4 g 5g-omg (1)

4G – 5G – OMG!The Sources and Effects of Electromagnetic and Radio Frequency Exposure on Physical and Mental Health
Speaker: Dr. Karen von Merveldt-Guevara, MD

Conflict of Interest Disclosure Statement
I have no financial interest or other relationships with the industry relevant to the topic being
discussed.
KvMG - VVMHC - 07/29/19 2

Medical Disclaimer
This presentation / PPP cannot and does not contain medical advice. The medical
information is provided for general information and educational purposes only, and is not a
substitute for professional medical advice.
Accordingly, before taking any actions based upon such information, we encourage you to
consult with the appropriate medical professionals. We do not provide any kind of medical
advice. The use or reliance of any information contained on this presentation / PPP is solely
at your own risk.
KvMG - VVMHC - 07/29/19 3

My Background
Degree – German MD graduated Friedrich-Alexander University in 1996, now working as
health consultant and Traditional Healer in Sedona, AZ
Current scope of practice: Environmental & Orthomolecular medicine in a setting as Health
Consultant by Appointment only
Karen von Merveldt-Guevara - Harmonia Mundi LLC
Sedona, AZ 86336 (928) 274-4801
www.harmoniamundi.info harmoniamundi@gmail.com
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Resources 1
Key Websites: Meters, Devices, Shielding and other Material
www.lessEMF.com
Key Websites: Local and Regional Home Services
www.lessenemf.com www.totalEMFsolutions.com
Key Websites: Educational and other Resources
https://www.emf-experts.com/ https://bioinitiative.org/ https://www.emfanalysis.com/
https://ehtrust.org/ https://theemfguy.com/ (Nick Pineault)
https://takebackyourpower.net/ (Josh Del Sol)
http://electromagnetichealth.org/category/electromagnetic-health-blog/
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Resources 2
Key Authors:
Stephen Genius: https://www.elettrosensibili.it/wp-content/uploads/2015/08/genuis-and-lipp-
2011.pdf – Case Study
Martin Pall: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3780531/ - Voltage gated calcium ion
channels
Magda Havas: https://magdahavas.com/ - Book: Public Health SOS – The Shadow Side of the Wireless
Revolution
Samuel Milham: http://www.sammilham.com/ - Book: Dirty Electricity
Steven Magee: https://www.amazon.com/Steven-
Magee/e/B00ITZU698%3Fref=dbs_a_mng_rwt_scns_share
Books: Toxic Electricity, Toxic Health, Electrical Forensics
Ann-Louise Gittleman: https://annlouise.com/ - Book: Zapped
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Reading Material
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Topics
• Sources of EMFs and RFs
• How they can affect our bodies head to toe
• Who is more at risk?
• How to reduce, avoid, and mitigate exposure
• How to support our bodies via proper mineralization
• What environmental factor put more of us at risk?
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Wavelengths
of different
Sources of
EMF-RF
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Effects of
Electromagnetic
Radiation
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Effects of
Electromagnetic
Radiation
• Nguyen H P et al.: The
effect of a high frequency
electromagnetic field in
the microwave range on
red blood cells, Scientific
Reports, Volume 7,
Article number: 10798
(2017)
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https://www.saferemr.com/2015/09/recent-research-on-wifi-
effects.html
Effects of Wifi on
voltage gated
Calcium channels
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RF affects Conductivity of the Heart
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Smart Meters create ”Dirty Electricity”
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RF Absorption
According to
Age
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Bluetooth Radiation
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Symptoms of Residents near Cell Phone Tower
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Effects - Summary
• EMFs interfere with natural signaling processes in the human body
• Both physical and mental consequences
• Electromagnetic Hypersensitivity Syndrome (EHS)
• Symptoms: sleep disturbance, fatigue, burning or prickling sensation on the skin, tinnitus,
headaches, feeling unwell near cell towers, psychological distress, depression, cardiac
issues, High Blood Pressure, Metabolic Syndrome, chronic inflammation,
Neurodegenerative Diseases like Alzheimer’s, Parkinson’s, DNA-breakage, Cancer
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How minerals are affected by EMF
• Loss patterns, inflammation due to calcium influx via calcium ion channels on cell
membrane, increased sodium/potassium/magnesium/boron loss
• Mercury, Copper, Aluminum, Lead, Iron and other metals undergo bond breakage by
“activation”  increase of Radical Oxygen Species  EHS reaction
• Livers cannot handle the “load”, liver pathways are exhausted or over-engaged, can lead to
cholestasis
• Direct Relationship between Cholestatic Liver and HPA-Axis
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“Road Map”
Support for
Glutathione
Complex:
Vitamin C
B2 and B3
Selenium
Lithium (GST)
Liposomal
Glutathione
cAMP (Mn)
Zn
Boron-----
Methyl-B12 / Co
Mo----
SOD1 (Cu/Zn)
SOD2 (Mn)
Mn / Arginase
Cu
CuCu
Heme
Fe/Cu
Heme
Fe/Cu
Se
Zn Fe
B3B3B3
B2 B2
B2
B2/B3/
Heme/BH4
Fe/Cu
SAMe
SAMe
Folate Cycle
Support:
5-MTHF only
Zinc and
Manganese
for ROS
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Consequence to Public Health 1
• Hypomagnesemia is associated with Insulin Dysregulation, Diabetes Type II, Inflammation,
Cardiac Arrhythmia, High Blood Pressure, Adrenal Hypofunction, Depression, Hyperactivity,
Metabolic Syndrome – increased in prevalence 1988-2012 (NHANES)
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Metabolic Syndrome NHANES 1988-2012
Analyzed data from the National Health and Nutrition Examination Survey (NHANES)
for 1988 through 2012. “We defined metabolic syndrome as the presence of at least 3 of
these components: elevated waist circumference, elevated triglycerides, reduced high-
density lipoprotein cholesterol, high blood pressure, and elevated fasting blood glucose”.
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Metabolic Syndrome
NHANES 1988-2012
• Moore JX, Chaudhary N,
Akinyemiju T. Metabolic
Syndrome Prevalence by
Race/Ethnicity and Sex in the
United States, National Health
and Nutrition Examination
Survey, 1988–2012. Prev
Chronic Dis 2017;14:160287.
DOI: http://dx.doi.org/10.588
8/pcd14.160287
24

