Hemolytic disease of newborn. GHS.pptx
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Hemolytic disease of the newborn (HDN), also called erythroblastosis fetalis, is a blood disorder that occurs when the mother and baby have incompatible blood types. During pregnancy, the baby's red blood cells can enter the mother's bloodstream through the placenta. If the mother's immune system sees the baby's blood type as foreign, it will produce antibodies to attack the baby's red blood cells. After birth, these antibodies cause the baby's red blood cells to break down too quickly. Symptoms of HDN include jaundice, swelling, enlarged liver or spleen, and yellowing of bodily fluids. Treatment may involve phototherapy, medications, transfusions, or in severe cases, an
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Hemolytic Disease of the Newborn.pptx
Hemolytic disease of the newborn (HDN), also called erythroblastosis fetalis, occurs when there is an incompatibility between the mother and baby's blood types. Specifically, it most often affects babies born to Rh-negative mothers who were sensitized during a previous pregnancy to an Rh-positive baby. The mother's antibodies then destroy the baby's red blood cells. This can cause anemia, jaundice, liver and spleen enlargement, fluid buildup, and in severe cases, brain damage or death in the baby. Treatment may involve blood transfusions before or after birth as well as medication to prevent Rh sensitization during future pregnancies.
Hemolytic disease in new born
Hemolytic disease of the newborn (HDN), also called erythroblastosis fetalis, occurs when there is an incompatibility between the mother and baby's blood types. The mother's immune system produces antibodies that destroy the baby's red blood cells. This can cause anemia, jaundice, liver and spleen enlargement, or fluid buildup in severe cases. Diagnosis involves blood tests of the mother and baby. Treatment may include intrauterine or postnatal blood transfusions. HDN is preventable by administering Rhogam to sensitized Rh-negative mothers during and after pregnancy.
Hemolytic Disease of Newborn (HDN)
Group C Hemolytic Disease of the Newborn (HDN) is caused by IgG antibodies produced by the mother that destroy the fetus or newborn's red blood cells. It can be caused by Rh, ABO, or other blood group incompatibilities. The antibodies cross the placenta and coat the fetus's red blood cells, which are then destroyed by the spleen and liver. This leads to anemia, liver and spleen enlargement, jaundice, and in severe cases hydrops fetalis or kernicterus. Diagnosis and management involves blood group testing of mother and baby, antibody screening and identification, amniocentesis to assess fetal anemia, and treatments like intrauterine transfusions, plasma
DISCUSSION ON HEMOLYTIC DISEASE IN NEWBORN.pptx
This document provides an overview of hemolytic disease of the newborn (HDN). It defines HDN as a condition where a fetus or neonate's red blood cells are destroyed by maternal IgG antibodies. The main causes are Rh and ABO incompatibility. Rh incompatibility occurs when a Rh-negative mother is pregnant with a Rh-positive baby. The document discusses the pathophysiology, symptoms, diagnosis and management of HDN. Prevention is through administration of Rh immune globulin to Rh-negative mothers to prevent sensitization to the Rh antigen.
assignmentbbgroupc-hdn-131025031937-phpapp01.pdf
This document discusses hemolytic disease of the newborn (HDN), specifically Rh incompatibility. It defines HDN as a condition where a fetus or neonate's red blood cells are destroyed by IgG antibodies produced by the mother. The document then covers the history of HDN, causes including Rh and ABO incompatibility, the pathophysiology whereby the mother's antibodies cross the placenta and destroy the baby's red blood cells, clinical symptoms ranging from jaundice to hydrops fetalis, prenatal and postnatal testing, and treatments such as intrauterine transfusions to prevent complications like hydrops fetalis.
HEMOLYTIC DISEASE.pdf
This document discusses hemolytic disease of the newborn, which occurs when maternal antibodies destroy the baby's red blood cells due to blood group incompatibility. The main types are Rh and ABO incompatibility. Rh incompatibility occurs when an Rh-negative mother has an Rh-positive baby. ABO incompatibility can occur when the mother's blood type is not compatible with the baby's. Symptoms range from jaundice to life-threatening hydrops fetalis. Diagnosis involves blood tests and ultrasound. Prevention focuses on Rh immune globulin for Rh-negative mothers.
