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Burns can cause  swelling, blistering, scarring and, in serious cases, shock and even death.  They also  can lead to infections  because they damage your skin's protective barrier. Antibiotic creams can prevent or treat infections.  After a third-degree burn, you need skin or synthetic grafts to cover exposed tissue and encourage new skin to grow. First- and second-degree burns usually heal without grafts.
Etiology   Thermal burns   may result from any external heat source (flame, liquids, solid objects, or gases).      Radiation burns   most commonly result from prolonged exposure to solar ultraviolet radiation (sunburn) but may result from prolonged or intense exposure to other sources of ultraviolet radiation (eg, tanning beds) or from exposure to sources of x-ray or other non solar radiation.
Etiology    Chemical burns   may result from strong acids, strong alkalis (e.g, lye, cement), phenols, cresols, mustard gas, or phosphorus. Skin and deeper tissue necrosis due to these agents may progress over several hours.    Electrical burns   result from the electrical generation of heat; they may cause extensive deep tissue damage despite minimal apparent cutaneous injury.
Risk Factors
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Diagnostic  Tests
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Pathophysiology
Burn Injury Local and Systemic edema Endothelial Injury Leukocyte sequestration Tissue Injury Tissue Ischemia Hypovolemia and hyperviscosity ,[object Object],[object Object],[object Object],Direct tissue injury  Capillary Permeability Systemic Injury Capillary Permeability
Estimating the Extent of a Burn ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
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9 18 9 18 9 18 1
Symptoms:
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Actual: ,[object Object],[object Object]
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Interventions
BURN INTERVENTIONS ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],Resuscitative phase - begins w/ the initiation of fluids and ends when capillary integrity returns to near normal levels and the large fluid shifts have decreased - the amount of fluid administered is based on the client’s weight and extent of injury - most fluid replacement formulas are calculated from the time of injury and not from the time of arrival at the hospital - the goal is to prevent shock by maintaining adequate circulating blood volume and maintaining vital organ perfusion Emergent phase  - begins at the time of injury and ends with the restoration of capillary permeability, usually at 48-72 hours after the injury - the 1 ˚ goal is to prevent hypovolemic shock and preserve vital organ functioning - includes prehospital care and emergency room care
Rehabilitative phase - final phase of burn care - overlaps the acute care phase and goes well beyond hospitalization - goals of this phase are designed so that the client can gain independence and achieve maximal function Acute phase - begins when the client is hemodynamically stable, capillary permeability is restored, and diuresis has begun - usually begins 48 - 72 hours after the time of injury - emphasis during this phase is placed on restorative therapy, and the phase continues until wound closure is achieved - the focus is on infection control, wound care, wound closure, nutritional support, pain management, and physical therapy
Management
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BurN baby Burn.

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  • 5. Burns can cause swelling, blistering, scarring and, in serious cases, shock and even death. They also can lead to infections because they damage your skin's protective barrier. Antibiotic creams can prevent or treat infections. After a third-degree burn, you need skin or synthetic grafts to cover exposed tissue and encourage new skin to grow. First- and second-degree burns usually heal without grafts.
  • 6. Etiology  Thermal burns may result from any external heat source (flame, liquids, solid objects, or gases).  Radiation burns most commonly result from prolonged exposure to solar ultraviolet radiation (sunburn) but may result from prolonged or intense exposure to other sources of ultraviolet radiation (eg, tanning beds) or from exposure to sources of x-ray or other non solar radiation.
  • 7. Etiology  Chemical burns may result from strong acids, strong alkalis (e.g, lye, cement), phenols, cresols, mustard gas, or phosphorus. Skin and deeper tissue necrosis due to these agents may progress over several hours.  Electrical burns result from the electrical generation of heat; they may cause extensive deep tissue damage despite minimal apparent cutaneous injury.
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  • 27. Rehabilitative phase - final phase of burn care - overlaps the acute care phase and goes well beyond hospitalization - goals of this phase are designed so that the client can gain independence and achieve maximal function Acute phase - begins when the client is hemodynamically stable, capillary permeability is restored, and diuresis has begun - usually begins 48 - 72 hours after the time of injury - emphasis during this phase is placed on restorative therapy, and the phase continues until wound closure is achieved - the focus is on infection control, wound care, wound closure, nutritional support, pain management, and physical therapy
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