Metabolic Syndrome
NHANES 1988-2012
18 – 29 year old male &
female
Chronic Dis 2017;14:160287.
8/pcd14.160287
25

Metabolic Syndrome
NHANES 1988-2012
female
Chronic Dis 2017;14:160287.
8/pcd14.160287
26

Metabolic Syndrome
NHANES 1988-2012
female
Chronic Dis 2017;14:160287.
8/pcd14.160287
27

Metabolic Syndrome
NHANES 1988-2012
>70 year old male &
female
Chronic Dis 2017;14:160287.
8/pcd14.160287
28

Economic Consequences
• “In 2018, the global Metabolic Syndrome market size
was 24,600 million [US: 24.6 billion] US$ and it is
expected to reach 36,700 million [US: 36.7 Billion]
US$ by the end of 2025, with a Compound Annual
Growth Rate of 5.1% during 2019-2025”
GLOBAL METABOLIC SYNDROME MARKET SIZE, STATUS AND FORECAST 2019-2025
SKU ID : QYR-14461456, Publishing Date : 19-Jul-2019, No. of pages : 105
https://www.researchreportsworld.com/global-metabolic-syndrome-market-size-status-
and-forecast-2019-2025-14461456
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Blue Cross Blue Shield Study:
“Millenials” 2014 – 2018
• Nearly 73 million people in the U.S. are millennials—people born between 1981 and 1996
and who were 21 to 36 years old in 2017.
• This report focuses on the 55 million millennial Americans that are commercially insured.
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Blue Cross Blue Shield
Study: “Millenials”
2014 – 2018
• BCBS: The Health of
Millenials, THE HEALTH OF
AMERICA REPORT, Published
April 24, 2019,
https://www.bcbs.com/the-
health-of-
america/reports/the-health-
of-millennials
31

Blue Cross Blue Shield
Study: “Millenials”
2014 – 2018
• BCBS: The Health of
Millenials, THE HEALTH OF
AMERICA REPORT, Published
April 24, 2019,
https://www.bcbs.com/the-
health-of-
america/reports/the-health-
of-millennials
32

Blue Cross Blue Shield Study: “Millenials” 2014 – 2018
• “BCBS Health Index data underscores the urgency for
the healthcare community to recognize that
millennial Americans are experiencing double-digit
increases in prevalence for eight of the top 10 health
conditions.”
• “Millennials had significantly higher prevalence rates
than did their Gen X counterparts at the same age.”
• ”Health status of millennials will likely have
substantial effects on the American economy over the
next two decades—including workplace productivity
and healthcare costs.”
KvMG - VVMHC - 07/29/19
BCBS: The Health of Millenials, THE HEALTH OF AMERICA REPORT, Published April 24, 2019,
https://www.bcbs.com/the-health-of-america/reports/the-health-of-millennials
33

