3. Critically Ill Patient (ABCDE Assessment)
o The ABCDE approach
ļ¼rapid bedside assessment of a deteriorating/ critically ill
patient
ļ¼designed to provide the initial management of life-threatening
conditions in order of priority
ļ¼used a structured method to keep the patient alive and to
achieve the first steps to improvement, rather than making a
definitive diagnosis (Smith 2003).
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4. The ABCDE approach: Underlying principles
ā¢ Use the ABCDE approach to assess and treat the patient.
ā¢ Do a complete initial assessment and re-assess regularly.
ā¢ Treat life-threatening problems before moving to the next part of
assessment.
ā¢ Assess the effects of treatment
ā¢ Recognize when you will need extra help
ā¢ Use all members of the team
ā¢ Communicate effectively
Ā» The aim of the initial treatment is to keep the patient alive, and achieve
some clinical improvement. This will buy time for further treatment and
making a diagnosis.
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5. The ABCDE approach: Underlying principles
3/19/2024 Critical care, Year II MSc Anesthesia, Jan 2019 5
6. The ABCDE approach: First steps
o Ensure personal safety
o First look at the patient in general
Ā» If the patient is awake, ask āHow are you?ā. If the patient appears unconscious, shake
him and ask āAre you alright?ā If he responds normally he has a patent airway, is
breathing and has brain perfusion.
Ā» If he speaks only in short sentences, he may have breathing problems. Failure of the
patient to respond is a clear marker of critical illness.
o Rapid āLook, Listen and Feelā of the patient should take about 30 s and will
often indicate a patient is critically ill and there is a need for urgent help.
Ā» If the patient is unconscious, unresponsive, and is not breathing normally start CPR
o Monitor the vital signs early.
Ā» Attach a pulse oximeter, ECG and a NIBP m to all critically ill patients, as soon as
possible.
o Insert an intravenous cannula as soon as possible.
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7. The ABCDE approach: First steps
3/19/2024 Critical care, Year II MSc Anesthesia, Jan 2019 7
8. Critically Ill Patient (ABCDE Assessment)
Airway (A)
ā¢ Airway obstruction is an emergency. Get expert help immediately.
Ā» Untreated, airway obstruction causes hypoxia and risks damage to the
brain, kidneys and heart, cardiac arrest, and death.
3/19/2024 Critical care, Year II MSc Anesthesia, Jan 2019 8
Immediate Management
A B C D E
9. Airway (A)
o The aim of the airway assessment is to establish the patency of the
airway and assess the risk of deterioration in the patientās ability to
protect their airways.
ļ¼patientās airway can be patent (if the patient is talking),
ļ¼partially obstructed (if air entry is diminished and often noisy) or
ļ¼completely obstructed (if there are no breath sounds at the mouth or
nose) (Resuscitation Council 2018).
3/19/2024 Critical care, Year II MSc Anesthesia, Jan 2019 9
10. Airway (A)
o Look for the signs of airway obstruction:
ļ¼Airway obstruction causes paradoxical chest and abdominal movements (āsee-sawā
respirations) and the use of the accessory muscles of respiration.
ļ¼Central cyanosis is a late sign of airway obstruction.
ļ¼In complete airway obstruction, there are no breath sounds at the mouth or
nose. In partial obstruction, air entry is diminished and often noisy.
ļ¼In the critically ill patient, depressed consciousness often leads to airway
obstruction
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11. Airway (A)
Decreased level of consciousness Patent airway
3/19/2024 Critical care, Year II MSc Anesthesia, Jan 2019 11
12. Airway (A)
o According to Resuscitation Council (2017), Treat airway obstruction as a
medical emergency:
ā Obtain expert help immediately. Untreated, airway obstruction causes
hypoxemia (low PaO2) with the risk of hypoxic injury to the brain, kidneys
and heart, cardiac arrest, and even death.
ā In most cases, only simple methods of airway clearance are required (e.g.
airway opening maneuvers, airways suction, insertion of an oropharyngeal or
nasopharyngeal airway).
ļ¶Tracheal intubation may be required when these fail.
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13. Airway (A)
ā¢ Give oxygen at high concentration:
ā Provide high-concentration oxygen using a mask with oxygen reservoir.
