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Electrocardiography
Arrhythmias
Review
R-R Interval to Measure
HR
If you’re using 25 mm/
sec:
•
•
•
Normal sinus impulse
formation
• Normal sinus rhythm
• Sinus arrhythmia
Disturbances from
sinus
• Sinus bradycardia
• Sinus tachycardia
Disturbances of atrial
impulse formation
• Atrial premature complexes
• Atrial tachycardia
• Atrial flutter
• Atrial fibrillation
• Disturbances of ventricular
impulse formation
• Ventricular premature
complexes
• Ventricular tachycardia
• Ventricular asystole- no
contraction
• Ventricular fibrillation
Disturbances of impulse
conduction
• Sinus arrest
• Atrial standstill
• First-degree AV block
• Second degree AV block
• Third degree AV block
Normal Sinus
Rhythm
• Normal ECG tracing depicting a normal rhythm of electrical
conductivity through the heart
(Respiratory)
Sinus Arrhythmia
• All criteria of normal rhythm except heart and pulse
rates increase with inspiration and decrease with
expiration
• Normal finding in brachycephalic breeds and in
chronic respiratory disease
• Increased number of cardiac cycles during inspiration;
decreased number during expiration
Sinus
Bradycardia
•
•
•
Regular sinus rhythm but heart rate
is below normal
Heart rate <60
Causes: Athletes,
Hypothermia,
Hypoglycemia,Hypothyroidism
Sinus
Tachycardia
• Regular sinus rhythm with increased
ventricular rate
• Heart rate >100
• Causes include: pain, fever,
excitement, hyperthyroidism
Atrial Premature Complexes
• Premature atrial impulses originating from atrial site other than
SA node
• Seen in electrolyte disturbances, drug reactions, congenital heart
disease, and neoplasia
• Premature P wave causes a heartbeat sooner than it should be
• QRS complexes are normal unless the P wave is so immature
that it overlaps to varying degrees
Atrial Flutter
•
•
•
•
•
Appears as a regular, “sawtooth” formation between
the mostly normal QRS complexes
Occurs when the ventricular rate differs from the
atrial rate
Single area in atrium other than SA node starts
impulse
AV node “gatekeeper” only allows some impulses
through to ventricles (lots of P waves, regular
QRS)
Atrial flutter is the precursor to atrial fibrillation
Atrial Fibrillation
•Fibrillation is the rapid, irregular, and unsynchronized
contraction of muscle fibers
•Caused by numerous disorganized atrial impulses
frequently bombarding the AV node
•Ventricular depolarization rate is irregular and rapid
•NO P waves are evident; replaced by
numerous f (fibrillation) waves
Premature Ventricular
Complexes (PVCs)
•
•
•
• “Premature beats” - cardiac impulses initiated
within the ventricles instead of the sinus node
Ventricle discharges before the arrival of the next
anticipated impulse from the SA node
Can occur at any rate but pose a greater
danger with tachycardia
Associated with congenital defects,
cardiomyopathy, GDV, drug reactions,
cardiac neoplasia, anemia, acidosis,
hyperthyroidism, hypokalemia
PVCs
(cont’d)
•
•
•
•
The P wave is often not seen on the ECG tracing
A wide, distorted/bizarre QRS complex is evident
The beat preceding the PVC and the beat following
are usually equal to the time of two normal beats
May treat with IV lidocaine
Ventricular Tachycardia
“V
- Tach”
• One strong ventricle impulse that hijacks the conduction system
of the heart. Patient may be “stable” with a pulse or unstable with
“no pulse”
• AV node is on its own and SA node is not working
• A series of three or more PVCs in a row
• Life threatening
• Treatment is reset heart via defibrillation
Ventricular Fibrillation
•The mechanical pumping of the heart is not evident on the
ECG
•Many weak impulses other than AV node present in
ventricles
•The ECG has bizarre baseline with prominent
undulations due to weak and uncoordinated ventricular
contractions
•Low to absent cardiac output
•Associated with shock, trauma,
electrolyte imbalances, drug reactions,
electric shock, hypothermia, cardiac sx
•Rapidly fatal
V Fib
cont.
•
•
•
There are no recognizable P or QRS
complexes Irregular, chaotic, deformed
reflections of
varying width, amplitude, and shape
Unless controlled immediately, ventricular
fibrillation will result in cardiac arrest
Atrial Standstill
•
•
•
•
SA node sends impulse but atria do notcontract
No P waves seen
Hyperkalemia is most common cause decrease
potassium
If not due to increased potassium, pace makeris
warranted
HEART
BLOCK
First Degree AV
Block
•
•
•
•
Delay in conduction of an impulse throughthe
AV junction and Bundle of His
The PR interval is longer thannormal
This type of heart block is a result of a minor
conduction defect
Seen in older patients secondary to
degenerative changes in the conduction system
Second Degree AV
Block
• Some atrial pulses are not conducted through the
AV node and therefore do not cause depolarization
of the ventricles
• There are two types:
• Type I (Wenckebach type I AV block): progressive
lengthening of the PR interval until no complex is conducted
• P waves occurring without QRS complexes “dropped beats”
Second Degree AV
Block (cont’d)
• Mobitz Type II: A intermittent block at the AV node,
that conducts some impulses but blocks others
• A constant PR interval that is usually of normal
duration with random dropped beats
• In the case of type 2 block, atrial contractions are
not regularly followed by ventricular contraction
• 2 or more dropped QRS in a row
Third degree AV block
(Complete Heart Block)
• The cardiac impulse is completely blocked in the
region of the AV junction and/or all bundle branches
• The most severe heart block
• No relationship between P waves and QRS
complexes; atria and ventricles each beat
independently and do not communicate at all
THANK
YOU

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Cardiac arrythmias -biochemistryconverted.pptx

  • 2. R-R Interval to Measure HR
  • 3.
