Efruzhu cancer laws com. 25.1.
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This document presents the "Efruzher Cancer (Carcinogenesis) Theory". It proposes that for a normal cell to become cancerous, it requires more than just mutations - it needs energy inefficiency, chronic inflammation, or suppression of normal DNA. Cancer cells take on a group identity defined by many mutations, seeking to live irregularly and being transferred from outside influences like macrophages. The malignancy level depends on energy ratios - low energy means more mutations and malignancy, while high energy means fewer mutations and potential benign conditions. Cancer cells are seen as selfish, parasitic, irregular and immortal due to their transferred mutational group identity from outside influences.
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The document discusses the "EFRUZHU Cancer (Carcinogenesis) Theory" which proposes that carcinogenesis is triggered by four main factors: 1) mutations that alter group identity, 2) inadequate or inefficient energy, 3) chronic inflammation, and 4) suppression of normal DNA sequences. It also suggests that carcinogenesis involves a person having both mutated and normal DNA identities that continuously recombine in a manner similar to crossover cloning, leading to metastatic conditions.
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The document outlines 4 laws of carcinogenesis theory related to efruzher cancer: 1) Mutations disrupt a cell's group identity. 2) Inadequate energy supply at the cellular level. 3) Chronic inflammation can promote cancerous growth. 4) Suppression of normal DNA functioning.
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This document discusses four theories of carcinogenesis or cancer development: 1) Mutations disrupt a cell's group identity, 2) Inadequate energy production leads to uncontrolled growth, 3) Chronic inflammation damages DNA, and 4) The suppression of normal DNA functions allows uncontrolled growth.
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The document discusses the "EFRUZHU Cancer (Carcinogenesis) Theory" which proposes that carcinogenesis is triggered by four main factors: 1) mutations that alter group identity, 2) inadequate or inefficient energy, 3) chronic inflammation, and 4) suppression of normal DNA sequences. It also suggests that carcinogenesis involves a person having both mutated and normal DNA identities that continuously recombine in a manner similar to crossover cloning, leading to metastatic conditions.
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This document discusses a theory about the causes of cancer known as the "EFRUZHU CANCER (CARCİNOGENESİS) THEORY". It also mentions "NORMAL DNA SUPPRESSİON" and includes a file name "Capture_20120813_2.wmv" but provides no other context or details about the document's contents.
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Cancer cells have a binary identity - one originating from outside immune cells like macrophages, making them non-self. Cancer cells have accumulated so many mutations that their group identity wants to live exceptionally and irregularly, transferred from outside. The main point is that cancer cells depend on inadequate energy, which increases mutations. Malignancy occurs with increasing mutations due to low energy. Benign conditions are possible with high energy and low mutations. Cancer cells are selfish, parasitic, irregular, immortal and immature due to their transferred mutated group identity from outside. Normal DNA sequences cannot be transcribed or expressed due to inadequate energy.
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Cancer cells have a binary identity - one originating from outside immune cells like macrophages, making them non-self. Cancer cell personalities become defined by many mutations that allow them to live exceptionally and irregularly. Malignancy occurs when energy levels are inadequate, increasing mutations, while benign conditions are possible when energy levels increase and mutations decrease. Cancer cells are selfish, parasitic, irregular, immortal, and immature due to their dependence on transferred group identities and mutations from outside influences, causing normal DNA sequences to not be transcribed or expressed due to inadequate energy levels.
Presentation4
Mutations from outside sources express a foreign identity in cancer cells. These mutation groups want to immortalize themselves by continuously signaling to bypass normal cell death mechanisms. Cancer cells have a dual identity with the mutation identity dominant over the normal DNA sequence. The normal DNA sequence is suppressed and unable to be transcribed and expressed due to inadequate energy and changes to histone proteins. The normal DNA wants to die through apoptosis but cell death is unsuccessful as there is not enough energy for apoptosis to occur. Apoptotic and anti-apoptotic molecules have dual roles that depend on the cancer cell's dual identity.
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A group of 10-100 thousand mutations collectively form a foreign identity that is transferred by immune cells from outside the body, unlike a cell's normal DNA sequence. This mutated identity wants to immortalize itself by continuously signaling from outside through things like nitric oxide and growth factors. The cancer cell has a dual identity with the mutated identity dominant and normal DNA suppressed, resulting in varying degrees of malignancy due to inadequate energy levels that prevent normal DNA transcription and expression. The normal DNA is affected by mutations and wants to die physiologically through apoptosis, but apoptosis is unsuccessful because of insufficient energy. Apoptotic and anti-apoptotic molecules have dual roles and actions depending on the cell's binary identity.