Ā» Ensure that the oxygen flow is sufficient (usually 15 L min-1) to prevent
collapse of the reservoir during inspiration.
Ā» In acute respiratory failure, aim to maintain an oxygen saturation of 94ā
98%.
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14. Breathing (B)
o Breathing function should only be assessed and managed after the airway
has been judged as adequate.
ļ¼ Assessment of breathing is designed to detect signs of respiratory distress or
inadequate ventilation (Smith 2003).
o During the immediate assessment of breathing, it is vital to diagnose and
treat immediately life-threatening conditions
e.g. acute severe asthma, pulmonary edema, tension pneumothorax, and massive
haemothorax.
3/19/2024 Critical care, Year II MSc Anesthesia, Jan 2019 14
15. Breathing (B)
o Look, listen and feel for the general signs of respiratory distress:
ļ¼Sweating, central cyanosis, use of the accessory muscles of respiration, and
abdominal breathing.
o Count the respiratory rate.
ļ¼An increasing respiratory rate is a marker of illness and a warning that the
patient may deteriorate suddenly.
o Assess the depth of each breath, the pattern (rhythm) of respiration and
whether chest expansion is equal on both sides.
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16. Breathing (B)
o Note any chest deformity; look for a raised JVP (e.g. in acute severe
asthma or a tension pneumothorax) and note the presence and patency of
any chest drains
o Record the inspired oxygen concentration (%) and the SpO2 reading of
the pulse oximeter.
o Listen to the patientās breath sounds a short distance from his face:
ļ¼ Rattling airway noises indicate the presence of airway secretions
ļ¼ Stridor or wheeze suggests partial, but significant, airway obstruction.
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17. Breathing (B)
o Percuss the chest:
ļ¼ hyper-resonance may suggest a pneumothorax; dullness usually indicates consolidation or
pleural fluid.
o Auscultate the chest:
ļ¼ bronchial breathing indicates lung consolidation; absent or reduced sounds suggest a
pneumothorax or pleural fluid or lung consolidation caused by complete obstruction.
o Check the position of the trachea in the suprasternal notch:
ļ¼ deviation to one side indicates mediastinal shift (e.g. pneumothorax, lung fibrosis or
pleural fluid).
o Feel the chest wall to detect surgical emphysema or crepitus;
ļ¼ suggesting a pneumothorax until proven otherwise.
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18. Breathing (B)
o The specific treatment of respiratory disorders depends upon the cause.
o Nevertheless, all critically ill patients should be given oxygen.
ļ¼In a subgroup of patients with COPD, high concentrations of oxygen may
depress breathing (i.e. they are at risk of hypercapnic respiratory failure
- often referred to as type 2 respiratory failure).
ļ¼These patients will also sustain end-organ damage or cardiac arrest if
their blood oxygen tensions are allowed to decrease.
ļ In this group, aim for a lower than normal PaO2 and oxygen saturation. Give oxygen via
a Venturi mask (4 L min-1) initially and reassess.
ļ Aim for target SpO2 range of 88ā92% in most COPD patients
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19. Breathing (B)
o If the patientās depth or rate of breathing is judged to be inadequate, or
absent, use bag-mask ventilation to improve oxygenation and ventilation,
whilst calling immediately for expert help.
ļ¼ In cooperative patients who do not have airway obstruction consider the use of
non-invasive ventilation (NIV).
ļ¼ In patients with an acute exacerbation of COPD, the use of NIV is often helpful
and prevents the need for tracheal intubation and invasive ventilation.
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21. Circulation (C)
ļ¼ Assessment of circulation should be
undertaken only once the airway and
breathing have been assessed and
appropriately treated.
ļ¼ The aim of assessing the circulatory
system is to determine the
effectiveness of the cardiac output.
(Mallet 2013).
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22. Circulation (C)
o Look at the color of the hands and digits
ļ¼ are they blue, pink, pale or mottled?
o Assess the limb temperature by feeling the patientās hands
ļ¼ are they cool or warm?
o Measure the capillary refill time (CRT). Apply cutaneous pressure for 5 s
on a fingertip held at heart level with enough pressure to cause
blanching. The normal value for CRT is usually < 2 s.
