SlideShare a Scribd company logo
1 of 117
BURNS AND ITS
PHYSIOTHERAPY MANAGEMENT
1
Dr. Hitiksha Bhalodia
(MPT in Women’s Health)
Assistant Professor , PIPT
Course Name: Physiotherapy in Medical and Surgical Condition
Course Code: 07101404
Course Objective: Select strategies for cure, care and prevention; adopt restorative and
rehabilitative measures for maximum possible functional independence of a patient at
home, work and in community. (CO2)
What is burns…?
⚫ It is a type of coagulative necrosis of tissue, caused
by thermal application transfer from source to body
or
⚫ Tissue injury caused by thermal, electrical or
chemical agents
2
Risk factors
⚫ Fire/combustion
□ Firefighter
□ Industrial workers
□ Occupant of burning structures
⚫ Chemical exposure
□ Industrial workers
⚫ Electrical exposure
□ Electrician
□ Electrical power distribution worker
3
Types of burn Injuries
1. thermal
burn
⚫ Skin injury
⚫ Inhalation injury
4
2. Chemical burn:
□ Skin injury
□ Inhalation injury
□ Mucous membrane injury
5
3. Electrical burn:
□ Skin injury’
□ Nerves, muscle and bone damage
4. Radiation burn
6
Effects:
● Burn injury causes destruction of tissue, usually the
skin from exposure to thermal extremes (either hot or
cold), electricity, chemical and radiation
⚫ The mucosa of the upper GI system (mouth,
esophagus, stomach) can be burned with ingestion of
chemicals
⚫ The respiratory system can be damaged if hot gases, smoke or toxic
chemical fumes are inhaled
⚫ Fat, muscle, bone and peripheral nerves can be affected in electrical
injuries or prolonged thermal or chemical exposure
⚫ Skin damage can result in altered ability to sense pain, touch and
temperature
7
Classification of burn
injury andits
pathophysiology
8
1. Depending upon depth of burn injury:
□ Previously used classification
First degree burn: only epidermis affected
Second degree burn: epidermis and dermal appendages
Third degree burn: epidermis and all dermis
Fourth degree burn: epidermis, dermis, and
subcutaneous tissues (fat, muscle, bone and peripheral
nerves)
9
Recent classification of burn depending
on depth of tissue injury
1. Epidermal burn
2. Superficial partial thickness burn
3. Deep partial thickness burn
4. Subdermal burn
10
11
Epidermal burn
⚫ Cell damage to epidermis
⚫ This depth of burns correlates to practice pattern 7B;
impaired integumentary integrity associated with
superficial skin involvement
e.g., sunburn
12
⚫ Clinically skin appears red / erythematous
⚫ Painful, dry, red burns which blanch with pressure
⚫ No blister
⚫ Slight edema may apparent
⚫ Spontaneous healing (within 7 days) & no scar
13
Superficial partial-thickness burn
⚫ Damage occurs through the epidermis and into the
papillary layer of dermis (second-degree burn)
⚫ The epidermal layer destroyed completely
⚫ This depth of burn corresponds to APTA pattern 7C;
impaired integumentary integrity associated with
partial thickness skin involvement and scar formation
14
⚫ Presence of intact blisters
⚫ Due to blister increased inflammatory response
⚫ Moist weeping or glistening surface when blisters
removed
⚫ Very painful
⚫ Sensitive to changes in temperature, exposure to air
currents, & light touch
⚫ Moderate edema, spontaneous healing, minimal
scarring & discoloration
⚫ Heal within 7-21 days
26
16
Deep partial thickness burn
⚫ Involves destruction of the epidermis and papillary dermis
with damage down into the reticular dermal layer
⚫ Impairment matches to APTA pattern 7C
⚫ Deep partial thickness burns appears as a mixed red or waxy
white color
17
⚫ Blanching and brisk capillary refill
⚫ Broken blisters, wet surface
⚫ Their color may range from patchy, cheesy white to red and
they do not blanch with pressure
⚫ Sensitive to pressure but insensitive to light touch or soft
pinprick
⚫ Affected area is depressed
⚫ Healing with skin graft or flap or scarring
18
Full thickness burn
⚫ All epidermal and dermal layers are destroyed
along with subcutaneous fat layer
⚫ This burn depth consistent with practice pattern 7D;
i.e., impaired integumentary integrity associated
with full-thickness skin involvement and scar
formation
⚫ A full-thickness burn is characterized by a hard,
parchment-like Escher covering the area.
19
⚫ Escher is devitalized tissue consisting of
designed coagulum of plasma & necrotic cells
⚫ Escher feels dry, leathery and rigid
⚫ the color of Escher can vary from black to deep
red to white ; latter indicates total ischemia of the
area
⚫ Hair follicles are completely destroyed
20
⚫ A major problem arises from deep burns is the damage
to the peripheral vascular system
⚫ Large amount of fluid leak into the interstitial space □
increase pressure in extracellular space □ constriction
to deep circulation
⚫ Very slow healing following this type of burn
21
22
Subdermal burn
⚫ Involves complete destruction of all tissue from the
epidermis down to and through the subcutaneous
tissue
⚫ Occurs following prolonged contact with a heat source
⚫ This depth of the injury correlates with APTA practice
pattern 7E; Impaired integumentary integrity associated
with skin involvement extending into fascia, muscle or
bone and scar formation
⚫ Extensive surgical and therapeutic management is
necessary to return a patient to some degree of function
23
Electrical burn
⚫ S/S of an electric burn may vary according to the
type of current, intensity of the current and the area
of the body the electric current passes through
⚫ Electric current follows the course of least resistance
offered by various tissues. i.e., nerves, followed by
blood vessels offers least resistance; bones offers most
resistance.
⚫ Tissue damage results from tissue resistance to the
passage of the current or by direct electrical current
24
⚫ Typically contact sites will exist where the patient
first came into contact with the electricity and a
second site where the patient was grounded
⚫ The affected skin appears yellow, ischemic and dry
⚫ Tissues along the pathway of the current may be
damaged owing to heat that developed as a result of
tissue resistance to current passage
⚫ Blood supply to surrounding tissues may be
altered and arteries undergo spasm
25
⚫ There can be other consequences of electricity
passing through the body such as
□ Cardiac arrhythmias and acute renal failure secondary
to fluid and electrolyte imbalances and release of
myoglobin into blood
□ The most severe complication following
electrical current damage is acute spinal cord
damage or vertebral fracture
□ Ventricular fibrillation and respiratory arrest
26
Burn wound Zones
⚫ Three zones:
1. Zone of coagulation
2. Zone of stasis
3. Zone of hyperemia
27
Zone of coagulation
⚫ In the zone of coagulation cells are
irreversibly damaged & skin death occurs
⚫ This area is equivalent to a full-thickness burn &
will require a skin graft to heal
⚫ Here, because of lack of viable tissue & the amount of
Escher, the risk of infection increased
⚫ This potential complication emphasizes the need for
careful monitoring, use of antibiotics & the
treatment of a burned pt. in specialized burn center
28
Zone of stasis
⚫ It contains injured cells that may die within 24 to
48 hrs. without diligent treatment
⚫ Here, in this zone; Infection, Drying & inadequate
perfusion of the wound □ conversion of potentially
salvageable tissue to completely necrotic tissue &
enlargement of the zone of coagulation
⚫ Splints or compression bandages, if applied too
tightly, can compromise this area
29
Zone of Hyperemia
⚫ Finally in this zone, there is minimal cell damage
⚫ Tissue should recover within several days with no
lasting effects
30
Extent of Burned Area
⚫ A major consideration when determining the
severity of a burn is the extent of body surface
involved
⚫ % of Total body surface area(TBSA) burned;
calculated by Rule of nines
31
32
33
34
35
⚫ The rule of nines divides the body surface into areas of
9% or multiples of 9% of TBSA
⚫ Modified:- according to continuum of age
accommodate for growth of different body segments
36
Total Burn Surface Area (TBSA): the greater the total burn
surface area the poorer the prognosis
Percentage chance of Survival =[100 – (Age in years + percentage
TBSA)]
37
38
39
Other Assessment Details:
40
Burn Patient Severity
Factors to Consider:
● Burn Depth classification
● Body surface area burned
● Age: adult v/s pediatric
● Preexisting medical conditions
● Associated trauma
- Blast injury
- Fall injury
- Airway compromise
- Child abuse
41
● Patient Age:
Less than 2 or greater than 55 have increased
incidence of complication
● Burn Configuration:
- Circumferential burns can cause total occlusion
of circulation to an area due to edema
- Restrict ventilation if encircle the chest
- Burns on joint area can cause disability due to
scar formation
42
Critical Burn
Criteria
⚫ 30 > 10% BSA
⚫ 20 > 30% BSA
> 20% pediatric
⚫ Burns with respiratory injury
⚫ Hands, face, feet or genitalia
⚫ Burns complicated by other trauma
⚫ Underlying health problems
⚫ Electrical & deep chemical burns
43
Moderate burn criteria
⚫ 30 : 2-10 % BSA
⚫ 20 : 15-30 % BSA
10-20% pediatric
⚫ Excluding hands, face, feet or genitalia
⚫ Without complicating factors
44
Minor Burn Criteria
⚫ 30< 2% BSA
⚫ 20< 15% BSA
10% BSA
⚫ 10< 15% BSA
45
Complications of Burn
injury
⚫ Depending on the extent of burn injury, the
depth of the burn & the type of burn, there may
be secondary systemic complications
⚫ In addition the health, age, & psychological status
of a patient who is burned will affect these
complications
46