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Mutations from outside sources accumulate in cells over time and express a foreign identity. This group of mutations wants to immortalize the cell by continuously signaling it to bypass normal apoptosis. The mutations situate themselves into the normal DNA sequence, making the cancer cell have a dual identity with the mutation identity dominant over the normal DNA. The normal DNA is suppressed and unable to be transcribed or expressed properly due to inadequate energy. Apoptosis is unsuccessful because the cell does not have enough energy to die physiologically as the normal DNA wants to, despite being affected by the mutations.
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Presentation12
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This document discusses the "Inadequate Energy Cancer (Carcinogenesis) Theory". It proposes that cancer may be triggered by mutations, but the main driver is inadequate or inefficient energy production within the cell. A variety of internal and external factors can disrupt energy production, especially oxidative processes, leading to inhibited or low-level energy generation over time. This stimulates an immune response and stresses the cell to find ways to survive, which can later lead to malignancy depending on the energy balance and accumulation of further mutations over time. Cancer cells mainly rely on anaerobic glycolysis for energy due to repression of normal aerobic pathways.
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Sunu 25.1.2
Cancer cells have a binary identity - one originating from outside immune cells like macrophages, making them non-self. Cancer cells have accumulated so many mutations that their group identity wants to live exceptionally and irregularly, transferred from outside. The main point is that cancer cells depend on inadequate energy, which increases mutations. Malignancy occurs with increasing mutations due to low energy. Benign conditions are possible with high energy and low mutations. Cancer cells are selfish, parasitic, irregular, immortal and immature due to their transferred mutated group identity from outside. Normal DNA sequences cannot be transcribed or expressed due to inadequate energy.
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Efruz Asiloglu is an independent researcher whose primary research interest is molecular cancer biochemistry. Their profile page provides information on their publications, books, talks and teaching documents related to their "Efruzhu Cancer (Carcinogenesis) Theory". They have been actively posting status updates discussing various aspects of cancer cell metabolism, signaling pathways, DNA mutations and potential treatment approaches.
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This document discusses two different styles of carcinogenesis or cancer development:
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Efruzhu cancer laws com. 25.1.
- 1. “EFRUZHU CANCER (CARCİNOGENESİS) THEORY” “MUTATİONS GROUP İDENTİTY” DETAİLED LAWS COMMENTS 1. NORMAL CELL THAT SHOULD BECOME CANCER CELL NOT ENOUGH İNTERACTİON BY MUTATİON ONLY THİS CONDİTİON NEEDS ENERGY İNEFFİCİENCY(MAİN DOMİNATİVE MECHANİSM) ,CHRONİC İNFLAMMATİON,NORMAL DNA SUPPRESSİON WOULD BECOME EXİSTENCE.CANCER CELL HAS BİNARY İDENTİTY ONE OF THEM ORİGİN FROM OUTSİDE BY İMMUN CEELS DOMİNANTLY MACROPHAGES (NONSELF=ECTOPİC). PERSONALİTY OF CANCER CELL HAS BECOME SO MANY MUTATİONS GROUP İDENTİTY THAT WANTS TO LİVE
- 2. EXCEPTİONAL-İRREGULAR,HAS BEEN TRANSFERRED FROM OUTSİDE MULTİPLY GROUP MUTATİONS İS MAİN POİNT THAT DEPENDENT ENERGY İNADEQUATE:DECREASİNG THE ENERGY RATİO İNCREASİNG THE MUTATİONS SO THAT MALİGNİTY HAVE BEEN DONE,İNCREASİNG THE ENERGY RATİO DECREASİNG THE MUTATİONS SO THAT BENİGN CONDİTİON İS POSSİBLE.OUR BODY HAS 100 TRİLLİON CELL AND HEALTYNESS NEED TO COOPERATİON, SHARENESS AND SUİTABLE RELATİONSHİP THAT SHOULD BE CONTİNUE.CANCER CELL SELFİSH,PARASİTİC,İRREGULAR,İMMORTAL,İMMATURE AND SO ON DEPENDENCE OF TRANSFERRED MUTATİONS GROUP İDENTİTY FROM OUTSİDE .NORMAL DNA SEQUENCE SHOULD NOT TRANSCRİPTİON EXPRESSİON BECAUSE OF İNADEQUATE ENERGY SO DOMİNANTİVE GOVERN OF CELL: MUTATİONS GROUP İDENTİTY ,ABERRANT SİGNALİNG PATHWAYS,MUTATİONAL RECEPTOR,SİGNALİNG ALL OF THESE COME FROM OUTSİDE ORİGİN. VOİCED LAWS COMMENTS 1. Capture_20120812.wmv