SICKEL CELL ANEMIA
Sickle cell anemia is a genetic blood disorder where red blood cells become rigid, sticky and form into a sickle or crescent shape. This is caused by a mutation in the hemoglobin gene. The sickle shaped cells can get stuck in blood vessels and slow or block blood flow. This causes symptoms like anemia, painful episodes and organ damage. It is diagnosed through a blood test. Treatments include antibiotics to prevent infection, pain medications, hydroxyurea to reduce symptoms, blood transfusions, and assessing risk of complications like stroke. Vaccinations, avoiding dehydration and staying warm in winter are also important for managing sickle cell anemia.
Presentation about sickle_cell_anemia_
Sickle cell anemia is a genetic blood disorder where red blood cells become rigid, sticky and crescent shaped. This causes blockages in blood vessels which can lead to pain, organ damage and other complications. It is caused by a mutation in the beta-globin gene that produces abnormal hemoglobin. Symptoms include anemia, pain crises, susceptibility to infection and organ damage. While there is no cure, treatments focus on managing pain, preventing infections, blood transfusions and newer therapies like hydroxyurea.
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Hemolytic Disease of the Newborn.pptx
Hemolytic disease of the newborn (HDN), also called erythroblastosis fetalis, occurs when there is an incompatibility between the mother and baby's blood types. Specifically, it most often affects babies born to Rh-negative mothers who were sensitized during a previous pregnancy to an Rh-positive baby. The mother's antibodies then destroy the baby's red blood cells. This can cause anemia, jaundice, liver and spleen enlargement, fluid buildup, and in severe cases, brain damage or death in the baby. Treatment may involve blood transfusions before or after birth as well as medication to prevent Rh sensitization during future pregnancies.
Hemolytic disease in new born
Hemolytic disease of the newborn (HDN), also called erythroblastosis fetalis, occurs when there is an incompatibility between the mother and baby's blood types. The mother's immune system produces antibodies that destroy the baby's red blood cells. This can cause anemia, jaundice, liver and spleen enlargement, or fluid buildup in severe cases. Diagnosis involves blood tests of the mother and baby. Treatment may include intrauterine or postnatal blood transfusions. HDN is preventable by administering Rhogam to sensitized Rh-negative mothers during and after pregnancy.
Hemolytic Disease of Newborn (HDN)
Group C Hemolytic Disease of the Newborn (HDN) is caused by IgG antibodies produced by the mother that destroy the fetus or newborn's red blood cells. It can be caused by Rh, ABO, or other blood group incompatibilities. The antibodies cross the placenta and coat the fetus's red blood cells, which are then destroyed by the spleen and liver. This leads to anemia, liver and spleen enlargement, jaundice, and in severe cases hydrops fetalis or kernicterus. Diagnosis and management involves blood group testing of mother and baby, antibody screening and identification, amniocentesis to assess fetal anemia, and treatments like intrauterine transfusions, plasma
DISCUSSION ON HEMOLYTIC DISEASE IN NEWBORN.pptx
This document provides an overview of hemolytic disease of the newborn (HDN). It defines HDN as a condition where a fetus or neonate's red blood cells are destroyed by maternal IgG antibodies. The main causes are Rh and ABO incompatibility. Rh incompatibility occurs when a Rh-negative mother is pregnant with a Rh-positive baby. The document discusses the pathophysiology, symptoms, diagnosis and management of HDN. Prevention is through administration of Rh immune globulin to Rh-negative mothers to prevent sensitization to the Rh antigen.
assignmentbbgroupc-hdn-131025031937-phpapp01.pdf
This document discusses hemolytic disease of the newborn (HDN), specifically Rh incompatibility. It defines HDN as a condition where a fetus or neonate's red blood cells are destroyed by IgG antibodies produced by the mother. The document then covers the history of HDN, causes including Rh and ABO incompatibility, the pathophysiology whereby the mother's antibodies cross the placenta and destroy the baby's red blood cells, clinical symptoms ranging from jaundice to hydrops fetalis, prenatal and postnatal testing, and treatments such as intrauterine transfusions to prevent complications like hydrops fetalis.