ļ¼ A prolonged CRT suggests poor peripheral perfusion. Other factors (e.g. cold
surroundings, poor lighting, old age) can prolong CRT.
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23. Circulation (C)
o Assess the state of the veins:
ļ¼they may be underfilled or collapsed when hypovolaemia is present.
o Count the patientās pulse rate.
ļ¼Palpate peripheral and central pulses, assessing for presence, rate, quality,
regularity and equality.
ļ¼Barely palpable central pulses suggest a poor cardiac output, whilst a bounding
pulse may indicate sepsis.
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24. Circulation (C)
o Auscultate the heart.
ļ¼ Is there a murmur or pericardial rub? Are the heart sounds difficult to hear? Look
for other signs of a poor cardiac output, such as reduced conscious level and, if the
patient has a urinary catheter, oliguria (urine volume < 0.5 mL kg-1 h-1).
o Look thoroughly for external hemorrhage from wounds or drains
ļ¼(e.g. thoracic, intra-peritoneal, retroperitoneal or into gut). Intra-thoracic,
intra-abdominal or pelvic blood loss may be significant, even if drains are
empty.
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25. Circulation (C)
o The specific treatment of cardiovascular collapse depends on the cause,
ļ¼should be directed at fluid replacement, hemorrhage control and restoration of
tissue perfusion.
o Seek the signs of conditions that are immediately life threatening
ļ¼e.g. cardiac tamponade, massive or continuing hemorrhage, septic shock), and
treat them urgently.
o Insert one or more large (14 or 16 G) intravenous cannula. Use short,
wide-bore cannula, because they enable the highest flow.
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26. Circulation (C)
o Give a bolus of 500 mL of warmed crystalloid solution (e.g. Hartmannās
solution or 0.9% sodium chloride) over less than 15 min if the patient is
hypotensive.
o Use smaller volumes (e.g. 250 mL) for patients with known cardiac failure or
trauma and use closer monitoring.
o Reassess the heart rate and BP regularly (every 5 min), aiming for the
patientās normal BP or, if this is unknown, a target > 100 mmHg systolic.
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27. Circulation (C)
o If symptoms and signs of cardiac failure (dyspnea, increased HR, raised
JVP, a third heart sound and pulmonary crackles on auscultation) occur,
decrease the fluid infusion rate or stop the fluids altogether.
o Seek alternative means of improving tissue perfusion (e.g. inotropes or
vasopressors).
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28. Circulation (C)
o According to the Resuscitation Council 2011, the specific treatment for
circulation problems depends on the cause
ļ¼fluid replacement, restoration of tissue perfusion and hemorrhage control will
usually be necessary.
o Remember to continuously reassess the patientās HR and BP, with the
target of restoring them to the patientās normal physiological state, or,
if this is not known, aim for >100mmHg systolic
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29. Disability (D)
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o This assessment involves reviewing the patientās neurological status, and should only be
undertaken once A, B and C above have been optimized, as these parameters can all
affect the patientās neurological condition.
o Level of consciousness: conduct a rapid assessment of the patientās level of
consciousness using the AVPU system (Mallet 2013)
30. Disability (D)
o If youāre concerned about the patientās level of consciousness, then use a
more in-depth assessment, such as GCS, and seek further help
(Resuscitation Council 2017).
ļ¼ Pupil reaction: examine the patientās pupils for size, shape and reaction to light.
ļ¼ Blood glucose levels: a blood glucose measurement should be taken to exclude
hypoglycemia. (Resuscitation Council UK 2017).
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31. Disability (D)
o Common causes of unconsciousness include
ļ¼ profound hypoxia, hypercapnia, cerebral hypoperfusion, or the recent administration
of sedatives or analgesic drugs.
o Check the patientās drug chart for reversible drug-induced causes of
depressed consciousness.
ļ¼ Give an antagonist where appropriate (e.g. naloxone for opioid toxicity).
ļ¼ Examine the pupils (size, equality and reaction to light).
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32. Disability (D)
o Measure the blood glucose to exclude hypoglycemia and follow local
protocols for management of hypoglycemia.
o For example, if the blood sugar is less than 4.0 mmol L-1 in an unconscious patient, give
an initial dose of 50 mL of 10% glucose solution intravenously. If necessary, give
further doses of intravenous 10% glucose every minute until the patient has fully
regained consciousness, or a total of 250 mL of 10% glucose has been given.
o Unconscious patients whose airways are not protected should be nursed
in the lateral position (Resuscitation Council 2011).