1. Infection
⚫ Infection is conjunction with organ system failure, is a
leading cause of mortality from burns
⚫ Some virulent strains of Pseudomonas aeruginosa &
Staphylococcus aureus are resistant to antibiotics &
have been responsible for epidemic infections in burn
centers
⚫ Systemic antibiotics are used to treat both burn
and general system infections once they have
been documented
47
2. Pulmonary complications
⚫ Inhalation injury: pt who has been burned in closed
space
⚫ Signs of an inhalation injury include facial burns,
singed nasal hairs, harsh cough, hoarsness, abnormal
breath sounds, respiratory distress, & carbonaceous
sputum & hypoxemia
⚫ The incidence of pulmonary complications is
extremely high after severe burns, & death due to
pneumonia
48
⚫ The primary complications associated with this
injury are carbon monoxide poisoning, tracheal
damage, upper airway obstruction, pulmonary
oedema, and pneumonia.
⚫ Lung damage from inhaling noxious gases and smoke
may be lethal
49
3. Metabolic Complications
⚫ Metabolic rates may increase up to 50% in a 25%
TBSA burn and much more as the burn size increases
⚫ The consequences of the increased metabolic and
catabolic activity following a burn are:-
- A rapid decrease in body weight
- Negative nitrogen balance and
- A decrease in energy stores that are vital the
healing process.
50
⚫ Following increased metabolic activity there will be
increase of 1.80F to 2.60F (10C to 20C) in core
temperature that seems to be due to a resetting of the
hypothalamic temperature centers in the brain
⚫ As a part of pt.'s altered metabolism, protein from
muscle tissue is preferentially used as a source of
energy muscle atrophy
51
4. Cardiovascular complications
⚫ Hemodynamic changes result from a shift in fluid to the
interstitium, which subsequently reduces the plasma &
intervascular fluid volume in a burnt pt.
⚫ This shift of fluid to the interstitium can result in
significant edema
⚫ Decrease cardiac output
● Hematological changes: alterations in platelet
concentration & function, clotting factors & white blood
cell components, RBC dysfunction & decrease hemoglobin
& hematocrit
52
Heterotopic Ossification
⚫ Pts. With more than 20% TBSA burn highly susceptible
⚫ Usually occurs in areas of full-thickness injury or sites that
remains unhealed for prolonged period
⚫ Symptoms: decrease ROM, point specific pain, generalized
pain
53
Neuropathy
⚫ Peripheral neuropathy: either polyneuropathy or local
neuropathy (depending on involvement of TBSA)
54
Pathological Scars
⚫ Burn scar occur in area of deep partial-thickness burn
□ where healing is with skin grafting
⚫ If maturing tissue demonstrates a greater rate of
collagen production than degradation, a scar become
raised and thick □ hypertrophic scar
55
⚫ Formation of Escher
⚫ Renal or hepatic failure
56
Psychological changes
⚫ Fear/ anxiety
⚫ Denial
⚫ Depression
⚫ Guilt
⚫ Grief & mourning
⚫ Loss of will to live
⚫ Apathy
⚫ Necrophilous orientation
⚫ anger
57
Management
Medical management of burns
● Initial treatment:
Goals: -
□ to address critical life-threatening problems & stabilize the
pt. through procedures designed to:
- Establish & maintain an airway
- prevent cyanosis, shock & hemorrhage
- establish baseline data on pt.; extent & depth of burn
injury
- Prevent or reduce fluid losses
- clean the pt. & wounds
- Examine injuries
- Prevent pulmonary & cardiac complications
58
⚫ Transport pt. to burn center from the site of
accident
● Assessment : demographic details, history,
calculation of TBSA
⚫ Fluid volume replacement therapy
⚫ Wound cleansing
⚫ Debridement
⚫ analgesics
59
Wound Care:
⚫ Remove Escher along with sharp debridement
⚫ Apply topical medications either with open
technique or closed technique
60
Surgical Management of burn wound
Primary Excision:
- Surgical removal of Escher
- With excision removal of peripheral layers of
Escher until vascular, viable tissue is exposed as the
site for skin graft placement
- Burn wound is closed with a skin graft at time
of primary excision
Grafts used are: autograft, allograft or xenograft
□ Now a days skin substitutes are used for coverage of
an excised wound
61
Skin grafting
⚫ Done under anesthesia
⚫ Skin used for a graft usually is removed with a
dermatome
⚫ It can either split-thickness skin graft (epidermis with
variable amount of dermis) or full-thickness skin graft
(full epidermis and dermis)
Correction of scar contracture
62
The Z-plasty servesto lengthen a scar
by interposing normal tissue in the line of the
scar.
63
Physical Therapy Management
● PT interventions are directed towards:-
□ Prevention of scar & contracture
□ Preservation of normal ROM
□ Prevention or minimization of hypertrophic scar formation
and cosmetic deformity
□ Maintenance or improvement in muscular strength
□ Improvement in cardiovascular endurance
□ Return to pre-burn function and performance of activities of
daily living
64
ASSESSMENT
⚫ Demographic details:
⚫ Review of medical records
⚫ History
⚫ Observation
⚫ Palpation
⚫ Examination
□ Initial examination for the depth of burn
□ Percentage of burn (TBSA) & percentage chance of
survival
65
● Screening/assessment for complications:
□ Hypovolemic
shock:-
Hypotension
Oliguria
Tachycardia
Sweating, Pallor
Clouding of consciousness
□ Septic Shock:- Increased
temperature
Hypotension
Oliguria
Dry & pink extremities
Altered pulmonary functions
66
⚫
67
● Skin Assessment:
□ Appearance
□ Temperature
□ Moisture/Dryness
□ Texture
□ Color
□ Size
□ Pulses
□ Sensations
68
⚫ Assessment of ROM
⚫ Tightness/contracture
⚫ Functional mobility
⚫ Gait and
⚫ functional status (FIM)
69
Special tests
● Pigmentation of the scar:
0 = normal
1 = hypopigmentation
2 = hyperpigmentation
● Height of scar:
0 = normal
1 =< 2mm
2 =<5
3 =>5
70
Vancouver
Scar
Scale(VSS)
71
⚫ ICF: Condition
□ Body structures and function
□ Activities
□ Function
Contextual factors: personal and environmental
facilitators and barriers
72
Anticipated Goals and
Expected outcomes (In
general)
⚫ Risk of infection and complications is reduced
⚫ Wound and soft tissue healing is enhanced.
⚫ Risk of secondary impairments is reduced.
⚫ Maximal range of motion is achieved.
⚫ Pre injury level of cardiovascular endurance is restored.
⚫ Good to normal strength is achieved.
73
⚫ Independent ambulation is achieved.
⚫ Independent function in ADL and IADL is increased.
⚫ Scar formation is minimized.
⚫ Patient, family, and caregivers’ understanding of
expectations and goals and outcomes is
increased.
⚫ Aerobic capacity is increased.
⚫ Self-management of symptoms is improved
74
Positioning And Splinting
⚫ Positioning begin on the day of admission
⚫ Concept: positioning burned areas in place and maintain
the body part in the opposite plane and direction to
which it will potentially contract
● Goals: - To minimize edema
- To prevent tissue destruction
- maintain soft tissues in elongated state
- preserve function
75
Positioning in bed of pt. with burn of the anterior
neck
76
Positioning in bed of pt. with burns of the axilla
77
Positioning of upper extremities to reduce edema while seated
78
● Splinting given to provide anti-deformity positions
● Indications:- Prevention of contracture
- Maintenance of ROM
- Reduction of developing contracture
- Protection of jt. or tendon
- Reduce pain
79
⚫ Splinting usually worn at night following skin grafting
⚫ Static or dynamic splint can be used
□ Dynamic splint apply low load, prolonged stress
that can be adjusted to pt.'s tolerance
80
81
82
83
Therapeutic Exercises
● Active and passive exercises:
□ Can be initiate from the day of admission
□ Before starting exercise look for the complications
and vital stability and consciousness/alertness
□ Perform AROM of all extremities and trunk
including unburned areas
□ Avoid ROM exercise of injured area in case of recent
skin grafting & allow the graft to adhere
84
Dosages:
□ AROM should be done twice daily
□ For sedated pts. PROM can be initiated and given thrice/day
□ Repetitions and degree of movement depends on
pt. tolerance and limit of stretch
85
Contraindications:
86
⚫ Active or Passive range of motion exercises should not
be carried out if there is suspected damage to any
tendons
e.g., flexion of PIP should be avoided at all to prevent
extensor tendon rupture
⚫ Range of motion exercises are also
contraindicated post skin grafting as a period of
3-5 days immobilisation is required to enable
graft healing
Practical considerations while mobilizing burn patient
⚫ Be aware of dressing clinic/daily dressing changes.
Mobilisation should coincide with this as it is important
to monitor the wound during AROM frequently.
⚫ Timing of pain relief. This should be timed appropriately
to ensure maximal benefit during treatment sessions.
⚫ Observe the patient carrying out the AROM and PROM
exercises prior to beginning treatment. Also observe the
patient taking on/off splints.
87
⚫ Always monitor for post exercise pain and wound
breakdown.
⚫ Avoid blanching for long period as you may
compromise vascularity.
⚫ The patient may present with a reduced capacity for
exercise secondary to increased metabolic rate,
altered thermoregulation and increased nutritional
demands.
⚫ Postural hypotension may be present due to prolonged
bed rest and low haemoglobin
88
Ambulation
⚫ It should be initiated at the earliest appropriate time
⚫ If LEs are skin grafted ambulation may be discontinued
until it is safe to resume
⚫ Wrap elastic bandages after skin grafting while
