This document summarizes a presentation on insomnia given at the AWP 2010 conference in Portland, OR. The presentation provided an overview of insomnia, discussing its definition, prevalence, and impacts. It also reviewed biological, psychological, social, and environmental factors that can predispose, precipitate, or perpetuate insomnia. Common sleep disorders that can cause insomnia symptoms, such as sleep apnea, restless leg syndrome, and circadian rhythm disorders were also outlined. The presentation emphasized a biopsychosocial model for conceptualizing insomnia and highlighted cognitive behavioral therapy as an effective treatment approach.
Hemostasis Physiology and Clinical correlations by Dr Faiza.pdf
Insomnia presentation
1. AWP 2010, Portland, OR
Conceptualization of Insomnia
A Holistic Approach
to Insomnia
Sovann Pen,
MA, Counseling
Kaiser Insomnia Clinic
2. The Importance of Sleep
• Sleep is vital to our health and well being.
• National Sleep Foundation reveal that 60 percent of adults report
having sleep problems a few nights a week or more.
• In addition, more than 40 percent of adults experience daytime
sleepiness severe enough to interfere with their daily activities
at least a few days each month - with 20 percent reporting problem
sleepiness a few days a week or more.
• At least 40 million Americans suffer from sleep disorders, yet
more than 60 percent of adults have never been asked about the
quality of their sleep by a physician and fewer than 20 percent ever
initiated a discussion.
• Poor sleep has a price.
• Millions of individuals struggle to stay alert at home, in school, on the
job - and on the road. Tragically, fatigue contributes to more than
100,000 police-reported highway crashes, causing 71,000
injuries and 1,500 deaths each year in the United States alone.
3. Insomnia
Insomnia is defined as
difficulty initiating sleep,
maintaining sleep,
final awakenings that occur much earlier
than desired
or sleep that is non-restorative and of poor
quality and
results in impairment in daytime function.
4. Epworth Sleepiness Scale
• Use the following scale to choose the most appropriate number for each
situation:
0 = no chance of dozing
• 1 = slight chance of dozing
• 2 = moderate chance of dozing
• 3 = high chance of dozing
• Sitting and reading____________
• Watching TV____________
• Sitting inactive in a public place (e.g a theater or a meeting)____________
• As a passenger in a car for an hour without a break____________
• Lying down to rest in the afternoon when circumstances permit____________
• Sitting and talking to someone____________
• Sitting quietly after a lunch without alcohol____________
• In a car, while stopped for a few minutes in traffic____________
23. • Pain and Sleep
• Stress and Pain
• Stress and Sleep
• Sleep and Mood
• Mood and motivation
• Mood and activity
• Activity and weight
• Caffeine and Sleep
24.
25. Compensatory strategy
• Go to bed early
“Give myself more of a chance to get
some sleep”
• Sleep in (wake up later)
“Catch up” “Only chance I have to sleep”
• Napping
• Cons: Deprimes sleep homeostat.
Dysregulation of circadian rhythm
26. Erratic sleeping patterns
• Your bedtime varies greatly depending
on your mood, favorite television
program, or the day of the week.
• This sends confusing messages to the
sleep-regulating centers of your brain
—a guarantee for all kinds of problems
with sleep.
28. Rituals and Strategies
• Increase in non-sleep in bedroom and
bed
• Sleep in other places
• “Rituals” for sleep
• Avoidance of behaviors thought to
inhibit sleep
• Cons: lack of stimulus control,
dependence, anticipatory anxiety
32. Cognitive vs Somatic
Lichstein & Rosenthal 1980
• Cognitive arousal 10x more likely
to be cited as major cause than
somatic arousal
33.
34. Unwanted intrusive thoughts
• Worry or Cognitive arousal
• Most Common - Racing thoughts
• “I am unable to empty my mind”
• “I can’t turn off my mind”
• “My mind keeps turning things over”
36. WHAT ARE YOU THINKING?
Watts, Coyle, East 1994
• Mental activity and rehearsal
• Thoughts about sleep
• Family and long-term concerns
• Positive plans and concerns
• Somatic preoccupations
• Work and recent concerns
40. Perception of sleep (memory)
• Subjective vs Objective Measures
• Overestimate sleep latency
• Underestimate Total Sleep Time (TST)
• Underestimate number of awakenings
41. Neitzer, Semler and Harvey
• Positive and Negative Feedback study
• Negative feedback increased: negative
thoughts, sleepiness, monitoring
sleep-threat and safety behaviors the
next day.