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33. Exposure (E)
o To examine the patient properly full exposure of the body may be
necessary.
ļ¼Respect the patientās dignity and minimize heat loss.
o By the time the assessment reaches this stage (exposure), there should
be a good understanding of the patientās problems (Mallet 2013).
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34. Exposure (E)
o Conduct a thorough examination of the patientās body for abnormalities,
checking the patientās skin for the presence of rashes, swelling, bleeding
or any excessive losses from drains.
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35. Exposure (E)
o Look at the patientās medical notes, medicine charts, observation charts
and results from investigations for any additional evidence that can
inform the assessment and ongoing plan of care for the patient.
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36. Exposure (E)
o Remember to document all the assessments, treatments and responses to
treatment in the patientās clinical notes.
o Always seek help from more senior or experienced practitioners if the
patient is continuing to deteriorate.
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37. 3/19/2024 Critical care, Year II MSc Anesthesia, Jan 2019 37
Immediate Management
A B C D E
Full patient assessment
Chart review
History and systematic examination
Available results
38. Chart review
CIRCULATION Heart rate, rhythm, BP
HYDRATION IV fluids, Urine output, fluid balance
ALERTNESS Glasgow Coma Score
RESPIRATORY Respiratory rate, Inspired oxygen
concentration, SaO2
TEMPERATURE Temperature
SPECIAL FEATURES Drains, special aspects of patients
disease
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39. Review medication chart
ā¢ Has their chronic medication been prescribed and given
ā¢ Have prescribed antibiotics and fluids been given
ā¢ Are they taking any medications with side effects that might be
influencing the current state
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40. History
o Take history from referring staff but do not rely completely on it ā
review the notes and talk to the relatives
o Many critically ill patients have co-morbidities which affect their
chances of survival
ļ¼ eg: COPD or IHD ā an exercise tolerance history is important and may not be documented
o Diabetes ā how well is it controlled, what systems are involved
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41. AMPLE history
o Allergies
o Medication
o Past medical history
o Last meal
o Events
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42. Examination
o Full examination
ā Vital signs
ā Systems
o Focused examination on systems/regions involved
ļ¼surgical site
ļ¼underlying disease
ļ¼identify problem areas
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43. Available results
o Results
ā Biochemistry ā U & Es, LFTs, glucose, ABGs
ā Haematology ā FBC and coagulation
ā Microbiology
ā Radiology ā reports and films
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44. 3/19/2024 Critical care, Year II MSc Anesthesia, Jan 2019 44
Immediate Management
A B C D E
Full patient assessment
Chart review
History and systematic examination
Available results
Decide and plan
45. Decide
o Is the patient stable or unstable?
o Do you have a diagnosis?
o Which organ systems are involved?
o Which organ systems need support? - eg.
ā Ventilation
ā Inotropic support
ā Dialysis/haemofiltration
o What specific investigations do you need?
3/19/2024 Critical care, Year II MSc Anesthesia, Jan 2019 45
46. Categories of organ support
3/19/2024 Critical care, Year II MSc Anesthesia, Jan 2019 46
47. Categories of organ support ā¦
3/19/2024 Critical care, Year II MSc Anesthesia, Jan 2019 47
48. 3/19/2024 Critical care, Year II MSc Anesthesia, Jan 2019 48
Immediate Management
A B C D E
Full patient assessment
Chart review
History and systematic examination
Available results
Decide and plan
Stable patient
Daily management plan
49. Stable patients
o Daily management plan
o Fluids & nutrition
o Drugs
ļ¼ Therapeutic and prophylactic
o Physiotherapy
o What level of care do they require?