ambulation
(supports new graft and promote venous return)
⚫ If orthostatic hypotension is there use tilt table to make
pt. standing gradually
89
“The rewards of a successful POC are
tremendous when a patient who has suffered a
life-threatening burn is able to walk out of the
hospital and return to productive community
90
involvement.”
Resistive and conditioning exercises
⚫ Pts. With major burns may lose body weight and lean
muscle mass can decrease rapidly
⚫ Exercise consist of isometric, isokinetic, isotonic or using
other resistive training devices can be started at rehabilitation
stage
⚫ General principles of exercise training and strength
improvement should be followed, but they may need to
be modified on the basis of a patient’s condition and
stage of wound healing
91
⚫ Initially Isometrics used to maintain muscle properties
& with that help to prevent further muscle atrophy
⚫ In progression use weights and resistive equipments to
improve muscle strength
⚫ Regular and accurate monitoring of vitals
(including PR,RR, BP, SPO2, RPE) before, during
and after exercises.
92
Scar Management
Pressure Garment Therapy (PGT)
⚫ Following wound closure, a skin graft or healed burn wound is
vascular, flat and soft.
⚫ To achieve this following 3-6 months, dramatic changes may
occur – the newly healed areas may become raised and firm
⚫ If wound healing takes longer than 10 to 14 days or skin
grated, pressure usually is indicated
⚫ Pressure has been used successfully to hasten scar
maturation and minimize hypertrophic scar formation
93
⚫
Pressure may exert control over hypertrophic scarring by;
- Pressure decreases blood flow
- Local hypoxia of hypervascular scars
- Reduction in collagen deposition
All three together causes:
o Decreases scar thickness
o Decreases scar redness
o Decreases swelling
o Reduces itch
o Protects new skin/grafts
o Maintains contours
⚫ The earlier the scar tissue is exposed to pressure, the better
result
94
Method of application:
For LE – fig. of eight pattern
For UE – spiral wraps
For trunk – circular wrap
Hand and toes – self-adherent bandages
95
96
Practical recommendations and safety considerations
⚫ Pressure exerted around 15 – 40 mmHg
⚫ Time : Worn up to 23 hrs./day
⚫ Worn as soon as wound closure has been obtained
(Post grafting after 10-14 days it is recommended and
it should be worn up to one year or until scar
maturation)
97
Possible complications/ confounding factors for
use of PGT
□ Lack of a scientific evidence to established optimum pressure
□ Non-Compliance ( due to comfort, movement, appearance)
□ Heat and perspiration
□ Swelling of extremities caused by inhibited venous return
□ Skin breakdown
□ Web space discomfort
□ Inconvenience
□ Personal hygiene difficulties possibility of infection
□ Allergies to material
98
Silicone Gel:
Sheets of silicone polymer gel may be applied directly over an
actively maturing scar.
⚫ Complication: local rash, rarely skin breakdown
99
Mechanism: unknown but possible mechanisms are as follow
□ Hydration: decreases capillary activity and collagen production, through
inhibition of proliferation of fibroblasts
□ Rise in temperature increases collagenase activity and scar
breakdown
□ polarized electric fields creates negative charge within silicone causes
polarization of scar tissue, resulting in involution of scar
□ Presence of silicone oil
□ Oxygen tension inhibit hypoxia signal
□ Increase in mast cell helps in remodeling of tissue
100
101
Massage:
Mechanisms on which Scar massage works:
- Prevent adherence
- Reduce redness
- Reduce elevation of scat tissue
- Relieve pruritus
- Maintain/restore moisture
102
Scar Massage Techniques:
⚫ Deep friction massage□ to loosen scar adhesions
⚫ Finger and thumb kneading □ mobilize the scar and surrounding
tissue
⚫ Effleurage □ to increase circulation
⚫ Skin rolling □ restore mobility to tissue interfaces
⚫ Wringing □ stretches scar and promote collagenous remodeling
⚫ Retrograde massage to aid venous return, increase lymphatic drainage,
mobilize fluid
114 □ Massage techniques can be used in conjunction with ROM exs
Guidelines for massage during 3 stages of healing:
1. Inflammatory phase: helps to decrease oedema and
increase blood supply
1. Proliferative phase: applies gentle stress to the
healing scar; to ensure correct alignment of
collagen tissues
1. Remodeling phase: massage should be progressed to
include prolonged stretching to minimize adhesions;
that aids in scar tissue breakdown
115
Practical considerations and safety:
□ Clean hands essential
□ Use non irritating lubricant
□ Modify practice according to pt. stage of healing and pain
levels
Contraindications:
- Compromised integrity of epidermis
- Acute infection
- Bleeding
- Graft failure
- Intolerable discomfort
116
Camouflage Make-up
⚫ Used for scars of face, neck and hands
⚫ Mostly used when a person has either
hyperpigmentation or hypopigmentation
⚫ It can also be used before scar maturation; so if pt.
wants to go out in public
⚫ The cosmetics are opaque, color-correct burn scars
and are available in multiple shades to accommodate
various skin colors
⚫ They also are waterproof and worn during all activities
106
The Role of Physiotherapist in rehabilitation
of the chronic Burn patient
⚫ Scar management is initiated in the sub-acute
phase, but it may need to be continued long term,
as many patient suffer from continuing limitation
of ROM
⚫ Healing process may continue for up to two years, or
more, as scar tissue remodels and matures
⚫ Patient may require functional retraining and
integration back into the community and
activities
107
Aerobic and Resistance Training Post Burn
Rationale for training:
□ Low cardiovascular endurance
□ Lower aerobic capacity measured by VO2 max and time to fatigue at one year
post burn compare to age matched healthy control (adults and children > 15%
TBSA)
□ Muscular strength and lean body mass has been found to be significantly less
in pts. Suffering from burns of 30% TBSA
□ Reduced lean body mass, endurance and strength has been associated with
limited standing/walking tolerance
□ Reduced upper limb function and lower health related QOL and ability to
participate in activities
108
Aerobic capacity and muscular strength is diminished by
the following factors:
⚫ Prolonged bed rest necessary in the early recovery process
109
⚫ Hyper metabolism; which may lead to:
- Exhaustion
- Protein catabolism
- Loss of lean body mass
⚫ Impaired thermoregulation
⚫ Inhalation injuries and compromised respiratory function
⚫ Fatigue
⚫ Pain
⚫ Psychological factors
Aerobic training recommendations for practice:
Frequency: 3 days/week; (3-5 days/week)
Intensity: moderate to high intensity i.e.,
65%% 85% of MHR
Type: Interval, continuous; using treadmill, walking,
running exercises
Time: 20-40 minutes/session
6-12 week programme
110
Resistance training recommendations for practice:
Frequency: 3 times/week
(break of more than 48 hrs. must be given between bouts of
resistance training as;
□resistance exs. Causes microtrauma to muscles already in
compromised state
□ resistance exs. In burned pts. stimulates protein synthesis)
Intensity/Type/time: 1 set of 50-60% of 3 RM at 1st week
70-75% of 3 RM for next 2-6 weeks
(4-10 reps)
80-85% of 3 RM for 7-12 weeks (2-6
reps)
111
Mixed and functional strengthening
112
□ Given for specific targeting muscle groups for desired
functional goal
□ Progression made by aiding resistive devices and finally
using free weight training
□ Initiate with 50-60% of 1 RM for 10-15 reps and than
progress further as per pt.’s need over 12 weeks
● Safety considerations for strength and aerobic training:
□ Post burn 6 months to 2 years time given before initiation
of programmes or
□ In case of 40% TBSA it can be started after;
95% healed wounds/scars
ambulatory
psychological status
□ Ensure patient is taking Optimal nutrition and other medications
□ Monitoring of HR, BP, SPO2 & RPE before during and after
124 exercises
Psychological Aspects of Burn patient
125
⚫ Depression and post traumatic stress disorder are most
common disorders following burn
⚫ Pain and cosmetic appearance of individual
Treatment
- Psychotherapy
- CBT
- Medications (SSRI)
- Social skills training and community intervention
- Vocational rehabilitation
Ultrasound Therapy after skin grafting
127
UST can be given on alternate day started from 14th day to
21st day after skin grafting
Dosage: 1MHz, 0.5-0.8 W/cm2, for 5 min
□ (EFFECTIVENESS OF PHYSIOTHERAPY AFTER
SKIN GRAFTING- Indian journal of physiotherapy &
occupational therapy, dec-16)
First Aid in burns
DO’s
128
⚫ Stop the burning process by removing clothing and jewelry
⚫ In electrical burns, put the main switch off as quick as possible
and use wooden chair to push victim away from electricity
⚫ Extinguish flames by pouring plain water; if water is not available
by applying a blanket & removing the blanket as soon the flames
are put off
⚫ In chemical burns, remove or dilute the chemical agent by
irrigating large amount of water
⚫ Use cool running water to reduce the temperature of the burn
⚫ Take care of fractures and probable injuries during transportation.
• Ensure A,B, C before transportation to higher center
Don’ts
129
⚫ Don’t start first aid before ensuring your own safety
⚫ Don’t apply ice it may damage the injured tissues
⚫ Avoid prolonged cooling with water it may
causes hypothermia
⚫ Don.t apply any ointments
⚫ Don’t open blisters with pin, needle until topical
antimicrobials can be applied