HEMOLYTIC DISEASE.pdf
This document discusses hemolytic disease of the newborn, which occurs when maternal antibodies destroy the baby's red blood cells due to blood group incompatibility. The main types are Rh and ABO incompatibility. Rh incompatibility occurs when an Rh-negative mother has an Rh-positive baby. ABO incompatibility can occur when the mother's blood type is not compatible with the baby's. Symptoms range from jaundice to life-threatening hydrops fetalis. Diagnosis involves blood tests and ultrasound. Prevention focuses on Rh immune globulin for Rh-negative mothers.
SICKEL CELL ANEMIA
Sickle cell anemia is a genetic blood disorder where red blood cells become rigid, sticky and form into a sickle or crescent shape. This is caused by a mutation in the hemoglobin gene. The sickle shaped cells can get stuck in blood vessels and slow or block blood flow. This causes symptoms like anemia, painful episodes and organ damage. It is diagnosed through a blood test. Treatments include antibiotics to prevent infection, pain medications, hydroxyurea to reduce symptoms, blood transfusions, and assessing risk of complications like stroke. Vaccinations, avoiding dehydration and staying warm in winter are also important for managing sickle cell anemia.
Presentation about sickle_cell_anemia_
Sickle cell anemia is a genetic blood disorder where red blood cells become rigid, sticky and crescent shaped. This causes blockages in blood vessels which can lead to pain, organ damage and other complications. It is caused by a mutation in the beta-globin gene that produces abnormal hemoglobin. Symptoms include anemia, pain crises, susceptibility to infection and organ damage. While there is no cure, treatments focus on managing pain, preventing infections, blood transfusions and newer therapies like hydroxyurea.
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Neonatal jaundice is a condition in newborns marked by high bilirubin levels, causing yellowing of the skin and eyes. Bilirubin is produced from the breakdown of red blood cells, but neonates have increased bilirubin production and immature liver function. This makes it difficult for newborns to eliminate bilirubin from their blood, potentially leading to hyperbilirubinemia and even brain damage if levels get too high. Special characteristics of neonates that contribute to jaundice include high red blood cell breakdown, low albumin and liver function, and an immature gastrointestinal system.
Sickle cell anemia
Sickle cell anemia is a genetic blood disorder where red blood cells become stiff and sticky and form clumps that can get stuck in blood vessels, blocking blood flow. This causes pain, infections, and organ damage. It occurs when someone inherits two sickle cell genes, one from each parent. Those with one sickle cell gene have sickle cell trait and do not have the disease. It most commonly affects those with ancestry from sub-Saharan Africa, South and Central America, the Caribbean, India, and Mediterranean countries. While there is no cure, treatments can help relieve symptoms and prevent complications.
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Disorder hereditary spherocytosis
Hereditary spherocytosis is a genetic blood disorder that causes red blood cells to assume a spherical shape rather than the normal disc shape. This makes it difficult for the spleen to filter the misshapen red blood cells from the bloodstream. Symptoms include anemia, jaundice, and an enlarged spleen. Treatment options depend on severity but may include spleen removal, folic acid supplements, blood transfusions, or light therapy for jaundice in infants. The condition is usually diagnosed based on family history, symptoms, and blood tests showing abnormal red blood cell shape and size.
sicklecellanemia-111229093154-phpapp01.pdf
Sickle cell anemia is a genetic blood disorder where red blood cells become sickle-shaped and have difficulty passing through small blood vessels. This causes pain, organ damage, and risk of infection. It occurs most commonly in those with ancestry from sub-Saharan Africa, South America, the Caribbean, Mediterranean, and Middle East. Symptoms begin in early childhood and include anemia, pain crises, infections, and other complications. Treatment focuses on pain management, prevention of infections and complications, and new therapies are being researched.
hemolyticdisease-190822150040 5.pdf
The document discusses hemolytic disease of the newborn (HDN), specifically Rh disease, ABO incompatibility, and their causes, pathophysiology, clinical manifestations, diagnosis, and antenatal management. Rh disease is caused by maternal alloimmunization to Rh+ fetal red blood cells, ranging from mild anemia to severe hydrops fetalis. ABO incompatibility also causes HDN when maternal antibodies attack fetal red blood cells of a different blood group than the mother's. Screening, antibody detection, and specialized care can help manage affected pregnancies and newborns.
