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Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
Acute scrotum is a general term referring to an emergency condition affecting the contents or the wall of the scrotum.
There are a number of conditions that present acutely, predominantly with pain and/or swelling
A careful and detailed history and examination, and in some cases, investigations allow differentiation between these diagnoses. A prompt diagnosis is essential as the patient may require urgent surgical intervention
Testicular torsion refers to twisting of the spermatic cord, causing ischaemia of the testicle.
Testicular torsion results from inadequate fixation of the testis to the tunica vaginalis producing ischemia from reduced arterial inflow and venous outflow obstruction.
The prevalence of testicular torsion in adult patients hospitalized with acute scrotal pain is approximately 25 to 50 percent
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
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NVBDCP.pptx Nation vector borne disease control programSapna Thakur
NVBDCP was launched in 2003-2004 . Vector-Borne Disease: Disease that results from an infection transmitted to humans and other animals by blood-feeding arthropods, such as mosquitoes, ticks, and fleas. Examples of vector-borne diseases include Dengue fever, West Nile Virus, Lyme disease, and malaria.
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Flu Vaccine Alert in Bangalore Karnatakaaddon Scans
As flu season approaches, health officials in Bangalore, Karnataka, are urging residents to get their flu vaccinations. The seasonal flu, while common, can lead to severe health complications, particularly for vulnerable populations such as young children, the elderly, and those with underlying health conditions.
Dr. Vidisha Kumari, a leading epidemiologist in Bangalore, emphasizes the importance of getting vaccinated. "The flu vaccine is our best defense against the influenza virus. It not only protects individuals but also helps prevent the spread of the virus in our communities," he says.
This year, the flu season is expected to coincide with a potential increase in other respiratory illnesses. The Karnataka Health Department has launched an awareness campaign highlighting the significance of flu vaccinations. They have set up multiple vaccination centers across Bangalore, making it convenient for residents to receive their shots.
To encourage widespread vaccination, the government is also collaborating with local schools, workplaces, and community centers to facilitate vaccination drives. Special attention is being given to ensuring that the vaccine is accessible to all, including marginalized communities who may have limited access to healthcare.
Residents are reminded that the flu vaccine is safe and effective. Common side effects are mild and may include soreness at the injection site, mild fever, or muscle aches. These side effects are generally short-lived and far less severe than the flu itself.
Healthcare providers are also stressing the importance of continuing COVID-19 precautions. Wearing masks, practicing good hand hygiene, and maintaining social distancing are still crucial, especially in crowded places.
Protect yourself and your loved ones by getting vaccinated. Together, we can help keep Bangalore healthy and safe this flu season. For more information on vaccination centers and schedules, residents can visit the Karnataka Health Department’s official website or follow their social media pages.
Stay informed, stay safe, and get your flu shot today!
Can You Get High from Second Hand Marijuana Smoke?
1. 6/9/2020 The Effect of Secondhand Marijuana Smoke
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SECONDHAND CANNABIS SMOKE
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2. 6/9/2020 The Effect of Secondhand Marijuana Smoke
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Secondhand smoke (SHS) comprises the smoke released from the burning tip of a cigarette (typically tobacco, but in this
case marijuana as well) between pu s and exhaled by the person smoking. It is also called ‘passive smoking,’
‘environmental smoke,’ and ‘involuntary smoking.’ These terms suggest that active smoking may be acceptable while
passive smoking is not. So why SHS faces such treatment?
Exposure to secondhand tobacco smoke is a serious public health threat worldwide, as many countries fail to protect
their citizens, especially women and children. According to a research paper
(https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3477960/pdf/AJPH.2012.300805.pdf) published by a group of scientists
from The Institute for Health & Aging at the University of California, approximately 603,000 deaths are caused by
secondhand tobacco smoke exposure every year.
Exposure to secondhand tobacco smoke takes a heavy death toll in developed countries as well. For example, the latest
statistics (https://www.cdc.gov/tobacco/data_statistics/fact_sheets/health_e ects/tobacco_related_mortality/index.htm)
provided by the Centers for Disease Control and Prevention suggest that exposure to SHS causes an estimated 41,000
deaths every year.
But what about secondhand marijuana smoke?
While some people believe that secondhand marijuana smoke is benign, there is some evidence suggesting otherwise.
For example, a recent study published in the Journal of the American Heart Association
(http://jaha.ahajournals.org/content/5/8/e003858.full) found that one minute of exposure to cannabis SHS signi cantly
impaired endothelial function in animals for at least 90 minutes, which is appreciably longer than the same treatment
with tobacco SHS.
Does that mean that Marijuana SHS is More Dangerous than Tobacco SHS?
Clearly, more research is needed to determine if negative e ect of marijuana SHS on health is the same in humans.
Right now, though, it is apparent that non-smokers should avoid exposure to marijuana SHS to be completely safe. The
same applies to tobacco SHS as well.