  • 4. If you’re using 25 mm/ sec:
  • 5.
  • 6. • • • Normal sinus impulse formation • Normal sinus rhythm • Sinus arrhythmia Disturbances from sinus • Sinus bradycardia • Sinus tachycardia Disturbances of atrial impulse formation • Atrial premature complexes • Atrial tachycardia • Atrial flutter • Atrial fibrillation • Disturbances of ventricular impulse formation • Ventricular premature complexes • Ventricular tachycardia • Ventricular asystole- no contraction • Ventricular fibrillation Disturbances of impulse conduction • Sinus arrest • Atrial standstill • First-degree AV block • Second degree AV block • Third degree AV block
  • 7.
  • 8. Normal Sinus Rhythm • Normal ECG tracing depicting a normal rhythm of electrical conductivity through the heart
  • 9. (Respiratory) Sinus Arrhythmia • All criteria of normal rhythm except heart and pulse rates increase with inspiration and decrease with expiration • Normal finding in brachycephalic breeds and in chronic respiratory disease • Increased number of cardiac cycles during inspiration; decreased number during expiration
  • 10. Sinus Bradycardia • • • Regular sinus rhythm but heart rate is below normal Heart rate <60 Causes: Athletes, Hypothermia, Hypoglycemia,Hypothyroidism
  • 11. Sinus Tachycardia • Regular sinus rhythm with increased ventricular rate • Heart rate >100 • Causes include: pain, fever, excitement, hyperthyroidism
  • 12. Atrial Premature Complexes • Premature atrial impulses originating from atrial site other than SA node • Seen in electrolyte disturbances, drug reactions, congenital heart disease, and neoplasia • Premature P wave causes a heartbeat sooner than it should be • QRS complexes are normal unless the P wave is so immature that it overlaps to varying degrees
  • 13. Atrial Flutter • • • • • Appears as a regular, “sawtooth” formation between the mostly normal QRS complexes Occurs when the ventricular rate differs from the atrial rate Single area in atrium other than SA node starts impulse AV node “gatekeeper” only allows some impulses through to ventricles (lots of P waves, regular QRS) Atrial flutter is the precursor to atrial fibrillation
  • 14. Atrial Fibrillation •Fibrillation is the rapid, irregular, and unsynchronized contraction of muscle fibers •Caused by numerous disorganized atrial impulses frequently bombarding the AV node •Ventricular depolarization rate is irregular and rapid •NO P waves are evident; replaced by numerous f (fibrillation) waves
  • 15.
  • 16.
  • 17.
  • 18.
  • 19.
  • 20. Premature Ventricular Complexes (PVCs) • • • • “Premature beats” - cardiac impulses initiated within the ventricles instead of the sinus node Ventricle discharges before the arrival of the next anticipated impulse from the SA node Can occur at any rate but pose a greater danger with tachycardia Associated with congenital defects, cardiomyopathy, GDV, drug reactions, cardiac neoplasia, anemia, acidosis, hyperthyroidism, hypokalemia
  • 21. PVCs (cont’d) • • • • The P wave is often not seen on the ECG tracing A wide, distorted/bizarre QRS complex is evident The beat preceding the PVC and the beat following are usually equal to the time of two normal beats May treat with IV lidocaine
  • 22. Ventricular Tachycardia “V - Tach” • One strong ventricle impulse that hijacks the conduction system of the heart. Patient may be “stable” with a pulse or unstable with “no pulse” • AV node is on its own and SA node is not working • A series of three or more PVCs in a row • Life threatening • Treatment is reset heart via defibrillation
  • 23. Ventricular Fibrillation •The mechanical pumping of the heart is not evident on the ECG •Many weak impulses other than AV node present in ventricles •The ECG has bizarre baseline with prominent undulations due to weak and uncoordinated ventricular contractions •Low to absent cardiac output •Associated with shock, trauma, electrolyte imbalances, drug reactions, electric shock, hypothermia, cardiac sx •Rapidly fatal
  • 24. V Fib cont. • • • There are no recognizable P or QRS complexes Irregular, chaotic, deformed reflections of varying width, amplitude, and shape Unless controlled immediately, ventricular fibrillation will result in cardiac arrest
  • 25. Atrial Standstill • • • • SA node sends impulse but atria do notcontract No P waves seen Hyperkalemia is most common cause decrease potassium If not due to increased potassium, pace makeris warranted
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  • 29. First Degree AV Block • • • • Delay in conduction of an impulse throughthe AV junction and Bundle of His The PR interval is longer thannormal This type of heart block is a result of a minor conduction defect Seen in older patients secondary to degenerative changes in the conduction system
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  • 31. Second Degree AV Block • Some atrial pulses are not conducted through the AV node and therefore do not cause depolarization of the ventricles • There are two types: • Type I (Wenckebach type I AV block): progressive lengthening of the PR interval until no complex is conducted • P waves occurring without QRS complexes “dropped beats”
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  • 33. Second Degree AV Block (cont’d) • Mobitz Type II: A intermittent block at the AV node, that conducts some impulses but blocks others • A constant PR interval that is usually of normal duration with random dropped beats • In the case of type 2 block, atrial contractions are not regularly followed by ventricular contraction • 2 or more dropped QRS in a row
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  • 35. Third degree AV block (Complete Heart Block) • The cardiac impulse is completely blocked in the region of the AV junction and/or all bundle branches • The most severe heart block • No relationship between P waves and QRS complexes; atria and ventricles each beat independently and do not communicate at all
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