hemorrhoids • Perianal Hematoma • Due to rupture of dilated anal vein as result of sever straining. • Sudden onset of painful lump at the anus. • Swelling tense & tender, bluish in colour covered with smooth shining skin.
  • 38. Treatment • Evacuation if the patient come within 48hours • If patient come late conservative treatment. • If untreated the haematoma undergoes: – resolution – ulceration – suppuration to forms in abscess – fibrosis which give rise to skin tag.
  • 39. Perianal Abscess • The infection usually starts in one of the crypts of Morgagni and extends along the related anal gland to the intersphincteric plane where it forms as abscess. • Soon it tracks in various directions to produce different types of abscesses .
  • 40. Types of Abscess • Perianal abscess (60%) • Ischiorectal abscess (30%) • Sub mucous abscess (5%) • Pelvirectal abscess
  • 41. Perianal Abscess • Patient with recurrent anorectal abscess always consider associated underlying diseases such as Crohn’s, UC, rectal cancer and active TB.
  • 42. Perianal Abscess • Symptoms • Acute pain • High fever • Signs • Swelling • Tenderness with induration • Treatment • Incision and drainage and covered by antibiotics.
  • 43. Fistula in ano • Defined as track lined by granulation tissues, which connects deeply in the anal canal or rectum and superficially on the skin around the anus. • It usually result from an anorectal abscess.
  • 44. Fistula in ano • Anal fistulas have well recognized association with crohn’s disease, UC, TB, colloid carcinoma of the rectum and lympho granuloma venercum.
  • 45. Types of Anal Fistulas • According to whether their natural opening is below or above the anorectal ring • Low level e.g., subcutaneous, low anal, sub mucous. • High level – open into anal canal at or above the anorectal ring e.g., high anal, pelvirectal
  • 46. Park’s Classification • Inter sphincteric (70%) low level anal fistula • Trans-sphincteric (25%) high level anal fistula • Supra sphincteric fistulae (4%). • Extra sphincteric (1%) rare type include the tract passes outside all sphincter muscles to open in the rectum.
  • 47. Good Sall's Rule • Fistulas with external opening in relation to the anterior ½ of the anus tend to be direct type.
  • 48. Clinical features • Persistent discharge which irritates the skin and causes discomfort at the anus may be associated with pain. • External opening may be seen with palpation the tracks is often palpable as cord.
  • 50. • Always sent track for biopsy.
  • 51. Anorectal Tumours • Benign tumours – Epithelial Tumours • Anal warts (virus) • Juvenile polyp • Adenomatous polyps • Villous papilloma • Familial polyposis • Pseudo polyps • Endometrioma
  • 52. Anorectal Tumours • Connective Tissue Tumours – Fibrous polyp – Lipoma – Myoma – Haemangioma – Benign Lymphoma
  • 53. Malignant Tumours of the Anal Canal • The lesion is usually squamous cell carcinoma. • Rarely adenocarcinoma, malignant melanoma or basal cell carcinoma.
  • 54. Squamous cell carcinoma • 5% of all anorectal malignancies. Arising from the stratified squamous epithelium of the lower ½ of the anal canal. • It is disease of elderly. • Squamous cell carcinoma more common in males. • The aetiology of anal carcinoma unknown but chronic irritation or infection may be predisposing factors.
  • 55. Clinical Features • Localized ulcer or raised growth with irregular ulcerated surface. • History of bleeding. • History of pain with discomfort. • Tenesmus with incontinence. • Discharge.
  • 56. Examination • On palpation squamous carcinoma feels hard and woody due to invasion of perianal tissues. • P/R examination may prove impossible because of stenosis or discomfort. • Inguinal LN are examined carefully as they receive lymph from the lower anal canal and perianal region and may be the site of metastasis.
  • 57. Treatment • Above the pectinate line – Abdomino perineal excision • Below the pertinate line – local excision. – If inguinal LN metastasis present should be removed by block dissection.
  • 59. Rare Malignant Anal Tumor's • Adenocarcinoma • Basal cell carcinoma • Malignant melanoma
  • 60. Benign strictures • Stricture of the anus and rectum may be: – Congenital – Postoperative – Inflammatory
  • 61. Clinical features • Progressive difficulty in defecation • In cases of inflammatory strictures – Bleeding – Discharge – Tenesmus • Late cases subacute intestinal obstruction
  • 62. Diagnosis • Rectal examination reveals the location type and degree of the stenosis. • Proctoscopy • Biopsy