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50. 3/19/2024 Critical care, Year II MSc Anesthesia, Jan 2019 50
Immediate Management
A B C D E
Full patient assessment
Chart review
History and systematic examination
Available results
Decide and plan
Stable patient
Daily management plan
Unstable / Unsure
Diagnosis required
Specific investigations
Definitive treatment
51. Unstable patient (or unsure)
o Investigations
ļ¼targeted not blind
ļ¼supports clinical assessment
o Definitive treatment
ļ¼start as soon as possible
ļ¼medical, surgical, radiological
o Co-ordinate and communicate
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52. Communication
o Case notes ā problem orientated approach
o Record plan and check progress
o Nursing staff - set parameters and agree plan
o Colleagues
o Patient and relatives
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54. FAST HUGS BID
o Objective:
ļ¼To introduce the Fast Hug mnemonic (Feeding, Analgesia, Sedation,
Thromboembolic prophylaxis, Head-of-bed elevation, stress Ulcer
prevention, and Glucose control) as a means of identifying and
checking some of the key aspects in the general care of all
critically ill patients.
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55. FAST HUGS BID
o Bowels
o Indwelling catheters removal
o De-escalation ā antibiotics, inotropes, FiO2 etc
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56. The seven components of the Fast Hug approach
3/19/2024 Critical care, Year II MSc Anesthesia, Jan 2019 56
57. FAST HUG
ā¢ Why feeding?
ā Adequate nutrition is vital for critically ill
ā¢ Poor wound healing
ā¢ Post operative complications
ā¢ Sepsis
ā Critically ill patients
ā¢ Are often already malnourished on admission
ā¢ Mechanically ventilated ICU patients have a malnutrition
rate of 38% to 100%.
ā¢ Have greatly increased calorie requirements
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58. FAST HUG ā¦
o 5.6 kJ/kg/day is an acceptable and achievable target
intake
ļ¼But patients with sepsis or trauma may require almost
twice as much energy during the acute phase of their
illness (Proc Nutr Soc 2004)
o If oral feeding is not possible, enteral nutrition is
preferred to parenteral nutrition and should be
started early, preferably within 24ā48 hrs of ICU
admission (Intensive Care Med 2008).
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59. FAST HUG ā¦
o The optimal constituents of feeding solutions remain under
debate, but literatures recommend that;
ļ¼ solutions containing fish oils, borage oils, and antioxidants
should be considered for patients with ARDS and
ļ¼ glutamine-enriched formulas should be considered for
patients with severe burns and trauma (JPEN J Parenter Enteral
Nutr 2003).
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60. FAST HUG
o Why Ensure Analgesia?
ļ Critically ill patients feel pain due to:
ā¢ Their primary illness e.g. pancreatitis / surgical wounds
ā¢ Routine procedures e.g. turning / suctioning / dressings
ļ Humanity & Compassion
ļ Psychological Effects
ļ Physiological Effects
ļ Metabolic effects
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61. FAST HUG ..
o Opioids are the most widely used, although they may be
combined with NSAIDS or acetaminophen for certain
patients.
ļ¼The most commonly used opioids are morphine, fentanyl, and
remifentanil.
o Continuous infusions of analgesic drugs or regularly
administered doses are more effective than bolus doses
given āas needed,ā
o Care should be taken to ensure analgesia is adequate
but not excessive.
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62. FAST HUG
o Why Sedate?
ļ¼Tolerance of invasive and uncomfortable monitoring and treatment
procedures
ļ¼Reduce oxygen consumption by reducing patient arousal and activity
ļ¼Promote amnesia for events in the ICU
ļ¼Specific treatment for conditions:
ļ¼ Epilepsy / Tetanus
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63. FAST HUG
o No rules governing how much to give and how often, and
sedative administration must be titrated to the individual.
o Increase the dose of sedative to have a calm and quiet
patient, oversedation is associated with harmful effects
o Using the āCCC (calm, comfortable, collaborative) ruleā to
help determine whether patients are appropriately
sedated.
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64. FAST HUG
o How to Sedate?
ļ¼Benzodiazepines (commonly midazolam)
ļ¼Intravenous anesthetic agents
ļ¼Major tranquilizers
ļ¼Analgosedation
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65. FAST HUG
o Oversedation
ļ¼Increased risk of nosocomial pneumonia
ļ¼Increased rate of Neurological Investigation (CT)
ļ¼Prolonged stay in the ICU + Polyneuropathy
ļ¼Increased incidence of post ā traumatic stress disorder and
depression
ļ¼Increased use of inotropes
o Monitoring Sedation
ļ¼Ramsay Sedation Score
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66. FAST HUG
o Why Ensure Thromboprophylaxis?