42. Mendelson 1990
• Another key study applying to
use of benzodiazepines
• Objectively: benzos decrease
SWS
• Subjectively: report better
sleep with benzos
43. Attention
Insomniacs more aware of:
• body sensations
• environment
• clock
• needing to use the bathroom
• mood
• performance: attention, memory,
concentration failing
44. Worry about negative
consequences of poor sleep
• Catastrophizing / awfulizing
• Negative prediction
Similar to Cognitive Distortions from
standard CBT –
“All-or-nothing”
“Black-and-white” thinking
45. Unhelpful beliefs in
Maintenance of insomnia
• Morin 1993
• Less Realistic about sleep
required
• Strongly endorse – negative
consequences of insomnia
• More likely to attribute insomnia to
external and stable causes
46. Rewards and reinforcement
• By rewarding yourself with your
favorite foods, beverages, or drug of
choice when you can’t sleep, you
ensure future nights of insomnia. The
pleasure centers of your brain have
great recall for this type of behavior.
They will continue to awaken you to
receive more of the same—night after
night, after night, after night.
47. Forcing the issue
• When unable to fall asleep, you try to
force sleep to happen with statements
such as, “I must get to sleep right
now,” or “If I can't get to sleep, I'll just
have to force myself to stay in bed until
I get to sleep.”
• Creating this negative association with
sleep will lead only to frustration.
48. Harvey 2003b
• Attempts to stop, modify, suppress
cognitive arousal may be counter
productive
• Other options (discuss cognitive
restructuring later)
• Suppress, distract (math problem
study, TV, sheep), neutralizing,
appraisal, punishment and worry
49. Catastrophic thinking
• Being unable to sleep, you predict that
tomorrow will be a disaster.
• You tell yourself things such as, “I
won't be able to function at all
tomorrow if I don't get to sleep.”
• This type of thinking creates so much
anxiety that you will most likely not be
able to return to sleep.
50. Rigid expectations
• You believe that sleep is dependent on
rigidly imposed expectations. You
create a flexible work schedule that
permits you to sleep in, expect a 100-
percent quiet sleep environment, and
strive for a stress-free life.
• If for some reason you cannot meet
these conditions, you begin to worry
that you will not be able to sleep.
52. Medication dependence
• You take a nightly sleeping pill “just in
case”—without first determining
whether you really need it.
• After a few weeks of this, you can lose
confidence in your ability to sleep
without the pill, creating the perfect
set-up for a pattern of medication
dependence.
53. Chronic, perpetual problems
• Pain, disability
• Are you managing or coping as best
as you can?
• Resources, trying new ways or
approaches, support group
• Mood: Depression and Bipolar D/o
63. Restless Leg Syndrome
• Do you feel a strong desire to move
your legs from time to time, often when
they make you uncomfortable?
• Do those sensations in your legs occur
or get stronger when you are inactive?
• Does moving around or stretching help
ease those uncomfortable sensations
in your legs?
• Do those uncomfortable sensations
feel their worst at night?
65. Periodic Limb Movement
Syndrome
• PLMS
• Prevalence of PLMS seems to increase with
age.
• 45% elderly adults aged 65 years and older
had PLMS, compared to 5% to 6% of the
younger adult population.
• 80% of those with RLS also had PLMS.
•
• Rule out SDB
Predisposing Precipitating Predisposing (See Sleep-interfering and Sleep-interpreting process)
What is the interfering? Why? What is the trigger? If answer is not to get up to use the bathroom, do you know what is waking you up? Internal or external trigger? Insomnia as a symptom
Mammalian circadian rhythms are generated within the neurons of the SCN of the hypothalmus SCN are 2 clusters of about 10,000 neurons Each SCN neuron keeps its own time – circadian rhythmicity is inherent in the biochemistry of the clock's most basic building blocks – the SCN's neurons Regulates a variety of circadian rhythms including melatonin, corticosterone, and core body temperature Intrinsic rhythm of the clock is slightly longer than 24 hours (circa = about; dian = day) Circadian clock modulates the timing of sleep and wakefulness through direct effects on sleep tendency (sleepiness/alertness) and on the neurophysiologic processes governing sleep state expression Demonstrated by lesion studies in rodents and squirrel monkeys and one case study of a human with lesions in this area Maintaining normal entrainment is a dynamic process that depends on regular adjustments of the circadian pacemaker via exposure to the relevant environmental time cues (zeitgebers) Melatonin is produced by the pineal gland. SCN control the timing of melatonin release. Light evokes an immediate decrease in melatonin secretion levels. Melatonin is able to cause a change in the phase of the circadian oscillator – a phase advance when administered late in the day and phase delay when administered in the morning (opposite of light). Melatonin sharpens the SCN waveform, and may potentiate sleep onset over a relatively narrow phase. Light is the most powerful entrainment factor for the circadian clock
Non-rod, non-cone photoreceptors have been identified in the retina as especially important for the entraining effects of light Most sensitive to blue wavelength light Light exposure in the morning resets the pacemaker to an earlier time Light exposure in the evening resets the pacemaker to a later time Time cues not related to light, such as schedule and activity, may have some influence on circadian timing, but their potency, compared to the solar light/dark schedule, remains to be defined and appears to be weak. For example, If I have a delayed sleep phase and so want to wake up earlier, exposure to bright light as soon as I wake up will shift the circadian clock. If I have an advanced sleep phase and want to stay up later, I can shift my clock by exposing myself to bright light up until bedtime.