Blue Cross Blue Shield Study: “Millenials” 2014 – 2018
KvMG - VVMHC - 07/29/19
Clinical Conditions associated with the consequences of magnesium deficiency
and their level of evidence.
• Magnesium and Depression (LOE = B).
• Magnesium, Sleep, and Restless Leg Syndrome (LOE = B).
• Magnesium: Metabolic Syndrome, Diabetes, and Prevention of Diabetic
Complications (LOE = A, B)
Gerry K. Schwalfenberg and Stephen J. Genuis: Review Article -The Importance of
Magnesium in Clinical Healthcare, Hindawi Scientifica Volume 2017, Article ID 4179326, 14
pages, https://doi.org/10.1155/2017/4179326
34

Meanwhile in a small rural community in
Marocco…
“Findings suggest that moderate to severe iodine
deficiency in overweight women elevates serum TSH
and produces a more atherogenic lipid profile and that
iodine supplementation in this group reduces the
prevalence of hypercholesterolemia. Thus, iodine
prophylaxis may reduce cardiovascular disease risk in
overweight adults."
KvMG - VVMHC - 07/29/19
Isabelle Herter-Aeberli, et al.: Iodine Supplementation Decreases Hypercholesterolemia in
Iodine-Deficient, Overweight Women: A Randomized Controlled Trial, The Journal of
Nutrition, online July 22, 2015; doi:10.3945/jn.115.213439
35

Consequence to Public Health (2)
• Lower sodium level becomes new “normal”
• Adrenal Hypofunction expressed by lowered sodium level is now “normal”
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• Lower potassium level becomes new “normal”
• Adrenal Hyperfunction expressed by increased aldosterone output and lowered potassium
level is now “normal”
• “Essential Hypertension” associated with Aldosterone response (RAA) as expression of
Na/K-Ratio >> and Magnesium-loss >> becomes more prevalent
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• Lower chloride level becomes new “normal”
• Adrenal Hypofunction expressed by lowered chloride level is now “normal”
• Lower sodium, potassium and chloride level reflect lower HCl output, decreased digestive
capacity, increase of mineral malabsorption, increase in dysbiosis and decrease in intestinal
health becomes more prevalent
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Adrenal Response (1)
• Adrenal Function under chronic stress
• Adrenal Hyperfunction  High Aldosterone output suite to Renin-Angiotensin-Aldosterone
upregulation
• High Cortisol output
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Adrenal Response (2)
• Renal Sodium Retention
• Renal Chloride Retention
• Renal Potassium Excretion
•  Increase in Uric Acid, “Inflammatory Potential”, e.g. Na+ in Q4 and K+ Q1 or Q2
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Adrenal Response & Further Consequences (1)
• Renal Magnesium Loss
• Renal Boron Loss
• Decrease in Iodine
• Dysregulation of Calcium
• Down Regulation of Vitamin D3
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• Increase in Ca/P ratio  decrease in metabolic turn-over
• Increase in Na/K ratio  increase of inflammatory potential, magnesium loss, boron loss,
increased anxiety and insomnia, fluid retention (swollen ankles), decrease of pain
threshold
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• Increase in Ca/K ratio decrease in thyroid function
• Increase in Na/Mg ratio  increase in aldosterone and cortisol output, followed by
• Decrease in Na/Mg ratio reflecting hypoadrenia
• Increase in Ca/Mg ratio  decrease in blood sugar regulation
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• Adrenal Hypofunction  Depleted Aldosterone output, low cortisone
• Antidiuretic Hormone (ADH), aka Vasopressin output to retain volume  “Dilution”
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Evaluating
Electrolytes
in Serum by
Distribution
in Quartiles
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Mineral Losses under Stress
• Magnesium is primarily occurring high in the
interstitium
• Potassium is primarily occurring high intracellularly
• Stress  increases Aldosterone to retain Na+ and
eliminate K+ via kidney, magnesium loss ensues
KvMG - VVMHC - 07/29/19
Weaver CM. Potassium and Health. Adv Nutr. 2013;4(3):368s-377s. (PubMed)
Faheemudin, A, Abdul, M: Magnesium: The Forgotten Electrolyte—A Review
onHypomagnesemia,Med.Sci. 2019, 7(4),56; https://doi.org/10.3390/medsci
7040056 46

Magnesium Physiology
Magnesium is involved in over 300 enzyme systems necessary for:
• Protein synthesis
• Muscle contraction
• Nerve function
• Blood glucose control
• Hormone receptor binding
• Blood pressure regulation
• Cardiac excitability
• Transmembrane ion flux
• Gating of calcium channels
Magnesium is involved in energy production:
• Crucial for ATP metabolism (adenylate cyclase)
• Oxidative phosphorylation
• Glycolysis Nucleic acid synthesis: Synthesis of RNA and DNA
KvMG - VVMHC - 07/29/19
Gerry K. Schwalfenberg and Stephen J. Genuis: Review Article -The Importance of Magnesium in
Clinical Healthcare, Hindawi Scientifica Volume 2017, Article ID 4179326, 14 pages,
https://doi.org/10.1155/2017/4179326
47