ļ¼Critically ill patients have numerous risk factors
ļ¼Without prophylaxis rates of deep-vein thrombosis range
between 13% and 31%
ļ May be much higher in trauma patients
ļ¼Must weigh up risks vs bleeding complications
o How Ensure Thromboprophylaxis?
ļ¼Mechanical
ļ¼Pharmacological
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67. FAST HUG
o Head of the bed inclined at 45 degrees
ļ¼Can decrease gastroesophageal reflux in mechanically ventilated
patients
ļ¼Can reduce rates of nosocomial pneumonia
ļ¼Is indicated (20 ā 30 degrees) in some patients e.g.
raised ICP
o Take care
o Donāt let the patient slide off the bed!
o Cerebral perfusion
o Pressure areas
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68. FAST HUG
o Peptic ulceration is related to protective barrier loss
ā Acid or biliary damage of the underlying mucosa
ā Barrier loss occurs secondary to critical illness
o Direct damage from feeding tubes
ā especially at the lower oesophagus
o Mucosal damage from tissue hypoperfusion
o Highest risk
ā Prolonged mechanical ventilation
ā Concurrent coagulopathy.
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69. FAST HUG
How to Ensure Ulcer Prophylaxis?
o Small-bore feeding tubes
o Enteral nutrition
o Adequate tissue perfusion (optimal haemodynamics)
o Prophylactic drug therapy (Controversial)
ā H2 antagonists (ranitidine)
ā Proton pump inhibitors (omeprazole, pantoprazole)
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70. FAST HUG
o Mayo Clinic 2004 Study: āKeeping blood glucose levels < 140
mg/dL ā
ā 29.3% decrease in hospital mortality
ā 10.8% reduction in length of ICU stay
o Care to avoid hypoglycemia ā disaster
o Many units now aim to keep blood sugar levels below about
150 mg/dL
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In patients at risk of hypercapnia respiratory failure aim for an oxygen saturation of 88ā92%.
Look for the general signs of respiratory distress such as sweating, the effort needed to breathe, abdominal breathing and central cyanosis. Count patientās respiratory rate: the normal respiratory rate in adults is between 12 ā 20 breaths/minute (Prytherch 2010). The respiratory rate should be measured by counting the number of breaths that a patient takes over one minute through observing the rise and fall of the chest. A high respiratory rate is a marker of illness or an early warning sign that the patient may be deteriorating (Resuscitation Council 2011).
Assess the depth of each breath the patient takes, the rhythm of breathing and whether chest movement is equal on both sides.
The pulse oximeter does not detect hypercapnia. If the patient is receiving supplemental oxygen, the SpO2 may be normal in the presence of a very high PaCO2.
Measure patientās peripheral oxygen saturation using pulse oximeter applied to the end of the patientās finger. The British Thoracic Society (OāDriscoll et al. 2008), recommends a target oxygen saturation of between 94%-98%, with a minimum level of 88%. However, the pulse oximeter does not detect hypercapnia (carbon dioxide retention) (Resuscitation Council UK 2011).
Rattling airway noises indicate the presence of airway secretions, usually caused by the inability of the patient to cough sufficiently or to take a deep breath.
Stridor or wheeze suggests partial, but significant, airway obstruction.
Blood gas analysis: This test provides a valuable respiratory assessment about the levels of oxygen, carbon dioxide in the blood and the blood PH. The test provides more in-depth information about the effectiveness of respiratory function than pulse oximetry (Mallet 2013).
Assess air entry using a stethoscope to confirm whether air is entering the lungs, whether both lungs have equal air entry and whether there are any additional abnormal breath sounds such as wheezing and crackles (Mallet 2013).
The specific treatment of respiratory disorders depends upon the cause.