3. 6/9/2020 The Effect of Secondhand Marijuana Smoke
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The Effect of Secondhand Marijuana
Smoke
Secondhand Cannabis Smoke Can Be Glorious or Trouble
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The E ect of Secondhand Marijuana Smoke
Second-Hand Marijuana Smoke - Can You Get High? Does it Show up on a Drug Test? (https://vimeo.com/352944950)
from CannabisNet (https://vimeo.com/user84347733) on Vimeo (https://vimeo.com).
Secondhand Smoke
Second-Hand Marijuana
Smoke - Can You Get
High? Does it Show up on
a Drug Test?
from CannabisNet
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4. 6/9/2020 The Effect of Secondhand Marijuana Smoke
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The current ndings are a clear cause for concern, said Dr. Seth Ammerman (https://med.stanford.edu/pro les/seth-
ammerman) from Stanford in a recent US News interview. “The notion that marijuana is natural and therefore safe is
"misleading," he pointed out. ”Cyanide comes from a plant, as an example. There are many deadly poisons that also
come from plants."
Can you get “Contact High” from Marijuana SHS?
A popular myth about marijuana smoking says that one can get high by inhaling secondhand smoke. Accordingly, you
may feel the same e ect even though you’re not smoking. While this sure sounds great for those who would like to
believe in it, it is pretty much impossible to get the same e ect, or get high, from inhaling marijuana SHS.
This statement is backed up by scholar evidence. A 2010 study completed by a group of Dutch researchers
(https://www.ncbi.nlm.nih.gov/pubmed/20465865) exposed eight healthy non-smokers to cannabis smoke in the
everyday setting in one of the co eeshops in a well-attended co ee shop in Maastricht, Netherlands. Every day, they
5. 6/9/2020 The Effect of Secondhand Marijuana Smoke
https://cannabis.net/blog/smoke/the-effect-of-secondhand-marijuana-smoke 6/16
were exposed to the smoke for three hours, and later blood and urine samples were collected to evaluate the exposure.
According to the results of this study, the participants’ samples showed minor traces of tetrahydrocannabinol (THC) in
their blood. However, the concentration was so insigni cant that every one of them would easily pass a standard drug
test. Therefore, they concluded that even a signi cant exposure to marijuana SHS leads to an extremely small amount
of THC in the system (http://www.ncsm.nl/english/thc-weed-detox), so the person will not feel any e ects, let alone get
“contact high.”
So does that mean I can sit with My Weed-Smoking Buddies on the Couch and Don’t Worry about a Drug Test the
Next Day?
Well, there’s the tricky part: you can’t. Until you make sure that the room is properly ventilated.
Yes, it was proven that a 3-hour exposure produced an incredibly small amount of THC in the blood. That study was
conducted in an everyday setting of a well-ventilated co ee shop, which is very di erent from a poorly ventilated or
unventilated environment. Every room in your house has a much poorer ventilation than in a co eeshop in the
Netherlands because the latter have to comply with extreme requirements of the government.
As it turns out, passive smoking can, in fact, cause you to fail a drug test. This unexpected nding was produced by a
2015 study conducted by the John Hopkins University (http://www.drugandalcoholdependence.com/article/S0376-
8716(15)00160-X/fulltext). Smokers and non-smokers were placed in both unventilated and ventilated rooms to see if
the exposure would be di erent.
Surprisingly, it was. In an unventilated environment, non-smokers obtained detectable THC levels in blood and urine, so
they would not pass a standard drug test. The reason for this, as you already guessed, was the lack of ventilation. In
addition to producing detectable THC levels, marijuana smoke also caused minor increases in heart rate, impaired
performance on the digit symbol substitution task (DSST), and mild to moderate self-reported sedative e ects.
Let’s wrap this up one last time: anyone exposed to marijuana smoke in an unventilated or poorly ventilated
environment can fail a drug test. So, you can experience some of the e ects of marijuana exposure. However, if you
keep a window open, it would be almost impossible for you to inhale enough marijuana smoke to fail a drug test.
6. 6/9/2020 The Effect of Secondhand Marijuana Smoke
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What Can Second Hand Cannabis Smoke Do To You? (//www.slideshare.net/binaryoptionsignals/what-can-second-hand-cannabis-
smoke-do-to-you)
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The Bottom Line
The known health risks of tobacco SHS raised questions about whether marijuana poses a similar threat. However, at
this point, very little research has been conducted in this area. There is some evidence suggesting an even more
harmful e ect of secondhand marijuana, but no human studies have been registered so far.
In terms of drug testing, it is clear that a person exposed to marijuana SHS is safe if the environment is properly
ventilated. Otherwise, the exposure may produce detectable levels of cannabinoids in blood and urine. Those saying
that one can get “contact high” are likely to be wrong because one would feel e ect from a lack of oxygen and the
smoke content much faster.
So, even though many recreational marijuana users would love the “contact high” to be true, sorry, it is nearly
impossible. This, however, should not stop vulnerable population such as women and children from taking precautions
to avoid exposure to to marijuana SHS.