Magnesium in Serum
• Subjective Indications of low Mg in serum:
Muscle Cramps
Chocolate Craving
Chronic Constipation
Dysrhythmia
• Association with epilepsy
• Lowered upper reference range (!)
• High in Interstitium, if in serum in Q1 or Q2:
Deficiency
Target with supplementation: ≥2.3 mg/dl
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Magnesium in Serum
• Check Albumin levels, as 35% of Mg bound to Protein, 75% of that to Albumin. If Alb <<,Mg
can be <<, and vice versa
• Check renal status: if BUN in Q4 or >25 mg/dl, Creatinine > 1.0 mg/dl, BUN/Creat ratio in
Q4 or >23, Uric Acid > 5.9, LDH in Q4 or >ref range, and AST >30
• Impaired renal Status: Supplement at low dose only
• If decreased along with AST <15 IU/l, MCH in Q1, MCV in Q1, magnesium needs B6, best as
P-5-P for cellular uptake
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Potassium Physiology
• Crucial electrolyte to maintain membrane potential:
K+ intracellular 30 times higher than extracellular; Na+
10 times lower intracellular compared to extracellular
– 2x K+ <-> 3x Na2+
• Essential for phosphoryl transfer enzymes (energy
production), like pyruvate kinase  carbohydrate
metabolism / Krebs Cycle, needed cofactors are 2x
Mg2+ or 2x Mn2+
KvMG - VVMHC - 07/29/19
Michael J. Page and Enrico Di Cera: Role of Na+ and K+ in Enzyme Function,
Physiol Rev 86: 1049–1092, 2006; doi:10.1152/physrev.00008.2006.
50

Potassium in Serum
• Subjective Indications of low K in serum:
Muscle weakness
Fatigue
Mental Confusion
High Blood Pressure
Heart disturbances
Problems in Nerve Conduction
Constipation
Anxiety
Insomnia
Edema
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51

Potassium in Serum
• A ”Nutrient of public health concern" according to the 2015-2020 Dietary Guidelines, found to be
“underconsumed” in US Population
https://health.gov/dietaryguidelines/2015/guidelines/
• Lowered upper reference range (!)
• High intracellular, if in serum in Q1 or Q2:
Deficiency
Target with supplementation: ≥ Median
• Watch for relationship with sodium:
Has to be K+ ≤ Na+, if K+ > Na+ = problems with Na/K-pumps!
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52

K+ > Na+ - Na/K Pump problems
• Post vaccination: interference by Hg and/or Al
 post-vaccination seizure disorder
• Treatment with fluoride containing drugs:
Diflucan (Fluconazole)
• Interference by other halides than Iodine, Bromine/Bromide, Chlorine / Perchlorate,
Fluorine, resp. Halide-containing drugs: ATBx, psychiatric drugs, Beta-blockers
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Drug-induced Hyperkalemia
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Drug-induced Hypokalemia – Part 1
KvMG - VVMHC - 07/29/19
Note: Fluconazole treatment can cause hypo- or hyperkalemia
depending on mechanism of interference
55

Drug-induced Hypokalemia – Part 2
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Other Causes of Hypokalemia
• Long-term effect: Opioids, Benzodiazepines, Cannabis  vicious cycle
• Long-term effect of birth-control: constant estrogen influx leads to increasing copper tissue
levels, Copper IUD: directly increases tissue copper levels
• “Copper Toxicity” by intracellular increase of copper  induces potassium loss
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57

Internal Hypokalemia: Effects of Triiodothyronine T3 (1)
Ca2+-ATPase & Na+/K+-ATPase induction:
- increased skeletal muscle contractility
- increased heart contractility
- restoration of proper intra- and extracellular
electrolyte distribution
- increased detoxification capacity
- aspect of adrenal recovery
- aspect of thyroid restitution
Everts, M. E. Effects of thyroid hormones on contractility and cation transport in skeletal muscle, Acta Physiol Scand 1996, 156, 325–333
Ismail-Beigi, Faramarz: Thyroid Hormone Regulation of Na+/K+-ATPase Expression, Trends Endocrinol Metab 1993;4:152-155
KvMG - VVMHC - 07/29/19 58