Nevertheless, all critically ill patients should be given oxygen. In a subgroup of patients with COPD, high concentrations of oxygen may depress breathing (i.e. they are at risk of hypercapnic respiratory failure - often referred to as type 2 respiratory failure). Nevertheless, these patients will also sustain end-organ damage or cardiac arrest if their blood oxygen tensions are allowed to decrease. In this group, aim for a lower than normal PaO2 and oxygen saturation. Give oxygen via a Venturi 28% mask (4 L min-1) or a 24% Venturi mask (4 L min-1) initially and reassess. Aim for target SpO2 range of 88ā92% in most COPD patients, but evaluate the target for each patient based on the patientās arterial blood gas measurements during previous exacerbations (if available). Some patients with chronic lung disease carry an oxygen alert card (that documents their target saturation) and their own appropriate Venturi mask.
The specific treatment of respiratory disorders depends upon the cause. However, regardless of the cause, expert help should be called immediately (Resuscitation Council 2011).
If the patientās breathing is compromised, position patient appropriately (usually in an upright position).
Cardiac output is the volume of blood ejected from the heart each minute
Measure the capillary refill time (CRT). Apply cutaneous pressure for 5 s on a fingertip held at heart level (or just above) with enough pressure to cause blanching. Time how long it takes for the skin to return to the colour of the surrounding skin after releasing the pressure. The normal value for CRT is usually < 2 s. A prolonged CRT suggests poor peripheral perfusion. Other factors (e.g. cold surroundings, poor lighting, old age) can prolong CRT.
Even in shock, the blood pressure may be normal, because compensatory mechanisms increase peripheral resistance in response to reduced cardiac output.
A low diastolic blood pressure suggests arterial vasodilation (as in anaphylaxis or sepsis).
A narrowed pulse pressure (difference between systolic and diastolic pressures; normally 35ā45 mmHg) suggests arterial vasoconstriction (cardiogenic shock or hypovolaemia) and may occur with rapid tachyarrhythmia.
Auscultate the heart. Is there a murmur or pericardial rub? Are the heart sounds difficult to hear? Does the audible heart rate correspond to the pulse rate?Look for other signs of a poor cardiac output, such as reduced conscious level and, if the patient has a urinary catheter, oliguria (urine volume < 0.5 mL kg-1 h-1).Look thoroughly for external haemorrhage from wounds or drains or evidence of concealed haemorrhage (e.g. thoracic, intra-peritoneal, retroperitoneal or into gut). Intra-thoracic, intra-abdominal or pelvic blood loss may be significant, even if drains are empty.
Blood pressure (BP): is an indication of the effectiveness of the cardiac output. Measure the patientās blood pressure as soon as possible; low blood pressure (relative to the normal blood pressure of the patient) is often a late sign in the deteriorating patient and can be an adverse clinical sign (Mallet 2013).Gauge the patientās peripheral skin temperature by feeling their hands to determine whether they are warm or cool. Take blood from the cannula for routine haematological, biochemical, coagulation and microbiological investigations, and cross-matching, before infusing intravenous fluid.
Feel and measure the patientās heart rate: assess the patientās heart rate relative to their normal physiological condition. Heart rate is usually felt by palpating the pulse from an artery that lies near the surface of the skin, such as the radial artery in the wrist. The pulse should be felt for presence, rate, quality and regularity (Smith 2003). If there are any abnormalities detected such as thread pulse, then a 12 lead electrocardiogram (ECG) should be undertaken (Mallet 2013). If the patient does not improve, repeat the fluid challenge. Seek expert help if there is a lack of response to repeated fluid boluses.
Patientās temperature: normal temperatures range from 36.8Oc to 37.9Oc. If a patient has a raised temperature, it is important to understand the reason for this, as the treatment will vary depending on the cause (Mallet 2013).Capillary refill time (CRT): a simple measure of peripheral circulation. The patientās hand should be at the level of their heart. Press the top of the patientās finger for 5 seconds to blanch the skin, and then release (Mangione 2000). The normal value for CRT is usually < 2 seconds. A prolonged CRT could indicate poor peripheral perfusion (Resuscitation Council UK 2011).Look for other signs of a poor cardiac output such as a decreased level of consciousness. If the patient has a urinary catheter, check for reduced urine output (urine output of < 0.5 mL kg/hr) and assess for any signs of external bleeding from wounds or drains (Resuscitation Council UK 2011). If the patient has primary chest pain and a suspected ACS, record a 12-lead ECG early. Immediate general treatment for ACS includes:Aspirin 300 mg, orally, crushed or chewed, as soon as possible. Nitroglycerine, as sublingual glyceryl trinitrate (tablet or spray).