Hemolytic disease
This document discusses hemolytic disease of the newborn (HDN), which is the destruction of fetal or newborn red blood cells by maternal antibodies. The main causes of HDN are Rh and ABO blood group incompatibilities between mother and fetus. In Rh disease, a Rh-negative mother produces antibodies against Rh-positive fetal blood cells. These antibodies can cross the placenta and destroy fetal red blood cells. The most severe manifestation is hydrops fetalis, where extensive hemolysis leads to fetal edema, ascites, heart failure and death. Diagnosis involves serological tests and ultrasound detection of edema. Treatment and prevention involves Rh immunoglobulin administration during pregnancy.
Hemolytic disease of newborn
Hemolytic disease of the newborn (HDN), also known as erythroblastosis fetalis, occurs when there is an incompatibility between the mother and baby's blood types. The mother's immune system produces antibodies that destroy the baby's red blood cells. This can cause anemia, liver and spleen enlargement, jaundice, and in severe cases hydrops fetalis or death. HDN is diagnosed through blood tests of both mother and baby. Management may include blood transfusions, phototherapy, IVIG, plasma exchange, and Rh immune globulin injections during pregnancy to prevent sensitization.
Neonatal Jaundice
Neonatal jaundice is a condition in newborns marked by high levels of bilirubin in the blood, causing yellowing of the skin and whites of the eyes. Bilirubin levels are often higher in neonates due to increased red blood cell breakdown, liver immaturity, and bacterial colonization. Without treatment, hyperbilirubinemia can cause permanent brain damage known as kernicterus. Proper monitoring and treatment is important to prevent dangerous bilirubin levels in newborns.
hdnnonmlt-151222085444 2.pdf
Hemolytic disease of newborn occurs when maternal IgG antibodies cross the placenta and destroy fetal red blood cells due to incompatibility between maternal and fetal blood groups. This can cause anemia, jaundice, liver and spleen enlargement in the fetus as well as risks after birth like respiratory distress and brain damage from high bilirubin levels. Proper screening and Rhogam administration in Rh-negative mothers can prevent Rh-HDN in future pregnancies.
chad
Sickle cell disease is caused by a genetic mutation that causes red blood cells to take on a sickle shape under certain conditions. This document summarizes the origins and genetics of sickle cell disease, describes how sickle cells can cause complications like pain crises, infections, acute chest syndrome and stroke by blocking blood flow, and outlines treatments and management strategies.
hdnnonmlt-151222085444.pptx
Hemolytic disease of the newborn (HDN) is a condition where maternal antibodies destroy the fetus or newborn's red blood cells. It occurs when the mother has antibodies against antigens on the father's red blood cells that her baby inherits. This leads to premature breakdown of the baby's red blood cells. Symptoms range from mild jaundice to life-threatening edema called hydrops fetalis. Testing identifies incompatibility between maternal and paternal blood groups, with the most common cause being Rh disease from an Rh negative mother's antibodies against Rh positive fetal blood. HDN can cause anemia, liver/spleen enlargement, and complications affecting several organ systems in the baby.
hemolytic disease of new born
hemolytic disease of new born is an aquire alla immune hemolytic anemia characterize by production extravascular destruction of RBC within the spleen of new born baby resulting anemia, positive coomb,s test
Haemolytic disease of new born
Hemolytic disease of the newborn (HDN) occurs when maternal antibodies destroy fetal red blood cells. It develops when an Rh-negative mother carries an Rh-positive fetus. During delivery, fetal red blood cells may enter the mother's circulation and cause her to produce IgG antibodies. These antibodies then cross the placenta and destroy fetal red blood cells, potentially causing jaundice, anemia, or heart failure in the newborn. Treatment may include intrauterine or postnatal blood transfusions. HDN can be prevented through Rh immunoglobulin injections during pregnancy to prevent maternal immunization against the Rh factor.