More Related Content

Similar to Burns & its Physiotherapy Management..pptx

Burn Lecture
Burn LectureBurn Lecture
Burn Lecture
LEDocDave
 

Similar to Burns & its Physiotherapy Management..pptx (20)

Burn management
Burn managementBurn management
Burn management
 
Peioperative Anaesthesia Management of Burn Patients.pptx
Peioperative Anaesthesia Management of Burn Patients.pptxPeioperative Anaesthesia Management of Burn Patients.pptx
Peioperative Anaesthesia Management of Burn Patients.pptx
 
BURNS
BURNSBURNS
BURNS
 
Burn
BurnBurn
Burn
 
BURNS MANAGEMENT 1.pptx
BURNS MANAGEMENT 1.pptxBURNS MANAGEMENT 1.pptx
BURNS MANAGEMENT 1.pptx
 
5. Burns. Frostbite. Electrical injury-1.pptx
5. Burns. Frostbite. Electrical injury-1.pptx5. Burns. Frostbite. Electrical injury-1.pptx
5. Burns. Frostbite. Electrical injury-1.pptx
 
Burn Lecture
Burn LectureBurn Lecture
Burn Lecture
 
evaluation and management of patient presenting with Burn.pptx
evaluation and management of patient presenting with Burn.pptxevaluation and management of patient presenting with Burn.pptx
evaluation and management of patient presenting with Burn.pptx
 
Critical care in burns patients
Critical care in burns patientsCritical care in burns patients
Critical care in burns patients
 
Burn
Burn Burn
Burn
 
burns-160419144753.pptx
burns-160419144753.pptxburns-160419144753.pptx
burns-160419144753.pptx
 
Burns
BurnsBurns
Burns
 
BURNS .pptx
BURNS .pptxBURNS .pptx
BURNS .pptx
 
Burn and Burn Rehabilitation by Dr shyam sunder sharma
Burn and Burn Rehabilitation by Dr shyam sunder sharmaBurn and Burn Rehabilitation by Dr shyam sunder sharma
Burn and Burn Rehabilitation by Dr shyam sunder sharma
 
BURN (1).pptx
BURN (1).pptxBURN (1).pptx
BURN (1).pptx
 
Burns -RBXY1.ppt
Burns -RBXY1.pptBurns -RBXY1.ppt
Burns -RBXY1.ppt
 
Presentation1.pptx
Presentation1.pptxPresentation1.pptx
Presentation1.pptx
 
Burn evaluation and management
Burn evaluation and managementBurn evaluation and management
Burn evaluation and management
 
3 Burn Management
3 Burn Management3 Burn Management
3 Burn Management
 
Presentation1.pptx
Presentation1.pptxPresentation1.pptx
Presentation1.pptx
 

More from HarishankarSharma27

Approach to Testicular Biopsy.........pptx
Approach to Testicular Biopsy.........pptxApproach to Testicular Biopsy.........pptx
Approach to Testicular Biopsy.........pptx
HarishankarSharma27
 
17_HBV_treatment_3 _special situation..............ppt
17_HBV_treatment_3 _special situation..............ppt17_HBV_treatment_3 _special situation..............ppt
17_HBV_treatment_3 _special situation..............ppt
HarishankarSharma27
 
Muscles of Forearm......................ppt
Muscles of Forearm......................pptMuscles of Forearm......................ppt
Muscles of Forearm......................ppt
HarishankarSharma27
 
mmmt ovary ppt final..................pptx
mmmt ovary ppt final..................pptxmmmt ovary ppt final..................pptx
mmmt ovary ppt final..................pptx
HarishankarSharma27
 
ACUTE LEUKEMIA CME FINAL............pptx
ACUTE LEUKEMIA CME FINAL............pptxACUTE LEUKEMIA CME FINAL............pptx
ACUTE LEUKEMIA CME FINAL............pptx
HarishankarSharma27
 
Template Review Meeting NVHCP.............-1.pptx
Template Review Meeting  NVHCP.............-1.pptxTemplate Review Meeting  NVHCP.............-1.pptx
Template Review Meeting NVHCP.............-1.pptx
HarishankarSharma27
 
FLOWCYTOMETRY........................pptx
FLOWCYTOMETRY........................pptxFLOWCYTOMETRY........................pptx
FLOWCYTOMETRY........................pptx
HarishankarSharma27
 
apoptosis.............................pptx
apoptosis.............................pptxapoptosis.............................pptx
apoptosis.............................pptx
HarishankarSharma27
 
AUTISM1..............................pptx
AUTISM1..............................pptxAUTISM1..............................pptx
AUTISM1..............................pptx
HarishankarSharma27
 
Brunnstrom Approach…..................pptx
Brunnstrom Approach…..................pptxBrunnstrom Approach…..................pptx
Brunnstrom Approach…..................pptx
HarishankarSharma27
 
DOC-20231211-WA0184..................pptx
DOC-20231211-WA0184..................pptxDOC-20231211-WA0184..................pptx
DOC-20231211-WA0184..................pptx
HarishankarSharma27
 
Worst is best good no one new..............
Worst is best good no one new..............Worst is best good no one new..............
Worst is best good no one new..............
HarishankarSharma27
 
New best dist .................do ur ppt
New best dist .................do ur pptNew best dist .................do ur ppt
New best dist .................do ur ppt
HarishankarSharma27
 
DENTAL CONDITIONS neww by Dr VR bhagat renowned pediateician.pptx
DENTAL CONDITIONS neww by Dr VR bhagat renowned pediateician.pptxDENTAL CONDITIONS neww by Dr VR bhagat renowned pediateician.pptx
DENTAL CONDITIONS neww by Dr VR bhagat renowned pediateician.pptx
HarishankarSharma27
 
module-15-unfccc-ipcc good characteristics features very nice presentation.ppt
module-15-unfccc-ipcc good characteristics features very nice presentation.pptmodule-15-unfccc-ipcc good characteristics features very nice presentation.ppt
module-15-unfccc-ipcc good characteristics features very nice presentation.ppt
HarishankarSharma27
 

More from HarishankarSharma27 (20)

Approach to Testicular Biopsy.........pptx
Approach to Testicular Biopsy.........pptxApproach to Testicular Biopsy.........pptx
Approach to Testicular Biopsy.........pptx
 
17_HBV_treatment_3 _special situation..............ppt
17_HBV_treatment_3 _special situation..............ppt17_HBV_treatment_3 _special situation..............ppt
17_HBV_treatment_3 _special situation..............ppt
 
Muscles of Forearm......................ppt
Muscles of Forearm......................pptMuscles of Forearm......................ppt
Muscles of Forearm......................ppt
 
mmmt ovary ppt final..................pptx
mmmt ovary ppt final..................pptxmmmt ovary ppt final..................pptx
mmmt ovary ppt final..................pptx
 
ACUTE LEUKEMIA CME FINAL............pptx
ACUTE LEUKEMIA CME FINAL............pptxACUTE LEUKEMIA CME FINAL............pptx
ACUTE LEUKEMIA CME FINAL............pptx
 
Template Review Meeting NVHCP.............-1.pptx
Template Review Meeting  NVHCP.............-1.pptxTemplate Review Meeting  NVHCP.............-1.pptx
Template Review Meeting NVHCP.............-1.pptx
 
topic seminar sudha-1...............pptx
topic  seminar sudha-1...............pptxtopic  seminar sudha-1...............pptx
topic seminar sudha-1...............pptx
 
FLOWCYTOMETRY........................pptx
FLOWCYTOMETRY........................pptxFLOWCYTOMETRY........................pptx
FLOWCYTOMETRY........................pptx
 
09_HBV_natural history......======....ppt
09_HBV_natural history......======....ppt09_HBV_natural history......======....ppt
09_HBV_natural history......======....ppt
 
2.CELLULAR DEGENERATION AND....... INFILTRATION-1.ppt
2.CELLULAR DEGENERATION AND....... INFILTRATION-1.ppt2.CELLULAR DEGENERATION AND....... INFILTRATION-1.ppt
2.CELLULAR DEGENERATION AND....... INFILTRATION-1.ppt
 
apoptosis.............................pptx
apoptosis.............................pptxapoptosis.............................pptx
apoptosis.............................pptx
 
AUTISM1..............................pptx
AUTISM1..............................pptxAUTISM1..............................pptx
AUTISM1..............................pptx
 
Brunnstrom Approach…..................pptx
Brunnstrom Approach…..................pptxBrunnstrom Approach…..................pptx
Brunnstrom Approach…..................pptx
 
GENETICS ppt...................... -KP.ppt
GENETICS ppt...................... -KP.pptGENETICS ppt...................... -KP.ppt
GENETICS ppt...................... -KP.ppt
 
DOC-20231211-WA0184..................pptx
DOC-20231211-WA0184..................pptxDOC-20231211-WA0184..................pptx
DOC-20231211-WA0184..................pptx
 
Anthropometry me achcha .............pptx
Anthropometry me achcha .............pptxAnthropometry me achcha .............pptx
Anthropometry me achcha .............pptx
 
Worst is best good no one new..............
Worst is best good no one new..............Worst is best good no one new..............
Worst is best good no one new..............
 