Awake (A): observe if the patient can open his/her eyes, takes interest and responds normally to his/her environment. This would be assessed as āawakeā.
Responding to voice (V): if the patient has his/her eyes closed and only opens them when spoken to, this would be assessed as āvoiceā. However, a judgement should be made when a patient is naturally sleeping, as physiologically this is not considered an altered level of consciousness.
Responding to pain (P): the patient who doesnāt respond to voice should be shaken gently to try to elicit a response. If there is still no response, then painful stimuli should be applied.
If the patient responds to painful stimuli, then the level of consciousness is assessed as āresponds to painā. Examples of painful stimuli include the ātrapezius squeezeā.
Unresponsive (U): a patient not responding to pain is āunresponsiveā.
Measure the blood glucose to exclude hypoglycaemia using a rapid finger-prick bedside testing method. In a peri-arrest patient use a venous or arterial blood sample for glucose measurement as finger prick sample glucose measurements can be unreliable in sick patients. Follow local protocols for management of hypoglycaemia. For example, if the blood sugar is less than 4.0 mmol L-1 in an unconscious patient, give an initial dose of 50 mL of 10% glucose solution intravenously. If necessary, give further doses of intravenous 10% glucose every minute until the patient has fully regained consciousness, or a total of 250 mL of 10% glucose has been given. Repeat blood glucose measurements to monitor the effects of treatment. If there is no improvement consider further doses of 10% glucose. Specific national guidance exists for the management of hypoglycaemia in adults with diabetes mellitus.
Unfortunately, there is no specific ānutritionā marker, and it is not practical to perform indirect calorimetryon all patients, but a clinical assessment including weight loss measurement is probably as reliable as more complex tests
Mechanically ventilated ICU patients have a malnutrition rate of 38% to 100%.[141] Therefore, nutritional support guidelines recommend starting enteral nutrition early, that is, within 24 to 48 hours of ICU admission, to reduce infectious complications and duration of hospitalization
Pain is not always easy to assess in critically ill patients, who may be unable to express themselves; for such patients, subjective
measures of pain-related behavior (e.g., facial expression, movement) and physiologic indicators (e.g., heart rate, blood
pressure) should also be used (26).
Pain is not always easy to assess in critically ill patients, who may be unable to express themselves; for such patients, subjective
measures of pain-related behavior (e.g., facial expression, movement) and physiologic indicators (e.g., heart rate, blood
pressure) should also be used (26).
Although it may be easier to increase the dose of sedative to have a calm and quiet patient, oversedation is associated with harmfuleffects, including an increased risk of venous thrombosis, decreased intestinal motility, hypotension, reduced tissue oxygen extraction capabilities, increasedrisk of ICU polyneuropathy, prolonged ICU stay, and increased costs.
have shown that daily transient discontinuation of sedation may reduce the length of ICU stay and the need for imaging procedures, althoughone may argue that if sedation is titrated continuously, as recommended in current guidelines (26), there should be noneed to discontinue it once a day. The use of sedation scales has been advocated
Although it may be easier to increase the dose of sedative to have a calm and quiet patient, oversedation is associated with harmfuleffects, including an increased risk of venous thrombosis, decreased intestinal motility, hypotension, reduced tissue oxygen extraction capabilities, increasedrisk of ICU polyneuropathy, prolonged ICU stay, and increased costs.
have shown that daily transient discontinuation of sedation may reduce the length of ICU stay and the need for imaging procedures, althoughone may argue that if sedation is titrated continuously, as recommended in current guidelines (26), there should be noneed to discontinue it once a day. The use of sedation scales has been advocated
Although it may be easier to increase the dose of sedative to have a calm and quiet patient, oversedation is associated with harmfuleffects, including an increased risk of venous thrombosis, decreased intestinal motility, hypotension, reduced tissue oxygen extraction capabilities, increasedrisk of ICU polyneuropathy, prolonged ICU stay, and increased costs.
daily transient discontinuation of sedation may reduce the length of ICU stay and the need for imaging procedures, althoughone may argue that if sedation is titrated continuously, as recommended in current guidelines (26), there should be noneed to discontinue it once a day. The use of sedation scales has been advocated