Immune hydrops
Immune hydrops fetalis is a condition where excess fluid builds up in fetal tissues due to maternal antibodies destroying fetal red blood cells. It can be caused by Rh incompatibility between mother and fetus. The excess hemolysis of fetal red blood cells leads to anemia, liver and spleen damage, heart failure and fluid buildup. Ultrasound is used to diagnose hydrops fetalis by detecting fluid in two fetal compartments. Treatment involves monitoring the fetus and performing intrauterine blood transfusions if needed to improve fetal hemoglobin levels and resolve hydrops. Routine Rh immunoprophylaxis can prevent Rh sensitization and immune hydrops in subsequent pregnancies.
Rh incom .ppt
Hemolytic disease of the fetus and newborn (HDFN), also known as erythroblastosis fetalis, is caused by maternal antibodies crossing the placenta and destroying fetal red blood cells. Rh incompatibility occurs when an Rh-negative mother has an Rh-positive baby. This can sensitize the mother's immune system and cause hemolytic anemia in subsequent Rh-positive babies. Management involves monitoring for signs of fetal anemia, performing intrauterine blood transfusions if needed, and delivering the baby when it is mature. After birth, affected babies may require phototherapy, exchange transfusions, or other treatments to prevent complications from hemolysis and jaundice. RhoGAM injections during and after pregnancy
Antimalady com-sickle-cell-disease-in-pregnancy-
Sickle cell disease also called sickle cell anemia is an autosomal recessive disorder. It is more likely to be a blood disorder passed from mother to child. This kind of blood disorder is very painful and intolerable for pregnant women with SCD(Sickle cell disease).
Hemolytic disease of the newborn
Hemolytic disease of the newborn (HDN), also known as erythroblastosis fetalis, occurs when maternal antibodies attack and destroy fetal red blood cells due to blood group incompatibilities between mother and fetus, most commonly Rh incompatibility, which can cause fetal anemia, jaundice, and in severe cases heart failure or death; diagnosis involves blood tests of the mother and newborn and management focuses on monitoring the pregnancy, preventing sensitization in unsensitized mothers, and treating the newborn if necessary.
Rh negative disease
Rh incompatibility is a condition that occurs during pregnancy if
a woman hasRh-negative blood and
her baby has Rh-positive blood.
"Rh-negative" and "Rh-positive" refer to
whether your blood has Rh factor. Rh factor is a protein on red blood cells. If
you have Rh factor, you're Rh-positive. If you don't have it, you're
Rh-negative. Rh factor is inherited (passed from parents to children through
the genes). Most people are Rh-positive.
Whether you have Rh factor doesn't affect your general health.
However, it can cause problems during pregnancy.
Sickle Cell Anemia. Student Presentation
Sickle cell anemia is a genetic blood disorder that affects red blood cells. It is most common among those of African descent. The disease causes red blood cells to become rigid and sickle-shaped, which can cause anemia, pain crises, and other serious complications. Symptoms vary from person to person but often include fatigue, shortness of breath, and pain. The disease is inherited when both parents carry the sickle cell trait, resulting in a 25% chance their child will have the disease.
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neonataljaundicemanaen-090706111645-phpapp02.pdf
Neonatal jaundice is a condition in newborns marked by high bilirubin levels, causing yellowing of the skin and eyes. Bilirubin is produced from the breakdown of red blood cells, but neonates have increased bilirubin production and immature liver function. This makes it difficult for newborns to eliminate bilirubin from their blood, potentially leading to hyperbilirubinemia and even brain damage if levels get too high. Special characteristics of neonates that contribute to jaundice include high red blood cell breakdown, low albumin and liver function, and an immature gastrointestinal system.