New best dist .................do ur ppt
New best dist .................do ur pptNew best dist .................do ur ppt
New best dist .................do ur ppt
 
DENTAL CONDITIONS neww by Dr VR bhagat renowned pediateician.pptx
DENTAL CONDITIONS neww by Dr VR bhagat renowned pediateician.pptxDENTAL CONDITIONS neww by Dr VR bhagat renowned pediateician.pptx
DENTAL CONDITIONS neww by Dr VR bhagat renowned pediateician.pptx
 
module-15-unfccc-ipcc good characteristics features very nice presentation.ppt
module-15-unfccc-ipcc good characteristics features very nice presentation.pptmodule-15-unfccc-ipcc good characteristics features very nice presentation.ppt
module-15-unfccc-ipcc good characteristics features very nice presentation.ppt
 

Recently uploaded

Beyond the EU: DORA and NIS 2 Directive's Global Impact
Beyond the EU: DORA and NIS 2 Directive's Global ImpactBeyond the EU: DORA and NIS 2 Directive's Global Impact
Beyond the EU: DORA and NIS 2 Directive's Global Impact
PECB
 
The basics of sentences session 3pptx.pptx
The basics of sentences session 3pptx.pptxThe basics of sentences session 3pptx.pptx
The basics of sentences session 3pptx.pptx
heathfieldcps1
 
The basics of sentences session 2pptx copy.pptx
The basics of sentences session 2pptx copy.pptxThe basics of sentences session 2pptx copy.pptx
The basics of sentences session 2pptx copy.pptx
heathfieldcps1
 
Making and Justifying Mathematical Decisions.pdf
Making and Justifying Mathematical Decisions.pdfMaking and Justifying Mathematical Decisions.pdf
Making and Justifying Mathematical Decisions.pdf
Chris Hunter
 

Recently uploaded (20)

Beyond the EU: DORA and NIS 2 Directive's Global Impact
Beyond the EU: DORA and NIS 2 Directive's Global ImpactBeyond the EU: DORA and NIS 2 Directive's Global Impact
Beyond the EU: DORA and NIS 2 Directive's Global Impact
 
This PowerPoint helps students to consider the concept of infinity.
This PowerPoint helps students to consider the concept of infinity.This PowerPoint helps students to consider the concept of infinity.
This PowerPoint helps students to consider the concept of infinity.
 
Web & Social Media Analytics Previous Year Question Paper.pdf
Web & Social Media Analytics Previous Year Question Paper.pdfWeb & Social Media Analytics Previous Year Question Paper.pdf
Web & Social Media Analytics Previous Year Question Paper.pdf
 
ICT Role in 21st Century Education & its Challenges.pptx
ICT Role in 21st Century Education & its Challenges.pptxICT Role in 21st Century Education & its Challenges.pptx
ICT Role in 21st Century Education & its Challenges.pptx
 
psychiatric nursing HISTORY COLLECTION .docx
psychiatric  nursing HISTORY  COLLECTION  .docxpsychiatric  nursing HISTORY  COLLECTION  .docx
psychiatric nursing HISTORY COLLECTION .docx
 
How to Give a Domain for a Field in Odoo 17
How to Give a Domain for a Field in Odoo 17How to Give a Domain for a Field in Odoo 17
How to Give a Domain for a Field in Odoo 17
 
ICT role in 21st century education and it's challenges.
ICT role in 21st century education and it's challenges.ICT role in 21st century education and it's challenges.
ICT role in 21st century education and it's challenges.
 
Key note speaker Neum_Admir Softic_ENG.pdf
Key note speaker Neum_Admir Softic_ENG.pdfKey note speaker Neum_Admir Softic_ENG.pdf
Key note speaker Neum_Admir Softic_ENG.pdf
 
The basics of sentences session 3pptx.pptx
The basics of sentences session 3pptx.pptxThe basics of sentences session 3pptx.pptx
The basics of sentences session 3pptx.pptx
 
Ecological Succession. ( ECOSYSTEM, B. Pharmacy, 1st Year, Sem-II, Environmen...
Ecological Succession. ( ECOSYSTEM, B. Pharmacy, 1st Year, Sem-II, Environmen...Ecological Succession. ( ECOSYSTEM, B. Pharmacy, 1st Year, Sem-II, Environmen...
Ecological Succession. ( ECOSYSTEM, B. Pharmacy, 1st Year, Sem-II, Environmen...
 
Class 11th Physics NEET formula sheet pdf
Class 11th Physics NEET formula sheet pdfClass 11th Physics NEET formula sheet pdf
Class 11th Physics NEET formula sheet pdf
 
TỔNG ÔN TẬP THI VÀO LỚP 10 MÔN TIẾNG ANH NĂM HỌC 2023 - 2024 CÓ ĐÁP ÁN (NGỮ Â...
TỔNG ÔN TẬP THI VÀO LỚP 10 MÔN TIẾNG ANH NĂM HỌC 2023 - 2024 CÓ ĐÁP ÁN (NGỮ Â...TỔNG ÔN TẬP THI VÀO LỚP 10 MÔN TIẾNG ANH NĂM HỌC 2023 - 2024 CÓ ĐÁP ÁN (NGỮ Â...
TỔNG ÔN TẬP THI VÀO LỚP 10 MÔN TIẾNG ANH NĂM HỌC 2023 - 2024 CÓ ĐÁP ÁN (NGỮ Â...
 
Asian American Pacific Islander Month DDSD 2024.pptx
Asian American Pacific Islander Month DDSD 2024.pptxAsian American Pacific Islander Month DDSD 2024.pptx
Asian American Pacific Islander Month DDSD 2024.pptx
 
Mixin Classes in Odoo 17 How to Extend Models Using Mixin Classes
Mixin Classes in Odoo 17  How to Extend Models Using Mixin ClassesMixin Classes in Odoo 17  How to Extend Models Using Mixin Classes
Mixin Classes in Odoo 17 How to Extend Models Using Mixin Classes
 
Energy Resources. ( B. Pharmacy, 1st Year, Sem-II) Natural Resources
Energy Resources. ( B. Pharmacy, 1st Year, Sem-II) Natural ResourcesEnergy Resources. ( B. Pharmacy, 1st Year, Sem-II) Natural Resources
Energy Resources. ( B. Pharmacy, 1st Year, Sem-II) Natural Resources
 
The basics of sentences session 2pptx copy.pptx
The basics of sentences session 2pptx copy.pptxThe basics of sentences session 2pptx copy.pptx
The basics of sentences session 2pptx copy.pptx
 
Introduction to Nonprofit Accounting: The Basics
Introduction to Nonprofit Accounting: The BasicsIntroduction to Nonprofit Accounting: The Basics
Introduction to Nonprofit Accounting: The Basics
 
Holdier Curriculum Vitae (April 2024).pdf
Holdier Curriculum Vitae (April 2024).pdfHoldier Curriculum Vitae (April 2024).pdf
Holdier Curriculum Vitae (April 2024).pdf
 
Making and Justifying Mathematical Decisions.pdf
Making and Justifying Mathematical Decisions.pdfMaking and Justifying Mathematical Decisions.pdf
Making and Justifying Mathematical Decisions.pdf
 
2024-NATIONAL-LEARNING-CAMP-AND-OTHER.pptx
2024-NATIONAL-LEARNING-CAMP-AND-OTHER.pptx2024-NATIONAL-LEARNING-CAMP-AND-OTHER.pptx
2024-NATIONAL-LEARNING-CAMP-AND-OTHER.pptx
 