Internal Hypokalemia: Effects of Triiodothyronine T3 (2)
“Active Na+/K+ transport across plasma membranes
(mediated by Na+/K+- ATPase) is stimulated by
triiodothyronine (T3) in all mammalian tissues
responsive to thyroid hormone, and this stimulation
has been proposed to account for a substantial fraction
of thyroid thermogenesis.
The enhancement of Na+/K+-ATPase activity by T3
results from increased biosynthesis of Na+/K+-ATPase
subunits and is associated with increased abundance of
their encoding mRNAs.”
Ismail-Beigi, Faramarz: Thyroid Hormone Regulation of Na,K-ATPase Expression, Trends Endocrinol Metab 1993;4:152-155
KvMG - VVMHC - 07/29/19 59

K+- A Potent Antihypertensive
KvMG - VVMHC - 07/29/19
”Achieving adequate dietary potassium may be the most important dietary
constituent in the tool box to lower blood pressure.”
Mark C. Houston:
The Importance of
Potassium
in Managing
Hypertension,
Springer,
Curr Hypertens Rep
(online March 15, 2011)
13:309–317
DOI 10.1007/s11906-011-0197-8
60

More Mineral Losses under Stress
• Parallel to potassium: lithium
• Often observed together as loss: potassium, magnesium, boron, lithium and rubidium.
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61

What causes “Stress” (1)
• Apart from obvious causes like emotional and psychological stress
• Caused by presence, mobilization and excretion of:
• Copper, mercury, lead, uranium, aluminum, arsenic, tin, zirconium and other metals
 RDW > 13.5
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• Bacterial Endotoxins, chronic low-grade bacterial infections (most commonly Root Canal or
Root Failure) – subacute; Lyme – chronic/protracted
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• Viral, chronic low-grade viral infections (e.g. EBV, CMV)
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• Mycotoxins and mold, yeast infestation
• Oxalates (Oxalic Acid), Lactic Acid, Uric Acid
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• Medications, “Uppers and Downers”, Drugs
• Sugar; refined, highly processed carbohydrates; HFCS, (Cow)Dairy, Gluten (Gliadin),
generally highly refined carbohydrate-based diet
• Chemical Toxins: preservatives, pesticides, herbicides, cleaning agents, personal hygiene
products, food additives, other environmental pollutants
• EMF-RFs – Dirty Electricity
KvMG - VVMHC - 07/29/19
66

• Any formation of Radical Oxygen Species, possible biomarkers:
KvMG - VVMHC - 07/29/19
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• Detoxification - a physically demanding (catabolic) process
• Detox affects the emotional and psychological experience of the patient, as nutrient
mineral levels become severely affected
• The value in mineral balancing lies in guiding your patient “smoothly” through the process
and to keep him / her able to function well.
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Minerals, Vitamins and Hormones
…to address Metabolic Syndrome with respect to the patient’s biochemical
individuality
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Minerals / Vitamins / Hormones
• Mg (360 mg – 700 mg): 1-0-2 to 1-0-2-2, ~120 mg in AM, most in PM, BID to QID
• K (200mg – 800 mg): 1-0-1 to 2-2-2-2, TID / QID
• B (4mg – 30 mg) - 2 mg: 1-0-1, 30 mg: ½-0-½, BID in AM and PM
KvMG - VVMHC - 07/29/19
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• Se (as -methionine 200 mcg – 400 mcg, avg. 300 mcg/d): 1(2)-0-0 qn in AM
• Li (as -orotate 5mg (500 mcg actual Li) 5-10 mg: 1-0-1, BID in AM and PM
• Iodine/Iodide 1 mg – 50 mg +, rather AM, but TID/QID at higher doses
• Zn low dose 15/25 mg - 30 mg: 0-0-1(2), qn in PM
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• Vitamin C 1000 mg ≥ 3g: 2-1-0 OR 3-0-0, QN OR BID, rather in AM
• B-Complex (activated): 1-(1)-0, qn/BID rather in AM
• Niacinamide (500 mg) 1000mg – 3000 mg/d: 1-0-1 to 2-2-2, BID/TID
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• Omega 3, 6 or combo (1000mg) 2-4g: 1-0-1 (2 or 3), largest part in PM for better sleep
• Vitamin K2 as MK4 (Menatetrenone) (1mg per drop in K2 liquid): x-0-x or x-x-x - BID/TID 
calcium deposition in bone and teeth, tightening of gut junctions, 1 drop per 10 lb
bodyweight
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"The Physician should look for the force and nature of illness at its source. He
is not to look to that which can be seen, for we are not called to extinguish
the smoke but the fire itself.”
Theophrastus Paracelsus, M.D., Switzerland (1493-1541)
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