Sickle cell anemia
Sickle cell anemia is a genetic blood disorder where red blood cells become stiff and sticky and form clumps that can get stuck in blood vessels, blocking blood flow. This causes pain, infections, and organ damage. It occurs when someone inherits two sickle cell genes, one from each parent. Those with one sickle cell gene have sickle cell trait and do not have the disease. It most commonly affects those with ancestry from sub-Saharan Africa, South and Central America, the Caribbean, India, and Mediterranean countries. While there is no cure, treatments can help relieve symptoms and prevent complications.
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Neonatal jaundice is a condition in newborns marked by high bilirubin levels, causing yellowing of the skin and whites of the eyes. This is commonly due to increased red blood cell breakdown, immature liver function, and inefficient bilirubin excretion in neonates. If bilirubin levels rise too high, it can cause permanent brain damage known as kernicterus by crossing the blood-brain barrier.
Disorder hereditary spherocytosis
Hereditary spherocytosis is a genetic blood disorder that causes red blood cells to assume a spherical shape rather than the normal disc shape. This makes it difficult for the spleen to filter the misshapen red blood cells from the bloodstream. Symptoms include anemia, jaundice, and an enlarged spleen. Treatment options depend on severity but may include spleen removal, folic acid supplements, blood transfusions, or light therapy for jaundice in infants. The condition is usually diagnosed based on family history, symptoms, and blood tests showing abnormal red blood cell shape and size.
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Sickle cell anemia is a genetic blood disorder where red blood cells become sickle-shaped and have difficulty passing through small blood vessels. This causes pain, organ damage, and risk of infection. It occurs most commonly in those with ancestry from sub-Saharan Africa, South America, the Caribbean, Mediterranean, and Middle East. Symptoms begin in early childhood and include anemia, pain crises, infections, and other complications. Treatment focuses on pain management, prevention of infections and complications, and new therapies are being researched.
hemolyticdisease-190822150040 5.pdf
The document discusses hemolytic disease of the newborn (HDN), specifically Rh disease, ABO incompatibility, and their causes, pathophysiology, clinical manifestations, diagnosis, and antenatal management. Rh disease is caused by maternal alloimmunization to Rh+ fetal red blood cells, ranging from mild anemia to severe hydrops fetalis. ABO incompatibility also causes HDN when maternal antibodies attack fetal red blood cells of a different blood group than the mother's. Screening, antibody detection, and specialized care can help manage affected pregnancies and newborns.
Hemolytic disease
This document discusses hemolytic disease of the newborn (HDN), which is the destruction of fetal or newborn red blood cells by maternal antibodies. The main causes of HDN are Rh and ABO blood group incompatibilities between mother and fetus. In Rh disease, a Rh-negative mother produces antibodies against Rh-positive fetal blood cells. These antibodies can cross the placenta and destroy fetal red blood cells. The most severe manifestation is hydrops fetalis, where extensive hemolysis leads to fetal edema, ascites, heart failure and death. Diagnosis involves serological tests and ultrasound detection of edema. Treatment and prevention involves Rh immunoglobulin administration during pregnancy.
Hemolytic disease of newborn
Hemolytic disease of the newborn (HDN), also known as erythroblastosis fetalis, occurs when there is an incompatibility between the mother and baby's blood types. The mother's immune system produces antibodies that destroy the baby's red blood cells. This can cause anemia, liver and spleen enlargement, jaundice, and in severe cases hydrops fetalis or death. HDN is diagnosed through blood tests of both mother and baby. Management may include blood transfusions, phototherapy, IVIG, plasma exchange, and Rh immune globulin injections during pregnancy to prevent sensitization.