Burns & its Physiotherapy Management..pptx

  • 1. BURNS AND ITS PHYSIOTHERAPY MANAGEMENT 1 Dr. Hitiksha Bhalodia (MPT in Women’s Health) Assistant Professor , PIPT Course Name: Physiotherapy in Medical and Surgical Condition Course Code: 07101404 Course Objective: Select strategies for cure, care and prevention; adopt restorative and rehabilitative measures for maximum possible functional independence of a patient at home, work and in community. (CO2)
  • 2. What is burns…? ⚫ It is a type of coagulative necrosis of tissue, caused by thermal application transfer from source to body or ⚫ Tissue injury caused by thermal, electrical or chemical agents 2
  • 3. Risk factors ⚫ Fire/combustion □ Firefighter □ Industrial workers □ Occupant of burning structures ⚫ Chemical exposure □ Industrial workers ⚫ Electrical exposure □ Electrician □ Electrical power distribution worker 3
  • 4. Types of burn Injuries 1. thermal burn ⚫ Skin injury ⚫ Inhalation injury 4
  • 5. 2. Chemical burn: □ Skin injury □ Inhalation injury □ Mucous membrane injury 5
  • 6. 3. Electrical burn: □ Skin injury’ □ Nerves, muscle and bone damage 4. Radiation burn 6
  • 7. Effects: ● Burn injury causes destruction of tissue, usually the skin from exposure to thermal extremes (either hot or cold), electricity, chemical and radiation ⚫ The mucosa of the upper GI system (mouth, esophagus, stomach) can be burned with ingestion of chemicals ⚫ The respiratory system can be damaged if hot gases, smoke or toxic chemical fumes are inhaled ⚫ Fat, muscle, bone and peripheral nerves can be affected in electrical injuries or prolonged thermal or chemical exposure ⚫ Skin damage can result in altered ability to sense pain, touch and temperature 7
  • 8. Classification of burn injury andits pathophysiology 8
  • 9. 1. Depending upon depth of burn injury: □ Previously used classification First degree burn: only epidermis affected Second degree burn: epidermis and dermal appendages Third degree burn: epidermis and all dermis Fourth degree burn: epidermis, dermis, and subcutaneous tissues (fat, muscle, bone and peripheral nerves) 9
  • 10. Recent classification of burn depending on depth of tissue injury 1. Epidermal burn 2. Superficial partial thickness burn 3. Deep partial thickness burn 4. Subdermal burn 10
  • 11. 11
  • 12. Epidermal burn ⚫ Cell damage to epidermis ⚫ This depth of burns correlates to practice pattern 7B; impaired integumentary integrity associated with superficial skin involvement e.g., sunburn 12
  • 13. ⚫ Clinically skin appears red / erythematous ⚫ Painful, dry, red burns which blanch with pressure ⚫ No blister ⚫ Slight edema may apparent ⚫ Spontaneous healing (within 7 days) & no scar 13
  • 14. Superficial partial-thickness burn ⚫ Damage occurs through the epidermis and into the papillary layer of dermis (second-degree burn) ⚫ The epidermal layer destroyed completely ⚫ This depth of burn corresponds to APTA pattern 7C; impaired integumentary integrity associated with partial thickness skin involvement and scar formation 14
  • 15. ⚫ Presence of intact blisters ⚫ Due to blister increased inflammatory response ⚫ Moist weeping or glistening surface when blisters removed ⚫ Very painful ⚫ Sensitive to changes in temperature, exposure to air currents, & light touch ⚫ Moderate edema, spontaneous healing, minimal scarring & discoloration ⚫ Heal within 7-21 days 26
  • 16. 16
  • 17. Deep partial thickness burn ⚫ Involves destruction of the epidermis and papillary dermis with damage down into the reticular dermal layer ⚫ Impairment matches to APTA pattern 7C ⚫ Deep partial thickness burns appears as a mixed red or waxy white color 17
  • 18. ⚫ Blanching and brisk capillary refill ⚫ Broken blisters, wet surface ⚫ Their color may range from patchy, cheesy white to red and they do not blanch with pressure ⚫ Sensitive to pressure but insensitive to light touch or soft pinprick ⚫ Affected area is depressed ⚫ Healing with skin graft or flap or scarring 18
  • 19. Full thickness burn ⚫ All epidermal and dermal layers are destroyed along with subcutaneous fat layer ⚫ This burn depth consistent with practice pattern 7D; i.e., impaired integumentary integrity associated with full-thickness skin involvement and scar formation ⚫ A full-thickness burn is characterized by a hard, parchment-like Escher covering the area. 19
  • 20. ⚫ Escher is devitalized tissue consisting of designed coagulum of plasma & necrotic cells ⚫ Escher feels dry, leathery and rigid ⚫ the color of Escher can vary from black to deep red to white ; latter indicates total ischemia of the area ⚫ Hair follicles are completely destroyed 20
  • 21. ⚫ A major problem arises from deep burns is the damage to the peripheral vascular system ⚫ Large amount of fluid leak into the interstitial space □ increase pressure in extracellular space □ constriction to deep circulation ⚫ Very slow healing following this type of burn 21
  • 22. 22
  • 23. Subdermal burn ⚫ Involves complete destruction of all tissue from the epidermis down to and through the subcutaneous tissue ⚫ Occurs following prolonged contact with a heat source ⚫ This depth of the injury correlates with APTA practice pattern 7E; Impaired integumentary integrity associated with skin involvement extending into fascia, muscle or bone and scar formation ⚫ Extensive surgical and therapeutic management is necessary to return a patient to some degree of function 23
  • 24. Electrical burn ⚫ S/S of an electric burn may vary according to the type of current, intensity of the current and the area of the body the electric current passes through ⚫ Electric current follows the course of least resistance offered by various tissues. i.e., nerves, followed by blood vessels offers least resistance; bones offers most resistance. ⚫ Tissue damage results from tissue resistance to the passage of the current or by direct electrical current 24
  • 25. ⚫ Typically contact sites will exist where the patient first came into contact with the electricity and a second site where the patient was grounded ⚫ The affected skin appears yellow, ischemic and dry ⚫ Tissues along the pathway of the current may be damaged owing to heat that developed as a result of tissue resistance to current passage ⚫ Blood supply to surrounding tissues may be altered and arteries undergo spasm 25
  • 26. ⚫ There can be other consequences of electricity passing through the body such as □ Cardiac arrhythmias and acute renal failure secondary to fluid and electrolyte imbalances and release of myoglobin into blood □ The most severe complication following electrical current damage is acute spinal cord damage or vertebral fracture □ Ventricular fibrillation and respiratory arrest 26
  • 27. Burn wound Zones ⚫ Three zones: 1. Zone of coagulation 2. Zone of stasis 3. Zone of hyperemia 27
  • 28. Zone of coagulation ⚫ In the zone of coagulation cells are irreversibly damaged & skin death occurs ⚫ This area is equivalent to a full-thickness burn & will require a skin graft to heal ⚫ Here, because of lack of viable tissue & the amount of Escher, the risk of infection increased ⚫ This potential complication emphasizes the need for careful monitoring, use of antibiotics & the treatment of a burned pt. in specialized burn center 28
  • 29. Zone of stasis ⚫ It contains injured cells that may die within 24 to 48 hrs. without diligent treatment ⚫ Here, in this zone; Infection, Drying & inadequate perfusion of the wound □ conversion of potentially salvageable tissue to completely necrotic tissue & enlargement of the zone of coagulation ⚫ Splints or compression bandages, if applied too tightly, can compromise this area 29
  • 30. Zone of Hyperemia ⚫ Finally in this zone, there is minimal cell damage ⚫ Tissue should recover within several days with no lasting effects 30
  • 31. Extent of Burned Area ⚫ A major consideration when determining the severity of a burn is the extent of body surface involved ⚫ % of Total body surface area(TBSA) burned; calculated by Rule of nines 31
  • 32. 32
  • 33. 33
  • 34. 34
  • 35. 35
  • 36. ⚫ The rule of nines divides the body surface into areas of 9% or multiples of 9% of TBSA ⚫ Modified:- according to continuum of age accommodate for growth of different body segments 36
  • 37. Total Burn Surface Area (TBSA): the greater the total burn surface area the poorer the prognosis Percentage chance of Survival =[100 – (Age in years + percentage TBSA)] 37
  • 38. 38
  • 39. 39
  • 41. Burn Patient Severity Factors to Consider: ● Burn Depth classification ● Body surface area burned ● Age: adult v/s pediatric ● Preexisting medical conditions ● Associated trauma - Blast injury - Fall injury - Airway compromise - Child abuse 41
  • 42. ● Patient Age: Less than 2 or greater than 55 have increased incidence of complication ● Burn Configuration: - Circumferential burns can cause total occlusion of circulation to an area due to edema - Restrict ventilation if encircle the chest - Burns on joint area can cause disability due to scar formation 42
  • 43. Critical Burn Criteria ⚫ 30 > 10% BSA ⚫ 20 > 30% BSA > 20% pediatric ⚫ Burns with respiratory injury ⚫ Hands, face, feet or genitalia ⚫ Burns complicated by other trauma ⚫ Underlying health problems ⚫ Electrical & deep chemical burns 43
  • 44. Moderate burn criteria ⚫ 30 : 2-10 % BSA ⚫ 20 : 15-30 % BSA 10-20% pediatric ⚫ Excluding hands, face, feet or genitalia ⚫ Without complicating factors 44
  • 45. Minor Burn Criteria ⚫ 30< 2% BSA ⚫ 20< 15% BSA 10% BSA ⚫ 10< 15% BSA 45
  • 46. Complications of Burn injury ⚫ Depending on the extent of burn injury, the depth of the burn & the type of burn, there may be secondary systemic complications ⚫ In addition the health, age, & psychological status of a patient who is burned will affect these complications 46
