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Iodine for Mother
and Fetus
IODINE
 It is an essential micro-nutrient ( Trace element ).
 It was discovered in 1811 by COURTOIS.
 Storage in- Thyroid gland It is necessary for the
synthesis of T3 & T4 thyroid hormones.
 Iodine present in food & water as- Inorganic iodide
 Absorbed from- Gut
IODINE
WENEEDIT.
WECAN’TLIVEWITHOUTIT!
SourceofIodine
IODIDES
TherapeuticUses
LOCATIONof
ThyroidGland
KeepingItRight:
TheBody’sNatural
ThyroidHormone
“Thermostat”
Synthesisand
Releaseofthyroid
hormones
Iodine
Deficiency – A
Disease of
The Soil
CAUSESOF
IODINE
DEFICIENCY
DietsThatMay
CauseIodine
Deficiency
 Diets without ocean fish or sea vegetables
 Inadequate use of iodized salt including low-sodium
diets
 Diets high in the consumption of bakery products (e.g.,
breads, pasta) which contain bromide
 Vegan and vegetarian diets
EffectofIodine
Deficiency
Disorder
Requirements
IodineDeficiency
Disorders
&HumanLife
Cycle
1) Unborn Child (Foetus)
2) Newborn Child (Neonate)
3) Child & Adolescent
4) Adult
Iodine Deficiency Iodine Sufficiency
Iodine Deficiency is the single most
common cause of preventable mental retardation
Brain Cell Growth
• Diminished brain cell branching due to iodine deficiency
• Diminished branching  Less connections  Lower IQ
Brain cell
Branching
And
IQ
Implications
of
Lossof I.Q.
1. Poor Scholastic performance
2. Frequent failures / grade repetitions
3. Absenteeism / Drop outs
4. Major implications: Education for All
5. Consequent economic & social effects
6. Drain on Human Resource Development
Iodine Deficiency = Goitre =
Visible Swelling
No Pain = Not a cause of
Mortality = Cosmetic problem
Cretinism rare
Historic
View
Current
View
Mental & Physical growth
Loss of Energy-hypothyroidism
Learning Disability, Poor Motivation
Child Development and Child Survival
Human Resource Development
IDD – The
Hourglass
CRETINISM
1% - 10%
Cretinism
5% - 30%
Some brain damage
30% - 70%
Loss of energy due to hypothyroidism
Iceberg
of
IDD
Assessingtheseverityofiodine deficiencyinthe
community:
CLASSIFICATION OF GOITER SIZE
Grade Description
0 no goiter
1 A thyroid lobes more than end of the thumb
B thyroid enlarged, visible when the head tilted back
2 thyroid enlarged, visible when neck in normal position
3 thyroid greatly enlarged, visible from about 10 meters
World Health Organisation
HYPOTHYROIDISM
Appropriate
ThyroidBlood
Testsand
ThyroidConditions
Consequences
ofPerchlorate
 Breast disease
 Hypothyroidism
 Immune System Problems
 Mental retardation in newborns
 Neurologic Problems
 Poor fetal development
 Poor neonatal development
 Thyroid cancer
NationalIodine
Deficiency
DisordersControl
Programme
 National Iodine Deficiency Disorders Control Programme
Launch of National Goitre Control Programme, 1962
 OBJECTIVES: Identification of Goitre endemic areas &
supply of iodised salt in these areas.
 To assess the impact of goitre control measures over a
period of time.
 Iodine Deficiency Disorder Control Programme, 1986 with
an objective to replace entire edible salt by iodine salt, in
phased manner by 1992.
 To Reduce the prevalence of Iodine Deficiency Disorders
below 10% in the entire country by 2012 Achieve
Universal Access to Iodized Salt.*
*Source: 11 th Five Year Plan, Govt. of India
• In 1983, Government of India took policy decision to iodize all salt meant for human consumption
Universal Salt Iodization (USI)
• Private sector was permitted and encouraged to produce iodized salt
• “Elimination of goiter” was included in then Prime Minister’s 20-point National Development Program
• Govt. of India issued notification banning the salt of non iodized salt for direct human consumption with
effect from 17th May, 2006.
Universal Salt Iodization
Iodine for mother and fetus

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