Neonatal Jaundice
Neonatal jaundice is a condition in newborns marked by high levels of bilirubin in the blood, causing yellowing of the skin and whites of the eyes. Bilirubin levels are often higher in neonates due to increased red blood cell breakdown, liver immaturity, and bacterial colonization. Without treatment, hyperbilirubinemia can cause permanent brain damage known as kernicterus. Proper monitoring and treatment is important to prevent dangerous bilirubin levels in newborns.
hdnnonmlt-151222085444 2.pdf
Hemolytic disease of newborn occurs when maternal IgG antibodies cross the placenta and destroy fetal red blood cells due to incompatibility between maternal and fetal blood groups. This can cause anemia, jaundice, liver and spleen enlargement in the fetus as well as risks after birth like respiratory distress and brain damage from high bilirubin levels. Proper screening and Rhogam administration in Rh-negative mothers can prevent Rh-HDN in future pregnancies.
chad
Sickle cell disease is caused by a genetic mutation that causes red blood cells to take on a sickle shape under certain conditions. This document summarizes the origins and genetics of sickle cell disease, describes how sickle cells can cause complications like pain crises, infections, acute chest syndrome and stroke by blocking blood flow, and outlines treatments and management strategies.
hdnnonmlt-151222085444.pptx
Hemolytic disease of the newborn (HDN) is a condition where maternal antibodies destroy the fetus or newborn's red blood cells. It occurs when the mother has antibodies against antigens on the father's red blood cells that her baby inherits. This leads to premature breakdown of the baby's red blood cells. Symptoms range from mild jaundice to life-threatening edema called hydrops fetalis. Testing identifies incompatibility between maternal and paternal blood groups, with the most common cause being Rh disease from an Rh negative mother's antibodies against Rh positive fetal blood. HDN can cause anemia, liver/spleen enlargement, and complications affecting several organ systems in the baby.
hemolytic disease of new born
hemolytic disease of new born is an aquire alla immune hemolytic anemia characterize by production extravascular destruction of RBC within the spleen of new born baby resulting anemia, positive coomb,s test
Haemolytic disease of new born
Hemolytic disease of the newborn (HDN) occurs when maternal antibodies destroy fetal red blood cells. It develops when an Rh-negative mother carries an Rh-positive fetus. During delivery, fetal red blood cells may enter the mother's circulation and cause her to produce IgG antibodies. These antibodies then cross the placenta and destroy fetal red blood cells, potentially causing jaundice, anemia, or heart failure in the newborn. Treatment may include intrauterine or postnatal blood transfusions. HDN can be prevented through Rh immunoglobulin injections during pregnancy to prevent maternal immunization against the Rh factor.
Immune hydrops
Immune hydrops fetalis is a condition where excess fluid builds up in fetal tissues due to maternal antibodies destroying fetal red blood cells. It can be caused by Rh incompatibility between mother and fetus. The excess hemolysis of fetal red blood cells leads to anemia, liver and spleen damage, heart failure and fluid buildup. Ultrasound is used to diagnose hydrops fetalis by detecting fluid in two fetal compartments. Treatment involves monitoring the fetus and performing intrauterine blood transfusions if needed to improve fetal hemoglobin levels and resolve hydrops. Routine Rh immunoprophylaxis can prevent Rh sensitization and immune hydrops in subsequent pregnancies.
Rh incom .ppt
Hemolytic disease of the fetus and newborn (HDFN), also known as erythroblastosis fetalis, is caused by maternal antibodies crossing the placenta and destroying fetal red blood cells. Rh incompatibility occurs when an Rh-negative mother has an Rh-positive baby. This can sensitize the mother's immune system and cause hemolytic anemia in subsequent Rh-positive babies. Management involves monitoring for signs of fetal anemia, performing intrauterine blood transfusions if needed, and delivering the baby when it is mature. After birth, affected babies may require phototherapy, exchange transfusions, or other treatments to prevent complications from hemolysis and jaundice. RhoGAM injections during and after pregnancy
Antimalady com-sickle-cell-disease-in-pregnancy-
Sickle cell disease also called sickle cell anemia is an autosomal recessive disorder. It is more likely to be a blood disorder passed from mother to child. This kind of blood disorder is very painful and intolerable for pregnant women with SCD(Sickle cell disease).