  • 47. 1. Infection ⚫ Infection is conjunction with organ system failure, is a leading cause of mortality from burns ⚫ Some virulent strains of Pseudomonas aeruginosa & Staphylococcus aureus are resistant to antibiotics & have been responsible for epidemic infections in burn centers ⚫ Systemic antibiotics are used to treat both burn and general system infections once they have been documented 47
  • 48. 2. Pulmonary complications ⚫ Inhalation injury: pt who has been burned in closed space ⚫ Signs of an inhalation injury include facial burns, singed nasal hairs, harsh cough, hoarsness, abnormal breath sounds, respiratory distress, & carbonaceous sputum & hypoxemia ⚫ The incidence of pulmonary complications is extremely high after severe burns, & death due to pneumonia 48
  • 49. ⚫ The primary complications associated with this injury are carbon monoxide poisoning, tracheal damage, upper airway obstruction, pulmonary oedema, and pneumonia. ⚫ Lung damage from inhaling noxious gases and smoke may be lethal 49
  • 50. 3. Metabolic Complications ⚫ Metabolic rates may increase up to 50% in a 25% TBSA burn and much more as the burn size increases ⚫ The consequences of the increased metabolic and catabolic activity following a burn are:- - A rapid decrease in body weight - Negative nitrogen balance and - A decrease in energy stores that are vital the healing process. 50
  • 51. ⚫ Following increased metabolic activity there will be increase of 1.80F to 2.60F (10C to 20C) in core temperature that seems to be due to a resetting of the hypothalamic temperature centers in the brain ⚫ As a part of pt.'s altered metabolism, protein from muscle tissue is preferentially used as a source of energy muscle atrophy 51
  • 52. 4. Cardiovascular complications ⚫ Hemodynamic changes result from a shift in fluid to the interstitium, which subsequently reduces the plasma & intervascular fluid volume in a burnt pt. ⚫ This shift of fluid to the interstitium can result in significant edema ⚫ Decrease cardiac output ● Hematological changes: alterations in platelet concentration & function, clotting factors & white blood cell components, RBC dysfunction & decrease hemoglobin & hematocrit 52
  • 53. Heterotopic Ossification ⚫ Pts. With more than 20% TBSA burn highly susceptible ⚫ Usually occurs in areas of full-thickness injury or sites that remains unhealed for prolonged period ⚫ Symptoms: decrease ROM, point specific pain, generalized pain 53
  • 54. Neuropathy ⚫ Peripheral neuropathy: either polyneuropathy or local neuropathy (depending on involvement of TBSA) 54
  • 55. Pathological Scars ⚫ Burn scar occur in area of deep partial-thickness burn □ where healing is with skin grafting ⚫ If maturing tissue demonstrates a greater rate of collagen production than degradation, a scar become raised and thick □ hypertrophic scar 55
  • 56. ⚫ Formation of Escher ⚫ Renal or hepatic failure 56
  • 57. Psychological changes ⚫ Fear/ anxiety ⚫ Denial ⚫ Depression ⚫ Guilt ⚫ Grief & mourning ⚫ Loss of will to live ⚫ Apathy ⚫ Necrophilous orientation ⚫ anger 57
  • 58. Management Medical management of burns ● Initial treatment: Goals: - □ to address critical life-threatening problems & stabilize the pt. through procedures designed to: - Establish & maintain an airway - prevent cyanosis, shock & hemorrhage - establish baseline data on pt.; extent & depth of burn injury - Prevent or reduce fluid losses - clean the pt. & wounds - Examine injuries - Prevent pulmonary & cardiac complications 58
  • 59. ⚫ Transport pt. to burn center from the site of accident ● Assessment : demographic details, history, calculation of TBSA ⚫ Fluid volume replacement therapy ⚫ Wound cleansing ⚫ Debridement ⚫ analgesics 59
  • 60. Wound Care: ⚫ Remove Escher along with sharp debridement ⚫ Apply topical medications either with open technique or closed technique 60
  • 61. Surgical Management of burn wound Primary Excision: - Surgical removal of Escher - With excision removal of peripheral layers of Escher until vascular, viable tissue is exposed as the site for skin graft placement - Burn wound is closed with a skin graft at time of primary excision Grafts used are: autograft, allograft or xenograft □ Now a days skin substitutes are used for coverage of an excised wound 61
  • 62. Skin grafting ⚫ Done under anesthesia ⚫ Skin used for a graft usually is removed with a dermatome ⚫ It can either split-thickness skin graft (epidermis with variable amount of dermis) or full-thickness skin graft (full epidermis and dermis) Correction of scar contracture 62
  • 63. The Z-plasty servesto lengthen a scar by interposing normal tissue in the line of the scar. 63
  • 64. Physical Therapy Management ● PT interventions are directed towards:- □ Prevention of scar & contracture □ Preservation of normal ROM □ Prevention or minimization of hypertrophic scar formation and cosmetic deformity □ Maintenance or improvement in muscular strength □ Improvement in cardiovascular endurance □ Return to pre-burn function and performance of activities of daily living 64
  • 65. ASSESSMENT ⚫ Demographic details: ⚫ Review of medical records ⚫ History ⚫ Observation ⚫ Palpation ⚫ Examination □ Initial examination for the depth of burn □ Percentage of burn (TBSA) & percentage chance of survival 65
  • 66. ● Screening/assessment for complications: □ Hypovolemic shock:- Hypotension Oliguria Tachycardia Sweating, Pallor Clouding of consciousness □ Septic Shock:- Increased temperature Hypotension Oliguria Dry & pink extremities Altered pulmonary functions 66
  • 68. ● Skin Assessment: □ Appearance □ Temperature □ Moisture/Dryness □ Texture □ Color □ Size □ Pulses □ Sensations 68
  • 69. ⚫ Assessment of ROM ⚫ Tightness/contracture ⚫ Functional mobility ⚫ Gait and ⚫ functional status (FIM) 69
  • 70. Special tests ● Pigmentation of the scar: 0 = normal 1 = hypopigmentation 2 = hyperpigmentation ● Height of scar: 0 = normal 1 =< 2mm 2 =<5 3 =>5 70
  • 72. ⚫ ICF: Condition □ Body structures and function □ Activities □ Function Contextual factors: personal and environmental facilitators and barriers 72
  • 73. Anticipated Goals and Expected outcomes (In general) ⚫ Risk of infection and complications is reduced ⚫ Wound and soft tissue healing is enhanced. ⚫ Risk of secondary impairments is reduced. ⚫ Maximal range of motion is achieved. ⚫ Pre injury level of cardiovascular endurance is restored. ⚫ Good to normal strength is achieved. 73
  • 74. ⚫ Independent ambulation is achieved. ⚫ Independent function in ADL and IADL is increased. ⚫ Scar formation is minimized. ⚫ Patient, family, and caregivers’ understanding of expectations and goals and outcomes is increased. ⚫ Aerobic capacity is increased. ⚫ Self-management of symptoms is improved 74
  • 75. Positioning And Splinting ⚫ Positioning begin on the day of admission ⚫ Concept: positioning burned areas in place and maintain the body part in the opposite plane and direction to which it will potentially contract ● Goals: - To minimize edema - To prevent tissue destruction - maintain soft tissues in elongated state - preserve function 75
  • 76. Positioning in bed of pt. with burn of the anterior neck 76
  • 77. Positioning in bed of pt. with burns of the axilla 77
  • 78. Positioning of upper extremities to reduce edema while seated 78
  • 79. ● Splinting given to provide anti-deformity positions ● Indications:- Prevention of contracture - Maintenance of ROM - Reduction of developing contracture - Protection of jt. or tendon - Reduce pain 79
  • 80. ⚫ Splinting usually worn at night following skin grafting ⚫ Static or dynamic splint can be used □ Dynamic splint apply low load, prolonged stress that can be adjusted to pt.'s tolerance 80
  • 81. 81
  • 82. 82
  • 83. 83
  • 84. Therapeutic Exercises ● Active and passive exercises: □ Can be initiate from the day of admission □ Before starting exercise look for the complications and vital stability and consciousness/alertness □ Perform AROM of all extremities and trunk including unburned areas □ Avoid ROM exercise of injured area in case of recent skin grafting & allow the graft to adhere 84
  • 85. Dosages: □ AROM should be done twice daily □ For sedated pts. PROM can be initiated and given thrice/day □ Repetitions and degree of movement depends on pt. tolerance and limit of stretch 85
  • 86. Contraindications: 86 ⚫ Active or Passive range of motion exercises should not be carried out if there is suspected damage to any tendons e.g., flexion of PIP should be avoided at all to prevent extensor tendon rupture ⚫ Range of motion exercises are also contraindicated post skin grafting as a period of 3-5 days immobilisation is required to enable graft healing
  • 87. Practical considerations while mobilizing burn patient ⚫ Be aware of dressing clinic/daily dressing changes. Mobilisation should coincide with this as it is important to monitor the wound during AROM frequently. ⚫ Timing of pain relief. This should be timed appropriately to ensure maximal benefit during treatment sessions. ⚫ Observe the patient carrying out the AROM and PROM exercises prior to beginning treatment. Also observe the patient taking on/off splints. 87
  • 88. ⚫ Always monitor for post exercise pain and wound breakdown. ⚫ Avoid blanching for long period as you may compromise vascularity. ⚫ The patient may present with a reduced capacity for exercise secondary to increased metabolic rate, altered thermoregulation and increased nutritional demands. ⚫ Postural hypotension may be present due to prolonged bed rest and low haemoglobin 88
