Ppt hai ek number ki ppt kl mil jaegi vasey tae grh hi vekhli roz roz mnuh kaan vich keh k liye yeh vedio call tae v nhi hai nah patni ko kyun nahi hai slow motion to dismiss the case of my life as a princess with the stars light bndh kri hai na tere liye yeh vedio call tae v nhi hai nah patni ko kyun nahi hai slow motion to dismiss the case u ever feel free hokey soa jandey c and i have seen a little more time with the flow jyada

3.  Formation of clotted mass of blood within the cardiovascular system.
 Clotted mass :- Thrombus (Singular)
Thrombi (plural)

4. Character Thrombus Blood clot
Involvement Wall of blood vessels
Elements of blood
Blood clotting system
Blood clotting system only
Composition Platelets and fibrin Fibrin only
Prognosis Life threatening Life saving

5. 1. Injury to the endothelium
2. Alteration in the blood flow
3. Alternation in the constituents of blood

6.  Trauma :- laceration, Contusion ,Rupture
 Toxins :- Streptococci and Erysipelothrix (endocardium vegatation)
 Viruses :- Hog chlorea virus (Spleen)
 Parasites:- Strongulus vulguris (Anterior mesnetric artery)
 Degeneration :- Atherosclerosis
 Tumours :- Invading tumours

7.  Chronic venous congestion
 Aneurysm
 Varicose veins
 Old Animals

8.  Increase in thrombocytes :- After Parturition and surgical operation
 Increase in adhesiveness of platelets :- After Parturition and surgical operation
 Decrease in heparin
 Increase amount of plasma fibrinogen and prothrombin
 Increase in viscosity of blood :- Dehydration
 Sludging of blood :- in trauma red cells get agglutinated intravascularly.
 Increased fragility of erythrocytes :- in hemolytic anemia.

9.  Blood flow normal - No thrombus
 Injury to endothelium. Endothelium rough. Platelets attached.
Murral Thrombus. Line of zahn. Plug of thrombocytes
Obstuct blood flow. Slow blood stream. White/pale Thrombus
Thromboplastin Homogeneous mass. Platelets loss their
morphology

10.  Thromboplastin converts fibrinogen into fibrin.
In sluggish blood fibrinogen. Fibrin. Meshes
Red thrombus. Red colur. Entangles erythrocytes
Thrombus size inc. In dir. Completely occlude the lumen
of blood flow
Occlusive thrombus
 Line of zahn:- When thrombocytes form a plug on endothelium then more and more
platelets that fall out get adhere to it at right angle to the flow of blood
 It takes minimum 4 minutes to complete clotting mechanism.

11.  According o Loacation:-
 Cardiac thombus
Valvular Thrombus –In valves – Swine erysipelas in pig
Mural thrombus - In wall of heart – In cattle by Claostridium chauvoei.
 Arterial thrombus – Strongylus vulgaris in horse.
 Venous thrombus
 Capillary thrombus

12.  According to location in blood vessels:-
 Lateral thrombi – one side of BV
 Occluding thrombi - entire circumference of BV
 Saddle thrombi – At bifurcation of BV
 Canalised thrombi – New blood channels through the clot
 According to Infective agent:-
 Septic thrombi
 Aseptic thrombi
 Parasitic thrombi

13.  According to colour :-
 Pale or white thrombi – in rapid blood flow ; only platelets
 Red thrombi – in slow blood glow;
fibrin attach to platelets
 Mixed thrombi – most common
 Laminated thrombi – alternate layers of
white and red

14. Fate depends on whether it is aseptic or septic.
1. Aseptic Thrombi:-
1. Contraction :- Fibrin of thrombus may contract due to action of thromasthenin.
2. Absorption:- leucocytes softened and absorbed to he thrombus
3. Organization :- fibroblasts elaborate collagen and elastin ; macrophages (thrombus
liquification
4. Canalisation :- endothelial cells invade thrombus anastomose with connective tissue
5. Calcification:- Phleobolith (stone of vein)

15. 2.Septic thrombi :-
 Inflammatary reaction from exudate
 Give rise to centre of infection where emboli lodge