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“EFRUZHU
CANCER (CARCİNOGENESİS) THEORY”
FEEDBACK CONTROL MECHANİSM İN CANCER CELL.
DETAİLED COMMENTS 18. =VOİCED 18.
AUTOCONTROL İS POSSİBLE BETWEEN SAME- İDENTİC DYNAMİCS.
BUT CANCER CELL HAS TWO DİFFERENT ORİGİN DYNAMİCS.
1.MUTATİONAL-FOREİGN ORİGİN DNA SEQUENCE THAT MAKE
DOMİNANTİVE EFFECT ONTO CELL METABOLİC REGULATİON BUT
2.NORMAL DNA SEQUENCE İS RESESSİVE EFFECT ONTO CELLULAR
METHABOLİCAL REGULATİON SO THESE SEPERATE ORİGİNS CAUSE
ASİMETRİC RELATİON AND COOPERATİON İMPOSSİBLE THAT
GOVERN TOGETHER AS PHYSİOLOGİC HOMEOSTATİC BALANCED.SO
FEEDBACK MECHANİSM İS WORKİNG UNSUCCESSFUL GENERALLY
RESESSİF DYNAMİC MAKES TRUE BEHAVİOUR.

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S18 presentation18

  • 1. “EFRUZHU CANCER (CARCİNOGENESİS) THEORY” FEEDBACK CONTROL MECHANİSM İN CANCER CELL. DETAİLED COMMENTS 18. =VOİCED 18. AUTOCONTROL İS POSSİBLE BETWEEN SAME- İDENTİC DYNAMİCS. BUT CANCER CELL HAS TWO DİFFERENT ORİGİN DYNAMİCS. 1.MUTATİONAL-FOREİGN ORİGİN DNA SEQUENCE THAT MAKE DOMİNANTİVE EFFECT ONTO CELL METABOLİC REGULATİON BUT 2.NORMAL DNA SEQUENCE İS RESESSİVE EFFECT ONTO CELLULAR METHABOLİCAL REGULATİON SO THESE SEPERATE ORİGİNS CAUSE ASİMETRİC RELATİON AND COOPERATİON İMPOSSİBLE THAT GOVERN TOGETHER AS PHYSİOLOGİC HOMEOSTATİC BALANCED.SO FEEDBACK MECHANİSM İS WORKİNG UNSUCCESSFUL GENERALLY RESESSİF DYNAMİC MAKES TRUE BEHAVİOUR.