  • 89. Ambulation ⚫ It should be initiated at the earliest appropriate time ⚫ If LEs are skin grafted ambulation may be discontinued until it is safe to resume ⚫ Wrap elastic bandages after skin grafting while ambulation (supports new graft and promote venous return) ⚫ If orthostatic hypotension is there use tilt table to make pt. standing gradually 89
  • 90. “The rewards of a successful POC are tremendous when a patient who has suffered a life-threatening burn is able to walk out of the hospital and return to productive community 90 involvement.”
  • 91. Resistive and conditioning exercises ⚫ Pts. With major burns may lose body weight and lean muscle mass can decrease rapidly ⚫ Exercise consist of isometric, isokinetic, isotonic or using other resistive training devices can be started at rehabilitation stage ⚫ General principles of exercise training and strength improvement should be followed, but they may need to be modified on the basis of a patient’s condition and stage of wound healing 91
  • 92. ⚫ Initially Isometrics used to maintain muscle properties & with that help to prevent further muscle atrophy ⚫ In progression use weights and resistive equipments to improve muscle strength ⚫ Regular and accurate monitoring of vitals (including PR,RR, BP, SPO2, RPE) before, during and after exercises. 92
  • 93. Scar Management Pressure Garment Therapy (PGT) ⚫ Following wound closure, a skin graft or healed burn wound is vascular, flat and soft. ⚫ To achieve this following 3-6 months, dramatic changes may occur – the newly healed areas may become raised and firm ⚫ If wound healing takes longer than 10 to 14 days or skin grated, pressure usually is indicated ⚫ Pressure has been used successfully to hasten scar maturation and minimize hypertrophic scar formation 93
  • 94. ⚫ Pressure may exert control over hypertrophic scarring by; - Pressure decreases blood flow - Local hypoxia of hypervascular scars - Reduction in collagen deposition All three together causes: o Decreases scar thickness o Decreases scar redness o Decreases swelling o Reduces itch o Protects new skin/grafts o Maintains contours ⚫ The earlier the scar tissue is exposed to pressure, the better result 94
  • 95. Method of application: For LE – fig. of eight pattern For UE – spiral wraps For trunk – circular wrap Hand and toes – self-adherent bandages 95
  • 96. 96
  • 97. Practical recommendations and safety considerations ⚫ Pressure exerted around 15 – 40 mmHg ⚫ Time : Worn up to 23 hrs./day ⚫ Worn as soon as wound closure has been obtained (Post grafting after 10-14 days it is recommended and it should be worn up to one year or until scar maturation) 97
  • 98. Possible complications/ confounding factors for use of PGT □ Lack of a scientific evidence to established optimum pressure □ Non-Compliance ( due to comfort, movement, appearance) □ Heat and perspiration □ Swelling of extremities caused by inhibited venous return □ Skin breakdown □ Web space discomfort □ Inconvenience □ Personal hygiene difficulties possibility of infection □ Allergies to material 98
  • 99. Silicone Gel: Sheets of silicone polymer gel may be applied directly over an actively maturing scar. ⚫ Complication: local rash, rarely skin breakdown 99
  • 100. Mechanism: unknown but possible mechanisms are as follow □ Hydration: decreases capillary activity and collagen production, through inhibition of proliferation of fibroblasts □ Rise in temperature increases collagenase activity and scar breakdown □ polarized electric fields creates negative charge within silicone causes polarization of scar tissue, resulting in involution of scar □ Presence of silicone oil □ Oxygen tension inhibit hypoxia signal □ Increase in mast cell helps in remodeling of tissue 100
  • 101. 101
  • 102. Massage: Mechanisms on which Scar massage works: - Prevent adherence - Reduce redness - Reduce elevation of scat tissue - Relieve pruritus - Maintain/restore moisture 102
  • 103. Scar Massage Techniques: ⚫ Deep friction massage□ to loosen scar adhesions ⚫ Finger and thumb kneading □ mobilize the scar and surrounding tissue ⚫ Effleurage □ to increase circulation ⚫ Skin rolling □ restore mobility to tissue interfaces ⚫ Wringing □ stretches scar and promote collagenous remodeling ⚫ Retrograde massage to aid venous return, increase lymphatic drainage, mobilize fluid 114 □ Massage techniques can be used in conjunction with ROM exs
  • 104. Guidelines for massage during 3 stages of healing: 1. Inflammatory phase: helps to decrease oedema and increase blood supply 1. Proliferative phase: applies gentle stress to the healing scar; to ensure correct alignment of collagen tissues 1. Remodeling phase: massage should be progressed to include prolonged stretching to minimize adhesions; that aids in scar tissue breakdown 115
  • 105. Practical considerations and safety: □ Clean hands essential □ Use non irritating lubricant □ Modify practice according to pt. stage of healing and pain levels Contraindications: - Compromised integrity of epidermis - Acute infection - Bleeding - Graft failure - Intolerable discomfort 116
  • 106. Camouflage Make-up ⚫ Used for scars of face, neck and hands ⚫ Mostly used when a person has either hyperpigmentation or hypopigmentation ⚫ It can also be used before scar maturation; so if pt. wants to go out in public ⚫ The cosmetics are opaque, color-correct burn scars and are available in multiple shades to accommodate various skin colors ⚫ They also are waterproof and worn during all activities 106
  • 107. The Role of Physiotherapist in rehabilitation of the chronic Burn patient ⚫ Scar management is initiated in the sub-acute phase, but it may need to be continued long term, as many patient suffer from continuing limitation of ROM ⚫ Healing process may continue for up to two years, or more, as scar tissue remodels and matures ⚫ Patient may require functional retraining and integration back into the community and activities 107
  • 108. Aerobic and Resistance Training Post Burn Rationale for training: □ Low cardiovascular endurance □ Lower aerobic capacity measured by VO2 max and time to fatigue at one year post burn compare to age matched healthy control (adults and children > 15% TBSA) □ Muscular strength and lean body mass has been found to be significantly less in pts. Suffering from burns of 30% TBSA □ Reduced lean body mass, endurance and strength has been associated with limited standing/walking tolerance □ Reduced upper limb function and lower health related QOL and ability to participate in activities 108
  • 109. Aerobic capacity and muscular strength is diminished by the following factors: ⚫ Prolonged bed rest necessary in the early recovery process 109 ⚫ Hyper metabolism; which may lead to: - Exhaustion - Protein catabolism - Loss of lean body mass ⚫ Impaired thermoregulation ⚫ Inhalation injuries and compromised respiratory function ⚫ Fatigue ⚫ Pain ⚫ Psychological factors
  • 110. Aerobic training recommendations for practice: Frequency: 3 days/week; (3-5 days/week) Intensity: moderate to high intensity i.e., 65%% 85% of MHR Type: Interval, continuous; using treadmill, walking, running exercises Time: 20-40 minutes/session 6-12 week programme 110
  • 111. Resistance training recommendations for practice: Frequency: 3 times/week (break of more than 48 hrs. must be given between bouts of resistance training as; □resistance exs. Causes microtrauma to muscles already in compromised state □ resistance exs. In burned pts. stimulates protein synthesis) Intensity/Type/time: 1 set of 50-60% of 3 RM at 1st week 70-75% of 3 RM for next 2-6 weeks (4-10 reps) 80-85% of 3 RM for 7-12 weeks (2-6 reps) 111
  • 112. Mixed and functional strengthening 112 □ Given for specific targeting muscle groups for desired functional goal □ Progression made by aiding resistive devices and finally using free weight training □ Initiate with 50-60% of 1 RM for 10-15 reps and than progress further as per pt.’s need over 12 weeks
  • 113. ● Safety considerations for strength and aerobic training: □ Post burn 6 months to 2 years time given before initiation of programmes or □ In case of 40% TBSA it can be started after; 95% healed wounds/scars ambulatory psychological status □ Ensure patient is taking Optimal nutrition and other medications □ Monitoring of HR, BP, SPO2 & RPE before during and after 124 exercises
  • 114. Psychological Aspects of Burn patient 125 ⚫ Depression and post traumatic stress disorder are most common disorders following burn ⚫ Pain and cosmetic appearance of individual Treatment - Psychotherapy - CBT - Medications (SSRI) - Social skills training and community intervention - Vocational rehabilitation
  • 115. Ultrasound Therapy after skin grafting 127 UST can be given on alternate day started from 14th day to 21st day after skin grafting Dosage: 1MHz, 0.5-0.8 W/cm2, for 5 min □ (EFFECTIVENESS OF PHYSIOTHERAPY AFTER SKIN GRAFTING- Indian journal of physiotherapy & occupational therapy, dec-16)
  • 116. First Aid in burns DO’s 128 ⚫ Stop the burning process by removing clothing and jewelry ⚫ In electrical burns, put the main switch off as quick as possible and use wooden chair to push victim away from electricity ⚫ Extinguish flames by pouring plain water; if water is not available by applying a blanket & removing the blanket as soon the flames are put off ⚫ In chemical burns, remove or dilute the chemical agent by irrigating large amount of water ⚫ Use cool running water to reduce the temperature of the burn ⚫ Take care of fractures and probable injuries during transportation. • Ensure A,B, C before transportation to higher center
  • 117. Don’ts 129 ⚫ Don’t start first aid before ensuring your own safety ⚫ Don’t apply ice it may damage the injured tissues ⚫ Avoid prolonged cooling with water it may causes hypothermia ⚫ Don.t apply any ointments ⚫ Don’t open blisters with pin, needle until topical antimicrobials can be applied