Hemolytic disease of the newborn
Hemolytic disease of the newborn (HDN), also known as erythroblastosis fetalis, occurs when maternal antibodies attack and destroy fetal red blood cells due to blood group incompatibilities between mother and fetus, most commonly Rh incompatibility, which can cause fetal anemia, jaundice, and in severe cases heart failure or death; diagnosis involves blood tests of the mother and newborn and management focuses on monitoring the pregnancy, preventing sensitization in unsensitized mothers, and treating the newborn if necessary.
Rh negative disease
Rh incompatibility is a condition that occurs during pregnancy if
a woman hasRh-negative blood and
her baby has Rh-positive blood.
"Rh-negative" and "Rh-positive" refer to
whether your blood has Rh factor. Rh factor is a protein on red blood cells. If
you have Rh factor, you're Rh-positive. If you don't have it, you're
Rh-negative. Rh factor is inherited (passed from parents to children through
the genes). Most people are Rh-positive.
Whether you have Rh factor doesn't affect your general health.
However, it can cause problems during pregnancy.
Sickle Cell Anemia. Student Presentation
Sickle cell anemia is a genetic blood disorder that affects red blood cells. It is most common among those of African descent. The disease causes red blood cells to become rigid and sickle-shaped, which can cause anemia, pain crises, and other serious complications. Symptoms vary from person to person but often include fatigue, shortness of breath, and pain. The disease is inherited when both parents carry the sickle cell trait, resulting in a 25% chance their child will have the disease.
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- 1. Hemolytic disease of new born baby Submited to:Ma'am Faiza Submited by :Hassan Saif Reg# 2021-Bs-Mls-080 Semester. 4th
- 2. Hemolytic Hemolytic disease of the newborn (HDN) — also called erythroblastosis fetalis. Erythro------------ Red cell Blast -------------- break down Fetails------------ fetus relate 0 1
- 4. Hemolytic disease of the newborn baby.. HDN is a blood disorder that occurs when the blood types of a mother and baby are incompatible. Normally, red blood cells (RBCs) last for about 120 days in the body. In this disorder, RBCs in the blood are destroyed quickly and thus do not last as long
- 6. Causes...... • There is more than one way in which the unborn baby's blood type may not match the mother's. • A, B, AB, and O are the 4 major blood group antigens or types. This is the most common form of a mismatch. In most cases, this is not very severe.
- 7. Rh blood group • Firstly discovered by Rhesus monkey. (Land steiner 1930) • The Rh blood group system is a human blood group system. It contains proteins on the surface of red blood cells. After the ABO blood group system, it is the most likely to be involved in transfusion reactions.
- 8. • HDN happens when an Rh negative mother has a baby with an Rh positive father. If the Rh negative mother has been sensitized to Rh positive blood, her immune system will make antibodies to attack her baby. When the antibodies enter the baby's blood, they will attack the red blood cells. This causes them to break down.
- 9. Causes........... What are the causes? Features During pregnancy, RBCs from the unborn baby can cross into the mother's blood through the placenta. HDN occurs when the immune system of the mother sees a baby's RBCs as foreign. Antibodies then develop against the baby's RBCs. These antibodies attack the RBCs in the baby's blood and cause them to break down too early.
- 11. Symptoms HDN can destroy the newborn baby's blood cells very quickly, which can cause symptoms such as: • Edema • (swelling under the surface of the skin) • Newborn jaundice which occurs sooner and is more severe than normal.
- 12. Symptoms • Yellowing of the amniotic fluid, umbilical cord, skin, and eyes. • enlarged liver or spleen. • severe swelling of the body.
- 14. Treatment • Light therapy (phototherapy) using special blue lights to convert bilirubin into a form which is easier for the baby's body to get rid of. • Antiserum injections. • Medicines to raise blood pressure if it drops too low. • In severe cases, an exchange transfusion may need to be performed. This involves removing a large amount of the baby's blood, and thus the extra bilirubin and antibodies.
- 15. Tests for HDN • Which tests are done depends on the type of blood group incompatibility and the severity of symptoms, but may include: • Complete blood count and immature red blood cell (reticulocyte) count • Bilirubin level • Blood typing
- 16. Thank You